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It is one of our weird flukes of medicine. They always knew it was a problem to

bleed to much (lack of clotting), but it never occured to them that it could be

a problem to have too much clotting. So they took the normal range all the way

to zero.

Also Sed is usually not used to determine clotting problems, more sophisticated

measures are used. The way sed rates are usually used is that a high sed rate

is associated with autoimmune diseases and other things I don't remember. So

that is what they are typically looking for when they do that test.

It turned out that everyone who has hypercoagulation also had very low sed

rates. Mine were always 0-2 since I have been sick which is all the data I

have, I hypercoagulate easily, and have genetic causes for it in my family. A

low sed rate means that the blood takes forever to settle out. In reality those

low numbers are not " normal " . A normal person will not have sed rates that low.

But it wasn't until they started studying people with CFS that they noticed this

correlation.

Thanks,

Doris

----- Original Message -----

From: winsomme

I was wondering if someone could illuminate something that has

confused me for some time.

over time, i have often read about a low sed rate in CFS being

indicative of hypercoagulation. i have always had low sed rates when

they were taken (between 0 and 3), but i always wondered why that

would indicate a problem.

i believe the reference range for a Sed Rate is 0-15, so how can

anybody have a low sed rate when the ref range starts at 0?

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Bill,

Reference ranges are almost worthless. Sometimes they call it “normal

range”. In pharmacy school, profs kept calling various health problems

“normal” and one day I asked how that could be. The prof replied that

“normal” means the norm or average. And the average American has health

problems!

This explains in part while doctors may tell you it’s normal, when you

complain of fatigue, PMS, pain, or whatever. And we think “normal means the

ideal, healthy state, to which we want to return.

For example, since the average American is somewhat anemic, the

reference range on your lab results shows anemic as “normal”. To compound

matters, the labs don’t use the whole population’s results for their

reference ranges, they use only the population consisting of people they’ve

tested. Which is mostly sick people. While everybody may get a CBC

(complete blood count), so that some non-anemic people are included, other

tests are done only on those who are suspected of having unhealthy levels

for that test. I almost used the words “abnormal results”. Even if a high

percentage of testees have unhealthy results, they are all counted in the

pool that determines what the “normal’ is.

Then, a very wide part of the resulting bell curve, is counted in the

reference, or “normal” range. Only the most extreme results are left out.

For your sed rate results, 0-3 is in the low end of “normal’, but

science has shown it to be unhealthy.

Hope this helps.

Barb

ESR/Sed Rate

I was wondering if someone could illuminate something that has

confused me for some time.

over time, i have often read about a low sed rate in CFS being

indicative of hypercoagulation. i have always had low sed rates when

they were taken (between 0 and 3), but i always wondered why that

would indicate a problem.

i believe the reference range for a Sed Rate is 0-15, so how can

anybody have a low sed rate when the ref range starts at 0?

any thoughts would be appreciated.

thanks

bill

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thanks for your posts. i guess i'm not so worried about the reference

range, but the idea of a " low sed rate. " could somebody point me to

some literature as to what makes a low sed rate abnormal.

it is my understanding that if you are hypercoaguable you would have

a " high " sed rate. and that technically there is no such thing as a

low sed rate because " 0 " is normal.

i totally understand that reference ranges aren't always the best

measure, but when the range tells you that there is no " LOW " side,

then what is the reasoning behind suggesting that it is abnormal?

anyway, these are just some questions circulating around in my head.

thanks

bill

> Bill,

> Reference ranges are almost worthless. Sometimes they call

it " normal

> range " . In pharmacy school, profs kept calling various health

problems

> " normal " and one day I asked how that could be. The prof replied

that

> " normal " means the norm or average. And the average American has

health

> problems!

> This explains in part while doctors may tell you it's normal,

when you

> complain of fatigue, PMS, pain, or whatever. And we think " normal

means the

> ideal, healthy state, to which we want to return.

> For example, since the average American is somewhat anemic, the

> reference range on your lab results shows anemic as " normal " . To

compound

> matters, the labs don't use the whole population's results for their

> reference ranges, they use only the population consisting of people

they've

> tested. Which is mostly sick people. While everybody may get a CBC

> (complete blood count), so that some non-anemic people are

included, other

> tests are done only on those who are suspected of having unhealthy

levels

> for that test. I almost used the words " abnormal results " . Even

if a high

> percentage of testees have unhealthy results, they are all counted

in the

> pool that determines what the " normal' is.

> Then, a very wide part of the resulting bell curve, is counted

in the

> reference, or " normal " range. Only the most extreme results are

left out.

> For your sed rate results, 0-3 is in the low end of " normal',

but

> science has shown it to be unhealthy.

> Hope this helps.

>

> Barb

>

>

> ESR/Sed Rate

>

> I was wondering if someone could illuminate something that has

> confused me for some time.

