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Posted

Just had a wild, wild thought but will say it loud anyway:

what if:

our (kids') bodies actually allow yeast to flourish so as to help keep viruses in check?

.... and once we start antivirals these fungi are no longer needed and

are allowed to 'roam' on their own and get out of hand? either that or

they are allowed by our bodies to multiply as to strengthen our lines

of attack...

mad one, I know, but had to let it out... what got me there was that

both brewers yeast (a non-pathogenic fungi) and Candida (pathogenic)

have similar polysaccharides in their cell structures - see last

abstract.

a bit more about brewers yeast:

http://www.ageless.co.za/herb-brewersyeast.htm

http://web-japan.org/trends01/article/020305sci.html

this one hopefully explains the 'mud' part:

http://216.239.59.104/search?q=cache:H_5nHxq_WnMJ:home.att.net/~p.caimi/Yeast.doc+brewers+yeast+from+yeast+mud & hl=en & gl=uk & ct=clnk & cd=8

and yet more relevant (?) abstracts:

Curr Med Chem. 2004 Sep;11(18):2399-419.

Related Articles,

Links

Sulfated seaweed polysaccharides as antiviral agents.Damonte EB, Matulewicz MC, Cerezo AS.Virologia,

Departamento de Quimica Biologica, CIHIDECAR-CONICET, Facultad de

Ciencias Exactas y Naturales, Universidad de Buenos Aires, Pabellon 2,

Ciudad Universitaria, 1428 Buenos Aires, Argentina.Several

sulfated seaweed polysaccharides show high antiviral activity against

enveloped viruses, including important human pathogens such as human

immunodeficiency virus, herpes simplex virus, human cytomegalovirus,

dengue virus and respiratory syncytial virus. They can be obtained in

major amounts and at low costs, have low toxicity and in some cases,

lack anticoagulant effects. Even if the systemic applications have many

drawbacks, their structure and mode of action indicate potential for

topical uses to prevent virus infection. The herpes simplex viruses

attach to cells by an interaction between the envelope glycoprotein C

and the cell surface heparan sulfate (HS). The virus-cell complex is

formed by ionic interactions between the anionic (mainly sulfate)

groups in the polysaccharide and basic amino acids of the glycoprotein,

and non-ionic ones depending on hydrophobic amino acids interspersed

between the basic ones in the glycoprotein-binding zone. Hypothesis are

advanced of the corresponding hydrophobic structures in the

polysaccharides. The antiviral activity of the sulfated seaweed

polysaccharides is based on the formation of formally similar complexes

that block the interaction of the viruses with the cells. Correlations

are established between different structural parameters and antiviral

activity. The minimal, ionic and hydrophobic, structures in the seaweed

polysaccharides were hypothesized by comparison of the polysaccharides

with the known minimal binding structure in HS/heparin, together with a

correlation between those structures of the polysaccharides and their

antiviral activity.Publication Types:

Review

PMID: 15379705 [PubMed - indexed for MEDLINE]

Dev Biol Stand. 1992;77:115-20.

Related Articles,

Links

Assessment of the anti-viral effect of a short-term oral treatment of mice with live Saccharomyces cerevisiae cells.Fattal-German M, Bizzini B.Unite de Toxinologie Moleculaire, Institut Pasteur, Paris, France.For

assessing the efficacy of antiviral treatments, influenza and herpes

virus HSV-1 infections of varying degrees of severity have been

produced. The infections proved to be reproducible with respect to both

their course and death rate. These infections also exhibited a course

slow enough to permit the assessment of treatments under conditions

mimicking human infections and lent themselves to the choice of the

best adapted strategy to treat an infection. A short-term oral

treatment with live cells of S. cerevisiae was efficacious in

protecting mice against mild influenza infection and partly but

significantly against severe infection. On the other hand, it did not

afford significant protection towards either mild or severe HSV-1

infections, but it significantly potentiated the effectiveness of the

antiviral drug vidarabin. S. cerevisiae treatment induced the synthesis

of IFN alpha but not that of TNF alpha.PMID: 1426651 [PubMed - indexed for MEDLINE]

1: Evid Based Complement Alternat Med. 2006 Mar;3(1):109-15. Epub 2006 Jan 23.

Related Articles,

Links

Beneficial effect of brewers' yeast extract on daily activity in a murine model of chronic fatigue syndrome.Takahashi T, Yu F, Zhu SJ, Moriya J, Sumino H, Morimoto S, Yamaguchi N, Kanda T.Department of General Medicine, Kanazawa Medical University, Kahoku-gun, Ishikawa, Japan.The

aim of this study was to assess the effect of Brewers' yeast extract

(BYE) on daily activity in a mouse model of chronic fatigue syndrome

(CFS). CFS was induced by repeated injection of Brucella abortus (BA)

antigen every 2 weeks. BYE was orally administered to mice in a dose of

2 g per kg per day for 2 weeks before injecting BA and for 4 weeks

thereafter. We evaluated daily running activity in mice receiving BYE

as compared with that in untreated mice. Weekly variation of body

weight (BW) and survival in both groups was monitored during the

observation period. Spleen weight (SW), SW/BW ratio, percent splenic

follicular area and expression levels of interferon-gamma (IFN-gamma)

and interleukin-10 (IL-10) mRNA in spleen were determined in both

groups at the time of sacrifice. The daily activity during 2 weeks

after the second BA injection was significantly higher in the treated

group than in the control. There was no difference in BW between both

groups through the experimental course. Two mice in the control died 2

and 7 days after the second injection, whereas no mice in the treated

group died. Significantly decreased SW and SW/BW ratio were observed in

the treated mice together with elevation of splenic follicular area.

There were suppressed IFN-gamma and IL-10 mRNA levels in spleens from

the treated mice. Our results suggest that BYE might have a protective

effect on the marked reduction in activity following repeated BA

injection via normalization of host immune responses.PMID: 16550231 [PubMed]

Mol Cell Biol. 1993 Jul;13(7):4331-41.

Related Articles,

Links

Evidence that the SKI antiviral system of Saccharomyces cerevisiae acts by blocking expression of viral mRNA.Widner WR, Wickner RB.Section

on Genetics of Simple Eukaryotes, National Institute of Diabetes and

Digestive and Kidney Diseases, Bethesda, land 20892.The

SKI2 gene is part of a host system that represses the copy number of

the L-A double-stranded RNA (dsRNA) virus and its satellites M and X

dsRNA, of the L-BC dsRNA virus, and of the single-stranded replicon 20S

RNA. We show that SKI2 encodes a 145-kDa protein with motifs

characteristic of helicases and nucleolar proteins and is essential

only in cells carrying M dsRNA. Unexpectedly, Ski2p does not repress M1

dsRNA copy number when M1 is supported by aN L-A cDNA clone;

nonetheless, it did lower the levels of M1 dsRNA-encoded toxin

produced. Since toxin secretion from cDNA clones of M1 is unaffected by

Ski2p, these data suggest that Ski2p acts by specifically blocking

translation of viral mRNAs, perhaps recognizing the absence of cap or

poly(A). In support of this idea, we find that Ski2p represses

production of beta-galactosidase from RNA polymerase I [no cap and no

poly(A)] transcripts but not from RNA polymerase II (capped)

transcripts.PMID: 8321235 [PubMed - indexed for MEDLINE]

Curr Drug Targets. 2006 Apr;7(4):505-12.

Related Articles,

Links

Getting in touch with Candida albicans: the cell wall of a fungal pathogen.Sohn K, Schwenk J, Urban C, Lechner J, Schweikert M, Rupp S.Fraunhofer-IGB, Nobelstr. 12, 70569 Stuttgart, Germany.The

cell wall of fungi is a highly complex structure consisting of a

network of polysaccharides in which a plethora of different proteins

are embedded. It is one of the major organelles of the cell surrounding

it like an armor which protects from environmental stresses like

osmotic pressure and defines the shape and physical strength of the

fungal cell. It is crucial for colonization and infection since it

defines the interface between host and pathogen. No similar structure

is present in the host, therefore it defines a prime target for drug

development. In this context, it has been shown that cell surface

proteins are required for adhesion to host cells. The fact, that both

pathogenic fungi, like Candida albicans as well as non-pathogenic

fungi, like Saccharomyces cerevisiae, in general, have a very similar

polysaccharide structure but differ significantly in their protein

composition which underscores the importance of cell wall proteins for

pathogenesis. However, cell wall proteomics of fungi is a highly

challenging task due to the complex biochemistry of these proteins. The

extensive post-translational modifications and covalent attachment to

the polysaccharide backbone of a large proportion of cell wall proteins

makes it a demanding task to isolate and identify them. In this

article, we describe the recent approaches that have been developed to

describe cell wall dynamics and to isolate and identify cell wall

proteins in the pathogenic yeast C. albicans.Publication Types:

Review

PMID: 16611038 [PubMed - indexed for MEDLINE]

> >> > > > 1: Zhongguo Zhong Yao Za Zhi. 1998 Mar;23(3):171-3,> > > > Antivirus effect of polysaccharides of brewer yeast in vitro> > > > The antivirus effect of polysaccharides of brewer yeast from yeast mud> > on 13> > kinds of viruses including DNA and RNA virus along with their mechanisms> > were> > studied. The result showed that this effect was remarkable on the> > infections> > with poliovirus III, adenovirus III, ECHO6 virus, enterovirus 71,> > vesicular> > stomatitis virus, herpesvirus I, II, coxsackie A16 virus and coxsackie> > B3 virus.> > The polysaccharides of brewer yeast could also inhibit the development> > of> > cytopathic effect(CPE) and protect cultural cells from being infected> > with the> > above viruses.> > > > PMID: 11596239 [PubMed - indexed for MEDLINE]> >>

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Guest guest

Guest guest

Posted
Posted

Just had a wild, wild thought but will say it loud anyway:

what if:

our (kids') bodies actually allow yeast to flourish so as to help keep viruses in check?

.... and once we start antivirals these fungi are no longer needed and

are allowed to 'roam' on their own and get out of hand? either that or

they are allowed by our bodies to multiply as to strengthen our lines

of attack...

mad one, I know, but had to let it out... what got me there was that

both brewers yeast (a non-pathogenic fungi) and Candida (pathogenic)

have similar polysaccharides in their cell structures - see last

abstract.

a bit more about brewers yeast:

http://www.ageless.co.za/herb-brewersyeast.htm

http://web-japan.org/trends01/article/020305sci.html

this one hopefully explains the 'mud' part:

http://216.239.59.104/search?q=cache:H_5nHxq_WnMJ:home.att.net/~p.caimi/Yeast.doc+brewers+yeast+from+yeast+mud & hl=en & gl=uk & ct=clnk & cd=8

and yet more relevant (?) abstracts:

Curr Med Chem. 2004 Sep;11(18):2399-419.

