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notan wrote:

> This message is for those wanting to know what end-stage achalasia is.

If forgot to add one very important point. Not everyone progress to

end-stage. Some don't progress, some progress slowly and some progress

quickly.

notan

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That is one thing that us definitely true about achalasia, you never know how

aggressive it is going to be. My first myotomy was December 1993. At that time

not much was being said, or heard of. My surgeon was very good in her treatment

with me, and it lasted 11years. 2004 my esophagus was in such bad shape that Dr

Rice wanted to do the ectomy but I wasn't mentally ready for it, and opted for a

2nd myotomy. From that point it progressed rapidly, and 3 years later I had the

esophagectmoy. This is a decision that I haven't regretted. It's like

said in her post, you get your life back and it seems normal, if you can call it

that. I have learned a lot from this board. Notan, you put in a lot of time

researching and trying to find answers that we all ask. I don't post much, but I

read each day and try to respond as I can. Between my 2 jobs and personal life

it gets a little hectic. I wish each and all the best of eating.

in West Virginia

Sent from 's iPhone

On Mar 3, 2011, at 11:44 PM, notan ostrich <notan_ostrich@...> wrote:

> notan wrote:

>> This message is for those wanting to know what end-stage achalasia is.

>

> If forgot to add one very important point. Not everyone progress to

> end-stage. Some don't progress, some progress slowly and some progress

> quickly.

>

> notan

>

>

>

> ------------------------------------

>

>

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Is there anything one could do after a myotomy to slow the progression fo the

achalasia?  After a myotomy, does the condition progress as a result of a new

obstruction?

 

> This message is for those wanting to know what end-stage achalasia is.

If forgot to add one very important point. Not everyone progress to

end-stage. Some don't progress, some progress slowly and some progress

quickly.

notan

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My doctor called my achalasia end stage because of the pig tail shaped

esophagus and pockets in it.It doesn't seem to be stretched too bad yet I don't

think. I now have Barretts also, so I must be scoped once a year.I am hoping I

can go at least another ten years before they even think about an Ectomy.I had

no idea I even had Achalasia until 1 1/2 years ago.

>

> This message is for those wanting to know what end-stage achalasia is.

>

> End-stage achalasia, typically is a massively dilated and tortuous

> oesophagus. It is also known as Stage IV achalasia or megaesophagus. The

> stages of achalasia are; Stage I-esophageal dilation up to 2-2.5 cm;

> Stage II-dilation up to 2.5-4 cm; Stage III up to 5-8 cm and Stage IV –

> lengthening and dilation of the esophagus over 8 cm. I have seen some

> paper refer to end-stage as starting a 6 cm. As the esophagus lengthens

> it bends (becomes tortuous) that often produces an S shape called

> sigmoid (the Greek letter for S is sigma). Stage IV is called End-Stage

> because there is no stage V and it is final result of lengthening and

> dilation. Sometimes, but not always, at this stage the esophagus is so

> dysfunctional and unresponsive to treatment that esophagectomy becomes

> the treatment option. End-stage does not mean it will kill you though

> for some tube feeding or esophagectomy are needed to survive.

>

> notan

>

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thanks so much for this info Notan. I had an endoscopy last year and the result

was 'tortuous oesophagus'. I didn't clarify this as a definition (unusual for me

as I ask a lot of questions!) but it would certainly explain my very severe

symptoms. I am due to see my consultant soon so will definately be asking more

then.

I am interested in people's comments and experiences of an esophagectomy. I am

not ready to consider this emotionally yet but would love to hear what people

think.

thanks so much.

Kay xx

>

> This message is for those wanting to know what end-stage achalasia is.

>

> End-stage achalasia, typically is a massively dilated and tortuous

> oesophagus. It is also known as Stage IV achalasia or megaesophagus. The

> stages of achalasia are; Stage I-esophageal dilation up to 2-2.5 cm;

> Stage II-dilation up to 2.5-4 cm; Stage III up to 5-8 cm and Stage IV –

> lengthening and dilation of the esophagus over 8 cm. I have seen some

> paper refer to end-stage as starting a 6 cm. As the esophagus lengthens

> it bends (becomes tortuous) that often produces an S shape called

> sigmoid (the Greek letter for S is sigma). Stage IV is called End-Stage

> because there is no stage V and it is final result of lengthening and

> dilation. Sometimes, but not always, at this stage the esophagus is so

> dysfunctional and unresponsive to treatment that esophagectomy becomes

> the treatment option. End-stage does not mean it will kill you though

> for some tube feeding or esophagectomy are needed to survive.

