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Sporotrichosis

http://www.cdc.gov/node.do/id/0900f3ec800075b1

Disease Listing | General Information | Technical Information |

Additional Information

Frequently Asked Questions

What is sporotrichosis?

Who gets sporotrichosis?

How is the fungus spread?

What are the symptoms of sporotrichosis?

Does sporotrichosis involve any other organs besides the skin?

How soon do symptoms appear?

How is sporotrichosis diagnosed?

If I have symptoms should I see my doctor?

How is sporotrichosis treated?

How can sporotrichosis be prevented?

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What is sporotrichosis?

Sporotrichosis is a fungal infection caused by a fungus called

Sporothrix schenckii. It usually infects the skin.

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Who gets sporotrichosis?

Persons handling thorny plants, sphagnum moss, or baled hay are at

increased risk of getting sporotrichosis. Outbreaks have occurred

among nursery workers handling sphagnum moss, rose gardeners,

children playing on baled hay, and greenhouse workers handling

bayberry thorns contaminated by the fungus. A number of cases have

recently occurred among nursery workers, especially workers handling

sphagnum moss topiaries.

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How is the fungus spread?

The fungus can be found in sphagnum moss, in hay, in other plant

materials, and in the soil. It enters the skin through small cuts or

punctures from thorns, barbs, pine needles, or wires. It is not

spread from person to person.

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What are the symptoms of sporotrichosis?

The first symptom is usually a small painless bump resembling an

insect bite. It can be red, pink, or purple in color. The bump

(nodule) usually appears on the finger, hand, or arm where the

fungus first enters through a break on the skin. This is followed by

one or more additional bumps or nodules which open and may resemble

boils. Eventually lesions look like open sores (ulcerations) and are

very slow to heal. The infection can spread to other areas of

the body.

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Does sporotrichosis involve any other organs besides the skin?

The majority of infections are limited to the skin. Cases of joint,

lung, and central nervous system infection have occurred but are

very rare. Usually they occur only in persons with previous

disorders of the immune system.

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How soon do symptoms appear?

The first nodule may appear any time from 1 to 12 weeks after

exposure to the fungus. Usually the nodules are visible within 3

weeks after the fungus enters the skin.

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How is sporotrichosis diagnosed?

Sporotrichosis can be confirmed when a doctor obtains a swab or a

biopsy of a freshly opened skin nodule and submits it to a

laboratory for fungal culture.

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If I have symptoms should I see my doctor?

Yes. It is important for the diagnosis to be confirmed by a doctor

so that proper treatment can be provided.

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How is sporotrichosis treated?

Sporotrichosis is generally treated with potassium iodide, taken by

mouth in droplet form. A new drug, called itraconazole (Sporanox),

is available for treatment, but experience with this drug is still

limited. Treatment is often extended over a number of weeks, until

the skin lesions are completely healed.

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How can sporotrichosis be prevented?

Control measures include wearing gloves and long sleeves when

handling wires, rose bushes, hay bales, conifer (pine) seedlings, or

other materials that may cause minor skin breaks. It is also

advisable to avoid skin contact with sphagnum moss. Moss has been

implicated as a source of the fungus in a number of outbreaks.

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Date: October 13, 2005

Content source: National Center for Infectious Diseases/Division of

Bacterial and Mycotic Diseases

Sporotrichosis

http://www.doctorfungus.org/mycoses/human/sporo/sporotrichosis

Definition

Sporotrichosis refers to the infection caused by the dimorphic

fungus Sporothrix schenckii. The disease has been described

worldwide, however it is more common in tropical and subtropical

America [1876, 2227]. Sporotrichosis is acquired through direct

inoculation into the skin and rarely via inhalation of conidia. As a

consequence, the majority of cases are localized lesions affecting

the skin and subcutaneous tissues with minimal if any systemic

manifestation. Skin lesions characteristically follow lymphatic

pathways, but the lymph nodes themselves are not usually involved.

