The Salk Vaccine (& Pesticide info)

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The Salk Vaccine ( & Pesticide info)

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Images Of Poliomyelitis

The Salk Vaccine

The success of the polio vaccines, like poliovirus causation, is

" proved " through interpretations of animal laboratory experiments,

and are thus subject to the same criticism as the animal experiments

for virus causation, i.e., animal experiments are extremely harsh and

thus are unrelated to the human polio disease condition.

Vaccination success in humans has also been " proved " through

statistics (epidemiology) gathered from hospitals which show polio

incidence declining as vaccination programs are implemented.

" Polio and the Salk Vaccine " is one of the most highly promoted

medical images, a typical example:

...the epidemics grew steadily worse each year, with the

number of new cases climbing from 5,000 in 1933 to 59,000 in 1952.

Salvation came in 1954 with the Salk vaccine...

" A Paralyzing Fear: The Story of Polio in America "

The New York Times, March 4, 1999 (Film Review)

The story of the Salk Polio Vaccine is highly touted by

teachers, medical representatives, and in popular, educational, and

news/medical media.

Salk Vaccine Timeline, U.S.

1952, Jonas Salk discovered his vaccine, though the technology

was not new, being based on vaccines utilized at least by the early 1930s.

March 26, 1953, the Salk vaccine announced, after evaluation of

600 vaccinated persons. (Jane , Patenting The Sun)

1954, during the large field trial, 423,000 second-grade

children were vaccinated (T. Francis, from son, Field's

Virology). At the height of the great polio epidemic, children of age

6-9 had become a primary susceptible group and this is said to be the

reason that second grade children were chosen. The high

susceptibility of infants below age 5 to toxic chemicals

(formaldehyde and merthiolate were used in the Salk vaccine) in

addition to the harsh method of receiving virus particles by

injection, could have been another reason to chose age 7, for age 7

is at the end of the range of infant nerve system vulnerability due

to a rapidly growth and myelination.

Nearing the height of the epidemic, the susceptibles range

extended from pre-school children to include the age 6-9 and beyond.

This is clearly a function of the extreme nature of the epidemic (in

terms of the toxic theory). This extension occurred before the height

and after the height. Regardless, infants below the age of 5 always

were a primary susceptible group.

The primary susceptible group of pre-school children (infants)

was not targeted for vaccination against infantile paralysis in 1954 or 1955.

Beddow, p8, writes that within two hours of release of the

Francis Report on April 12, 1955, the vaccine was licensed. JAMA,

vol 158, no 14, p1249 reports April 12, 1955. However, A Paralyzing

Fear, Seavey, 1977, states that the Salk vaccine was licensed by HEW

in March, 1955. Seavey is apparently incorrect.

Large scale vaccination begins April 12, 1955. The target age

group is 6-9 years (Scheele, A.S., , J.A., " Health

Implications In A Program Of Vaccination Against Poliomyelitis " ,

JAMA, August 6, 1955).

The Cutter Laboratory disaster is discovered 13 days later,

April 25, 1955. Documented cases caused by the Cutter vaccine were

79. The EIS went on to find 204 polio cases with 11 deaths out of the

approximately 423,000 persons inoculated with Cutter's Salk vaccine

(Jane , Patenting The Sun, 1990). Contagion patterns are very

difficult to discover with polio, yet the EIS expanded the original

79 cases to 204 by assuming contagion patterns. Tremendous publicity

was given to these cases.

April 27, 1955, Surgeon General calls Cutter for a recall of its

vaccines (Jane , Op. Cit.).

May 8, 1955, the entire U.S. vaccination program was cancelled

by the Surgeon General ( J. Rutty, Dept. History, U. of

Toronto, URL:http://www.eskimo.com/~dempt/salk.htm). The program was

soon resumed and 5,394,000 persons were vaccinated during 1955.

During 1956, approximately 25,000,000 persons were vaccinated.

The 1959 CDC Polio Packet indicates only the cc's of vaccine shipped

per month, not persons vaccinated. However, it gives enough

information so that the persons vaccinated can be interpolated from

these shipment figures.

