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Re: glutathione?

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no, your body needs N-acetylcysteine, L glutamine and glycine to make

glutathione according to an old post I had from Andy. He said that giving

glutathione by itself is not absorbed well, it's digested in the gut. I am

confused why it it listed in the new protocol to be given that way that it

is, doesn't make much sense, Val

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The only thing is know about cysteine is that sometimes it builds up and

supplimenting it makes things worse. It is an excitory amimo. Why it builds

up in certain cases I don't know . someone else may know and could tell

both of us. kelly

<< no, your body needs N-acetylcysteine, L glutamine and glycine to make

glutathione according to an old post I had from Andy. He said that giving

glutathione by itself is not absorbed well, it's digested in the gut. I am

confused why it it listed in the new protocol to be given that way that it

is, doesn't make much sense, Val >>

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In a message dated 10/17/2000 11:47:33 PM Eastern Daylight Time,

happymomto4@... writes:

<<

Does anyone know--is glutathione the same thing as L-glutamine?

Thanks. M >>

No wendy it is different. gluthathione is a peptide that rids the body of

unwanted stuff . so technical I can't stand it. L-glutamime is an amino

acid that is a component of gluthatione as is glycine and cysteine. kelly

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In a message dated 10/18/00 7:51:19 AM Central Daylight Time, scromb@...

writes:

<< no, your body needs N-acetylcysteine, L glutamine and glycine to make

glutathione according to an old post I had from Andy. He said that giving

glutathione by itself is not absorbed well, it's digested in the gut. I am

confused why it it listed in the new protocol to be given that way that it

is, doesn't make much sense, Val >>

They're probably talking about Reduced L-Glutathione which is a mixture of

the ingredients you listed above that are used by the body to make

l-glutathione.

Gaylen

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In a message dated 10/18/2000 10:43:33 AM Eastern Daylight Time,

Nomoremetals@... writes:

<< hey're probably talking about Reduced L-Glutathione which is a mixture of

the ingredients you listed above that are used by the body to make

l-glutathione.

Gaylen >>

t hat what was asked How to make L-gluthatione. glycine, cysteine and

glutamine makes gluthathione.

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  • 7 years later...

HI,

I'm not familiar with Nutrical (when I Googled it, it came up as an animal

product)

But as far as glutathione, my naturopath recommended I take 600mg pills 3X

daily of NAC, N-acetylCysteine, instead. She said that the human body uses

the NAC to make its own glutathione, whereas if you buy glutathione itself,

it is prone to easy oxidation, which negates its affect as an anti-oxidant.

(I do know someone who was sicke nough, however, to get glutathione IV for

two weeks straight, but it was obviously under a doctor's care.

The reason I take it is that I have MCS, and that the glutathione levels in

the liver of someone with MCS are low, and can't help get toxins out of

there well enough.

I would love to hear others' comments on this.

Deb

On Fri, Oct 17, 2008 at 1:39 AM, healthyhayashis <amhayashi@...>wrote:

> Have any of you heard of Nutrical? I may be WAY behind, I just found

> it online and was wondering what you thought of it? Does it work?

>

>

>

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Gosh, I am such a spaz!! I don't know where the heck the name

nutrical came from!!!! The product I meant to inquire about is

called immunocal!!!!! Sorry for the mix up!!!

Adrean

>

> HI,

>

> I'm not familiar with Nutrical (when I Googled it, it came up as an

animal

> product)

>

> But as far as glutathione, my naturopath recommended I take 600mg

pills 3X

> daily of NAC, N-acetylCysteine, instead. She said that the human

body uses

> the NAC to make its own glutathione, whereas if you buy glutathione

itself,

> it is prone to easy oxidation, which negates its affect as an anti-

oxidant.

> (I do know someone who was sicke nough, however, to get glutathione

IV for

> two weeks straight, but it was obviously under a doctor's care.

>

> The reason I take it is that I have MCS, and that the glutathione

levels in

> the liver of someone with MCS are low, and can't help get toxins

out of

> there well enough.

>

> I would love to hear others' comments on this.

>

> Deb

>

>

> On Fri, Oct 17, 2008 at 1:39 AM, healthyhayashis

<amhayashi@...>wrote:

>

> > Have any of you heard of Nutrical? I may be WAY behind, I just

found

> > it online and was wondering what you thought of it? Does it work?

> >

> >

> >

>

>

>

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Deb,

You might want to check this paper out.

I am sure that NAC is very useful to people recovering from sick buildings..

It has been for me..

A few months ago I saw ths paper which describes a very interesting aspect

of NAC in toxicology..

Its very technical but you might want to check it out..

Chemically Diverse Toxicants Converge on Fyn and c-Cbl to Disrupt Precursor

Cell Function

http://biology.plosjournals.org/perlserv/?request=get-document & doi=10.1371/journ\

al.pbio.0050035

Zaibo Li, Tiefei Dong, Pröschel, Mark

Noble*<http://biology.plosjournals.org/perlserv/?request=get-document & doi=10.137\

1/journal.pbio.0050035#cor1>

*1* Department of Biomedical Genetics, University of Rochester Medical

Center, Rochester, New York, United States of America

Identification of common mechanistic principles that shed light on the

action of the many chemically diverse toxicants to which we are exposed is

of central importance in understanding how toxicants disrupt normal cellular

function and in developing more effective means of protecting against such

effects. Of particular importance is identifying mechanisms operative at

environmentally relevant toxicant exposure levels. Chemically diverse

toxicants exhibit striking convergence, at environmentally relevant exposure

levels, on pathway-specific disruption of receptor tyrosine kinase (RTK)

signaling required for cell division in central nervous system (CNS)

progenitor cells. Relatively small toxicant-induced increases in oxidative

status are associated with Fyn kinase activation, leading to secondary

activation of the c-Cbl ubiquitin ligase. Fyn/c-Cbl pathway activation by

these pro-oxidative changes causes specific reductions, in vitro and in

vivo, in levels of the c-Cbl target platelet-derived growth factor

receptor-α and other c-Cbl targets, but not of the TrkC RTK (which is not a

c-Cbl target). Sequential Fyn and c-Cbl activation, with consequent

pathway-specific suppression of RTK signaling, is induced by levels of

methylmercury and lead that affect large segments of the population, as well

as by paraquat, an organic herbicide. Our results identify a novel

regulatory pathway of oxidant-mediated Fyn/c-Cbl activation as a shared

mechanism of action of chemically diverse toxicants at environmentally

relevant levels, and as a means by which increased oxidative status may

disrupt mitogenic signaling. These results provide one of a small number of

general mechanistic principles in toxicology, and the only such principle

integrating toxicology, precursor cell biology, redox biology, and signaling

pathway analysis in a predictive framework of broad potential relevance to

the understanding of pro-oxidant–mediated disruption of normal development.

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