>

> over time, i have often read about a low sed rate in CFS being

> indicative of hypercoagulation. i have always had low sed rates when

> they were taken (between 0 and 3), but i always wondered why that

> would indicate a problem.

>

> i believe the reference range for a Sed Rate is 0-15, so how can

> anybody have a low sed rate when the ref range starts at 0?

>

> any thoughts would be appreciated.

>

> thanks

> bill

>

>

>

>

>

>

>

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Try www.hemex.com

The reasoning is empirical. People with low sed rates also are found to have

excess fibrin (hypercoagulation). When treated, the sed rates come up. I don't

believe you are correct that hypercoagaguable would be a high sed rate. It is

the opposite. Sed rate is how fast the blood settles. If you have thick blood

it does not settle fast. As I said before, high sed rate is usually associated

with autoimmune problems such as lupus, although I don't know why those types of

illnesses cause that.

A 0 sed rate means the blood doesn't settle out, so I don't see how anyone could

think that " 0 " is normal. " Normal " is probably something like 5-10. But they

took the range down to 0 because they didn't know it was a problem.

Thanks,

Doris

----- Original Message -----

From: winsomme

thanks for your posts. i guess i'm not so worried about the reference

range, but the idea of a " low sed rate. " could somebody point me to

some literature as to what makes a low sed rate abnormal.

it is my understanding that if you are hypercoaguable you would have

a " high " sed rate. and that technically there is no such thing as a

low sed rate because " 0 " is normal.

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Bill, what I’m saying is that the medical definition of “normal” is less

than worthless. Forget normal/abnormal. Go for the science, and think

healthy/unhealthy. If you keep leaning on the fact that the lab said it’s

normal, you’re stuck.

Barb

ESR/Sed Rate

>

> I was wondering if someone could illuminate something that has

> confused me for some time.

>

> over time, i have often read about a low sed rate in CFS being

> indicative of hypercoagulation. i have always had low sed rates when

> they were taken (between 0 and 3), but i always wondered why that

> would indicate a problem.

>

> i believe the reference range for a Sed Rate is 0-15, so how can

> anybody have a low sed rate when the ref range starts at 0?

>

> any thoughts would be appreciated.

>

> thanks

> bill

>

>

>

>

>

>

>

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I think my question is getting confused here. i am trying to figure

out why a sed rate of " 0 " is considered unhealthy and why medicine

does not acknowledge this....

normal, abnormal, healthy, unhealthy. whatever the terminology, i'm

trying to figure out the reasoning.

as my understanding of the test goes, an ESR is done to check for

signs of inflamation. a " high " ESR indicates the presence of

inflamation.

the idea of hypercoagulation, as i understand it, is that due to

immune system activation there is a build up of fibrin in the blood.

thus, i would think that the ESR should be high when the blood is in

a hypercoaguable state.

but that's not what i'm reallly confused about. what i really want to

know is why a low sed rate is considered bad, and what the arguement

is by mainstream medicine as to why a low sed rate is not bad.

thanks

bill

> Try www.hemex.com

> The reasoning is empirical. People with low sed rates also are

found to have excess fibrin (hypercoagulation). When treated, the

sed rates come up. I don't believe you are correct that

hypercoagaguable would be a high sed rate. It is the opposite. Sed

rate is how fast the blood settles. If you have thick blood it does

not settle fast. As I said before, high sed rate is usually

associated with autoimmune problems such as lupus, although I don't

know why those types of illnesses cause that.

>

> A 0 sed rate means the blood doesn't settle out, so I don't see how

anyone could think that " 0 " is normal. " Normal " is probably

something like 5-10. But they took the range down to 0 because they

didn't know it was a problem.

> Thanks,

> Doris

> ----- Original Message -----

> From: winsomme

>

> thanks for your posts. i guess i'm not so worried about the

reference

> range, but the idea of a " low sed rate. " could somebody point me

to

> some literature as to what makes a low sed rate abnormal.

>

> it is my understanding that if you are hypercoaguable you would

have

> a " high " sed rate. and that technically there is no such thing as

a

> low sed rate because " 0 " is normal.

>

>

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Bill -

I went through this some time ago, here is what I remember and found...

ESR is used as a measure of inflammation based on the fact that some

proteins produced by inflammation cause the red cells to clump together (in

nice neat little stacks of discs) more than normal. Because they are

clumped they tend to fall out of suspension and drop to the bottom. The

faster they drop, the higher the " sedimentation Rate " . So the reasoning is

- higher sed rate means they are more clumped which means that more of this

inflammatory protein must be present which means that there is

inflammation. This is the normal way of looking at it.