1956, the federal government rigorously tested the vaccine, and

began to take over NFIP's role.

1957, the vaccination program resumed under CDC/EIS control and

was highly promoted (Jane , Op. Cit.). Approximately 31,300,000

persons were vaccinated in U.S. (CDC, Polio Packet, 1959)

1958, approximately 15,700,000 persons were vaccinated.

As of 1959, less than half of the usual primary susceptible

group (infants below age 5) were vaccinated. Only 55% of those below

age 40 were vaccinated, according to the CDC's Polio Packet. 28% of

the vaccinations had been given to non-susceptibles, the group of 40 and above.

However...

Polio incidence began its sharp plummet in 1952. This is 3 years

before the vaccine was licensed by HEW in 1955. Thus, the Salk polio

vaccination did not significantly correlate with declining polio

incidence. However, a significant correlation with declining polio

incidence is found with reports in 1952 that nursing cow calves were

dying with of symptoms and physiology when their mother's were fed

fodder that had been treated with DDT.

The plummet also correlates with the 1951 government/industry

debate over pesticide safety.

The plummet also correlates with the phase-out of persistent

pesticides production in the early 1950s. ( " Persistent " pesticides

are those which do not easily biodegrade, such as DDT, BHC, arsenic

compounds, and lead compounds.)

Dr. Francis did not mention in his key evaluation of the

1954 Salk field trials that those who contracted polio after their

first inoculation and before their second inoculation were placed in

the " not-inoculated " list.' (Maurice B. Bayly, The Story Of The Salk

Anti-poliomyelitis Vaccine, 1956). During the 1954 field trials

second graders only were inoculated, and 1st and 3rd graders used as

controls, yet susceptibility to polio (or toxicity) can be highly

variable between the ages of 6 to 7 to 8, and as such, this made

proof of success difficult.

During the 1954 field trials only volunteers were inoculated,

yet the control groups included non-volunteers. This enters even more

complex variables into the trial. (Beddow, p14)

Jonas Salk worked for Francis in his laboratory. Francis was a

former Army epidemiologist who was involved in U.S. studies of

Nagasaki and Hiroshima after the nuclear bombardment. Those studies

have been characterized as inaccurate:

...the National Academy of Science... released a new

report... the 'BEIR V' report, concludes that cancer and leukemia

risks for the survivors... has been underestimated by a factor of

three to four, due to faulty dose estimates and insufficient

follow-up study of the survivors. "

...the BEIR report suggests... legitimate questions about

the validity of the currently accepted estimates. (Jay Gould et al,

Deadly Deceit: Low-Level Radiation, High-Level Cover-up (1991), p179)

Though vaccination safety was said to be a matter of record, the

weekly polio case rates during 1955 show a definite rise in polio

case rates during the period of vaccination (April and May), unlike

previous or subsequent years. This is not discussed, however,

downward movements in the age 7 group (in the age distribution

graphs) were used by the CDC to show evidence of vaccination efficacy

(CDC, Polio Packet, 1959).

The Detroit Epidemic, 1958

In 1958, laboratory analyses of the epidemics in New Jersey and

Detroit associated poliovirus with only 56% of diagnosed cases. The

Detroit analysis associated only 51% of diagnosed polio with poliovirus.

Regardless of these associations, a positive finding for the

poliovirus would not by itself prove poliovirus causation because

this virus is often found during epidemics in those who have no

disease symptoms or in those who have disease symptoms that can be

attributed to other microbes (Scobey, " Is Human Poliomyelitis Caused

By An Endogenous Virus? " , Science, v71, 1954).

Detroit Epidemic, Microbe Analysis

47% of the diagnosed polio cases in the Detroit epidemic were

found to have been inoculated at least once, 34% at least twice, 22%

at least thrice. Non-white cases had been found vaccinated 54% as

much as white cases, however, according to the CDC's Polio Packet,

non-white to white paralytic case incidence was 18 to 1, or 1800%:

The epidemic was the second worst in Detroit history.