The initial erythematous papulonodular lesions evolve into either

smooth or verrucose painless nodules of about 3 cm that may ulcerate

and drain [1887]. There is also a clinical variety called " the fixed

form " [239, 1259]. The most common form of extracutaneous

sporotrichosis is osteoarthritis. Pulmonary sporotrichosis has also

been described. Disseminated forms of disease are seen on occasion,

and these classically affect immunosuppressed individuals [1380,

2406]. With the advent of the HIV epidemic the frequency of this

presentation has increased [37, 280, 599].

Forms of the disease

CATEGORIES NOTES

Cutaneous Varieties:

Lymphocutaneous sporotrichosis: affects the skin, subcutaneous

tissues and regional lymphatics

Fixed sporotrichosis: skin lesion evolves locally without spread

Osteoarticular Arthritis and tenosynovitis that may affect joints of

the hands, elbows, ankles or knees. The spine, hips and shoulders

are usually not involved.

Sinus tracts and local spread to adjacent bones may develop.

Pulmonary Classically produce enlarging cavitary lesions,

unilateral or bilateral usually with pulmonary infiltrates.

May produce pleural effusion and hilar lymphadenopathy

Associated with alcoholism, diabetes, corticosteroid use,

sarcoidosis and tuberculosis

Meningitis Rare

Produce a typical chronic meningitis picture.

CSF cultures have a low sensitivity, serologic testing may be

necessary

Disseminated Varieties:

Multifocal tenosynovitis and arthritis that mimic gonococcal

infection or seronegative spondyloarthropathy

Widespread visceral involvement associated with fungemia. Many

organs may be involved, including the central nervous system

(meninges or parenchyma), liver, spleen, bone marrow, and colon

Prognosis and therapy

Special resource: You may also want to refer to the Infectious

Disease Society of America-Mycoses Study Group (IDSA-MSG) Practice

Guidelines for this disease. It is available at the IDSA website.

Sporotrichosis is in general an indolent infection that requires

prolonged therapy but usually responds [1156]. Due to the

temperature sensitivity of the fungus, local application of

hyperthermia can be used for the treatment of cutaneous lesions.

However, systemic therapy, especially with oral azoles such as

itraconazole, has shown to be very effective [1876, 2062]. Therapy

with iodide (given as a saturated solution of potassium iodide, or

SSKI) is also effective [1156]. Treatment is continued for 3 to 6

months in cases of skin lesions and at least 12 months for

osteoarticular forms [1156]. Prognosis of disseminated disease is

grave. Amphotericin B is reserved to treat relapsed lymphocutaneous

disease, pulmonary, disseminated sporotrichosis as well as for

serious infections presenting during pregnancy [1156]. Antibacterial

antibiotics are useful when secondary bacterial infections occur.

Histopathology

The pattern of inflammation is characteristically well circumscribed

and granulomatous with central areas of acute suppuration. In the

skin, this pattern is similar to that seen in blastomycosis and

coccidioidomycosis. Demonstration of the organism in tissue may be

difficult because the fungi are not numerous. It may be necessary to

search through many serial sections before observing the organism.

The fungus is yeast-like, subglobose to ovoid, and 3-5 µm in

diameter. The organisms are often described as cigar-shaped. The

yeasts are not encapsulated. An asteroid body may be present and

consists of globose to ovoid, basophilic cells, 3-5 µm in diameter

with radiating eosinophilic rays up to 10 µm in diameter. An

asteroid body formation appears to be more common in secondary

lesions than in primary ones.

Laboratory

Direct examination

Typically unrewarding. Fluorescent antibody staining techniques may

be helpful. Diastase digestion prior to staining with H & E or PAS

may be helpful.

Isolation

Inoculate the aspirates, material from Culturettes, or swabbings

from open lesions onto Sabouraud dextrose agar and/or Inhibitory

Mould Agar and/or BHI with Gentamycin and Chloramphenicol with 10%

sheep blood, and a medium containing cycloheximide. Incubate the

media at 30°C. Growth is usually present in 3-5 days.

