A Theory as to Why KT helps Diabetes

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Hi Everybody,

KT is a known antiviral, antifungal, and antibacterial. See

http://www.gaiaresearch.co.za/kombucha.html

Here is a medical article by Koch, M.D. as to the infectious etiology

(causation) of diabetes, which would explain why KT helps eradicate it. Dr. Koch

invented stabilized liquid oxygen as substitute for hydrogen peroxide since H202

is hard on the stomach, and also did pioneering work on cancer.

He too, felt that cancer was caused by a fungal infection, which would explain

why KT works on it.

AN EFFICIENT SINGLE DOSE TREATMENT FOR DIABETES,

On A Full Carbohydrate Diet Without Insulin

By F. Koch, Ph.D., M.D.

http://www.williamfkoch.com/textframe.html

COPYRIGHTED 1941

The period of observation includes scattered cases treated since 1922 and recent

systematic studies. The cases treated cover about every type known, including a

few of diabetes insipidus.

The treatment material consists of catalytic dilutions of the carriers of

aerobic oxidation which we have described in the past elsewhere. (1) These

substances are 1:4 B Benzoquinone and its transition products Glyoxylide,

(O=C=C=O), and Malonide, (O=C=C=C=O), and also Ketene. Their chemical structures

conform to the rules we have laid down as requisite to the production of

immunity against disease; namely, they possess the smallest molecular weight

possible; they possess carbonyl groups that share ethylene linkages or carbonyl

groups that are conjugated with ethylene linkages in molecules that can yield

carbonyl groups sharing ethylene linkages. (2)

Substances of this type can be extracted from the heart and brain. Such extracts

demonstrate the same curative results as the synthetic products and, like them,

correct a definite blood coagulation deficiency. Because of this physiological

property, I named them " Tissue Thrombin " and showed that they are able to cure

cancer, in a paper published in the New York Medical Record in October, 1920.

The synthetic products can be used with good control while the tissue extracts

are very unreliable and have only academic interests. Vitamin K offers very weak

protective properties in addition to its influence on blood coagulation and in

its little way confirms what we have been teaching for years with, reference to

carbonyl and ethylene groups in the catalysis of the oxidations. There is

beginning to be a general appreciation of the significance of these findings.

This is important, since it is our mission to demonstrate that the common basis

of disease of all kinds; including diabetes mellitus and diabetes insipidus, is

a specific defect in the oxidation catalysis.

The clinical data indicate that the cause of diabetes is the prolonged poisoning

of the tissues by bacterial products liberated in scars that have scanty

circulation. These products are intended to serve the nutrition of the germs

that produce them and are originally of fairly small molecular weight,

diffusible, and fully oxidizable by the oxidation mechanism of healthy tissues.

However, when secreted under the anaerobic conditions prevailing in scars they

are not burned, but instead some of their free valencies yield to

polymerizations by which the molecular weight increases progressively while the

photochemic properties vary also with the different stages in the

polymerizations. The different photochemic values have different pathogenic

powers, and so as the polymerizations progress, the patient passes through a

series of different pathogenic influences that produce different

symptomatologies and physical changes.

Diabetes is one of these effects, with a predilection to express itself in

certain persons and races. Neuritis, various degenerative diseases, psoriasis

and other changes may be exhibited, but the final change in all, if the patient

lives long enough to develop it, is cancer. Here too the hereditary factor plays

a part, for we have observed that successive generations tend to develop

malignant growths earlier and earlier in life and the longer the disease has

expressed itself in the ancestry the shorter is the pre-growth, toxic period

that exhibits diabetes or the other changes. (3)

The oxidation catalysts we use therapeutically are de-polymerizing agents and,

while the toxin is being depolymerized to its simple burnable structure, it

passes through, the various phases that were active during the pathogenesis, and

so fleeting recurrence of these changes come, and disappear in the reversed

order while recovery is going on. Thus, when the patient is treated in the

cancer stage of the poisoning and retraces the symptomatology of the

pathogenesis, the last toxic expression to come is the first to go and the first

to come is the last to go. Therefore, an acute inflammation in some old cicatrix

that imprisoned the causative infection is the last change to take place. After

the germ's nutritional agents have been oxidized the germ dies and the scar

which imprisoned it becomes obsolete and is absorbed. Thus, with the destruction

of the etiological factor, the disease is completely cured. When some form of

diabetes is one of the pre-growth symptoms it is overcome before the scar is

cleaned out and, since this happens soon after the cancer growth is absorbed, we

conclude that the molecular weight of the toxin at this stage is quite great.