However there are some things that stop sedimentation, and lead to low ESRs

as a result. Firstly, extra thick blood. This I think is the main reason

PWCs score a low sed rate. THis is Berg's theory, there is a quote

below. It is also shown in the second quote as " hyperviscosity " .

A second reason is if the red cells are distorted and not in their normal

nice disk shape. In this case, they are unable to clump in the neat stacks

becasue they don't fit together, so they don't clump as much and thus they

don't sediment as fast.

Here are two references that tell you a bit about the subject generally,

google has plenty more under CFS ESR.

http://www.aafp.org/afp/991001ap/1443.html

http://www.fpnotebook.com/HEM113.htm

here's a quote from Berg...

We are writing a new journal article addressing the Normal Range of Sed

Rates (ESRs). <5 test values are indicative of a hypercoagulable state. The

only time this is not true is a cancer called Multiple Myeloma where there

is a lot of extra protein producced by the cancer cells. In either case,

because of the Soluble Fibrin or extra proteins, the RBCs cannot settle out

of the plasma and thus you have rates of 0-4. The lower the Sed Rate, the

more SFM and the more hypercoagulable the patient is

and from http://www.aafp.org/afp/991001ap/1443.html

A decreased ESR is associated with a number of blood diseases in which red

blood cells have an irregular or smaller shape that causes slower

settling.1,3 In patients with polycythemia, too many red blood cells

decrease the compactness of the rouleau network and artifactually lower the

ESR. An extreme elevation of the white blood cell count as observed in

chronic lymphocytic leukemia has also been reported to lower the ESR.1,5

ypofibrinogenemia, hypergammaglobulinemia associated with dysproteinemia,

and ******************hyperviscosity***************** may each cause a

marked decrease in the ESR. Although it has been reported that drug therapy

with aspirin or other nonsteroidal anti-inflammatory agents may decrease

the ESR, this has been disputed.2,3

Cheers,

n

At 07:40 27/05/03, you wrote:

>thanks for your posts. i guess i'm not so worried about the reference

>range, but the idea of a " low sed rate. " could somebody point me to

>some literature as to what makes a low sed rate abnormal.

>

>it is my understanding that if you are hypercoaguable you would have

>a " high " sed rate. and that technically there is no such thing as a

>low sed rate because " 0 " is normal.

>

>i totally understand that reference ranges aren't always the best

>measure, but when the range tells you that there is no " LOW " side,

>then what is the reasoning behind suggesting that it is abnormal?

>

>anyway, these are just some questions circulating around in my head.

>

>thanks

>bill

>

>

>

>

>

>

>

> > Bill,

> > Reference ranges are almost worthless. Sometimes they call

>it " normal

> > range " . In pharmacy school, profs kept calling various health

>problems

> > " normal " and one day I asked how that could be. The prof replied

>that

> > " normal " means the norm or average. And the average American has

>health

> > problems!

> > This explains in part while doctors may tell you it's normal,

>when you

> > complain of fatigue, PMS, pain, or whatever. And we think " normal

>means the

> > ideal, healthy state, to which we want to return.

> > For example, since the average American is somewhat anemic, the

> > reference range on your lab results shows anemic as " normal " . To

>compound

> > matters, the labs don't use the whole population's results for their

> > reference ranges, they use only the population consisting of people

>they've

> > tested. Which is mostly sick people. While everybody may get a CBC

> > (complete blood count), so that some non-anemic people are

>included, other

> > tests are done only on those who are suspected of having unhealthy

>levels

> > for that test. I almost used the words " abnormal results " . Even

>if a high

> > percentage of testees have unhealthy results, they are all counted

>in the

> > pool that determines what the " normal' is.

> > Then, a very wide part of the resulting bell curve, is counted

>in the

> > reference, or " normal " range. Only the most extreme results are

>left out.

> > For your sed rate results, 0-3 is in the low end of " normal',

>but

> > science has shown it to be unhealthy.

> > Hope this helps.

> >

> > Barb

> >

> >

> > ESR/Sed Rate

> >

> > I was wondering if someone could illuminate something that has

> > confused me for some time.

> >

> > over time, i have often read about a low sed rate in CFS being

> > indicative of hypercoagulation. i have always had low sed rates when

> > they were taken (between 0 and 3), but i always wondered why that

> > would indicate a problem.

> >

> > i believe the reference range for a Sed Rate is 0-15, so how can

> > anybody have a low sed rate when the ref range starts at 0?

> >

> > any thoughts would be appreciated.

> >

> > thanks

> > bill

> >

> >

> >

> >

> >

> >

> >

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Bill,

The erythrocyte sedimentation test is a very old test. You're

right, a high value of the ESR indicates inflammation.