Related Articles,

Links

Sulfated seaweed polysaccharides as antiviral agents.Damonte EB, Matulewicz MC, Cerezo AS.Virologia,

Departamento de Quimica Biologica, CIHIDECAR-CONICET, Facultad de

Ciencias Exactas y Naturales, Universidad de Buenos Aires, Pabellon 2,

Ciudad Universitaria, 1428 Buenos Aires, Argentina.Several

sulfated seaweed polysaccharides show high antiviral activity against

enveloped viruses, including important human pathogens such as human

immunodeficiency virus, herpes simplex virus, human cytomegalovirus,

dengue virus and respiratory syncytial virus. They can be obtained in

major amounts and at low costs, have low toxicity and in some cases,

lack anticoagulant effects. Even if the systemic applications have many

drawbacks, their structure and mode of action indicate potential for

topical uses to prevent virus infection. The herpes simplex viruses

attach to cells by an interaction between the envelope glycoprotein C

and the cell surface heparan sulfate (HS). The virus-cell complex is

formed by ionic interactions between the anionic (mainly sulfate)

groups in the polysaccharide and basic amino acids of the glycoprotein,

and non-ionic ones depending on hydrophobic amino acids interspersed

between the basic ones in the glycoprotein-binding zone. Hypothesis are

advanced of the corresponding hydrophobic structures in the

polysaccharides. The antiviral activity of the sulfated seaweed

polysaccharides is based on the formation of formally similar complexes

that block the interaction of the viruses with the cells. Correlations

are established between different structural parameters and antiviral

activity. The minimal, ionic and hydrophobic, structures in the seaweed

polysaccharides were hypothesized by comparison of the polysaccharides

with the known minimal binding structure in HS/heparin, together with a

correlation between those structures of the polysaccharides and their

antiviral activity.Publication Types:

Review

PMID: 15379705 [PubMed - indexed for MEDLINE]

Dev Biol Stand. 1992;77:115-20.

Related Articles,

Links

Assessment of the anti-viral effect of a short-term oral treatment of mice with live Saccharomyces cerevisiae cells.Fattal-German M, Bizzini B.Unite de Toxinologie Moleculaire, Institut Pasteur, Paris, France.For

assessing the efficacy of antiviral treatments, influenza and herpes

virus HSV-1 infections of varying degrees of severity have been

produced. The infections proved to be reproducible with respect to both

their course and death rate. These infections also exhibited a course

slow enough to permit the assessment of treatments under conditions

mimicking human infections and lent themselves to the choice of the

best adapted strategy to treat an infection. A short-term oral

treatment with live cells of S. cerevisiae was efficacious in

protecting mice against mild influenza infection and partly but

significantly against severe infection. On the other hand, it did not

afford significant protection towards either mild or severe HSV-1

infections, but it significantly potentiated the effectiveness of the

antiviral drug vidarabin. S. cerevisiae treatment induced the synthesis

of IFN alpha but not that of TNF alpha.PMID: 1426651 [PubMed - indexed for MEDLINE]

1: Evid Based Complement Alternat Med. 2006 Mar;3(1):109-15. Epub 2006 Jan 23.

Related Articles,

Links

Beneficial effect of brewers' yeast extract on daily activity in a murine model of chronic fatigue syndrome.Takahashi T, Yu F, Zhu SJ, Moriya J, Sumino H, Morimoto S, Yamaguchi N, Kanda T.Department of General Medicine, Kanazawa Medical University, Kahoku-gun, Ishikawa, Japan.The

aim of this study was to assess the effect of Brewers' yeast extract

(BYE) on daily activity in a mouse model of chronic fatigue syndrome

(CFS). CFS was induced by repeated injection of Brucella abortus (BA)

antigen every 2 weeks. BYE was orally administered to mice in a dose of

2 g per kg per day for 2 weeks before injecting BA and for 4 weeks

thereafter. We evaluated daily running activity in mice receiving BYE

as compared with that in untreated mice. Weekly variation of body

weight (BW) and survival in both groups was monitored during the

observation period. Spleen weight (SW), SW/BW ratio, percent splenic

follicular area and expression levels of interferon-gamma (IFN-gamma)

and interleukin-10 (IL-10) mRNA in spleen were determined in both

groups at the time of sacrifice. The daily activity during 2 weeks

after the second BA injection was significantly higher in the treated

group than in the control. There was no difference in BW between both

groups through the experimental course. Two mice in the control died 2

and 7 days after the second injection, whereas no mice in the treated

group died. Significantly decreased SW and SW/BW ratio were observed in

the treated mice together with elevation of splenic follicular area.

There were suppressed IFN-gamma and IL-10 mRNA levels in spleens from

the treated mice. Our results suggest that BYE might have a protective

effect on the marked reduction in activity following repeated BA

injection via normalization of host immune responses.PMID: 16550231 [PubMed]

Mol Cell Biol. 1993 Jul;13(7):4331-41.

Related Articles,

Links

Evidence that the SKI antiviral system of Saccharomyces cerevisiae acts by blocking expression of viral mRNA.Widner WR, Wickner RB.Section

on Genetics of Simple Eukaryotes, National Institute of Diabetes and

Digestive and Kidney Diseases, Bethesda, land 20892.The

SKI2 gene is part of a host system that represses the copy number of

the L-A double-stranded RNA (dsRNA) virus and its satellites M and X

dsRNA, of the L-BC dsRNA virus, and of the single-stranded replicon 20S

RNA. We show that SKI2 encodes a 145-kDa protein with motifs

characteristic of helicases and nucleolar proteins and is essential

only in cells carrying M dsRNA. Unexpectedly, Ski2p does not repress M1

dsRNA copy number when M1 is supported by aN L-A cDNA clone;

nonetheless, it did lower the levels of M1 dsRNA-encoded toxin

produced. Since toxin secretion from cDNA clones of M1 is unaffected by

Ski2p, these data suggest that Ski2p acts by specifically blocking

translation of viral mRNAs, perhaps recognizing the absence of cap or

poly(A). In support of this idea, we find that Ski2p represses

production of beta-galactosidase from RNA polymerase I [no cap and no

poly(A)] transcripts but not from RNA polymerase II (capped)

transcripts.PMID: 8321235 [PubMed - indexed for MEDLINE]

Curr Drug Targets. 2006 Apr;7(4):505-12.

Related Articles,

Links

Getting in touch with Candida albicans: the cell wall of a fungal pathogen.Sohn K, Schwenk J, Urban C, Lechner J, Schweikert M, Rupp S.Fraunhofer-IGB, Nobelstr. 12, 70569 Stuttgart, Germany.The

cell wall of fungi is a highly complex structure consisting of a

network of polysaccharides in which a plethora of different proteins

are embedded. It is one of the major organelles of the cell surrounding

it like an armor which protects from environmental stresses like

osmotic pressure and defines the shape and physical strength of the

fungal cell. It is crucial for colonization and infection since it

defines the interface between host and pathogen. No similar structure

is present in the host, therefore it defines a prime target for drug

development. In this context, it has been shown that cell surface

proteins are required for adhesion to host cells. The fact, that both

pathogenic fungi, like Candida albicans as well as non-pathogenic

fungi, like Saccharomyces cerevisiae, in general, have a very similar

polysaccharide structure but differ significantly in their protein

composition which underscores the importance of cell wall proteins for

pathogenesis. However, cell wall proteomics of fungi is a highly

challenging task due to the complex biochemistry of these proteins. The

extensive post-translational modifications and covalent attachment to

the polysaccharide backbone of a large proportion of cell wall proteins

makes it a demanding task to isolate and identify them. In this

article, we describe the recent approaches that have been developed to

describe cell wall dynamics and to isolate and identify cell wall

proteins in the pathogenic yeast C. albicans.Publication Types:

Review

PMID: 16611038 [PubMed - indexed for MEDLINE]

> >> > > > 1: Zhongguo Zhong Yao Za Zhi. 1998 Mar;23(3):171-3,> > > > Antivirus effect of polysaccharides of brewer yeast in vitro> > > > The antivirus effect of polysaccharides of brewer yeast from yeast mud> > on 13> > kinds of viruses including DNA and RNA virus along with their mechanisms> > were> > studied. The result showed that this effect was remarkable on the> > infections> > with poliovirus III, adenovirus III, ECHO6 virus, enterovirus 71,> > vesicular> > stomatitis virus, herpesvirus I, II, coxsackie A16 virus and coxsackie> > B3 virus.> > The polysaccharides of brewer yeast could also inhibit the development> > of> > cytopathic effect(CPE) and protect cultural cells from being infected> > with the> > above viruses.> > > > PMID: 11596239 [PubMed - indexed for MEDLINE]> >>

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Posted
Posted

forgot to say Saccharomyces cerevisiae = brewer's yeast (for those

bothering to read on...)

> > >

> > >

> > > 1: Zhongguo Zhong Yao Za Zhi. 1998 Mar;23(3):171-3,

> > >

> > > Antivirus effect of polysaccharides of brewer yeast in vitro

> > >

> > > The antivirus effect of polysaccharides of brewer yeast from yeast

> mud

> > > on 13

> > > kinds of viruses including DNA and RNA virus along with their

> mechanisms

> > > were

> > > studied. The result showed that this effect was remarkable on the

> > > infections

> > > with poliovirus III, adenovirus III, ECHO6 virus, enterovirus 71,

> > > vesicular

> > > stomatitis virus, herpesvirus I, II, coxsackie A16 virus and

> coxsackie

> > > B3 virus.

> > > The polysaccharides of brewer yeast could also inhibit the

> development

> > > of

> > > cytopathic effect(CPE) and protect cultural cells from being

> infected

> > > with the

> > > above viruses.

> > >

> > > PMID: 11596239 [PubMed - indexed for MEDLINE]

> > >

> >

>

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Share on other sites
Guest guest

Guest guest

Posted
Posted

forgot to say Saccharomyces cerevisiae = brewer's yeast (for those

bothering to read on...)

> > >

> > >

> > > 1: Zhongguo Zhong Yao Za Zhi. 1998 Mar;23(3):171-3,

> > >

> > > Antivirus effect of polysaccharides of brewer yeast in vitro

> > >

> > > The antivirus effect of polysaccharides of brewer yeast from yeast

> mud

> > > on 13

> > > kinds of viruses including DNA and RNA virus along with their

> mechanisms

> > > were

> > > studied. The result showed that this effect was remarkable on the

> > > infections

> > > with poliovirus III, adenovirus III, ECHO6 virus, enterovirus 71,

> > > vesicular

> > > stomatitis virus, herpesvirus I, II, coxsackie A16 virus and

> coxsackie

> > > B3 virus.

> > > The polysaccharides of brewer yeast could also inhibit the

> development

> > > of

> > > cytopathic effect(CPE) and protect cultural cells from being

> infected

> > > with the

> > > above viruses.

> > >

> > > PMID: 11596239 [PubMed - indexed for MEDLINE]

> > >

> >

>

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Posted
Posted

Interesting. Don't worry, I like wild discussion like this. Continuing the

discussion..

something we would need to fit in is why are our kids the only ones that seem to

react to

antivirals with these fungal flair? Why not NT folks with the same HSV viruses?

Are there other viruses involved that we are not aware of and they act

differently than the

STD HSV community? (stealth viruses? other hsv we don't know about yet.)

Is there some relationship to metals that may be in our kids?

Is there some other biological difference?

I need to think this through a few times, but this was my first thought.