>

> notan

>

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Sorry Notan, one more thing. Is there evidence that end stage reverts on it's

own. My deterioration since the myotomy has been gradual and the severity can

fluctuate.

thanks so much for all your info.

Kay

> >

> > This message is for those wanting to know what end-stage achalasia is.

> >

> > End-stage achalasia, typically is a massively dilated and tortuous

> > oesophagus. It is also known as Stage IV achalasia or megaesophagus. The

> > stages of achalasia are; Stage I-esophageal dilation up to 2-2.5 cm;

> > Stage II-dilation up to 2.5-4 cm; Stage III up to 5-8 cm and Stage IV –

> > lengthening and dilation of the esophagus over 8 cm. I have seen some

> > paper refer to end-stage as starting a 6 cm. As the esophagus lengthens

> > it bends (becomes tortuous) that often produces an S shape called

> > sigmoid (the Greek letter for S is sigma). Stage IV is called End-Stage

> > because there is no stage V and it is final result of lengthening and

> > dilation. Sometimes, but not always, at this stage the esophagus is so

> > dysfunctional and unresponsive to treatment that esophagectomy becomes

> > the treatment option. End-stage does not mean it will kill you though

> > for some tube feeding or esophagectomy are needed to survive.

> >

> > notan

> >

>

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wrote:

> ... I don't post much, ...

Completely reasonable, but it is always good to get your posts and

updates. I would hate for this site to be just the kind of posts I make.

One of the reasons I will miss Isabella's posts is that they were so

real and easy to relate to. People like Isabella and you provide a kind

of vitality that makes this like being in the presence of others. Who

needs TV drama? This is real life and we can help each other. Your story

is important and we are thankful for as much of it as we get.

notan

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Mat T wrote:

> Is there anything one could do after a myotomy to slow the progression fo the

achalasia?

Your esophagus is pretty much just a hollow tube of muscle. It is a lot

like an earth worm. When a worm dies it becomes longer and very flimsy.

This is because the muscles loose their tone. It is possible that

something like that happens as the muscles of the esophagus atrophy and

loose tone. The esophagus become dilated, longer and easier to distend.

Others here have reported a surgeon describing doing surgery on an

end-stage esophagus as being like working on wet paper. If, and I say

IF, there is nothing you can do to prevent the loss of the nerves that

leads to atrophy then there isn't much you can do to stop the atrophy.

Sometimes in achalasia the LES (Lower Esophageal Sphincter) is not

atrophied it hypertrophied from all of the intense spasming it does. I

suspect that those that have strong enough spasms in the lower esophagus

above the LES may be more likely to avoid the atrophy and dilation than

those with little or no muscle action in the lower esophagus. I don't

really know though. So, if there is progressive nerve damage that is

caused by an infection, toxin, immune disorder, or genetic flaw and it

leads to decreased muscle action of the esophagus then you would have to

find a way to stop that process. A number of studies have shown more

nerve loss in end-stage achalasia than in earlier stages.

It may also be that distention itself can lead to more nerve damage.

Nerves don't like to be stretched too far. When we fight to get food

down but it stays in the esophagus and we continue to eat more that

causes the esophagus to distend. The farther, longer and more often the

esophagus is distended may increase the risk of increasing nerve damage.

In older, decades ago, papers air trapped in the esophagus due to a

failure of the UES (Upper Esophageal Sphincter) was given as a possible

cause for esophageal distention and dilation. I don't remember seeing

that in more recent papers. Maybe they gave up on that idea. It does

make me wonder about the gas from carbonated drinks. There is enough

pressure there to be a concern while a myotomy is healing. Is it enough

to cause nerve damage? Maybe, maybe not. I don't really know. How the

UES functions is going to make a difference. So, if distention does

cause nerve damage then you want to prevent distention or at least

reduce the time and distance the esophagus is distended.