Cases of diabetes cured by this treatment, before they can develop cancer, may

pass through other expressions of the intoxication producible by greater or

lesser molecular weights, thus the neuritis will get well before the diabetes

and the obliterative endarteritis will start to heal before the blood sugar is

normal. But a psoriasis or other change may show up transiently after the blood

sugar has become normal. Therefore, diabetes must be studied as a phase of a

systemic intoxication and cure cannot be fully established until the focus of

infection has been wiped out. Thus the recovery is not measured alone by the

return of the blood sugar to normal.

Polymerizations of pathogenic toxins, therefore, appear to us to account for the

different stages of chronic diseases, such as the several stages of syphilis,

the different phases of malaria and of the " Fourth Disease, " etc. It accounts

also for the creation of pathogenic viruses. Certain polymerization phases

have-specific pathogenic action, while others have no action at all. The

rapidity of the recovery from virus caused disease after one dose of our

Benzoquinone, solution or one of the transition forms, Glyoxylide or Malonide,

can only be accounted for by this assumption, for recovery from early acute

infantile paralysis has taken place in twenty-four, hours and measles recovers

regularly in twelve hours. The dreaded tropical pemphigus begins to improve in

twenty-four hours and recovers in a few weeks, with ultimate full restoration of

the skin. The virus action is probably destroyed in the first three or four

days, so far as the " Wild-fire " itself is concerned.

Obliterative endarteritis, which is a specific effect of the poison that causes

diabetes, we regard as the lesion that specifically indicates a depression of

the oxidation catalysis of the tissues, general or local. Therefore, this lesion

is a definite indication that the oxidation catalysts we have contributed to

medicine should be employed to remove the basic cause of many diseases,

including Buerger's disease, leprosy, tuberculosis, syphilis, cancer, and so

many more. There can belittle doubt that the endothelial hyperplasia is a

compensatory attempt to increase the surface for filtration of oxygen from blood

to, the tissues, even though it defeats its own purpose. The demand for

activated oxygen on the part of the tissues is also expressed by poor sugar

oxidation as observed in diabetes mellitus, cancer, the thyroid diseases, and

others. By removing the basic pathology, that is, by restoring the oxidation

catalysis to normal or better, all expressions of the intoxication are removed,

and the metabolism, the blood pressure, and the tissue functions are again able

to run along as they should. The focus of infection is wiped out and the disease

is cured in its totality.

After an intramuscular injection of our Benzoquinone solution or one of the

other catalysts mentioned is given in diabetes, recovery begins very quickly and

its progress can be measured by the decrease in the blood sugar. The amount of

sugar eaten will affect this reading, but not the recovery mechanism. We feed

the patient an ordinary amount of sugar and starches and as he recovers he is

more and more able to use them. Where the sugar intake is controlled, however,

it is usual to observe a drop of twenty mgms. percent every week until normal is

reached. But we have seen both slower and more rapid restoration to normal.

Sometimes a severe case is encountered in which the blood sugar is over four

hundred mgms. percent and the vital organs have undergone fatty degeneration to

a fatal extent. In such cases the blood sugar may come to normal and the

gangrene heal, but the patient may, die from heart failure after excessive

exertion, either very early or very late after recovery from the diabetes.

In one series of cases, where the patients could be watched closely, the four

per cent of failures belonged to this group only, but in each instance the blood

sugar came to normal before death. Acidosis is not a serious factor either,

because with the restoration of the oxidation of sugar, the fatty acids are also

burned. The recovery is not simply the reduction of the blood sugar to normal.

It involves the correction of the whole pathology and the removal of its cause.

Therefore, the use of insulin is not essential. Indeed, it is better as a rule

to do without it from the commencement of treatment so as to avoid a

hypoglycemia in cases making rapid recoveries. The few cases of diabetes

insipidus treated so far have also recovered.