- Stan

> > >

> > >

> > > 1: Zhongguo Zhong Yao Za Zhi. 1998 Mar;23(3):171-3,

> > >

> > > Antivirus effect of polysaccharides of brewer yeast in vitro

> > >

> > > The antivirus effect of polysaccharides of brewer yeast from yeast

> mud

> > > on 13

> > > kinds of viruses including DNA and RNA virus along with their

> mechanisms

> > > were

> > > studied. The result showed that this effect was remarkable on the

> > > infections

> > > with poliovirus III, adenovirus III, ECHO6 virus, enterovirus 71,

> > > vesicular

> > > stomatitis virus, herpesvirus I, II, coxsackie A16 virus and

> coxsackie

> > > B3 virus.

> > > The polysaccharides of brewer yeast could also inhibit the

> development

> > > of

> > > cytopathic effect(CPE) and protect cultural cells from being

> infected

> > > with the

> > > above viruses.

> > >

> > > PMID: 11596239 [PubMed - indexed for MEDLINE]

> > >

> >

>

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Share on other sites
Guest guest

Guest guest

Posted
Posted

Interesting. Don't worry, I like wild discussion like this. Continuing the

discussion..

something we would need to fit in is why are our kids the only ones that seem to

react to

antivirals with these fungal flair? Why not NT folks with the same HSV viruses?

Are there other viruses involved that we are not aware of and they act

differently than the

STD HSV community? (stealth viruses? other hsv we don't know about yet.)

Is there some relationship to metals that may be in our kids?

Is there some other biological difference?

I need to think this through a few times, but this was my first thought.

- Stan

> > >

> > >

> > > 1: Zhongguo Zhong Yao Za Zhi. 1998 Mar;23(3):171-3,

> > >

> > > Antivirus effect of polysaccharides of brewer yeast in vitro

> > >

> > > The antivirus effect of polysaccharides of brewer yeast from yeast

> mud

> > > on 13

> > > kinds of viruses including DNA and RNA virus along with their

> mechanisms

> > > were

> > > studied. The result showed that this effect was remarkable on the

> > > infections

> > > with poliovirus III, adenovirus III, ECHO6 virus, enterovirus 71,

> > > vesicular

> > > stomatitis virus, herpesvirus I, II, coxsackie A16 virus and

> coxsackie

> > > B3 virus.

> > > The polysaccharides of brewer yeast could also inhibit the

> development

> > > of

> > > cytopathic effect(CPE) and protect cultural cells from being

> infected

> > > with the

> > > above viruses.

> > >

> > > PMID: 11596239 [PubMed - indexed for MEDLINE]

> > >

> >

>

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Posted
Posted

I think one big connection to the yeast and our kids is liver

dysfunction. Since the liver is the center of detox in the body and

affects every function in the body, and since our kids are heavy

metal toxic which causes liver dysfunction, I think it's something

to really look at. From what I've been hearing from parents who are

restoring liver functions with Liver Life, I think yeast/gut issues

have so much to do with the liver. The Liver Life restores pH,

drains acids, decongests the kidneys and liver, normalizes liver

enzymes, rejuvenates the liver restoring it's functions.

The liver filters out 99% of yeast and bacteria that enter the blood

through the gut. If your kidneys and liver are congested, they

will not drain acids efficiently. Acids build up in our tissues.

(Acids are toxins like heavy metals, chemicals, pesticides, yeast,

bacteria.) An acidic environment is a breeding ground for yeast,

bacteria, parasites. Viruses and metals are also acid forming.

Other factors of course include our diets. Here is two good lists

of alkaline forming foods vs acid forming foods. For someone trying

to restore their body's alkaline state, it's really helpful to eat

more alkaline forming foods. Lemons and watermelons being the

best. (if tolerated, of course) One little girl who had a really

bad yeast infection got immediate relief by adding in fresh squeezed

lemon juice to her drinks.

http://home.bluegrass.net/~jclark/alkaline_foods.htm

http://www.essense-of-life.com/info/foodchart.htm

My boss, Ray said in clinic, they noticed that virals

usually exascerbates the yeast. And since virals are acid forming,

that would make sense.

I thought Gavin's liver was functioning fine. We never had any

indications of liver dysfunction until I learned about urine pH. IF

your urine pH is high it means that your body is not draining

acids. We drain acids through our urine so the urine should be

acidic, which helps our saliva pH become alkaline. Gavin's urine pH

was an 8.0. An appropriate range is 5.8-6.2 for a healthy person

and 5.5-5.8 for someone going through detox.

His saliva pH was a 6.0. Healthy range is 6.8-7.2, 7.0 being

perfect.

OF course you could definitely still have liver dysfunction if the

urine pH was in healthy range. The liver has over 500 functions and

I don't know if we have a way to test for all those functions.

Things we've noticed after addressing his liver with LL are:

-able to discontinue many of his supps and decrease others

-yeast and bacteria finally under control without the addition of 4-

5 antifungals/antibacterials (alternating every 3-4 days)

-went from knowing 19/100 of his fry words for 1st grade to 100/100

in just a few months

-went up three reading levels

-social skills through the roof (he had really good skills to start

but is now just amazing!)

-his teachers are amazed at his progress. Went from being really

inconsistent to knowing how to do these things every day.( He knew

how to count to 100 one day, not the next. Or knew how to spell

words one day but not the next.)

-calm, less explosive anger (he's still affected by seasonal

allergies but much less severe)

-was able to increase his dose of NDF Plus (mine too) without

anxiety issues. Before LL, I had only tolerated 3-4 drops, now I

can tolerate a full dropperful (26 drops).

-better digestion and tolerance of foods

I am sure there are tons more that I have forgotten.

Stan, I don't know if you remember when I asked you about the

connection between MB12 and acids many months back. You had made a

statement in a post that MB12 does not do well in an acidic

environment and that's why some autistic kids do better with the

nasal because the mucous in the nasal is much less acidic than the

tissues. My son was able to discontinue all MB12 after draining the

acids in his body and restoring his liver functions. (I don't think

we are done. I think it's a process) Even oral MB12. I think

restoring the liver will help these kids get better so much faster

cause they could utilize the other things much more efficiently.

It's amazing to not have to deal with yeasty behaviors every day. I

am sure we may need to address it here and there, but it's amazing

how much the liver works to battle yeast.

My best,

Tami W

Gavin's mom

BioRay customer support

for the ASD community

BioRaynaturaldetox/

> > > >

> > > >

> > > > 1: Zhongguo Zhong Yao Za Zhi. 1998 Mar;23(3):171-3,

> > > >

> > > > Antivirus effect of polysaccharides of brewer yeast in vitro

> > > >

> > > > The antivirus effect of polysaccharides of brewer yeast from

yeast

> > mud

> > > > on 13

> > > > kinds of viruses including DNA and RNA virus along with their

> > mechanisms

> > > > were

> > > > studied. The result showed that this effect was remarkable

on the

> > > > infections

> > > > with poliovirus III, adenovirus III, ECHO6 virus,

enterovirus 71,

> > > > vesicular

> > > > stomatitis virus, herpesvirus I, II, coxsackie A16 virus and

> > coxsackie

> > > > B3 virus.

> > > > The polysaccharides of brewer yeast could also inhibit the

> > development

> > > > of

> > > > cytopathic effect(CPE) and protect cultural cells from being

> > infected

> > > > with the

> > > > above viruses.

> > > >

> > > > PMID: 11596239 [PubMed - indexed for MEDLINE]

> > > >

> > >

> >

>

Link to comment
Share on other sites
Guest guest

Guest guest

Posted
Posted

I think one big connection to the yeast and our kids is liver

dysfunction. Since the liver is the center of detox in the body and

affects every function in the body, and since our kids are heavy

metal toxic which causes liver dysfunction, I think it's something

to really look at. From what I've been hearing from parents who are

restoring liver functions with Liver Life, I think yeast/gut issues

have so much to do with the liver. The Liver Life restores pH,

drains acids, decongests the kidneys and liver, normalizes liver

enzymes, rejuvenates the liver restoring it's functions.

The liver filters out 99% of yeast and bacteria that enter the blood

through the gut. If your kidneys and liver are congested, they

will not drain acids efficiently. Acids build up in our tissues.

(Acids are toxins like heavy metals, chemicals, pesticides, yeast,

bacteria.) An acidic environment is a breeding ground for yeast,

bacteria, parasites. Viruses and metals are also acid forming.

Other factors of course include our diets. Here is two good lists

of alkaline forming foods vs acid forming foods. For someone trying

to restore their body's alkaline state, it's really helpful to eat

more alkaline forming foods. Lemons and watermelons being the

best. (if tolerated, of course) One little girl who had a really

bad yeast infection got immediate relief by adding in fresh squeezed

lemon juice to her drinks.

http://home.bluegrass.net/~jclark/alkaline_foods.htm

http://www.essense-of-life.com/info/foodchart.htm

My boss, Ray said in clinic, they noticed that virals

usually exascerbates the yeast. And since virals are acid forming,

that would make sense.

I thought Gavin's liver was functioning fine. We never had any

indications of liver dysfunction until I learned about urine pH. IF

your urine pH is high it means that your body is not draining

acids. We drain acids through our urine so the urine should be

acidic, which helps our saliva pH become alkaline. Gavin's urine pH

was an 8.0. An appropriate range is 5.8-6.2 for a healthy person

and 5.5-5.8 for someone going through detox.

His saliva pH was a 6.0. Healthy range is 6.8-7.2, 7.0 being

perfect.

OF course you could definitely still have liver dysfunction if the

urine pH was in healthy range. The liver has over 500 functions and

I don't know if we have a way to test for all those functions.

Things we've noticed after addressing his liver with LL are:

-able to discontinue many of his supps and decrease others

-yeast and bacteria finally under control without the addition of 4-

5 antifungals/antibacterials (alternating every 3-4 days)

-went from knowing 19/100 of his fry words for 1st grade to 100/100

in just a few months

-went up three reading levels

-social skills through the roof (he had really good skills to start

but is now just amazing!)

-his teachers are amazed at his progress. Went from being really

inconsistent to knowing how to do these things every day.( He knew

how to count to 100 one day, not the next. Or knew how to spell

words one day but not the next.)

-calm, less explosive anger (he's still affected by seasonal

allergies but much less severe)

-was able to increase his dose of NDF Plus (mine too) without

anxiety issues. Before LL, I had only tolerated 3-4 drops, now I

can tolerate a full dropperful (26 drops).

-better digestion and tolerance of foods

I am sure there are tons more that I have forgotten.

Stan, I don't know if you remember when I asked you about the

connection between MB12 and acids many months back. You had made a

statement in a post that MB12 does not do well in an acidic

environment and that's why some autistic kids do better with the

nasal because the mucous in the nasal is much less acidic than the

tissues. My son was able to discontinue all MB12 after draining the

acids in his body and restoring his liver functions. (I don't think

we are done. I think it's a process) Even oral MB12. I think

restoring the liver will help these kids get better so much faster

cause they could utilize the other things much more efficiently.

It's amazing to not have to deal with yeasty behaviors every day. I

am sure we may need to address it here and there, but it's amazing

how much the liver works to battle yeast.