Repeated distention of the esophagus may cause it to become permanently

dilated even without any nerve damage simply because of the intense

stretching. This is a bit like creating a loop in an ear lobe for ear

gauge jewelery. You stretch it a little more each time over a long time.

When the esophagus is holding food it becomes heavy and may try to sag

and lengthen. Again air trapped in the esophagus may be a factor in

stretching, or maybe not. It seems like keeping the esophagus clear is a

good idea. Once the esophagus starts to sag that is going to be hard to do.

> After a myotomy, does the condition progress as a result of a new obstruction?

A new obstruction could lead to food being trapped causing distention. A

number of studies have reported that damage to the esophagus from acid

reflux (causing strictures) is a major cause of long-term myotomy

failure. I think one thing you can do is try to avoid acid reflux. Hence

my position on another topic.

Selected links of interest:

Repetitive proximal esophageal contractions: a new manometric finding

and a possible further link between Parkinson's disease and

achalasia.http://www.ncbi.nlm.nih.gov/pubmed/6088351

http://www.ncbi.nlm.nih.gov/pubmed/11453565

" We speculate that the common link between all three disease processes

may be poor distensibility of the esophagus. "

Evolutive radiological changes of the esophagus in patients with

achalasia who did not receive treatment.

http://www.ncbi.nlm.nih.gov/pubmed/17342353

" These results suggest that there is a progressive deterioration in the

radiological parameters of the esophagus in patients with achalasia not

treated over a 5-year period of observation. "

Achalasia - An Update

http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2912115/?tool=pubmed

" Early disease has more of an inflammatory component, with some of the

ganglion cells appearing to be intact, while end stage disease is

associated with complete loss of ganglion cells and replacement with

myenteric fibrosis. "

Ear gauge

http://www.gettyimages.com/detail/83589021/Photodisc

notan

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I too will miss Isabella. She helped me so much when having all my issues. We

talked daily to see how each of us were doing. The one thing I have found is

when you actually put a face to the people you talk with so much. Getting to

meet and understand what each of us have gone thru is so interesting. I recently

noticed a post from someone who is around a 3 hour drive from me. This disease

is so strange. I'm currently fighting an acid reflux issue. I've gained more

weight than what Dr Rice has advised me to be. I feel that between that and some

of my current meds are the issue. Enjoy the day, I'm off to relax for a few

hours.

in WV

Sent from 's iPhone

On Mar 6, 2011, at 12:48 PM, notan ostrich <notan_ostrich@...> wrote:

> wrote:

> > ... I don't post much, ...

>

> Completely reasonable, but it is always good to get your posts and

> updates. I would hate for this site to be just the kind of posts I make.

> One of the reasons I will miss Isabella's posts is that they were so

> real and easy to relate to. People like Isabella and you provide a kind

> of vitality that makes this like being in the presence of others. Who

> needs TV drama? This is real life and we can help each other. Your story

> is important and we are thankful for as much of it as we get.

>

> notan

>

>

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Thanks Notan, really helpful. Kay

> > ... Is there evidence that end stage reverts on it's own.

>

> Sorry, no, not that I know of.

>

> > My deterioration since the myotomy has been gradual and the severity can

fluctuate.

>

> I assume you are talking about your symptoms. Like how easy it is to

> eat. That is not always a good indication of the progression. The

> esophagus can dilate and lengthen without causing much change in

> symptoms. Then one day it seems to not work and you get a barium X-ray

> and find it is now sigmoid. If you get regular checkups of the esophagus

> you can probably avoid the surprise but you may not be able to avoid the

> progression. Many of us have better days and worse days or even months

> at a time. It can make knowing what long-term changes are actually

> happening. There are probably people who have end-stage and have no idea

> that they do. They just fight on as they have for years.

>

> The good news is that most people don't ever make it to an end-stage

> diagnosis. Some because they either just stop progressing or do so

> slowly and others because they are older when they get achalasia. And

> possibly some are never diagnosed because they stopped going to doctors

> for their achalasia and just live with it still being able to get by

> somehow.

>

> notan

>

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