Normally red blood cells repel each other electrostatically because

of their common zeta potential. Anything that helps to cancel the

zeta potential out will assist the red blood cells in clumping

together and in forming rouleaux, which are like stacks of poker

chips. When the red cells are joined together, it decreases their

effective surface-to-volume ratio, and thus increases their weight-

to-drag ratio. They are thus able to fall out of suspension in the

viscous medium of plasma faster. This is what happens in

inflammation states and in certain diseases. Thus, a high ESR has

traditionally been an indicator of inflammation or these other

diseases.

In the case of hypercoagulation, what happens is that the

fibrinogen, which normally would increase the ESR if present in

excess because of its effect on cancelling the zeta potential, is

joined together to produce fibrin fibers. These larger fibers act

as mechanical barriers and impede the settling of red blood cells,

producing a lower ESR.

As I understand it this is something that was discovered rather

recently by Berg. It isn't yet accepted by the conventional

medical community, and probably won't be unless someone who

is " properly placed " in the medical establishment publishes a

statistally significant double-blind paper studying it in one of the

respected journals, and it is verified by a second study, etc.

Berg is a guy with a master's degree who runs a private lab in

Phoenix, Arizona. The alternative practitioners pay attention to

him, and he sponsors a booth at their conferences and gives talks.

But to get it into medical textbooks to be taught at medical

schools, the information has to have a more sophisticated pedigree

than that. It needs to come from s Hopkins, or Mayo Clinic or

UC San Francisco, or Duke or etc. There may not be much incentive

for the drug companies, who fund most biomedical research, to fund

such a study. There probably aren't big profits in selling heparin,

because it's a natural substance and thus can't be patented to

achieve a monopoly position, as is normally done with

pharmaceuticals. So I think that's the nitty-gritty behind this

situation.

Rich

> > Try www.hemex.com

> > The reasoning is empirical. People with low sed rates also are

> found to have excess fibrin (hypercoagulation). When treated, the

> sed rates come up. I don't believe you are correct that

> hypercoagaguable would be a high sed rate. It is the opposite.

Sed

> rate is how fast the blood settles. If you have thick blood it

does

> not settle fast. As I said before, high sed rate is usually

> associated with autoimmune problems such as lupus, although I

don't

> know why those types of illnesses cause that.

> >

> > A 0 sed rate means the blood doesn't settle out, so I don't see

how

> anyone could think that " 0 " is normal. " Normal " is probably

> something like 5-10. But they took the range down to 0 because

they

> didn't know it was a problem.

> > Thanks,

> > Doris

> > ----- Original Message -----

> > From: winsomme

> >

> > thanks for your posts. i guess i'm not so worried about the

> reference

> > range, but the idea of a " low sed rate. " could somebody point

me

> to

> > some literature as to what makes a low sed rate abnormal.

> >

> > it is my understanding that if you are hypercoaguable you

would

> have

> > a " high " sed rate. and that technically there is no such thing

as

> a

> > low sed rate because " 0 " is normal.

> >

> >

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thanks for the info.

the reason i was posting this question is my most recent sed rate

was " 0 " , but my ISAC from HEMEX was all normal.

thanks

bill

>

>

> > Rich,

> >

> > Does this then resemble what Les Sipsoms work describes about

the 'clumping' blood cells that are not able to get through the

capilaries?

>

> That 'should' read Les Simpson....shouldn't type when I'm this

tired....sigh...

>

> Marcia

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Bill,

I think that's interesting. Have you had your blood examined by

live blood dark field microscopy? It might be possible to actually

see what's in the blood that is preventing settling of the red cells.

Rich

> thanks for the info.

>

> the reason i was posting this question is my most recent sed rate

> was " 0 " , but my ISAC from HEMEX was all normal.

>

> thanks

> bill

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> Anything that helps to cancel the

> zeta potential out will assist the red blood cells in clumping

> together and in forming rouleaux, which are like stacks of poker

> chips. When the red cells are joined together, it decreases their

> effective surface-to-volume ratio, and thus increases their weight-

> to-drag ratio. They are thus able to fall out of suspension in the

> viscous medium of plasma faster. This is what happens in

> inflammation states and in certain diseases. Thus, a high ESR has

> traditionally been an indicator of inflammation or these other

> diseases.

Rich,

Does this then resemble what Les Sipsoms work describes about the 'clumping'

blood cells that are not able to get through the capilaries?

I don't know if they did SED rates on me in my early years since CFS hadn't been

invented yet, but the last 4 or so yrs my SED rate is now high.

Marcia

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Marcia,

Les doesn't really directly observe " clumping. " What he does is to

examine the shapes of individual red blood cells that have

been " fixed " with glutaraldehyde so that their shapes are " frozen "

and don't change during shipment to his lab in New Zealand. He

finds that there are more abnormally shaped red cells in people with

CFS than in normal, healthy people, particularly more " flat " cells,

which have flat sides, without the usual dimples.