My best,

Tami W

Gavin's mom

BioRay customer support

for the ASD community

BioRaynaturaldetox/

> > > >

> > > >

> > > > 1: Zhongguo Zhong Yao Za Zhi. 1998 Mar;23(3):171-3,

> > > >

> > > > Antivirus effect of polysaccharides of brewer yeast in vitro

> > > >

> > > > The antivirus effect of polysaccharides of brewer yeast from

yeast

> > mud

> > > > on 13

> > > > kinds of viruses including DNA and RNA virus along with their

> > mechanisms

> > > > were

> > > > studied. The result showed that this effect was remarkable

on the

> > > > infections

> > > > with poliovirus III, adenovirus III, ECHO6 virus,

enterovirus 71,

> > > > vesicular

> > > > stomatitis virus, herpesvirus I, II, coxsackie A16 virus and

> > coxsackie

> > > > B3 virus.

> > > > The polysaccharides of brewer yeast could also inhibit the

> > development

> > > > of

> > > > cytopathic effect(CPE) and protect cultural cells from being

> > infected

> > > > with the

> > > > above viruses.

> > > >

> > > > PMID: 11596239 [PubMed - indexed for MEDLINE]

> > > >

> > >

> >

>

Link to comment
Share on other sites
Guest guest

Guest guest

Posted
Posted

Hi Tami,

Good memory about the MB12 and acid. Many of our kids are acidic because of the

increased fungal and bacterial levels. Now, is this downstream or upstream from

a

potential liver problem? My guess is it could be either depending on the child.

Every virus

listed on the Valtrex PDR can take up residence in the liver. I have a file

about this in the

FILES section of the group. That said, one example impairment could have

started with a

virus in the liver which may lead to increased bacterial and fungal levels which

raise acid

levels which tax the entire body. Add to that a chronic viral infection will

tax methylation

and likely cause oxidative stress. Add metals in there however you'd like

considering

healthy liver function is important for metals detox and metals are more

damaging with

lowered glutathione and the related higher testosterone.

This, IMO, is another reason to go after fungus at the same time as treating

viruses.

Lastly, about MB12... it's true. The molecule doesn't do well in acidic

environments.

Possibly one of the reasons that oral MB12 doesn't work (in addition to the

active

transport, intrisic factor, and possible genitic SNP mutation issues).

- Stan

> > > > >

> > > > >

> > > > > 1: Zhongguo Zhong Yao Za Zhi. 1998 Mar;23(3):171-3,

> > > > >

> > > > > Antivirus effect of polysaccharides of brewer yeast in vitro

> > > > >

> > > > > The antivirus effect of polysaccharides of brewer yeast from

> yeast

> > > mud

> > > > > on 13

> > > > > kinds of viruses including DNA and RNA virus along with their

> > > mechanisms

> > > > > were

> > > > > studied. The result showed that this effect was remarkable

> on the

> > > > > infections

> > > > > with poliovirus III, adenovirus III, ECHO6 virus,

> enterovirus 71,

> > > > > vesicular

> > > > > stomatitis virus, herpesvirus I, II, coxsackie A16 virus and

> > > coxsackie

> > > > > B3 virus.

> > > > > The polysaccharides of brewer yeast could also inhibit the

> > > development

> > > > > of

> > > > > cytopathic effect(CPE) and protect cultural cells from being

> > > infected

> > > > > with the

> > > > > above viruses.

> > > > >

> > > > > PMID: 11596239 [PubMed - indexed for MEDLINE]

> > > > >

> > > >

> > >

> >

>

Link to comment
Share on other sites
Guest guest

Guest guest

Posted
Posted

Hi Tami,

Good memory about the MB12 and acid. Many of our kids are acidic because of the

increased fungal and bacterial levels. Now, is this downstream or upstream from

a

potential liver problem? My guess is it could be either depending on the child.

Every virus

listed on the Valtrex PDR can take up residence in the liver. I have a file

about this in the

FILES section of the group. That said, one example impairment could have

started with a

virus in the liver which may lead to increased bacterial and fungal levels which

raise acid

levels which tax the entire body. Add to that a chronic viral infection will

tax methylation

and likely cause oxidative stress. Add metals in there however you'd like

considering

healthy liver function is important for metals detox and metals are more

damaging with

lowered glutathione and the related higher testosterone.

This, IMO, is another reason to go after fungus at the same time as treating

viruses.

Lastly, about MB12... it's true. The molecule doesn't do well in acidic

environments.

Possibly one of the reasons that oral MB12 doesn't work (in addition to the

active

transport, intrisic factor, and possible genitic SNP mutation issues).

- Stan

> > > > >

> > > > >

> > > > > 1: Zhongguo Zhong Yao Za Zhi. 1998 Mar;23(3):171-3,

> > > > >

> > > > > Antivirus effect of polysaccharides of brewer yeast in vitro

> > > > >

> > > > > The antivirus effect of polysaccharides of brewer yeast from

> yeast

> > > mud

> > > > > on 13

> > > > > kinds of viruses including DNA and RNA virus along with their

> > > mechanisms

> > > > > were

> > > > > studied. The result showed that this effect was remarkable

> on the

> > > > > infections

> > > > > with poliovirus III, adenovirus III, ECHO6 virus,

> enterovirus 71,

> > > > > vesicular

> > > > > stomatitis virus, herpesvirus I, II, coxsackie A16 virus and

> > > coxsackie

> > > > > B3 virus.

> > > > > The polysaccharides of brewer yeast could also inhibit the

> > > development

> > > > > of

> > > > > cytopathic effect(CPE) and protect cultural cells from being

> > > infected

> > > > > with the

> > > > > above viruses.

> > > > >

> > > > > PMID: 11596239 [PubMed - indexed for MEDLINE]

> > > > >

> > > >

> > >

> >

>

Link to comment
Share on other sites
Guest guest

Guest guest

Posted
Posted

Hi Tami,

Good memory about the MB12 and acid. Many of our kids are acidic because of the

increased fungal and bacterial levels. Now, is this downstream or upstream from

a

potential liver problem? My guess is it could be either depending on the child.

Every virus

listed on the Valtrex PDR can take up residence in the liver. I have a file

about this in the

FILES section of the group. That said, one example impairment could have

started with a

virus in the liver which may lead to increased bacterial and fungal levels which

raise acid

levels which tax the entire body. Add to that a chronic viral infection will

tax methylation

and likely cause oxidative stress. Add metals in there however you'd like

considering

healthy liver function is important for metals detox and metals are more

damaging with

lowered glutathione and the related higher testosterone.

This, IMO, is another reason to go after fungus at the same time as treating

viruses.

Lastly, about MB12... it's true. The molecule doesn't do well in acidic

environments.

Possibly one of the reasons that oral MB12 doesn't work (in addition to the

active

transport, intrisic factor, and possible genitic SNP mutation issues).

- Stan

> > > > >

> > > > >

> > > > > 1: Zhongguo Zhong Yao Za Zhi. 1998 Mar;23(3):171-3,

> > > > >

> > > > > Antivirus effect of polysaccharides of brewer yeast in vitro

> > > > >

> > > > > The antivirus effect of polysaccharides of brewer yeast from

> yeast

> > > mud

> > > > > on 13

> > > > > kinds of viruses including DNA and RNA virus along with their

> > > mechanisms

> > > > > were

> > > > > studied. The result showed that this effect was remarkable

> on the

> > > > > infections

> > > > > with poliovirus III, adenovirus III, ECHO6 virus,

> enterovirus 71,

> > > > > vesicular

> > > > > stomatitis virus, herpesvirus I, II, coxsackie A16 virus and

> > > coxsackie

> > > > > B3 virus.

> > > > > The polysaccharides of brewer yeast could also inhibit the

> > > development

> > > > > of

> > > > > cytopathic effect(CPE) and protect cultural cells from being

> > > infected

> > > > > with the

> > > > > above viruses.

> > > > >

> > > > > PMID: 11596239 [PubMed - indexed for MEDLINE]

> > > > >

> > > >

> > >

> >

>

Link to comment
Share on other sites
Guest guest

Guest guest

Posted
Posted

Hi Stan,

What's interesting about the MB12/acid connection and why it sticks

so well in my mind is that we were on MB12 shots for a year. We had

really good results in all areas. When we started the liver life,

we noticed (after a very short time) that he was having really bad

days every three days. Thank GOD we weren't on HDD otherwise I

wouldn't have thought it was the MB12. I would have figured the LL

just was causing awful behaviors in him. It was actually your post

that made that connection click in my brain. Since I was draining

the acids in his body, and making him more alkaline, it would make

sense that the MB12 wouldn't be needed as much....or for us, at

all. INteresting thing about the oral MB12 is that my son even

reacted to that after addressing his liver and pH. By restoring the

liver and pH, the body is better able to assimilate nutrients from

the foods we eat or supps we take. We have seen this happening for

many asd kids.

Since the b12 is stored in the liver (as well as the kidneys)if the

liver is not functioning properly, the b12 may not be stored or

maybe can't be utilized. I'd be curious to know which one. :) I

think the yeast overgrowth is a downstream from the liver problems.

(course I don't know) When working properly, the liver clears ninety-

nine percent of the bacteria and other toxins from the blood before

it is allowed to re-enter the general circulation. However, when the

liver is damaged this filtration system breaks down.

I do think, like you said, the viral/heavy metal/chemical overloads

is what puts too much stress on the liver and cause the dysfunction

in many cases. Or maybe some kids were born with a sluggish liver

from the parent's toxicity. (I believe this was my son's case.)

I wonder if the biological difference starts with the liver. I was

looking over medical records a while back and saw doc notes that my

son was born with an enlarged liver. I was never told this. Nothing

was ever done, no tests were taken. Had he not been a c-section

baby, we would never have known cause they took a chest xray to look

at his lungs. If parents, namely mothers, are toxic and have

sluggish livers, the extra toxins would go from the mom to the

baby. So it's not unthinkable that the liver could be impacted

before our kids ever took a breath on this Earth.

For some kids, unless you address the liver dysfunction, it may be

very hard to get the rest of the junk out of the body. It's like

trying to drive home during traffic hour. If there's congestion on

the highways, you won't be getting home too fast no matter how big a

truck you drive. The more people that try to push through, the

bigger the mess. But as soon as the congestion clears, you can

drive home pretty darn fast. (Hoping no cops are in sight!) It's

the same thing when you are trying to detox with a congested liver

and kidneys. THe more you try to push through (chelators,

antifungals, allergy meds), the more backed up they get until

eventually not much is getting through at all. I believe this is

the case for many asd kids. The background of antibiotics, tylenol,

vaccines, chemical toxicities. IT's just so much that the liver

can't keep up.

