We're wrong about, CJD and mad cows

December 11, 2002

Letter Daily Mail Dec 9

I'M APPALLED by the unfounded assumptions of 'experts' about the

hyper-infectious nature of the BSE/CJD agent (Mail). Basic evidence suggests

variant CJD didn't arise from eating BSE-affected cattle. Third World countries

imported even greater quantities of the incriminated British meat than we

consumed here, but no cases of vCJD have occurred in those countries. I've

travelled worldwide carrying out analyses of areas with prion diseases, of which

vCJD is one. These diseases increase only in areas with a high manganese/low

copper imbalance. Experiments at Cambridge and Case Western, Cleveland,

universities confirmed my findings that misshapen prions develop whenever brain

cells are exposed to this imbalance.

I found one organophosphate - poured on cows to counteract warble fly and on

humans for headlice - which bonded with copper in the brain, starving the prion

protein of its copper partner. When this happens, foreign metals, such as

manganese, are more likely to bond with prions, which can have devastating

consequences. Manganese is increasing in the environment due to its use as a

fertiliser and fungicide, a lead substitute in petrol, and emission from

factories. It was fed to UK cattle in calf milk replacement powder (at levels up

to 1,000 times higher than in normal cow's milk) and as supplements for bone

growth. Manganese is also fed to humans (in health food supplements), deer (for

antler growth) and cats -and all have had prion disease. The totalitarian

mindset about the origins of prion diseases betrays an agenda. It's more about

global profits for the GM soya protein empires than about the illusory health

risks of animal proteins to humans.

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