The treatment procedure is to stop all medication and cleanse the bowels for a

few days. Then one dose of one of the oxidation catalysts is injected

intramuscularly. Except for the use of plenty of animal fats, the dietary regime

is vegetarian entirely. No animal proteins whatever are permitted. Thus the

production of nitrogenous negative oxidation catalysts in the intestine is

retarded: Colon lavage is helpful. The diet should be reasonable and include

plenty of vitamins and tissue salts. Foods containing quinones and terpenes that

serve as negative oxidation catalysts must also be eliminated from the diet.

Therefore, coffee, tea, mangoes, and citrus fruits are not, used, and exposure

to pint solvents, perfumes and automobile or furnace gases is avoided. Food

should not be cooked in aluminum. Adequate colon lavage should be employed to

assure proper elimination. Alcohol, tobacco, and spices are forbidden. One dose

of the remedy is usually sufficient where cure is possible.

After the remedy is injected one should watch for periodic reactions which play

their part in the recovery process. These have already been described. (4) They

generally come at three and a half day or three-week intervals until recovery is

complete: If an interfering factor prevents recovery it should be identified and

removed and the dose repeated.

Diabetic Mechanism

To orthodox medicine the mechanism of diabetes still remains a puzzle. It is

observed, however, that after a person dies of diabetes, his pancreas can be

removed and from it a normal amount of insulin is extractable. (Of, course,

syphilitic and malignant destruction of the pancreas belong to different

categories.) The difficulties must lie with the function of secreting or

transferring the hormone into the blood stream. This is a function which like

muscle contraction requires the expenditure of energy. We may say that two

possibilities can exist in the production of diabetes, the inability to produce

the energy within the secreting fibrillae, and the inhibition of the secretory

function through allergic action of the " sugar center " in the brain or an

allergic action of the pituitary gland. Both the anergic and allergic

suppression of Islet function exist on the same chemical basis and have one

means of correction. A few explanatory words are in order.

We have given a thorough description of the allergy mechanism in various

writings and lectures in the past. (8) Briefly we may say that in this condition

the cell functional elements are made to work " under forced draft " beyond

physiological control. They have adsorbed into their colloidal surfaces, a

fluorescent toxin which transfers the exothermic energy which is constantly

evolved in living cells, into the functional element affected. The specificity

is determined by the similarity in the spectral absorption range of the

functional unit and the emission range of the fluorescent substance. So the

energy transferred passes right into the chemical processes of the functional

unit and forces its activity to proceed without the usual control. Thus are

produced the hyper-secretion of hay fever, the contraction of the bronchial

musculature in asthma, the conduction of a constant series of impulses through

the neurones associated in some thought complex resulting in the fixed ideas and

delusions of in. sanity and. the phenomena of hysteria. Thus also mitotic units

are forced into uncontrolled hyperactivity in the cell multiplications of

malignancy.

When energy is prevented from being evolved in a cell functional unit, the work

of the affected unit cannot be performed. The mechanism of blocking the

functional process is very much of the same nature as that of forcing allergic

behavior. Both depend upon the free valencies of the offending substance

(toxin). In one instance energy is transferred from the cell substance to a

functional unit where it does not belong and forces function; in the other, the

energy evolved within the cell is absorbed by the fluorescent substance and

emitted at a range that does not correspond to the energy absorption range of

the colloids of the functional unit, and hence it is dissipated without being

used. Thus the functional unit is inactivated. It appears that the

polymerization is less advanced in the allergenic than in the anergenic phase.

Since the free valencies of the offending molecule in both instances are subject

to oxidations and the molecule itself is a polymer of a much simpler oxidizable

structure, both effects can be corrected and really completely cured by

completely de-polymerizing and oxidizing the fluorescent materials that cause

the interferences.

As was stated earlier the origins of these toxins are imprisoned germs living

under anaerobic conditions in scar tissue, occluded tonsilar crypts, intestinal

diverticula, etc. The toxin is the germ's nutritional agent and its destruction

is fatal to the germ.