My best,

TamiW

Gavin's mom

> > > > > >

> > > > > >

> > > > > > 1: Zhongguo Zhong Yao Za Zhi. 1998 Mar;23(3):171-3,

> > > > > >

> > > > > > Antivirus effect of polysaccharides of brewer yeast in

vitro

> > > > > >

> > > > > > The antivirus effect of polysaccharides of brewer yeast

from

> > yeast

> > > > mud

> > > > > > on 13

> > > > > > kinds of viruses including DNA and RNA virus along with

their

> > > > mechanisms

> > > > > > were

> > > > > > studied. The result showed that this effect was

remarkable

> > on the

> > > > > > infections

> > > > > > with poliovirus III, adenovirus III, ECHO6 virus,

> > enterovirus 71,

> > > > > > vesicular

> > > > > > stomatitis virus, herpesvirus I, II, coxsackie A16 virus

and

> > > > coxsackie

> > > > > > B3 virus.

> > > > > > The polysaccharides of brewer yeast could also inhibit

the

> > > > development

> > > > > > of

> > > > > > cytopathic effect(CPE) and protect cultural cells from

being

> > > > infected

> > > > > > with the

> > > > > > above viruses.

> > > > > >

> > > > > > PMID: 11596239 [PubMed - indexed for MEDLINE]

> > > > > >

> > > > >

> > > >

> > >

> >

>

Link to comment
Share on other sites
Guest guest

Guest guest

Posted
Posted

Hi Stan,

What's interesting about the MB12/acid connection and why it sticks

so well in my mind is that we were on MB12 shots for a year. We had

really good results in all areas. When we started the liver life,

we noticed (after a very short time) that he was having really bad

days every three days. Thank GOD we weren't on HDD otherwise I

wouldn't have thought it was the MB12. I would have figured the LL

just was causing awful behaviors in him. It was actually your post

that made that connection click in my brain. Since I was draining

the acids in his body, and making him more alkaline, it would make

sense that the MB12 wouldn't be needed as much....or for us, at

all. INteresting thing about the oral MB12 is that my son even

reacted to that after addressing his liver and pH. By restoring the

liver and pH, the body is better able to assimilate nutrients from

the foods we eat or supps we take. We have seen this happening for

many asd kids.

Since the b12 is stored in the liver (as well as the kidneys)if the

liver is not functioning properly, the b12 may not be stored or

maybe can't be utilized. I'd be curious to know which one. :) I

think the yeast overgrowth is a downstream from the liver problems.

(course I don't know) When working properly, the liver clears ninety-

nine percent of the bacteria and other toxins from the blood before

it is allowed to re-enter the general circulation. However, when the

liver is damaged this filtration system breaks down.

I do think, like you said, the viral/heavy metal/chemical overloads

is what puts too much stress on the liver and cause the dysfunction

in many cases. Or maybe some kids were born with a sluggish liver

from the parent's toxicity. (I believe this was my son's case.)

I wonder if the biological difference starts with the liver. I was

looking over medical records a while back and saw doc notes that my

son was born with an enlarged liver. I was never told this. Nothing

was ever done, no tests were taken. Had he not been a c-section

baby, we would never have known cause they took a chest xray to look

at his lungs. If parents, namely mothers, are toxic and have

sluggish livers, the extra toxins would go from the mom to the

baby. So it's not unthinkable that the liver could be impacted

before our kids ever took a breath on this Earth.

For some kids, unless you address the liver dysfunction, it may be

very hard to get the rest of the junk out of the body. It's like

trying to drive home during traffic hour. If there's congestion on

the highways, you won't be getting home too fast no matter how big a

truck you drive. The more people that try to push through, the

bigger the mess. But as soon as the congestion clears, you can

drive home pretty darn fast. (Hoping no cops are in sight!) It's

the same thing when you are trying to detox with a congested liver

and kidneys. THe more you try to push through (chelators,

antifungals, allergy meds), the more backed up they get until

eventually not much is getting through at all. I believe this is

the case for many asd kids. The background of antibiotics, tylenol,

vaccines, chemical toxicities. IT's just so much that the liver

can't keep up.

My best,

TamiW

Gavin's mom

> > > > > >

> > > > > >

> > > > > > 1: Zhongguo Zhong Yao Za Zhi. 1998 Mar;23(3):171-3,

> > > > > >

> > > > > > Antivirus effect of polysaccharides of brewer yeast in

vitro

> > > > > >

> > > > > > The antivirus effect of polysaccharides of brewer yeast

from

> > yeast

> > > > mud

> > > > > > on 13

> > > > > > kinds of viruses including DNA and RNA virus along with

their

> > > > mechanisms

> > > > > > were

> > > > > > studied. The result showed that this effect was

remarkable

> > on the

> > > > > > infections

> > > > > > with poliovirus III, adenovirus III, ECHO6 virus,

> > enterovirus 71,

> > > > > > vesicular

> > > > > > stomatitis virus, herpesvirus I, II, coxsackie A16 virus

and

> > > > coxsackie

> > > > > > B3 virus.

> > > > > > The polysaccharides of brewer yeast could also inhibit

the

> > > > development

> > > > > > of

> > > > > > cytopathic effect(CPE) and protect cultural cells from

being

> > > > infected

> > > > > > with the

> > > > > > above viruses.

> > > > > >

> > > > > > PMID: 11596239 [PubMed - indexed for MEDLINE]

> > > > > >

> > > > >

> > > >

> > >

> >

>

Link to comment
Share on other sites
Guest guest

Guest guest

Posted
Posted

When you say NT folks with the same viruses, do you mean adults only or

adults AND children? I am not aware of NT kids having viral problems of

that nature - how would they be manifesting (??). It could be (or could

it be?) that child's immune system has a different set up than that of

an adult. Not just ASD children, but kids in general... ??

Also, have a look at one of the abstract below, I found the that VERY

intriguing that brewers yeast helped with mouse-model symptoms of CFS,

especially in the light of newer, and very credible, theories of CFS

being just the 'adult' manifestation of ASD.

more ??? : could the IgG reaction towards brewers yeast that comes up in

test have sth to do with labs/testing methods having it mistaken for

Sac.boulardii (which is very similar, if not identical to Sac.crevisae)?

Would you know if those kids with those test results ever took Sac.b for

any length of time?

Natasa

> > > >

> > > >

> > > > 1: Zhongguo Zhong Yao Za Zhi. 1998 Mar;23(3):171-3,

> > > >

> > > > Antivirus effect of polysaccharides of brewer yeast in vitro

> > > >

> > > > The antivirus effect of polysaccharides of brewer yeast from

yeast

> > mud

> > > > on 13

> > > > kinds of viruses including DNA and RNA virus along with their

> > mechanisms

> > > > were

> > > > studied. The result showed that this effect was remarkable on

the

> > > > infections

> > > > with poliovirus III, adenovirus III, ECHO6 virus, enterovirus

71,

> > > > vesicular

> > > > stomatitis virus, herpesvirus I, II, coxsackie A16 virus and

> > coxsackie

> > > > B3 virus.

> > > > The polysaccharides of brewer yeast could also inhibit the

> > development

> > > > of

> > > > cytopathic effect(CPE) and protect cultural cells from being

> > infected

> > > > with the

> > > > above viruses.

> > > >

> > > > PMID: 11596239 [PubMed - indexed for MEDLINE]

> > > >

> > >

> >

>

Link to comment
Share on other sites
Guest guest

Guest guest

Posted
Posted

When you say NT folks with the same viruses, do you mean adults only or

adults AND children? I am not aware of NT kids having viral problems of

that nature - how would they be manifesting (??). It could be (or could

it be?) that child's immune system has a different set up than that of

an adult. Not just ASD children, but kids in general... ??

Also, have a look at one of the abstract below, I found the that VERY

intriguing that brewers yeast helped with mouse-model symptoms of CFS,

especially in the light of newer, and very credible, theories of CFS

being just the 'adult' manifestation of ASD.

more ??? : could the IgG reaction towards brewers yeast that comes up in

test have sth to do with labs/testing methods having it mistaken for

Sac.boulardii (which is very similar, if not identical to Sac.crevisae)?

Would you know if those kids with those test results ever took Sac.b for

any length of time?

Natasa

> > > >

> > > >

> > > > 1: Zhongguo Zhong Yao Za Zhi. 1998 Mar;23(3):171-3,

> > > >

> > > > Antivirus effect of polysaccharides of brewer yeast in vitro

> > > >

> > > > The antivirus effect of polysaccharides of brewer yeast from

yeast

> > mud

> > > > on 13

> > > > kinds of viruses including DNA and RNA virus along with their

> > mechanisms

> > > > were

> > > > studied. The result showed that this effect was remarkable on

the

> > > > infections

> > > > with poliovirus III, adenovirus III, ECHO6 virus, enterovirus

71,

> > > > vesicular

> > > > stomatitis virus, herpesvirus I, II, coxsackie A16 virus and

> > coxsackie

> > > > B3 virus.

> > > > The polysaccharides of brewer yeast could also inhibit the

> > development

> > > > of

> > > > cytopathic effect(CPE) and protect cultural cells from being

> > infected

> > > > with the

> > > > above viruses.

> > > >

> > > > PMID: 11596239 [PubMed - indexed for MEDLINE]

> > > >

> > >

> >

>

Link to comment
Share on other sites
Guest guest

Guest guest

Posted
Posted

Hi Tami,

We're on the same page.

You wrote:

" I do think, like you said, the viral/heavy metal/chemical overloads

is what puts too much stress on the liver and cause the dysfunction

in many cases. Or maybe some kids were born with a sluggish liver

from the parent's toxicity. (I believe this was my son's case.) "

Response:

Us too. I totally agree with you. My son had jaundice and very fair skin when

he was born.

I believe he started having some liver detox problems prior to birth.

About the liver:

When Binstock and I found a bunch of references to herpes, Valtrex

(valacyclovir)

and hepatic function we were quite jazzed about it. Valtrex is broken down into

acyclovir

in the liver, so it's a very potent antiviral for the liver.

One more element to think about is that gut fungus creates alcohol when any type

of

undigested sugars are around (just go to Napa Valley and do a wine tour and

you'll quickly

learn the ingredients are fungus and sugar). Of course the liver doesn't

respond very well

to chronic alcohol.

So, yes. I agree with you, the liver is likely a central element in some,

possibly many of our

kid's conditions.

- Stan

> > > > > > >

> > > > > > >

> > > > > > > 1: Zhongguo Zhong Yao Za Zhi. 1998 Mar;23(3):171-3,

> > > > > > >

> > > > > > > Antivirus effect of polysaccharides of brewer yeast in

> vitro

> > > > > > >

> > > > > > > The antivirus effect of polysaccharides of brewer yeast

> from

> > > yeast

> > > > > mud

> > > > > > > on 13

> > > > > > > kinds of viruses including DNA and RNA virus along with

> their

> > > > > mechanisms

> > > > > > > were

> > > > > > > studied. The result showed that this effect was

> remarkable

> > > on the

> > > > > > > infections

> > > > > > > with poliovirus III, adenovirus III, ECHO6 virus,

> > > enterovirus 71,

> > > > > > > vesicular

> > > > > > > stomatitis virus, herpesvirus I, II, coxsackie A16 virus

> and

> > > > > coxsackie

> > > > > > > B3 virus.

> > > > > > > The polysaccharides of brewer yeast could also inhibit

> the

> > > > > development

> > > > > > > of

> > > > > > > cytopathic effect(CPE) and protect cultural cells from

> being

> > > > > infected

> > > > > > > with the

> > > > > > > above viruses.