Paralytic cases were 18 times more frequent among non-whites...

(Sect. VII, p17, 1959)

A check on these calculations shows the incidence ratio of

non-whites to whites actually to be just over 13 to 1, which is

intriguing enough. The base numbers are as follows. For those who

would like to check for themselves, the non-white to white paralytic

cases were 246 to 66. Non-white to white population was 420,000 to

1,480,000). The result, an incidence ratio of 13 to 1, has been

applied to the considerations below, which gives a break to

orthodoxy. At the time the Polio Packet was published, during the

epidemic, the " 18 times more frequent " phrase enhanced the imperative

for vaccination, since these non-whites were lagging in their

vaccination status.

The poliovirus now appears to be unreasonably selective for

non-whites. There are many mitigating factors that could be brought

to argue against these general impressions. Thus it is necessary to

proceed into detailed comparisons.

Age Group 5-9: White vs Non-White (Detroit)

Consider a comparison of the most similar age group, e.g.,

school children in the age group 5-9. These children were similar in

education level, age, and vaccination status. Only the paralytic

cases are utilized in this comparison because they were much more

frequently associated with poliovirus: 77% of the paralytic cases

were associated with poliovirus, yet only 24% of the non-paralytic

cases were associated with the poliovirus.

In the 1950s, clinical doctors diagnosed polio according to

symptoms and physiological evidence. Only a small percent of total

cases in the U.S. were analysed in the laboratory for presence of the

poliovirus. If the poliovirus could not be associated with a case

then it was set aside as non-polio. Suddenly, much of the disease

called polio was technically not polio. How is that so much

non-poliovirus polio rose and declined concurrently with poliovirus

polio during the Great Epidemic? No vaccine existed for non-poliovirus polio.

Without pursuing that quackmire, we continue here within the

slippery orthodox definitions of polio and limit our focus to

paralytic polio in order to deal within a set of cases which can at

least be assumed to be mostly poliovirus caused -- just in order to

be able to argue the shifting orthodox assumptions.

Regarding the comparison of non-white vs white age group 5-9, we

first find that the non-white to white paralytic case ratio is 3.2 to

1. Converted to incidence, which accounts for population differences,

we have an incidence ratio of 11.3 to 1.

This vast disparity cannot be explained by vaccination status

because these were quite similar. Non-whites were found to be

vaccinated 88% as much as whites. Based on this, if polio vaccination

was valid, we would expect an incidence ratio between the two groups

to be closer to 1.1 to 1, than 11.3 to 1.

The vaccination profile of these cases is provided in the following table:

Age Group 0-4: White vs Non-White (Detroit)

The second comparison group is pre-school children in the age

group 0-4. These polio cases are, at the least, similar in age, and

not very dissimilar in vaccination status. Another similarity is that

they both represent the traditionally highest susceptible age group,

from whence comes the name " Infantile Paralysis " .

The non-white to white case ratio is very different, 6.3 to 1.

Converted to incidence, an even more stunning incidence ratio is

found, 22.4 to 1.

Non-whites were found to be vaccinated 58% as much as whites. If

vaccination were valid then we would expect an incidence ratio closer

to 1.7 to 1 than 22.4 to 1.

The vaccination profile of the 0-4 cases is provided in the

following table:

Poliovirus theory creates massive contradictions between public

images, epidemiological data, and laboratory data.

The toxic theory can easily resolve these contradictions.

For instance, with regards to the Detroit epidemic a study might

begin with a focus upon the industrial pollution of drinking water,

extraction solvents in cooking oils, and pesticides in cooking oils

(particularly those, which in fine print show their ingredients to

include cottonseed oil). Pesticides and herbicides in fatty meat and

dairy products should also be analyzed for toxic chemicals. The polio

victims should be studied for levels of industrial toxins in their

urine, tissue, and in nursing mothers, breast milk. The Polio Packet

shows the epidemic peaks in Detroit occurring somewhat later for

non-whites, which is consistent with the later harvest time of the

cotton crop vs other crops. A similar epidemic shift occurred in a

white community in Massachusetts in 1908, where an epidemic centered

around three cotton mills.