Laboratory confirmation

The mould-to-yeast form conversion is necessary since other fungi

are morphologically similar. Transfer the fungus to brain heart

infusion agar and incubate at 37°C in 5-10 % CO2

Natural habitat

Plant material

Susceptibility testing

Standardized testing procedures are not available. Microbiological

resistance has not been demonstrated.

Yeast phase of Sporothrhix schenckii

lymphocutaneous lesions spreading up the arm

Typical colony of Sporothrix schenckii in its mould phase

References

37. Al-Tawfiq, J. A., and K. K. Wools. 1998. Disseminated

sporotrichosis and Sporothrix schenckii fungemia as the initial

presentation of human immunodeficiency virus infection. Clin Infect

Dis. 26:1403-1406.

239. Bickley, L. K., I. J. Berman, and A. F. Hoodd. 1985. Fixed

cutaneous sporotrichosis: Unusual histopathology following

intralesional corticosteroid administration. J. Amer. Acad.

Dermatol. 12:1007-1012.

280. Bolao, F., D. Podzamczer, M. Ventin, and F. Gudiol. 1994.

Efficacy of acute phase and maintenance therapy with itraconazole in

an AIDS patient with sporotrichosis. Eur. J. Clin. Microbiol.

Infect. Dis. 13:609-12.

599. Donabedian, H., E. O'Donnell, C. Olszewski, R. D. MacArthur,

and N. Budd. 1994. Disseminated cutaneous and meningeal

sporotrichosis in an AIDS patient. Diagn. Microbiol. Infect. Dis.

18:111-115.

1156. Kauffman, C. A., R. Hajjeh, S. W. Chapman, and Mycoses Study

Group. 2000. Practice guidelines for the management of patients with

sporotrichosis. Clin. Infect. Dis. 30:684-687.

1259. Kwon-Chung, K. J. 1979. Comparison of isolates of Sporothrix

schenckii obtained from fixed cutaneous lesions with isolates from

other types of lesions. J. Infect. Dis. 139:424-431.

1380. Lynch, P. J., J. J. Voorhees, and E. R. Harrell. 1970.

Systemic sporotrichosis. Ann. Intern. Med. 73:23-30.

1876. Restrepo, A., J. Robledo, I. Gomez, A. M. Tabares, and R.

Gutierrez. 1986. Itraconazole therapy in lymphangitic and cutaneous

sporotrichosis. Arch. Dermatol. 122:413-417.

1887. Rex, J. H., and P. C. Okhuysen. 2000. Sporothrix schenckii, p.

2695-2698. In G. L. Mandell, J. E. , and R. Dolin (ed.),

Mandell, and 's Principles and Practice of Infectious

Diseases, 5th ed. Churchill Livingstone, New York.

2062. Sharkey-Mathis, P. K., C. A. Kauffman, J. R. Graybill, D. A.

s, J. S. Hostetler, G. Cloud, W. E. Dismukes, and Other

Members of the NIAID Mycoses Study Group. 1993. Treatment of

sporotrichosis with itraconazole. Am. J. Med. 95:279-285.

2227. Travassos, L. R., and K. O. Lloyd. 1980. Sporothrix schenckii

and related species of Ceratocystis. Microbiol. Rev. 44:683-721.

2406. , D. E., J. J. Mann, J. E. , and J. P. Utz. 1967.

Clinical features of extracutaneous sporotrichosis. Medicine. 46:265-

279.

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Sporotrichosis

http://www.mycology.adelaide.edu.au/Mycoses/Subcutaneous/Sporotrichos

is/

Description:

Sporotrichosis is primarily a chronic mycotic infection of the

cutaneous or subcutaneous tissues and adjacent lymphatics

characterized by nodular lesions which may suppurate and ulcerate.

Infections are caused by the traumatic implantation of the fungus

into the skin, or very rarely, by inhalation into the lungs.

Secondary spread to articular surfaces, bone and muscle is not

infrequent, and the infection may also occasionally involve the

central nervous system, lungs or genitourinary tract.

