Guest guest Posted May 14, 2012 Report Share Posted May 14, 2012 Maybe this starts to explains why my K has always been normal. These 8 PA/SCS patients were significantly older and had larger tumor, higher serum potassium levels, lower basal plasma levels of aldosterone, ACTH and DHEA-S as well as lower response of aldosterone after ACTH stimulation than those in 12 patients with aldosterone-producing adenoma without hypercortisolism. Source: https://www.jstage.jst.go.jp/article/endocrj/advpub/0/advpub_K10E-402/_article " Clinicopathological features of primary aldosteronism associated with subclinical Cushing's syndrome " Quote Link to comment Share on other sites More sharing options...
Guest guest Posted May 15, 2012 Report Share Posted May 15, 2012 Interesting but I would expect if one had both one would have lower K as both cortisol and aldo can lower K.Of course in SC there should be no low K as is true of the normokalemic phase of PA. So we must wait and see I guess. CE Grim MDOn May 14, 2012, at 7:17 PM, wrote: Maybe this starts to explains why my K has always been normal. These 8 PA/SCS patients were significantly older and had larger tumor, higher serum potassium levels, lower basal plasma levels of aldosterone, ACTH and DHEA-S as well as lower response of aldosterone after ACTH stimulation than those in 12 patients with aldosterone-producing adenoma without hypercortisolism. Source: https://www.jstage.jst.go.jp/article/endocrj/advpub/0/advpub_K10E-402/_article "Clinicopathological features of primary aldosteronism associated with subclinical Cushing's syndrome" Quote Link to comment Share on other sites More sharing options...
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