It is unlikely, in my opinion, but if dietary and toxicology

studies were found negative, then a bacterial hunt could begin. If

that proved negative, then viropathologists could possibly find

rogue, nucleic acid as the culprit. And lastly, if all else fails, it

could be asserted that the polio epidemic was caused by genetic

defects in the population, with packaged salvation available from

biotech industry's nearest representative.

Pesticides vs Polio vs Salk

In essence, the Salk Vaccination program appears irrelevant to

the Great Polio Epidemic, as shown in the following graph which

represents vaccination data from the above timeline.

Notes

Only the older generation of pesticides, the persistent

(low biodegradable) pesticides are shown. DDT production is not

included past1954 because it was being re-directed heavily into

underdeveloped countries. Vaccinated population numbers are derived

from the timeline. The Salk Effective Index is a HARpub concept,

determined as follows:

Salk protection probability

X's

Percent of poliovirus caused polio

X's

Accumulated percent of inoculated population.

Stated otherwise: 70% x 56% x accumulated percent of

inoculated population per year. The height of the Salk Efficacy Index

is adjusted to be equal to a percent of the highest value represented

in the graph, which is the apex of the pesticide or polio lines. The

pesticide and polio lines have been proportioned so that their high

points are equal.

Can we now presume that if Salk vaccinations correlated with

polio incidence, as do pesticides in the above graph, then such a

graph would be impressed upon everyone's minds from birth, by every

available media source? (As is done by pro-vaccinationists now.)

With regard to polio incidence, the Salk vaccine shows less

impact (if any) than we have been lead to believe, especially when

compared to the nearly perfect correlation of the persistent

pesticides, which spans a much longer period, and maintains a direct

correlation throughout a complex series of oscillations, and not only

in terms of this composite graph, but in terms of each pesticide.

Internationally, the pesticides, polio, and vaccination programs

co-exist under an organized central leadership, such as in Mexico,

which is decades behind the U.S. in terms of pesticide regulations.

In 1994, W.H.O. gave Mexico a low rating for polio,

characterizing it as a " Stage C " country. That is, it has ongoing

polio cases and ongoing vaccination programs. (Van Nostrand's

Encyclopedia of Science and Engineering, Van Nostrand Reinhold, 1995, p2492)

Coincidentally, the North American Working Group on Sound

Management of Chemicals, a NAFTA group,

...will finalize action plans aimed at reducing mercury,

PCBs, DDT and chlordane in the North American environment. The action

plan requires Mexico to reduce and eliminate, within a specific

timeframe, the use of DDT in malaria control. Chlordane will also be

phased out in Mexico. ( G. Hall, Journal of Commerce and

Commercial, Nov 4, 1996 v410)

The phrase " DDT in malaria control " indicates the presence of W.H.O.

Worldwide polio vaccination programs and industrial pollution

continue concurrently.

Post-Polio

If polio vaccination theory were valid, then in terms of

orthodoxy, former victims of polio would not be able be infected with

poliovirus, except when infected with a poliovirus type to which they

have not been previously exposed. However, several studies have found

evidence of poliovirus (including the common Type I) in post-polio

victims (see PubMed listings). It is likely that these post-polio

victims were previously exposed to the Type I poliovirus because most

polio victims suffer from post-polio (approximately two-thirds) and

Type I is common.

Generally, virus association with disease can be expected

because viruses are more apt to proliferate in poisoned biological systems.

The most consistent correlation which contributes understanding

to post-polio is pesticides -- just previous to the first reports of

post-polio (March, 1984), U.S. legislation allowed for the re-entry

of DDT in pesticide blends (June, 1983).

Conclusion

Because

Pesticide symptoms and physiology are identical to poliomyelitis.

and pesticide dosage arguments alone suffice,

and epidemiological proofs for pesticide causation correlate perfectly.