He infers that these abnormally shaped red cells would have

difficulty in travelling through the smaller diameter capillaries,

and thus would not be able to deliver oxygen as well as normal cells.

As n said in his post about this, abnormally shaped red cells

can't " clump " together to form rouleaux as readily as normal cells,

so having abnormally shaped red cells slows down their sedimentation

and gives a lower ESR.

I don't think Les has done a correlation of ESR with percentage of

flat cells, but that would be interesting to see. My impression is

that Les pretty much sticks with examining red cells with his

scanning electron microscope and reporting the results. He doesn't

wander too far afield from this activity, for example by trying to

understand the biochemistry of what's going on and offering

hypotheses to explain his observations. He leaves that to others.

Rich

> Rich,

>

> Does this then resemble what Les Simpson's work describes about

the 'clumping' blood cells that are not able to get through the

capilaries?

>

> I don't know if they did SED rates on me in my early years since

CFS hadn't been invented yet, but the last 4 or so yrs my SED rate

is now high.

>

> Marcia

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> Rich,

>

> Does this then resemble what Les Sipsoms work describes about the 'clumping'

blood cells that are not able to get through the capilaries?

That 'should' read Les Simpson....shouldn't type when I'm this tired....sigh...

Marcia

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Hi Marcia,

The little I know of Les Simpson's findings is that red blood cells

are misshapen and his hypothesis is that they cannot easily flow

through the capillaries or that they may have reduced oxygen carrying

capacity. The bi-concave shape of the RBC does aid in its ability to

carry oxygen so this does make sense.

If blood volume is reduced due to loss of water. The blood would

be more hypertonic than normal. The website below shows what a red

blood cell looks like in a hypertonic solution (scroll down toward

the bottom of the page and clik on the photo of the RBC in a

hypertonic solution as seen through a microscope.

http://cwx.prenhall.com/bookbind/pubbooks/hillchem3/medialib/media_por

tfolio/12.html

I sometimes wonder if this could not account for the misshapen red

blood cells found by Dr. Simpson, but he being a Dr. I would imagine

that he would know about the effects of the solution upon the cell.

I would like to know more about what he is finding.

Anyway, take a look, its pretty interesting.

All the best,

Jim

>

>

> > Anything that helps to cancel the

> > zeta potential out will assist the red blood cells in clumping

> > together and in forming rouleaux, which are like stacks of poker

> > chips. When the red cells are joined together, it decreases their

> > effective surface-to-volume ratio, and thus increases their

weight-

> > to-drag ratio. They are thus able to fall out of suspension in

the

> > viscous medium of plasma faster. This is what happens in

> > inflammation states and in certain diseases. Thus, a high ESR has

> > traditionally been an indicator of inflammation or these other

> > diseases.

>

> Rich,

>

> Does this then resemble what Les Sipsoms work describes about

the 'clumping' blood cells that are not able to get through the

capilaries?

>

> I don't know if they did SED rates on me in my early years since

CFS hadn't been invented yet, but the last 4 or so yrs my SED rate is

now high.

>

> Marcia

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Jim,

That really is a pretty picture, but the blood in PWCs is usually

not found to be hypertonic, and that type of cell shape is not what

Les predominately sees. Blood chemistry panels usually show the

electrolytes and proteins to be in the normal ranges in PWCs. It's

important to keep in mind that the concentration of each of these

solutes is controlled, and when the blood plasma volume decreases by

excessive urination, for example, the separate control systems

continue to control the concentrations of the major electrolytes.

Rich

> >

> >

> > > Anything that helps to cancel the

> > > zeta potential out will assist the red blood cells in clumping

> > > together and in forming rouleaux, which are like stacks of

poker

> > > chips. When the red cells are joined together, it decreases

their

> > > effective surface-to-volume ratio, and thus increases their

> weight-

> > > to-drag ratio. They are thus able to fall out of suspension

in

> the

> > > viscous medium of plasma faster. This is what happens in

> > > inflammation states and in certain diseases. Thus, a high ESR

has

> > > traditionally been an indicator of inflammation or these other

> > > diseases.

> >

> > Rich,

> >

> > Does this then resemble what Les Sipsoms work describes about

> the 'clumping' blood cells that are not able to get through the

> capilaries?

> >

> > I don't know if they did SED rates on me in my early years since

> CFS hadn't been invented yet, but the last 4 or so yrs my SED rate

is

> now high.

> >

> > Marcia

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Rich,

Thanks for the further explanation. I'm sure that the hypertonic

solution that produced the cell structure deformations in that

picture was much more hypertonic than would be found in blood. Per

info in the " Cell Physiology Sourcebook " an acidic pH in the cell

could possibly affect the cytoskeletal structure of the cell.

