RUSSEL BACTERIA AND CANCER

December 15, 2003

In a message dated 11/18/03 6:49:20 PM Eastern Standard Time,

michaelmarty787@... writes:

> michaelmarty787@...

> Reply-to:cures for cancer

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> The Body

> The Forgotten Clue To

> The Bacterial Cause Of Cancer

> 11-17-3

>

> The twentieth century was indeed the century of Modern Medicine with

> tremendous strides made in the understanding and control of

> infectious diseases, as well as the introduction of life-saving

> antibiotics and vaccines. Unfortunately, along with these advances

> came the perils of genetic engineering, the increasing threat of

> newly emerging viruses, biowarfare, and bioterrorism

>

> Despite these scientific achievements, the cause of cancer remains a

> mystery. Scientists suspect genetic susceptibility, possible cancer-

> causing viruses, and environmental factors might play a role in some

> cancers, but none of these factors explain why millions of people die

> yearly from a variety of malignancies.

>

> 1852-1940, as pictured in

> The British Medical Journal, August 24, 1940.

>

> How could scientists put men on the moon, but remain so ignorant

> about cancer and its origin? How can the infectious causes of

> tuberculosis, leprosy, syphilis, smallpox, polio, malaria, and other

> viral and bacterial and parasitic diseases be understood, but the

> cause of cancer be unknown? Could the cause of cancer conceivably be

> an infectious agent that has been overlooked, ignored, or

> unrecognized by medical doctors in the twentieth century? Could the

> germ of cancer be hidden in the body? - a large microscopic

> form known to every pathologist for over a century!

>

> (1852-1940) and " the parasite of cancer "

>

> On December 3, 1890 , a pathologist in the School of

> Medicine at the Royal Infirmary in Edinburgh, gave an address to the

> Pathological Society of London in which he outlined his

> histopathologic findings of " a characteristic organism of cancer "

> that he observed microscopically in fuchsine-stained tissue sections

> from all forms of cancer that he examined, as well as in certain

> cases of tuberculosis, syphilis and skin infection.

>

> The parasite was seen within the tissue cells (intracellular) and

> outside the cells (extracellular). The size of 's parasite

> ranged from barely visible, up to " half again as large as a red blood

> corpuscle. " The largest round forms were easily seen microscopically.

> The large size of some of these bodies suggested a fungal or yeast-

> like parasite. provisionally classified the parasite as a

> possible " blastomycete " (a type of fungus); and called the

> forms " fuchsine bodies " because of their bluish-red staining

> qualities.

>

> Microbiology was still in its infancy in 's era, and it was

> generally thought that each microbe could only give rise to a single

> disease. Thus, the idea of a cancer germ (especially one that could

> also be identified in TB and syphilis) was received cautiously. Nine

> years later in 1899, in yet another report on " The parasite of

> cancer " appearing in The Lancet (April 29), admitted that

> finding cancer parasites in diseases other than cancer was indeed

> a " stumbling block. " By this time a considerable number of scientists

> concluded that bodies were merely the result of cellular

> degeneration of one kind or another. Furthermore, no consistent

> microbe was cultured from tumors; and the inoculation of these

> microbes into animals produced conflicting and often negative

> results.

>

> was trained as a pathologist, not as a microbiologist, and he

> avoided getting into the bacteriologic controversies regarding

> various microbes grown from cancer. He simply concluded, " It seems

> almost needless to add that there remains abundant work to be done in

> this important and attractive field. "

>

> After three years' work at the New York State Pathological Laboratory

> of the University of Buffalo, Harvey Gaylord confirmed 's

> research in a 36 page report titled " The protozoon of cancer " ,

> published in May, 1901, in the American Journal of the Medical

> Sciences. Gaylord found the small forms and the large sacs

> characteristic of bodies in every cancer he examined. Some

> large spherical bodies were four times the diameter of a leukocyte

> (white blood cell). Red blood cells measure about 7 micron in

> diameter and leukocytes are 2 to 3 times larger than red blood cells.

> Thus, some of the bodies that Gaylord observed attained the amazing

> size of around 50 micron in diameter. In addition, he found evidence

> of internal segmentation within the larger bodies " after the manner

> recognized in malarial parasites. " The tiniest forms appeared the

> size of ordinary staphylococci.

>

> bodies in a lymph node of Hodgkin's disease.

> Gram's stain, magnified 1000 times, (in oil).

>

> 's 1899 paper ended his writings of a cancer parasite, but his

> discovery quickly became known to pathologists as bodies.

> These bodies continue to fascinate researchers and physicians (like

> myself) up to the present time.