And because

Poliovirus proofs, based on animal brain injection experiments,

are artifacts of the laboratory and cannot transcend this limitation,

and polio contagion among animals in the laboratory is unknown,

and poliovirus can easily infect benignly, yet the paralytic

disease shows no pattern of contagion,

and vaccination proofs are based on harsh laboratory experiments

that do not reflect the human experience,

and vaccination epidemiological evidence is relatively irrelevant,

and poliovirus presence in post-polio invalidates vaccination theory,

and poliovirus presence is not required for the polio disease,

and in spite of an extremely biased scientific environment

ranging from textbook warning statements regarding the mere

consideration of pesticide causation, exclusively biased public

health laws, exclusively biased funding, and dramatically charged

global propaganda for the public and medical professional,

We are able to conclude with the most direct explanation:

Pesticide causation.

Addenda: Testing The Pesticide Theory

A theory is only as good as its ability to predict events.

Accordingly, the poliovirus theory is a non-theory, because the

poliovirus has always been associated with humans and thus its

presence predicts nothing.

The pesticide theory, however, correlates perfectly in all data

areas. It correlates a historically new event (pesticide poisoning),

with another new event (poliomyelitis). It simply correlates dosage

with physiology and symptoms.

Billions of dollars have funded the development of the

poliovirus theory since Landsteiner and Popper in 1908. Zero dollars

have funded the pesticide theory. Yet, after the smoke has settled,

the simple pesticide argument remains strong.

The pesticide theory can be used by historians as a tool for the

discovery of evidence of mass poisoning. In the table below are

examples of such use:

Polio Epidemic Pesticide Event

1887: The first polio epidemic (Sweden) 1873: Patent of

first pesticide sprayer

1874: invention of DDT

1874-1887: first relatively large group of pesticide developments.

1908: A polio epidemic within northwestern, Massachusetts that

occurred in three manufacturing towns, all within 20 miles of each

other. A thorough study in 1909 concluded that polio is

non-contagious, and that the port of entry for the virus is probably

food, milk, and water. No exclusively breastfed infant acquired

polio. By far the highest polio incidence was in an upstream town

with 3 cotton mills. No toxicological investigation was made. As

usual, the gathering of data was based upon the infectious disease

model. 1907: The first high-volume production of carbon

tetrachloride begins in the U.S. Carbon tet was used as a fumigant,

insecticide, herbicide, and cleaning solvent. Used in cottonseed oil

extraction. Cottonseed byproducts can be used in dairy fodder.

1916: The polio epidemic in NYC region 1915, Hooker

Chemical and DOW begin first high-volume production of chloral

benzene, at Niagara Falls. 8,500 metric tons per year.

1921: FDR acquired polio in the Bay of Fundy, off the remote

island of Campobello, Canada. Campobello is directly downstream from

several major industries that dumped organochlorines, lead, arsenic,

and mercury. 1972, the Bay of Fundy was the chosen as the site for an

study of high DDT levels in porpoises.

1942-1962: The Great U.S. Polio Epidemic 1942-1962: Referred

to as the era of " Pesticides As Panacea " .

1945: U.S. troops in the Philippines: Polio epidemics took

casualties second only to battle casualties. Troops in

Philippines were doused with DDT. Surrounding population was

unaffected by polio.

1958: Polio epidemic (136 victims) in New Jersey (Essex, Hudson,

Bergen Counties) The CDC PolioPacket (1959) omitted describing the

environment, which included the area of the Bayonne and Linden

petroleum refineries. Nevertheless, the prime focus (lab work)

revealed that the poliovirus could only be found in 65% of the

victims. Their " polio " had to be recategorized, thus driving the

polio incidence down immediately.

1952: Philippines, polio epidemics.

1964: Philippines, 383 humans and 25,000 dogs per year died of

rabies. Rabies is a paralytic CNS disease, physiologically and

symptomatically similar to polio, with a different virus assigned to

it. Landsteiner was originally quoted as observing the similarities

and has been criticized for that opinion. Philippines, under

W.H.O. policy, continues with DDT/BHC for agriculture and DDT for

malaria anti-mosquito campaigns.