Whether or not the toxin has polymerized to the very advanced stage where it

acts directly upon the secretory filaments of the Islet cells inhibitively, or

has polymerized only to the lesser degree where it can act allergically upon the

nerve centers that inhibit Islet function, or upon the pituitary to accomplish

the same result, is, therefore, of no consequence therapeutically. The same

treatment measure is successful, for it removes the pathogenic agent completely

and thereby causes the death of the germ that produces and depends, upon the

poison. Since the scar prison is no longer needed after the death of the germ,

it too is disposed of and recovery is completed. Recovery is complete too for

another reason. The lack of oxidation capacity that permitted acute infection to

become chronic and produce its poisons in any tissue is corrected by the new

vigorous oxidations instituted by, the treatment and since they are of the

normal type that conduct aerobic glycolysis, it is the normal protective

procedure that is re-established. Thus we have overcome a serious disease by a

physiological procedure. The same system is used in securing true recoveries in

all of the other incurable diseases and lent infections so far encountered for

the chemistry of their pathogenesis is the same.

Relation to Other Therapies

The curative agents, 1:4 Benzoquinone, and its transition products, Glyoxylide

and Malonide, are the unrecognized active principles of the favorite sulfonamide

chemotherapies of the present day. But the active principles themselves are far

superior because the sulfo drugs must be changed to 1:4 Benzoquinone first at

the expense of the tissue vitality before they can do service. In complicated

and serious sickness the vitality may be too depleted to conduct the oxidations

required to accomplish this change and then the toxic effects of the drug added

to the infection toxins have a good chance to prove fatal. This situation is

eliminated and direct curative action only is had by the use of 1:4 Benzoquinone

or Glyoxylide or Malonide. (6) It is thus evident that in diabetes and

malignancy, sulfa drugs offer certain dangers.

Vitamins B, C, and K and some other useful agents depend primarily upon the

carbonyl group for their specific action. In each instance the rest of the

molecular structure determines the specific position at which the carbonyl

activity fits into the body chemistry and what its intensity should be. The free

valency of the carbonyl group exerts a photochemic action that influences the

formation of oxidizable ethylene groups and activates oxygen and other carbonyl

groups. (10) Therefore these vitamins, and some others are special oxidation

agents that play a part in the recovery from diabetes and cancer. Their forced

feeding gives a good boost to the oxidations, enough even to produce temporary

approximate recoveries in a few instances.

However, for true curative efficiency their structures would have to be changed

to conform to the laws of chemical structure we have announced.

The very infrequent good results from irradiation we attribute to the accidental

dehydration of tissue inosite to " Hexylene " (9) and its further oxidation to

Glyoxylide and Malonide with 1:4 Benzoquinone as a possible intermediary. The

carcinogenic action of irradiation is far better known than its curative action

in cancer which critical investigation shows to be nearly nonexistent. We,

therefore, suspect that irradiation produces a reduction of the normal catalysts

of sugar oxidation, Hexylene, Glyoxylide, and Malonide, to 1:4 Benzoquinone, for

in fairly concentrated solution 1:4 Benzoquinone has proved to be carcinogenic.

We have attributed all carcinogenic action to the quinone group. (7)

The ultimate of all curative therapies are Glyoxylide, Malonide, Hexylene, and

their relative 1:4 Benzoquinone because of their critical physiological

positions. Benzoquinone is indicated in the acute infections and lighter

allergies, while Glyoxylide serves best in the more chronic infections, the

deeper allergies like cancer, and the degenerative diseases.

Case Histories

One interesting observation is that of a physician of about fifty years of age

whose mother we cured of an enormous cancer of the breast in 1920. She was about

eighty years of age. The son, about fifty years old, came in 1926 with profound

diabetes of two years' standing. His diet was carefully managed and, insulin was

used regularly. Still in the last ix months he developed a general multiple

neuritis that affected both legs and hips most severely so that he was nearly

bedfast and needed crutches to move about. The Glyoxylide solution we were using

at that time was given intramuscularly. The neuritis and general nutrition

improved very quickly. It was much better in a week and in seven weeks it had

disappeared entirely. Still the blood sugar had only dropped from 190 to 160 in

that time and he still took some insulin. Recovery was not completed until after

the twelfth week. Here we see the changes disappear in reverse order to their

coming, and it appears that the stage of polymerization of the toxin causing the

neuritis was farther along than that which affected the pancreas islets.