> > > > > > >

> > > > > > > PMID: 11596239 [PubMed - indexed for MEDLINE]

> > > > > > >

> > > > > >

> > > > >

> > > >

> > >

> >

>

Link to comment
Share on other sites
Guest guest

Guest guest

Posted
Posted

Hi Tami,

We're on the same page.

You wrote:

" I do think, like you said, the viral/heavy metal/chemical overloads

is what puts too much stress on the liver and cause the dysfunction

in many cases. Or maybe some kids were born with a sluggish liver

from the parent's toxicity. (I believe this was my son's case.) "

Response:

Us too. I totally agree with you. My son had jaundice and very fair skin when

he was born.

I believe he started having some liver detox problems prior to birth.

About the liver:

When Binstock and I found a bunch of references to herpes, Valtrex

(valacyclovir)

and hepatic function we were quite jazzed about it. Valtrex is broken down into

acyclovir

in the liver, so it's a very potent antiviral for the liver.

One more element to think about is that gut fungus creates alcohol when any type

of

undigested sugars are around (just go to Napa Valley and do a wine tour and

you'll quickly

learn the ingredients are fungus and sugar). Of course the liver doesn't

respond very well

to chronic alcohol.

So, yes. I agree with you, the liver is likely a central element in some,

possibly many of our

kid's conditions.

- Stan

> > > > > > >

> > > > > > >

> > > > > > > 1: Zhongguo Zhong Yao Za Zhi. 1998 Mar;23(3):171-3,

> > > > > > >

> > > > > > > Antivirus effect of polysaccharides of brewer yeast in

> vitro

> > > > > > >

> > > > > > > The antivirus effect of polysaccharides of brewer yeast

> from

> > > yeast

> > > > > mud

> > > > > > > on 13

> > > > > > > kinds of viruses including DNA and RNA virus along with

> their

> > > > > mechanisms

> > > > > > > were

> > > > > > > studied. The result showed that this effect was

> remarkable

> > > on the

> > > > > > > infections

> > > > > > > with poliovirus III, adenovirus III, ECHO6 virus,

> > > enterovirus 71,

> > > > > > > vesicular

> > > > > > > stomatitis virus, herpesvirus I, II, coxsackie A16 virus

> and

> > > > > coxsackie

> > > > > > > B3 virus.

> > > > > > > The polysaccharides of brewer yeast could also inhibit

> the

> > > > > development

> > > > > > > of

> > > > > > > cytopathic effect(CPE) and protect cultural cells from

> being

> > > > > infected

> > > > > > > with the

> > > > > > > above viruses.

> > > > > > >

> > > > > > > PMID: 11596239 [PubMed - indexed for MEDLINE]

> > > > > > >

> > > > > >

> > > > >

> > > >

> > >

> >

>

Link to comment
Share on other sites
Guest guest

Guest guest

Posted
Posted

Regarding the virus in kids...

Yes, I mean both adults and kids regarding the PDR. Neither shows a rash as a

side effect.

also there is no reference in the medical literature about one.

and Yes, I have written responses about the rash effect not happening in the

adults within

the STD community, but there isn't a herpes virus parents group.

There are many neuronatal references in the medical literature about herpes

strain viruses.

How they manifest I would have to read more thoroughly, but from what I

understand they

more are more likely to manifest in the brain or the liver based on the less

develped

immune systems, but I also saw references about eyes and heart and csf as well.

Here I'll

grab some stuff that might help.

------

Here's a recent one:

Reprod Toxicol. 2006 May;21(4):436-45. Epub 2005 Jan 8. Related Articles, Links

Herpes simplex virus and Epstein-Barr virus infections in pregnancy:

consequences of

neonatal or intrauterine infection.

Avgil M, Ornoy A.

Laboratory of Teratology, Hebrew University Hadassah Medical School, Jerusalem,

Israel.

Herpes simplex virus (HSV) type 1/2 and Epstein-Barr virus (EBV) belong to the

human

herpes viruses and are among the most ubiquitous viruses in the adult

population. In spite

of the fact that a large proportion of women at childbearing age are

seropositive to these

viruses, especially to HSV, primary or secondary infections with these viruses

may occur

during pregnancy. Genital HSV, especially in primary infections, may be

dangerous to the

neonate if infected during delivery, as it can cause a severe neonatal disease.

Intrauterine

infection causing abortion or stillbirth as well as skin scars (cutaneous

manifestations),

ophthalmologic findings (chorioretinitis, microphtalmia), and neurological

involvement

(causing brain damage) is also possible, but relatively rare. Primary infection

with EBV

during pregnancy with apparent transplacental transmission is rare and only few

cases

were reported. They main damage was in the heart, eyes and liver. The actual

damage to

the developing embryo and fetus from maternal HSV and EBV seems to be very

small. It

should be mentioned that both HSV and EBV seem to be able to cross the placenta

and

cause, as described by several investigators, placental infection manifested by

deciduitis

and villitis. These placental pathological changes may increase fetal damage.

PMID: 16580943 [PubMed - in process]

--- This one includes a liver failure reference.

J Pediatr Gastroenterol Nutr. 2006 Mar;42(3):282-6. Related Articles, Links

Neonatal herpes simplex virus infection presenting as acute liver failure:

prevalent role of

herpes simplex virus type I.

Verma A, Dhawan A, Zuckerman M, Hadzic N, Baker AJ, Mieli-Vergani G.

Department of Medical Microbiology and Virology, Health Protection Agency,

London,

King's College Hospital, London, UK. anita.verma@...

BACKGROUND: Acute liver failure (ALF) in neonates is rare but carries a high

mortality

without liver transplantation. Herpes simplex virus (HSV) is one of the microbes

that more

commonly causes ALF and is potentially treatable; hence, early diagnosis and

treatment

are important to avoid progression to liver failure. PATIENTS AND RESULTS: We

have

analysed retrospectively the case notes of 11 patients with HSV-induced ALF. A

history of

possible herpes infection was elicited in 5 parents, but HSV had not been

suspected

clinically. All patients were asymptomatic when discharged from postnatal units

and were

presented with nonspecific symptoms of poor feeding and lethargy within 2 weeks

from

birth. Seven of the 11 patients had HSV-1 infection, 4 HSV-2. Only 2 patients

who received

early treatment with intravenous acyclovir survived. CONCLUSIONS: HSV-related

ALF in the

neonatal period carries high morbidity and mortality and needs a high index of

suspicion

so that life-saving treatment can be started promptly. Both HSV-1 and HSV-2 can

cause

severe neonatal infection. It is important to recognise HSV infection in women

of

childbearing age and their sexual partners.

PMID: 16540797 [PubMed - in process]

---- This one mentions chicken pox

Hautarzt. 2006 Mar;57(3):207-16. Related Articles, Links

[infections with herpes simplex and varicella-zoster viruses during pregnancy.]

[Article in German]

Marculescu R, Richter L, Rappersberger K.

Abteilung fur Dermatologie, Krankenanstalt Rudolfstiftung, Wien.

Primary infections with herpes simplex virus (HSV) and varicella-zoster virus

(VZV) may

lead to severe illness in pregnancy. Both diseases may be associated with

transplacental

virus transmission and fetal infection. Such infections can lead to intrauterine

death,

severe malformations and premature birth; the fetal/congenital varicella

syndrome is well-

defined. Herpes genitalis and varicella at the time of labor may lead to life

threatening

neonatal-herpes or varicella of the newborn. Currently neither active

immunization nor

neutralizing immunoglobulin is available for HSV infections. VZV-seronegative

women in

child-bearing age can be vaccinated and pregnant women exposed to VZV can be

given

specific immunoglobulins. While an infection is rarely blocked, the severity is

generally

reduced. For severe disease antiviral treatment is necessary, with valacyclovir

and acyclovir

represents the drugs of choice. Primary or recurrent overt disease of the

genital tract at

the time of delivery an indication for caesarean section. Suppression of

recurrent genital

herpes during the last weeks of pregnancy with valacyclovir and acyclovir

reduces the need

for surgical intervention. Neonates exposed to VZV should receive specific

immunoglobulin. If neonates show signs of either infection, immediate treatment

with

acyclovir must be initiated.

PMID: 16514526 [PubMed - in process]

---- This one mentions csf involvement.

Arch Pediatr. 2005 Oct;12(10):1483-6. Epub 2005 Aug 2. Related Articles, Links

[Herpetic neonatal hepatitis]

[Article in French]

Boumahni B, Djemili S, Gerardin P, Kauffmann E, -Wolter M, Robillard PY,

Fourmaintraux A.

Service de reanimation neonatale et infantile, groupe hospitalier Sud Reunion,

BP 350,

97410 Saint-Pierre, La Reunion, France. boumahni.b@...

Herpes simplex virus (HSV) infection can affect various organs-systems in the

neonatal

period. Herpetic hepatitis was seldom reported in the literature. We report on 2

cases.

Firstly, a 16 day-old newborn infant was admitted because of haemorrhagic

syndrome and

shock. Biological assessment showed a severe hepatic insufficiency. Antibiotic

and

aciclovir therapy was started as HSV infection was suspected. Five days later,

the herpetic

attack was confirmed by polymerase chain reaction (PCR) in blood and

cerebrospinal fluid

(CSF). The genotye of the virus in the CSF was HSV1. Treatment included

aciclovir for 21

days intravenously and 2 months orally. At 10 months, the clinical and

biological

examinations were normal. Secondly, a 4 day-old newborn was hospitalised because

of

fever and polypnea. Pulmonary X rays showed heterogeneous opacities of the right

base.

Serum C reactive protein was 30 mg/l. Antibiotic therapy was started. Two days

later, the

fever persisted while a severe hepatic insufficiency developed. The diagnosis of

herpetic

hepatitis was evoked and the child was given aciclovir. Forty-eight hours later,

the PCR

confirmed a HSV in blood, while viral culture of a mouth swab found HSV 2.

Evolution was

favourable after 21 days of specific and symptomatic treatment. Aciclovir

treatment was

continued orally for six months. Herpetic hepatitis is rare in the neonatal

period. Diagnosis

must be evoked early when facing severe neonatal hepatic insufficiency. Provided

specific

treatment, prognosis is good.

Publication Types:

Case Reports

PMID: 16061366 [PubMed - indexed for MEDLINE]

--- This informative website talks about the possible risks of herpes,

espeically in the

case of immune impairment. It also mentions transmission to newborns.

http://www.gotherpes.com/doc52severity.sht

....Herpes simplex in any patient with a seriously compromised immune system can

cause

serious and even life-threatening complications, including the following:

Pneumonia.

Liver damage, including hepatitis. Hepatitis caused by primary or recurrent HSV

can

sometimes develop into a life-threatening condition called fulminant liver

failure.

Inflammation of the esophagus.

Encephalitis.

Destruction of the adrenal glands.

Increased risk for disseminated herpes and herpes encephalitis.

Less serious conditions include stomach and anal ulcers, inflammation in the

colon, and

eczema herpeticum.

