Re: A bit of caution on vitamin D (study in goats)

[Guest guest]

Two other things:

The risk factor for osteo is age. That's a medical problem.

The risk factor for PCa is ancestry. I don't believe the latitude studies hold water. Might be ALA (alpha linolenic acid), which makes more sense because Americans consume a LOT of soy oil.

And the risk factor for essential HTN is genetic, I believe. Again a medical problem mostly treatable.

I believe the risk factor for athero is probably larger than all others and it's something difficult to treat.

The biggest problem is figuring out who to believe. That's why I have a dr.

Regards.

[ ] A bit of caution on vitamin D (study in goats)

Regardless of what Vieth and the IOM people are saying, though, thisis the type of study that tends to make one a bit cautious:-------------------------------http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve & db=PubMed & list_uids=3968586 & dopt=CitationJ Nutr. 1985 Feb;115(2):167-7.Dietary calcium and vitamin D: risk factors in the development ofatherosclerosis in young goats.Hines TG, son NL, Beitz DC, Littledike ET.Effects of supplemental dietary calcium and vitamin D on lipiddistribution and aortic mineralization were examined in young goats.Twenty-four goats, 2-4 wk of age, were allotted one of four dietarytreatments for a 20-wk period and fed a basal milk diet (Basal), acalcium-supplemented diet (Basal + Ca), a cholecalciferol-enricheddiet (Basal + D3) or a diet with both calcium and cholecalciferol(Basal + Ca + D3). Goats in the Basal + Ca group had plasmacholesterol concentrations that were 16.6% of those of the Basalgroup. Percentage absorption and fecal excretion of total lipids wereunaffected by dietary treatment. Generally, total lipid andcholesterol concentrations were unaltered in liver, other viscera andcarcass tissues. Dietary cholecalciferol increased concentrations ofcholesterol and total lipid in aortas, whereas dietary calciumdecreased total lipids in aortas. Concentrations of calcium, magnesiumand total ash were increased in aortas by dietary treatment, with amarked increase observed in the Basal + Ca + D3 group. Sudan IV andgross calcium staining in aortas revealed both lipid and mineraldeposition that confirmed composition data. A high intake of vitamin Daccompanied by excessive intake of calcium seems to accelerate thedevelopment of atherosclerosis. Supplemental calcium with normalamounts of vitamin D, however, is hypocholesterolemic and seemsprotective against the atherogenic process.So you may prevent osteoporosis, and you may not get prostate cancer(I'm sure the women on the board are sighing in relief at this), butyou may calcify all your arteries.So go figure. It pays to be cautious with the vitamin D. Mepersonally I'm staying below 800 IU/day, and only in latefall/winter/early spring (Chicago).

[Guest guest]

Two other things:

The risk factor for osteo is age. That's a medical problem.

The risk factor for PCa is ancestry. I don't believe the latitude studies hold water. Might be ALA (alpha linolenic acid), which makes more sense because Americans consume a LOT of soy oil.

And the risk factor for essential HTN is genetic, I believe. Again a medical problem mostly treatable.

I believe the risk factor for athero is probably larger than all others and it's something difficult to treat.

The biggest problem is figuring out who to believe. That's why I have a dr.

Regards.

[ ] A bit of caution on vitamin D (study in goats)

Regardless of what Vieth and the IOM people are saying, though, thisis the type of study that tends to make one a bit cautious:-------------------------------http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve & db=PubMed & list_uids=3968586 & dopt=CitationJ Nutr. 1985 Feb;115(2):167-7.Dietary calcium and vitamin D: risk factors in the development ofatherosclerosis in young goats.Hines TG, son NL, Beitz DC, Littledike ET.Effects of supplemental dietary calcium and vitamin D on lipiddistribution and aortic mineralization were examined in young goats.Twenty-four goats, 2-4 wk of age, were allotted one of four dietarytreatments for a 20-wk period and fed a basal milk diet (Basal), acalcium-supplemented diet (Basal + Ca), a cholecalciferol-enricheddiet (Basal + D3) or a diet with both calcium and cholecalciferol(Basal + Ca + D3). Goats in the Basal + Ca group had plasmacholesterol concentrations that were 16.6% of those of the Basalgroup. Percentage absorption and fecal excretion of total lipids wereunaffected by dietary treatment. Generally, total lipid andcholesterol concentrations were unaltered in liver, other viscera andcarcass tissues. Dietary cholecalciferol increased concentrations ofcholesterol and total lipid in aortas, whereas dietary calciumdecreased total lipids in aortas. Concentrations of calcium, magnesiumand total ash were increased in aortas by dietary treatment, with amarked increase observed in the Basal + Ca + D3 group. Sudan IV andgross calcium staining in aortas revealed both lipid and mineraldeposition that confirmed composition data. A high intake of vitamin Daccompanied by excessive intake of calcium seems to accelerate thedevelopment of atherosclerosis. Supplemental calcium with normalamounts of vitamin D, however, is hypocholesterolemic and seemsprotective against the atherogenic process.So you may prevent osteoporosis, and you may not get prostate cancer(I'm sure the women on the board are sighing in relief at this), butyou may calcify all your arteries.So go figure. It pays to be cautious with the vitamin D. Mepersonally I'm staying below 800 IU/day, and only in latefall/winter/early spring (Chicago).