India: Continues with high polio incidence. Human rabies deaths

are estimated to be 10,000 per year (circa 1980). India as of 1980

used persistent pesticides and has lagged far behind developing

countries in banning these.

Underdeveloped and developing countries continue with polio

epidemics. Underdeveloped and developing countries continue with

persistent pesticides.

Sri Lanka has high polio rates and the highest rabies rate in

the world, 140 human deaths out of 11.5 million population per year

(circa 1980). In 1980, the U.S. had a population of approximately 360

million with a rabies incidence per year of from 0 to 2. Sri Lanka

ranks among countries with highest rates of acute pesticide

poisoning, and in 1980 allowed DDT imports. As of 1995, DDT is listed

as banned in Sri Lanka, according to PANNA.

June, 1983: U.S. legislation allows re-entry of DDT for use in

insecticide blends. March, 1984: Post-polio is recognized as an

emerging epidemic in former polio victims. Post-polio cases today are

estimated to be 600,000.

Nigeria (Africa): 2003-2004, highest polio case count

worldwide. WHO designates Nigeria its #1 target (worldwide) for DDT

malaria campaigns. These campaigns were not in effect for many years prior.

PANUPS (5/19/95) reports:

In Paraguay, an extensive 1990/91 study of paralysis of the

limbs in children, originally thought to have been brought on by

polio, suggested that monocrotophos [OP pesticide] drift from nearby

cotton fields was the most likely culprit.

Other Examples

The irrationality of vaccination programs extends far beyond

polio. Smallpox, claimed to be virus caused and the first disease

conquered by vaccination, provides us with data for a strong

anti-vaccination stance. The following graph is generated from two

tables found in the bibliography of Pitcairn's, The Fallacy Of

Vaccination (1911):

If one is searching for evidence of causation then vaccination

obviously causes disease. Similarly, this is found to be so in the

next example.

Before discovering the following smallpox data from Emerson's

epidemiological compilations of New York City, I was under the

impression that Western pesticide usage had begun approximately 1873.

The following epidemiological chart convinced me that the date must

have been closer to 1868. Later, I found in Zimmerman et al, DDT,

Killer of Killers that 1869 is the date for first pesticide

implementation in the West (though there are earlier instances, see

Pesticide Introductions), thus demonstrating again how epidemiology

can be used to discover data regarding pesticide production and

population exposure. To this day, pesticide production data is

repressed by government and industry.

Summary Hypothesis

Vaccinations, and proofs of virus causation, are similar

phenomena. They both use injections of virus material to provide

images of a context of impending toxic doom to the body. Before the

wholly artificial event of injection existed, impending toxic doom

was the only interpretation available to the body for the presence of

large amounts of virus -- in view of the cellular S.O.S. response to

toxicity, described in the Overview. Thus, the presence of

exceedingly large amounts of injected virus material should be

expected to generate a radical response: inflammation, proliferation

of viruses, genetic recombination, and the self destruction of

tissue, tissue which would be regarded as portals of entry -- for

poisons. It should not be expected to generate such a radical

response repeatedly, because the body learns not to be tricked. Thus

we have " immunization " and artificial proofs of its efficacy.

The Challenge

Modern Medicine is hereby challenged to find a polio epidemic

that was not preceded by mass poisoning. It is further challenged to

include toxicological data in its reporting of disease. The chemical

industry is challenged to allow tracking of its production and

distribution. The laws forbidding dissemination of pesticide

production data should be repealed. All pesticide use (place, date,

type, quantity, purpose) must be registered and toxic chemical

content in food should be labeled. The same goes for genetically

engineered crops. Pesticide registration would facilitate

epidemiology, making it truly meaningful and useful.

--------------------------------------------------------

Sheri Nakken, R.N., MA, Hahnemannian Homeopath

Vaccination Information & Choice Network, Nevada City CA & Wales UK

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