HERPES IN THE PREGNANT WOMAN AND THE NEWBORN

HSV infection in a newborn is a very serious and even-life threatening condition

if it goes

undiagnosed and untreated. Fortunately, since the introduction of acyclovir the

outlook for

these children has significantly improved.

Transmission of HSV to Newborns

HSV infection is uncommon, occurring in between one in 3,500 to 20,000 depending

on

the population group. The greatest danger to the newborn occurs in women who

have a

primary genital infection with an outbreak at the time of delivery. In such

cases, about half

of the newborns become infected. The lowest risk (less than 4%) to the child of

an infected

mother is from shedding during a recurring non-symptomatic infection.

Unfortunately, many women whose newborn infants develop HSV infection have no

history

of herpes and or fail to recognize symptoms at the time of delivery.

Occasionally, lesions

on the mother's buttocks may help indicate the presence of the virus. The risk

for

transmission increases if infants with infected mothers are born prematurely or

instruments are required during vaginal delivery. Transmission can occur if the

amniotic

membrane of an infected woman ruptures prematurely, or as the infant passes

through an

infected birth canal. Very rarely, the virus is transmitted across the placenta,

a form of the

infection known as congenital herpes.

- Stan

> > > > >

> > > > >

> > > > > 1: Zhongguo Zhong Yao Za Zhi. 1998 Mar;23(3):171-3,

> > > > >

> > > > > Antivirus effect of polysaccharides of brewer yeast in vitro

> > > > >

> > > > > The antivirus effect of polysaccharides of brewer yeast from

> yeast

> > > mud

> > > > > on 13

> > > > > kinds of viruses including DNA and RNA virus along with their

> > > mechanisms

> > > > > were

> > > > > studied. The result showed that this effect was remarkable on

> the

> > > > > infections

> > > > > with poliovirus III, adenovirus III, ECHO6 virus, enterovirus

> 71,

> > > > > vesicular

> > > > > stomatitis virus, herpesvirus I, II, coxsackie A16 virus and

> > > coxsackie

> > > > > B3 virus.

> > > > > The polysaccharides of brewer yeast could also inhibit the

> > > development

> > > > > of

> > > > > cytopathic effect(CPE) and protect cultural cells from being

> > > infected

> > > > > with the

> > > > > above viruses.

> > > > >

> > > > > PMID: 11596239 [PubMed - indexed for MEDLINE]

> > > > >

> > > >

> > >

> >

>

Link to comment
Share on other sites
Guest guest

Guest guest

Posted
Posted

Regarding the virus in kids...

Yes, I mean both adults and kids regarding the PDR. Neither shows a rash as a

side effect.

also there is no reference in the medical literature about one.

and Yes, I have written responses about the rash effect not happening in the

adults within

the STD community, but there isn't a herpes virus parents group.

There are many neuronatal references in the medical literature about herpes

strain viruses.

How they manifest I would have to read more thoroughly, but from what I

understand they

more are more likely to manifest in the brain or the liver based on the less

develped

immune systems, but I also saw references about eyes and heart and csf as well.

Here I'll

grab some stuff that might help.

------

Here's a recent one:

Reprod Toxicol. 2006 May;21(4):436-45. Epub 2005 Jan 8. Related Articles, Links

Herpes simplex virus and Epstein-Barr virus infections in pregnancy:

consequences of

neonatal or intrauterine infection.

Avgil M, Ornoy A.

Laboratory of Teratology, Hebrew University Hadassah Medical School, Jerusalem,

Israel.

Herpes simplex virus (HSV) type 1/2 and Epstein-Barr virus (EBV) belong to the

human

herpes viruses and are among the most ubiquitous viruses in the adult

population. In spite

of the fact that a large proportion of women at childbearing age are

seropositive to these

viruses, especially to HSV, primary or secondary infections with these viruses

may occur

during pregnancy. Genital HSV, especially in primary infections, may be

dangerous to the

neonate if infected during delivery, as it can cause a severe neonatal disease.

Intrauterine

infection causing abortion or stillbirth as well as skin scars (cutaneous

manifestations),

ophthalmologic findings (chorioretinitis, microphtalmia), and neurological

involvement

(causing brain damage) is also possible, but relatively rare. Primary infection

with EBV

during pregnancy with apparent transplacental transmission is rare and only few

cases

were reported. They main damage was in the heart, eyes and liver. The actual

damage to

the developing embryo and fetus from maternal HSV and EBV seems to be very

small. It

should be mentioned that both HSV and EBV seem to be able to cross the placenta

and

cause, as described by several investigators, placental infection manifested by

deciduitis

and villitis. These placental pathological changes may increase fetal damage.

PMID: 16580943 [PubMed - in process]

--- This one includes a liver failure reference.

J Pediatr Gastroenterol Nutr. 2006 Mar;42(3):282-6. Related Articles, Links

Neonatal herpes simplex virus infection presenting as acute liver failure:

prevalent role of

herpes simplex virus type I.

Verma A, Dhawan A, Zuckerman M, Hadzic N, Baker AJ, Mieli-Vergani G.

Department of Medical Microbiology and Virology, Health Protection Agency,

London,

King's College Hospital, London, UK. anita.verma@...

BACKGROUND: Acute liver failure (ALF) in neonates is rare but carries a high

mortality

without liver transplantation. Herpes simplex virus (HSV) is one of the microbes

that more

commonly causes ALF and is potentially treatable; hence, early diagnosis and

treatment

are important to avoid progression to liver failure. PATIENTS AND RESULTS: We

have

analysed retrospectively the case notes of 11 patients with HSV-induced ALF. A

history of

possible herpes infection was elicited in 5 parents, but HSV had not been

suspected

clinically. All patients were asymptomatic when discharged from postnatal units

and were

presented with nonspecific symptoms of poor feeding and lethargy within 2 weeks

from

birth. Seven of the 11 patients had HSV-1 infection, 4 HSV-2. Only 2 patients

who received

early treatment with intravenous acyclovir survived. CONCLUSIONS: HSV-related

ALF in the

neonatal period carries high morbidity and mortality and needs a high index of

suspicion

so that life-saving treatment can be started promptly. Both HSV-1 and HSV-2 can

cause

severe neonatal infection. It is important to recognise HSV infection in women

of

childbearing age and their sexual partners.

PMID: 16540797 [PubMed - in process]

---- This one mentions chicken pox

Hautarzt. 2006 Mar;57(3):207-16. Related Articles, Links

[infections with herpes simplex and varicella-zoster viruses during pregnancy.]

[Article in German]

Marculescu R, Richter L, Rappersberger K.

Abteilung fur Dermatologie, Krankenanstalt Rudolfstiftung, Wien.

Primary infections with herpes simplex virus (HSV) and varicella-zoster virus

(VZV) may

lead to severe illness in pregnancy. Both diseases may be associated with

transplacental

virus transmission and fetal infection. Such infections can lead to intrauterine

death,

severe malformations and premature birth; the fetal/congenital varicella

syndrome is well-

defined. Herpes genitalis and varicella at the time of labor may lead to life

threatening

neonatal-herpes or varicella of the newborn. Currently neither active

immunization nor

neutralizing immunoglobulin is available for HSV infections. VZV-seronegative

women in

child-bearing age can be vaccinated and pregnant women exposed to VZV can be

given

specific immunoglobulins. While an infection is rarely blocked, the severity is

generally

reduced. For severe disease antiviral treatment is necessary, with valacyclovir

and acyclovir

represents the drugs of choice. Primary or recurrent overt disease of the

genital tract at

the time of delivery an indication for caesarean section. Suppression of

recurrent genital

herpes during the last weeks of pregnancy with valacyclovir and acyclovir

reduces the need

for surgical intervention. Neonates exposed to VZV should receive specific

immunoglobulin. If neonates show signs of either infection, immediate treatment

with

acyclovir must be initiated.

PMID: 16514526 [PubMed - in process]

---- This one mentions csf involvement.

Arch Pediatr. 2005 Oct;12(10):1483-6. Epub 2005 Aug 2. Related Articles, Links

[Herpetic neonatal hepatitis]

[Article in French]

Boumahni B, Djemili S, Gerardin P, Kauffmann E, -Wolter M, Robillard PY,

Fourmaintraux A.

Service de reanimation neonatale et infantile, groupe hospitalier Sud Reunion,

BP 350,

97410 Saint-Pierre, La Reunion, France. boumahni.b@...

Herpes simplex virus (HSV) infection can affect various organs-systems in the

neonatal

period. Herpetic hepatitis was seldom reported in the literature. We report on 2

cases.

Firstly, a 16 day-old newborn infant was admitted because of haemorrhagic

syndrome and

shock. Biological assessment showed a severe hepatic insufficiency. Antibiotic

and

aciclovir therapy was started as HSV infection was suspected. Five days later,

the herpetic

attack was confirmed by polymerase chain reaction (PCR) in blood and

cerebrospinal fluid

(CSF). The genotye of the virus in the CSF was HSV1. Treatment included

aciclovir for 21

days intravenously and 2 months orally. At 10 months, the clinical and

biological

examinations were normal. Secondly, a 4 day-old newborn was hospitalised because

of

fever and polypnea. Pulmonary X rays showed heterogeneous opacities of the right

base.

Serum C reactive protein was 30 mg/l. Antibiotic therapy was started. Two days

later, the

fever persisted while a severe hepatic insufficiency developed. The diagnosis of

herpetic

hepatitis was evoked and the child was given aciclovir. Forty-eight hours later,

the PCR

confirmed a HSV in blood, while viral culture of a mouth swab found HSV 2.

Evolution was

favourable after 21 days of specific and symptomatic treatment. Aciclovir

treatment was

continued orally for six months. Herpetic hepatitis is rare in the neonatal

period. Diagnosis

must be evoked early when facing severe neonatal hepatic insufficiency. Provided

specific

treatment, prognosis is good.

Publication Types:

Case Reports

PMID: 16061366 [PubMed - indexed for MEDLINE]

--- This informative website talks about the possible risks of herpes,

espeically in the

case of immune impairment. It also mentions transmission to newborns.

http://www.gotherpes.com/doc52severity.sht

....Herpes simplex in any patient with a seriously compromised immune system can

cause

serious and even life-threatening complications, including the following:

Pneumonia.

Liver damage, including hepatitis. Hepatitis caused by primary or recurrent HSV

can

sometimes develop into a life-threatening condition called fulminant liver

failure.

Inflammation of the esophagus.

Encephalitis.

Destruction of the adrenal glands.

Increased risk for disseminated herpes and herpes encephalitis.

Less serious conditions include stomach and anal ulcers, inflammation in the

colon, and

eczema herpeticum.

HERPES IN THE PREGNANT WOMAN AND THE NEWBORN

HSV infection in a newborn is a very serious and even-life threatening condition

if it goes

undiagnosed and untreated. Fortunately, since the introduction of acyclovir the

outlook for

these children has significantly improved.

Transmission of HSV to Newborns

HSV infection is uncommon, occurring in between one in 3,500 to 20,000 depending

on

the population group. The greatest danger to the newborn occurs in women who

have a

primary genital infection with an outbreak at the time of delivery. In such

cases, about half

of the newborns become infected. The lowest risk (less than 4%) to the child of

an infected

mother is from shedding during a recurring non-symptomatic infection.