A case of diabetes with considerable obliterative endarteritis throws some light

on the subject. This patient, age 50, was first seen by us in July 1928. For

about a year the obliterative endarteritis in both legs and feet increased in

severity so that walking became difficult. One of our best hospitals found the

blood sugar to be 380; gave insulin, morphine to control the pain, and advised

him to prepare for amputation of both legs at the knees. He took our treatment

instead and made a full recovery. In twelve weeks the legs were normal and at

the sixth month the blood sugar was again estimated at the same hospital and

found to be 90 mgms. percent. No insulin was used after the injection of

Glyoxylide and he ate all he wished. He was a fatty type and suffered from

dyspnoea on slight exertion. The, heart muscle sounds were weak and its action

flabby. A fatty degeneration of the myocardium was suspected therefore. Six

years later after prolonged exertion he died from acute cardiac dilatation.

Evidently better care and a longer time were required to permit the heart muscle

to be repaired with good healthy muscle tissue.

A simple case of diabetes which illustrates the vast majority one encounters is

illustrated by the woman of 73 who was discovered by her grandson, a diabetes

expert, to be carrying a blood sugar of 220 mgms. percent. He did not give

insulin, but our 1:4 Benzoquinone solution instead. After one injection the

blood sugar steadily decreased by about twenty mgms. percent per week until

normal was reached. Thus it took her about six weeks to recover. Her diet was

not arranged to restrict carbohydrates rigorously. She simply ate carefully as a

woman her age should, but all animal proteins were forbidden.

We have encountered diabetes complicated with rapidly advancing tuberculosis.

Such cases do as well as though no tuberculosis were present and the

tuberculosis undergoes recovery just as rapidly as though no diabetes were

present, whereas, before treatment the two conditions definitely aggravated each

other or rather were both aggravated by the basic crippling of the oxidation

mechanism.

The time relations may be obscured sometimes as in a case of diabetes where

cancer is present, if for instance the size of the growth is very large. Such a

case is that of a woman of 57 whom we treated in 1927. The diabetes was mild,

the blood sugar rarely exceeding 200 mgms. percent. This condition was known for

a year before a growth was found in the left breast. The growth developed

rapidly producing large metastases in the axilla, and was found inoperable at

the first examination. At her first visit to me the volume of the growth was

about a liter and a half, the axillary growths included. Two injections of

Glyoxylide were given, but recovery was very slow, which is contrary to the

rule, for the greater the growth rate, the greater the autolysis rate. Recovery

from the growth was not completed until seventy-two weeks had passed. However,

the diabetes started to improve after the thirty-sixth week and the blood sugar

became normal before the sixtieth week. Thus the time required for the

organization, autolysis, and absorption of the growth exceeded the time required

to get rid of the pathogenic toxin by something like nine months.

The whole profession is invited to make observations with the four remedies we

have been using most effectively, especially Benzoquinone and Glyoxylide. We

prepare each dose as carefully as possible for every patient that is chosen for

treatment and at the most reasonable cost possible. Case discussion is welcomed,

and advice is offered whenever desired; and all progress reports are of the

utmost interest to us.

That the earliest possible overwhelming statistical support to this thesis is

highly desirable is certainly evident, and we ask for your cooperation.

BIBLIOGRAPHY

1. The Chemistry of Natural Immunity, Koch, American Printing Co., 1936, pp.

155-163.

2. Lecture, " The Laws of Chemical Structure that Determine Immunity to Disease " ,

Koch, 1939, pp. 4.

3. Cancer and Its Allied Diseases, Koch, American Printing Co., 1927, pp. 52.

4. Cancer and Its Allied Diseases, Koch, 2nd edition American Printing Co.,

1929, pp. 151.

6. Lectures, " Laws of Chemical Structure that Control Immunity " , Koch, Dr. Wm.

F., pp. 6 and 7, Simon, Rio de Janeiro, Brazil, 1941.

7. Chemistry of Natural Immunity, Koch, Dr. Wm. F., pp. 67, 68, and 70,

Publishing Company, 1938.

8. Ibid, pp. 47.

9. Ibid, pp. 5 1, 54.

10. Ibid, pp. 33, 37, 38.