Clinical manifestations:

Fixed cutaneous sporotrichosis: Primary lesions develop at the site

of implantation of the fungus, usually at more exposed sites mainly

the limbs, hands and fingers. Lesions often start out as a painless

nodule which soon become palpable and ulcerate often discharging a

serous or purulent fluid. Importantly, lesions remain localised

around the initial site of implantation and do not spread along the

lymphangitic channels. Isolates from these lesions usually grow well

at 35C, but not at 37C.

Fixed cutaneous sporotrichosis showing

an ulcerating lesion on the leg.

Lymphocutaneous sporotrichosis: Primary lesions develop at the site

of implantation of the fungus, but secondary lesions also appear

along the lymphangitic channels which follow the same indolent

course as the primary lesion ie they start out as painless nodules

which soon become palpable and ulcerate. No systemic symptoms are

present. Isolates from these lesions usually grow well at both 35C

and 37C.

Lymphocutaneous sporotrichosis showing typical elevated

subcutaneous nodules developing along the regional lymphatics of the

forearm

Lymphocutaneous sporotrichosis showing more advanced,

ulcerating lesions developing along the lymph system of the forearm.

Pulmonary sporotrichosis: This is a rare entity usually caused by

the inhalation of conidia but cases of haematogenous dissemination

have been reported. Symptoms are nonspecific and include cough,

sputum production, fever, weight loss and upper-lobe lesion.

Haemoptysis may occur and it can be massive and fatal. The natural

course of the lung lesion is gradual progression to death.

Osteoarticular sporotrichosis: Most patients also have cutaneous

lesions and present with stiffness and pain in a large joint,

usually the knee, elbow, ankle or wrist. Osteomyelitis seldom occurs

without arthritis; the lesions usually confined to the long bones

near affected joints.

Other rare forms of sporotrichoisis include endophthalmitis,

chorioretinitis and meningitis.

Laboratory diagnosis:

1. Clinical material: A tissue biopsy is the best specimen.

2. Direct Microscopy: Tissue sections should be stained using PAS

digest, Grocott's methenamine silver (GMS) or Gram stain.

Section from a fixed cutaneous lesion showing round

Periodic Acid-Schiff (PAS) positive budding yeast-like cells.

Sporothrix schenckii is a dimorphic fungus and this

is the typical yeast-like form seen in tissue.

Interpretation: Look for small narrow base budding yeast cells (2-

5um). Note they are often present in very low numbers and may be

difficult to find. PAS and GMS stains are essential.

3. Culture: Clinical specimens should be inoculated onto primary

isolation media, like Sabouraud's dextrose agar and Brain heart

infusion agar supplemented with 5% sheep blood.

Interpretation: A positive culture from a biopsy should be

considered significant.

4. Serology: Serological tests are of limited value in the diagnosis

of Sporotrichosis.

5. Identification: Hyphomycete characterized by thermal dimorphism

and clusters of ovoid, denticulate conidia produced sympodially on

short conidiophores.

Causative agents:

Sporothrix schenckii

Management:

Cutaneous lesions respond well to saturated potassium iodide [4-6 ml

three time a day for 2-4 months], however itraconazole [400 mg/day]

and terbinafine [250 mg twice daily] have both proved to be

effective, although treatment times may be long. Ideally, treatment

needs to be maintained for at least a month after clinical cure is

achieved. Local heat has also been shown to improve cutaneous

lesions. Extracutaneous forms of sporotrichosis may need a

combination of antifungal treatment with Amphotericin B or

itraconazole together with surgical debridement.

Further reading:

Ajello L and R.J. Hay. 1997. Medical Mycology Vol 4 Topley &

's Microbiology and Infectious Infections. 9th Edition, Arnold

London.

Elewski BE. 1992. Cutaneous fungal infections. Topics in

dermatology. Igaku-Shoin, New York and Tokyo.

Kwon-Chung KJ and JE 1992. Medical Mycology Lea & Febiger.

MD and DW Warnock. 1993. Fungal Infection: Diagnosis and

Management. Blackwell Scientific Publications, London.

Rippon JW. 1988. Medical Mycology WB Saunders Co.

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