>

> Solitary " giant " body in a lymph node of Hodgkin's disease

> (cancer), magnified 1000 times. Gram's stain, magnified 1000 times.

>

> When died at the age of 89 in 1940, the British Medical

> Journal published a large obituary noting that he was universally

> respected and embued with the dignity and highest ideals of his

> profession, and that he had served at one time as President of the

> Royal College of Physicians. No mention was made of his " parasites "

> or his " bodies " , except to remark that " in his earlier years

> devoted much time to the study of the cancer cell. " Similarly, a

> large obituary appeared in the Edinburgh Medical Journal along with a

> full-page photo. His published books on Clinical Methods and widely

> read texts on circulation and gastro-intestinal diseases were cited,

> but not a word about his discovery in cancer.

>

> The heresy of " the cancer microbe "

>

> By the early part of the twentieth century the top cancer experts had

> all rejected so-called " cancer parasites " as the cause of cancer. The

> most influential physician to speak against it was Ewing, an

> American pathologist and author of the widely-read textbook,

> Neoplastic Diseases. In 1919 Ewing wrote that " few competent

> observers consider it (the parasitic theory) as a possible

> explanation in cancer. " According to Ewing and other authorities,

> cancer did not act like an infection. Therefore, microbes could not

> possibly cause cancer. He concluded, " The general facts of the

> genesis of tumors are strongly against the possibility of a parasitic

> origin. "

>

> As a result, the parasitic theory was totally discarded and few

> doctors dared to contradict Ewing's dogma by continuing to search for

> an infectious agent in cancer. Nevertheless, a few die-hard

> physicians remained convinced microbes were at the root cause of

> cancer and wrote about it convincingly in medical journals. The long

> history of this research is recorded in my book, The Cancer Microbe

> (1990) and anyone with internet access can do a Google search (type

> in " cancer microbe " ) and obtain a wealth of information on the

> microbiology of cancer. Another excellent history of cancer

> microbiology and the suppression of this controversial research is

> contained in Hess' Can Bacteria Cause Cancer? (1997).

>

> Photo #5: Larger bodies (RB) in a lymph node showing non-

> cancerous " reactive lymph node hyperplasia " from a fatal case of

> AIDS. The arrow points to nearby bacterial-sized intracellular

> coccoid smaller forms from which the bodies are derived. Fite

> (acid-fast) stain, magnified 1000 times.

>

> In the 1920s Young, an obstetrician from Scotland, repeatedly

> grew pleomorphic (having many forms) bacteria from various cancers.

> The microbes had a " specific life cycle " and " spore stages " comprised

> of exceedingly tiny and barely visible spores. In the laboratory

> these tiny spores transformed into larger coccoid (round) forms, rod-

> forms and yeast-like forms (similar in size to bodies).

> Nuzum, a Chicago physician, reported a pleomorphic coccus he

> repeatedly isolated from breast cancer. The tiniest forms were virus-

> like and passed through a filter designed to hold back bacteria.

>

> In 1925 Northwest Medicine published two papers by , a

> Montana surgeon who learned about the cancer microbe in TJ Glover's

> lab in 1921. 's microbe was similar to Young's. The parasite had

> a life cycle composed of three stages: a coccus, a rod, and a " spore

> sac " stage. believed cancer was an infection like tuberculosis

> and attempted a vaccine treatment, but his treatment methods were

> quickly suppressed by the medical establishment.

>

> In the 1930s in Germany the controversial Wilhelm von Brehmer

> described microbes in the blood of cancer patients, evoking the wrath

> of his scientific colleagues and prompting an intervention by Adolf

> Hitler. (See Proctor's The Nazi War on Cancer [1999]) s Mazet,

> a French physician, also found pleomorphic bacteria in Hodgkin's

> disease in 1941. Hodgkin's is a type of lymphoma cancer involving the

> lymphatic system. Mazet later reported similar acid-fast (red

> staining) bacteria in many different kinds of cancer, including

> leukemia.

>

> In the 1950s, 60s, and 70s, a quartet of women further refined the

> microbiology of cancer, emphasizing the extreme pleomorphism of the

> organism and its detection in tissue with the acid-fast stain. The

> published research of Virginia Livingston, Eleanor -,

> Irene Diller and Florence Seibert, is essential reading for the most

> updated understanding of the microbiology of cancer.

>

> In the late 1970s Guido Tedeschi and other Italian microbiologists at

> the University of Camerino discovered " granules " in the red blood

> cells of healthy and ill people that turned out to be bacteria that

> could be cultured in the laboratory. Some of the staphylococcal and

> corynebacteria-like bacteria cultured from the red blood cells were

> acid-fast and cell wall-deficient, a staining and growth

> characteristic shared with the cancer microbe. This research has been

> confirmed by newer studies suggesting that bacteria reside in blood

> from healthy as well as sick individuals. These findings of tiny

> blood bacteria (nanobacteria) provide further evidence to support the

> theory that microbes can cause cancer.