Another possibility that I've thought about is that when

constructing the RBC maybe their was not sufficient resources to

build it properly. But with the tight controls that the body has on

everything I doubt that that may be the case. It would probably not

build the RBC until all required ingredients were available.

All the best,

Jim

> Jim,

>

> That really is a pretty picture, but the blood in PWCs is usually

> not found to be hypertonic, and that type of cell shape is not what

> Les predominately sees. Blood chemistry panels usually show the

> electrolytes and proteins to be in the normal ranges in PWCs. It's

> important to keep in mind that the concentration of each of these

> solutes is controlled, and when the blood plasma volume decreases

by

> excessive urination, for example, the separate control systems

> continue to control the concentrations of the major electrolytes.

>

> Rich

>

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I went to bed last night and i was still wrestling with this topic.

here's my quandry: what is it about this test (ESR) that it does not

have a low level?

Forget about CFS for a second. just think about this test by itself,

with no diagnosis attatched to it. there must be some reason (whether

it is right or not) to think that a Sed Rate of " 0 " or " 1 " is

healthy.

it's not a complicated test, right?

what would a hematologist say if you asked them to explain what a Sed

Rate of " 0 " means?

the reason i'm harping on this is because one of the things we are

looking for are markers that can demonstrate that PWC's have a

physiological problem. and if a large portion of us have a sed rate

<4, and a sed rate of <4 is bad, then wouldn't a portion of our

problem be picked up right away. An ESR was one of the first tests i

had done four years ago.

There must be some reason that science does not recognize that a low

sed rate is bad. i'm not saying that it is accurate, but they must

have a reason. forget about CFS for a second, just think about the

the sed rate test.

a low sed rate means (according to Berg et al) that the RBC's are not

moving properly through the blood, right? So, what is the counter

arguement?

just think back to that first DR appt you had, and think what it

would have like if the DR said, " Your sed rate is LOW. I'm not sure

what is causing it to be low, but it is LOW none-the-less. "

thanks

bill

> > Jim,

> >

> > That really is a pretty picture, but the blood in PWCs is usually

> > not found to be hypertonic, and that type of cell shape is not

what

> > Les predominately sees. Blood chemistry panels usually show the

> > electrolytes and proteins to be in the normal ranges in PWCs.

It's

> > important to keep in mind that the concentration of each of these

> > solutes is controlled, and when the blood plasma volume decreases

> by

> > excessive urination, for example, the separate control systems

> > continue to control the concentrations of the major electrolytes.

> >

> > Rich

> >

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and correct me if i'm wrong, but in order to demontstrate this fact

(low sed rate in CFS), wouldn't you just have to take a group of

PWC's and a group of healthy controls and show that healthy people

don't have low sed rates and PWC's do....?

i mean the idea that if you have a low sed rate, you won't feel well

should be really easy to demonstrate.

> > > Jim,

> > >

> > > That really is a pretty picture, but the blood in PWCs is

usually

> > > not found to be hypertonic, and that type of cell shape is not

> what

> > > Les predominately sees. Blood chemistry panels usually show

the

> > > electrolytes and proteins to be in the normal ranges in PWCs.

> It's

> > > important to keep in mind that the concentration of each of

these

> > > solutes is controlled, and when the blood plasma volume

decreases

> > by

> > > excessive urination, for example, the separate control systems

> > > continue to control the concentrations of the major

electrolytes.

> > >

> > > Rich

> > >

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> i mean the idea that if you have a low sed rate, you won't feel well

> should be really easy to demonstrate.

This is true about low blood pressure too. Its obviously low, but back in the

late 70's when I first became ill (before CFS was a recognized illness) my BP

was 90/50. My Dr. just said, well, if it

stays like that you should live forever !(meaning, how lucky for me, no high

BP). I just grumped 'You call this living????'. Then he admitted it would

probably not be associated with a lot of

energy! Thats as far as it ever went. Over the years it did go up, unhelped by

him. I think the avg. dr. just doesn't 'think' sometimes.

Marcia

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Bill,

I can imagine that it might be a bit chagrining to have a zero sed

rate with normal coagulation tests, and not know what that means.

As I understand it, the red blood cells in the plasma act like a

colloid. In a colloid, a particulate solid is suspended within a

liquid. The particles have a higher density than the liquid, so

that by Archimedes principle they would settle to the bottom, with

their rate of settling determined by the balance between the force

of gravity, determined by the density difference, and the drag force

due to the viscosity of the liquid. However, in a colloid, the

particles are small enough that the electrostatic forces that are

always present become significant compared to the gravitational

force. The electrostatic forces arise from the fact that the

quantum mechanically determined electron affinity is different for

each substance, and when two substances are placed in contact, there

is a net transfer of electrons from the one with the lower affinity

to the one with the higher affinity, until the resulting

electrostatic force balances the force due to the difference in

affinity. Then the net current ceases, and there is a residual net

charge, setting up what's called the zeta potential. The result of

this is that the colloidal particles all reach the same zeta

potential, if they are composed of the same substance. The are thus

charged with the same polarity, and since like charges repel, they

repel each other. If these electrostatic forces are sufficiently

strong, they can overbalance the gravitational force, and the

colloid will remain in suspension. This is called a stable colloid.