Unfortunately, many women whose newborn infants develop HSV infection have no

history

of herpes and or fail to recognize symptoms at the time of delivery.

Occasionally, lesions

on the mother's buttocks may help indicate the presence of the virus. The risk

for

transmission increases if infants with infected mothers are born prematurely or

instruments are required during vaginal delivery. Transmission can occur if the

amniotic

membrane of an infected woman ruptures prematurely, or as the infant passes

through an

infected birth canal. Very rarely, the virus is transmitted across the placenta,

a form of the

infection known as congenital herpes.

- Stan

> > > > >

> > > > >

> > > > > 1: Zhongguo Zhong Yao Za Zhi. 1998 Mar;23(3):171-3,

> > > > >

> > > > > Antivirus effect of polysaccharides of brewer yeast in vitro

> > > > >

> > > > > The antivirus effect of polysaccharides of brewer yeast from

> yeast

> > > mud

> > > > > on 13

> > > > > kinds of viruses including DNA and RNA virus along with their

> > > mechanisms

> > > > > were

> > > > > studied. The result showed that this effect was remarkable on

> the

> > > > > infections

> > > > > with poliovirus III, adenovirus III, ECHO6 virus, enterovirus

> 71,

> > > > > vesicular

> > > > > stomatitis virus, herpesvirus I, II, coxsackie A16 virus and

> > > coxsackie

> > > > > B3 virus.

> > > > > The polysaccharides of brewer yeast could also inhibit the

> > > development

> > > > > of

> > > > > cytopathic effect(CPE) and protect cultural cells from being

> > > infected

> > > > > with the

> > > > > above viruses.

> > > > >

> > > > > PMID: 11596239 [PubMed - indexed for MEDLINE]

> > > > >

> > > >

> > >

> >

>

Link to comment
Share on other sites
Guest guest

Guest guest

Posted
Posted

Thanks Stan for your insight. I've ordered the Valtrex and am

impatiently waiting for it to get here. :) I am hoping that since

we've been addressing his liver, he won't have " that bad " of a

regression. I suspect he's a big viral kid. Whenever we try to

increase LDN to full dose, he breaks out in cold sores all over his

mouth. If he didn't have such painful reactions (gut issues like

constipation and bloating but very severe), I would of course, let

that happen and run it's course. But he physically just can't handle

it.

You said:

" Valtrex is broken down into acyclovir in the liver, so it's a very

potent antiviral for the liver. "

I read that in the files where you said Valtrex turns into three

times the amount of acyclovir in the body but less toxic. That's

very exciting. I also " think " I read somewhere that the " Valtrex

hasn't been hard on the liver like docs were expecting. It seems to

be helping the liver functions. " Did you write something about

that. I might have read it on another site. I've been trying to

find it with no luck! The fact that it crosses the blood brain

barrier is exciting too!

Thanks for the alcohol info. It makes a lot of sense. Maybe our kids

are all " weird " while they have yeast is because they're drunk! :)

My best,

TamiW

Gavin's mom

> > > > > > > >

> > > > > > > >

> > > > > > > > 1: Zhongguo Zhong Yao Za Zhi. 1998 Mar;23(3):171-3,

> > > > > > > >

> > > > > > > > Antivirus effect of polysaccharides of brewer yeast

in

> > vitro

> > > > > > > >

> > > > > > > > The antivirus effect of polysaccharides of brewer

yeast

> > from

> > > > yeast

> > > > > > mud

> > > > > > > > on 13

> > > > > > > > kinds of viruses including DNA and RNA virus along

with

> > their

> > > > > > mechanisms

> > > > > > > > were

> > > > > > > > studied. The result showed that this effect was

> > remarkable

> > > > on the

> > > > > > > > infections

> > > > > > > > with poliovirus III, adenovirus III, ECHO6 virus,

> > > > enterovirus 71,

> > > > > > > > vesicular

> > > > > > > > stomatitis virus, herpesvirus I, II, coxsackie A16

virus

> > and

> > > > > > coxsackie

> > > > > > > > B3 virus.

> > > > > > > > The polysaccharides of brewer yeast could also

inhibit

> > the

> > > > > > development

> > > > > > > > of

> > > > > > > > cytopathic effect(CPE) and protect cultural cells

from

> > being

> > > > > > infected

> > > > > > > > with the

> > > > > > > > above viruses.

> > > > > > > >

> > > > > > > > PMID: 11596239 [PubMed - indexed for MEDLINE]

> > > > > > > >

> > > > > > >

> > > > > >

> > > > >

> > > >

> > >

> >

>

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Guest guest

Guest guest

Posted
Posted

Thanks Stan for your insight. I've ordered the Valtrex and am

impatiently waiting for it to get here. :) I am hoping that since

we've been addressing his liver, he won't have " that bad " of a

regression. I suspect he's a big viral kid. Whenever we try to

increase LDN to full dose, he breaks out in cold sores all over his

mouth. If he didn't have such painful reactions (gut issues like

constipation and bloating but very severe), I would of course, let

that happen and run it's course. But he physically just can't handle

it.

You said:

" Valtrex is broken down into acyclovir in the liver, so it's a very

potent antiviral for the liver. "

I read that in the files where you said Valtrex turns into three

times the amount of acyclovir in the body but less toxic. That's

very exciting. I also " think " I read somewhere that the " Valtrex

hasn't been hard on the liver like docs were expecting. It seems to

be helping the liver functions. " Did you write something about

that. I might have read it on another site. I've been trying to

find it with no luck! The fact that it crosses the blood brain

barrier is exciting too!

Thanks for the alcohol info. It makes a lot of sense. Maybe our kids

are all " weird " while they have yeast is because they're drunk! :)

My best,

TamiW

Gavin's mom

> > > > > > > >

> > > > > > > >

> > > > > > > > 1: Zhongguo Zhong Yao Za Zhi. 1998 Mar;23(3):171-3,

> > > > > > > >

> > > > > > > > Antivirus effect of polysaccharides of brewer yeast

in

> > vitro

> > > > > > > >

> > > > > > > > The antivirus effect of polysaccharides of brewer

yeast

> > from

> > > > yeast

> > > > > > mud

> > > > > > > > on 13

> > > > > > > > kinds of viruses including DNA and RNA virus along

with

> > their

> > > > > > mechanisms

> > > > > > > > were

> > > > > > > > studied. The result showed that this effect was

> > remarkable

> > > > on the

> > > > > > > > infections

> > > > > > > > with poliovirus III, adenovirus III, ECHO6 virus,

> > > > enterovirus 71,

> > > > > > > > vesicular

> > > > > > > > stomatitis virus, herpesvirus I, II, coxsackie A16

virus

> > and

> > > > > > coxsackie

> > > > > > > > B3 virus.

> > > > > > > > The polysaccharides of brewer yeast could also

inhibit

> > the

> > > > > > development

> > > > > > > > of

> > > > > > > > cytopathic effect(CPE) and protect cultural cells

from

> > being

> > > > > > infected

> > > > > > > > with the

> > > > > > > > above viruses.

> > > > > > > >

> > > > > > > > PMID: 11596239 [PubMed - indexed for MEDLINE]

> > > > > > > >

> > > > > > >

> > > > > >

> > > > >

> > > >

> > >

> >

>

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Guest guest

Guest guest

Posted
Posted

Hi Tami,

Yep. Many parents say their kids look drunk... hyper, aggressive, impulsive,

poor motor

skills.....

Yes, I may have said that about Valtrex and the liver.

- Stan

> > > > > > > > >

> > > > > > > > >

> > > > > > > > > 1: Zhongguo Zhong Yao Za Zhi. 1998 Mar;23(3):171-3,

> > > > > > > > >

> > > > > > > > > Antivirus effect of polysaccharides of brewer yeast

> in

> > > vitro

> > > > > > > > >

> > > > > > > > > The antivirus effect of polysaccharides of brewer

> yeast

> > > from

> > > > > yeast

> > > > > > > mud

> > > > > > > > > on 13

> > > > > > > > > kinds of viruses including DNA and RNA virus along

> with

> > > their

> > > > > > > mechanisms

> > > > > > > > > were

> > > > > > > > > studied. The result showed that this effect was

> > > remarkable

> > > > > on the

> > > > > > > > > infections

> > > > > > > > > with poliovirus III, adenovirus III, ECHO6 virus,

> > > > > enterovirus 71,

> > > > > > > > > vesicular

> > > > > > > > > stomatitis virus, herpesvirus I, II, coxsackie A16

> virus

> > > and

> > > > > > > coxsackie

> > > > > > > > > B3 virus.

> > > > > > > > > The polysaccharides of brewer yeast could also

> inhibit

> > > the

> > > > > > > development

> > > > > > > > > of

> > > > > > > > > cytopathic effect(CPE) and protect cultural cells

> from

> > > being

> > > > > > > infected

> > > > > > > > > with the

> > > > > > > > > above viruses.

> > > > > > > > >

> > > > > > > > > PMID: 11596239 [PubMed - indexed for MEDLINE]

> > > > > > > > >

> > > > > > > >

> > > > > > >

> > > > > >

> > > > >

> > > >

> > >

> >

>

Link to comment
Share on other sites
Guest guest

Guest guest

Posted
Posted

Hi Tami,

Yep. Many parents say their kids look drunk... hyper, aggressive, impulsive,

poor motor

skills.....

Yes, I may have said that about Valtrex and the liver.

- Stan

> > > > > > > > >

> > > > > > > > >

> > > > > > > > > 1: Zhongguo Zhong Yao Za Zhi. 1998 Mar;23(3):171-3,

> > > > > > > > >

> > > > > > > > > Antivirus effect of polysaccharides of brewer yeast

> in

> > > vitro

> > > > > > > > >

> > > > > > > > > The antivirus effect of polysaccharides of brewer

> yeast

> > > from

> > > > > yeast

> > > > > > > mud

> > > > > > > > > on 13

> > > > > > > > > kinds of viruses including DNA and RNA virus along

> with

> > > their

> > > > > > > mechanisms

> > > > > > > > > were

> > > > > > > > > studied. The result showed that this effect was

> > > remarkable

> > > > > on the

> > > > > > > > > infections

> > > > > > > > > with poliovirus III, adenovirus III, ECHO6 virus,

> > > > > enterovirus 71,

> > > > > > > > > vesicular

> > > > > > > > > stomatitis virus, herpesvirus I, II, coxsackie A16

> virus

> > > and

> > > > > > > coxsackie

> > > > > > > > > B3 virus.

> > > > > > > > > The polysaccharides of brewer yeast could also

> inhibit

> > > the

> > > > > > > development

> > > > > > > > > of

> > > > > > > > > cytopathic effect(CPE) and protect cultural cells

> from

> > > being

> > > > > > > infected

> > > > > > > > > with the

> > > > > > > > > above viruses.

> > > > > > > > >

> > > > > > > > > PMID: 11596239 [PubMed - indexed for MEDLINE]

> > > > > > > > >

> > > > > > > >

> > > > > > >

> > > > > >

> > > > >

> > > >

> > >

> >

>

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