In the case of the red blood cells, they become negatively charged

relative to the plasma. There are proteins in the plasma that are

unsymmetrical and carry electrical charges as well. These can have

dipoles, which consist of a positive and a negative charge separated

by some distance on the molecule. These dipoles will orient in the

electrostatic field of the red blood cells, and in doing so will

tend to cancel out the field. If this is sufficiently effective,

the red cells will not only be able to fall out of the plasma under

the action of gravity, but they will also be able to join together,

bound by shorter-range Van der Waals forces, and stack together,

forming rouleaux. These forces don't come into play unless the

particles are able to get close together, i.e., unless the forces

arising from the zeta potential are sufficiently decreased.

If the red cells stack together, they decrease their effective

surface to volume ratio. They can thus fall out more rapidly under

the force of gravity, because the drag force will be minimized. So

the effect of the protein dipoles is two-fold: first, it

destabilized the colloid and allows the red blood cells to fall out

of the plasma, and second, it allows them to aggregate and thus to

drop out faster, producing a high sed rate.

These are two separate issues. As I understand it, when a person

has a hypercoagulation problem, the fibrinogen, which would normally

act to increase the sed rate by helping to cancel out the zeta

potential, actually binds together to form larger fibrin fibers, and

these act as mechanical barriers to the sedimentation of the red

cells. This thus produces a low sed rate, but not a zero sed rate.

In order to have a truly zero sed rate, it means that the zeta

potential is not sufficiently cancelled out, and the red blood cells

are thus repelling each other enough to preserve a stable colloid.

They thus remain in suspension indefinitely. What could cause

this? I think that one possibility is that there is a deficiency of

proteins in the blood that would normally balance the zeta

potential. Have you had your blood proteins measured, i.e.,

globulins and albumin?

I think you are also asking why doctors haven't paid any attention

to low sed rates, or zero sed rates in the past. I think it's just

a matter of the significance of this not having been understood

until Berg pointed it out a few years ago, and the difficulty

in getting something new tested, published, and accepted as medical

doctrine by the huge medical establishment. In principle you are

right that it shouldn't be too difficult to test this idea.

However, this takes someone with the right credentials and stature

in the scientific community, funding, motivation and great care to

make sure it is done in a convincing and statistally significant

way, and factors that may confound the interpretation must be

thought out and compensated for. It really is a lot of work to do a

scientific experiment in a way that will convince the (rightly)

critical scientific community of something new. Believe me, I

worked in scientific research for nearly 40 years, and I have some

feeling for this, having been involved in a lot of scientific

debates over the interpretation of experiments. It may seem very

simple, but it isn't.

There are actually lots of things in medicine that are not well

proven for many of these same reasons, but are used anyway. A few

years ago the medical establishment started emphasizing the concept

of " evidence-based medicine " to try to counter this problem, but it

has revealed that there are many things that are done that aren't

based on documented evidence. They have just been " grandfathered

in " by experience and long practice. The sed rate test is one of

these very old lab tests that continues to be used, even though it

isn't always well understood on a " first principles " basis.

I hope this helps.

Rich

> > > > Jim,

> > > >

> > > > That really is a pretty picture, but the blood in PWCs is

> usually

> > > > not found to be hypertonic, and that type of cell shape is

not

> > what

> > > > Les predominately sees. Blood chemistry panels usually show

> the

> > > > electrolytes and proteins to be in the normal ranges in

PWCs.

> > It's

> > > > important to keep in mind that the concentration of each of

> these

> > > > solutes is controlled, and when the blood plasma volume

> decreases

> > > by

> > > > excessive urination, for example, the separate control

systems

> > > > continue to control the concentrations of the major

> electrolytes.

> > > >

> > > > Rich

> > > >

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Bill,

I have a sed rate of around 10 or 12, but have two genetic coagulation

defects and an abnormal ISAC panel. When I stop my heparin for more than a

few days or so, I can definitely tell the difference. So I wonder why my sed

rate isn't low like most pwc's. Any ideas, Rich?

Donna in NC

Re: ESR/Sed Rate

> thanks for the info.

>

> the reason i was posting this question is my most recent sed rate was " 0 "

, but my ISAC from HEMEX was all normal.

>

> thanks

> bill

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Donna,

Take a look at my message #21812 to Bill. My guess is that you have

some proteins in your blood that are cancelling the zeta potential

of the red blood cells and overwhelming the mechanical barrier

effect of the fibrin molecules. I think we should keep in mind that

Berg has not said that a low sed rate is an unequivocal sign

that there is hypercoagulation, or that a high sed rate is an

unequivocal sign that there is no hypercoagulation. I have a copy

of the slides he used at the recent OHM meeting in San Francisco,

and one of them says, " Sed Rates < 5 are indicative of a hypercoag

state. " He has always said that to really be sure, you have to take

the types of tests that he offers.

Rich

> Bill,

> I have a sed rate of around 10 or 12, but have two genetic

coagulation

> defects and an abnormal ISAC panel. When I stop my heparin for

more than a

> few days or so, I can definitely tell the difference. So I wonder

why my sed

> rate isn't low like most pwc's. Any ideas, Rich?

>

> Donna in NC

>

> Re: ESR/Sed Rate

>

>

> > thanks for the info.

> >

> > the reason i was posting this question is my most recent sed

rate was " 0 "

> , but my ISAC from HEMEX was all normal.

> >

> > thanks

> > bill

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rich

thanks for posting so much info on this topic.

i'm not so much frustrated by my DR's interpretation of the ESR, but

as to why the reference range is for the test is 0-15 (0-20, i have

seen it that too).

To me, that means that when they developed the test, for some reason

they determined that you can be healthy and have a sed rate of " 0 "

what we seem to be saying here is that you can't have a sed rate

of " 0 " and be healthy because with a sed rate of 0, your RBC's are

not settling properly.

i can see why interpreting the cause of a low sed rate would be

complicated, but i can't see why recognizing that a Sed rate of " 0 "

is unhealthy is....

At some point the developers of this test or those who set up

guidelines for the test looked at it and said that a sed rate of " 0 "

is healthy.

and i'm just trying to figure out what that rationale is....

thanks

bill

> > > > > Jim,

> > > > >

> > > > > That really is a pretty picture, but the blood in PWCs is

> > usually

> > > > > not found to be hypertonic, and that type of cell shape is

> not

> > > what

> > > > > Les predominately sees. Blood chemistry panels usually

show

> > the

> > > > > electrolytes and proteins to be in the normal ranges in

> PWCs.

> > > It's

> > > > > important to keep in mind that the concentration of each of

> > these

> > > > > solutes is controlled, and when the blood plasma volume

> > decreases

> > > > by

> > > > > excessive urination, for example, the separate control

> systems

> > > > > continue to control the concentrations of the major

> > electrolytes.

> > > > >

> > > > > Rich

> > > > >

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Donna,

Sorry, I meant message #58212. Can I blame it on brain fog?

Rich

> > Bill,

> > I have a sed rate of around 10 or 12, but have two genetic

> coagulation

> > defects and an abnormal ISAC panel. When I stop my heparin for

> more than a

> > few days or so, I can definitely tell the difference. So I

wonder

> why my sed

> > rate isn't low like most pwc's. Any ideas, Rich?

> >

> > Donna in NC

> >

> > Re: ESR/Sed Rate

> >

> >

> > > thanks for the info.

> > >

> > > the reason i was posting this question is my most recent sed

> rate was " 0 "

> > , but my ISAC from HEMEX was all normal.

> > >

> > > thanks

> > > bill

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Bill,

In principle, I don't think it's necessary for red blood cells to

settle out of standing blood in order for them to do their job of

transporting oxygen to the other cells of the body. After all, in

the body, you want them to stay mixed with plasma so they can be

transported around the circulatory system by it. So I don't see an

intrinsic reason why a person could not be healthy while having a

zero sed rate. It's just that most people's red cells do settle out

at some rate, and Berg has noticed that there is a fairly good

correlation between having a sed rate less than 5 and being

hypercoagulated. He has also mentioned that having cold hands and

feet all the time is suggestive of hypercoagulation. But these

aren't 100% one-to-one correspondences, just indicators that it

might be worthwhile to test for hypercoagulation.

Rich

> rich

>

> thanks for posting so much info on this topic.

>

> i'm not so much frustrated by my DR's interpretation of the ESR,

but

> as to why the reference range is for the test is 0-15 (0-20, i

have

> seen it that too).

>

> To me, that means that when they developed the test, for some

reason

> they determined that you can be healthy and have a sed rate of " 0 "

>

> what we seem to be saying here is that you can't have a sed rate

> of " 0 " and be healthy because with a sed rate of 0, your RBC's are

> not settling properly.

>

> i can see why interpreting the cause of a low sed rate would be

> complicated, but i can't see why recognizing that a Sed rate

of " 0 "

> is unhealthy is....

>

>

> At some point the developers of this test or those who set up

> guidelines for the test looked at it and said that a sed rate

of " 0 "

> is healthy.

>

> and i'm just trying to figure out what that rationale is....

>

> thanks

> bill

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