> Some other well-known scientists in the field of cancer microbiology

> include Gunther Enderlein, Royal Rife, Gaston Naessens and

> Wilhelm Reich. All have web sites devoted to their cancer research.

>

> bodies and their Origin

>

> More than a century has passed since 's discovery and although

> electron microscopes (which have been used since the 1950s) have the

> ability to magnify objects tens of thousands of times, the

> significance and function of his bodies still remains unknown.

>

> What is well-known is that bodies can be found, not only in

> cancer, but in the majority of inflamed tissues throughout the body.

> Distinguishing large bodies from actual fungal forms of

> Blastomyces can still be difficult, particularly when a pathologist

> encounters a true case of fungal infection due to Blastomyces.

>

> In 1954 RG White, in " Observations on the formation and nature of

> bodies " , produced bodies in animals by injecting them

> with different species of bacteria. He then studied the ensuing

> development of these bodies in the spleen, lymph nodes and plasma

> cells of the injected animals. Plasma cells are specialized forms of

> white blood cells that normally produce antibodies.

>

> EM Schleicher, in his 1965 paper on " Giant bodies " , discusses

> the various theories of origin. Possibilities include origin from the

> lymphocyte, origin in plasma cells with later degeneration, origin

> from the mitochondria of cells, and even an origin from a red blood

> cell (erythrocyte) swallowed up by a plasma cell.

>

> Most researchers currently believe bodies are essentially

> immunoglobulins (proteins that acts as antibodies), but an electron

> microscopic study by SM Hsu et al. in 1981 has cast some doubt on

> this belief.

>

> None of these studies mention the possibility that bodies

> might represent unusual large growth forms of bacteria. However, if

> bodies prove to be tiny intracellular microbes that grow and

> enlarge within leukocytes, it would be natural to expect these white

> blood cells (especially the plasma cell) to produce an antibody

> attack against these invading organisms, resulting in the production

> of immunoglobulin-coated cells and organisms.

>

> Bacterial transformation into Giant forms (L-form " large bodies')

>

> There are many different kinds of bacteria but only one type that has

> been consistently observed and studied in cancer for over a century.

> The cancer microbe has many forms, some of which appear as ordinary

> staphylococci or larger yeast-like forms that further enlarge to the

> size of bodies. As mentioned, some bodies enlarge to

> truly gigantic proportions, one hundred times the diameter of small

> cocci. One can liken this growth potential to an empty balloon that

> is then blown up to full-size. In addition, the microbe has

> exceedingly small filterable submicroscopic forms approaching the

> size of viruses, visible only by use of the electron microscope.

>

> Scientists who have extensively studied the cancer microbe claim it

> most closely resembles the type bacteria that cause tuberculosis and

> leprosy- the so-called mycobacteria. Mycobacteria are closely related

> to fungi; and some microbiologists claim mycobacteria are essentially

> derived from the " higher " fungi. " Myco " in Greek means fungus. Ergo,

> mycobacteria are considered fungus-like bacteria.

>

> Photo #6: Extremely large " super-giant-sized " solitary body

> in the skin of " cutaneous lupus erythematosus " , a so-called " collagen

> disease. " The perfectly round shape, except for one area, suggests

> this large body is developing inside a cell that is readly to burst.

> Kinyoun's (acid-fast) stain, magnification x 1000.

>

> During the 1960s microbiologist Louis Dienes popularized the

> terms " cell wall-deficient " and " L form " to encompass bacterial

> growth stages that exist at one extreme as small filterable virus-

> sized forms, and at the opposite extreme as large (50 micron or

> larger) spherical forms that he termed " large bodies. " These so-

> called large bodies are what I believe bodies represent.

>

> It must be understood that microbes are partially " classified " in

> microbiology according to size. Viruses are submicroscopic and cannot

> be visualized with an ordinary light microscope. Unlike bacteria,

> viruses can only replicate inside a cell. Bacteria can be seen

> microscopically, but smaller submicroscopic and filterable bacterial

> forms (now known as nanobacteria) are also known. Fungi and yeast

> forms are much larger than bacteria, and " mold " can obviously be seen

> with the naked eye.

>

> Larger bodies are indeed similar in size to certain spore

> forms of fungi. However, what is generally not appreciated is that

> bacteria can grow into fungal-sized large bodies, depending on

> certain laboratory conditions. Thus, bacteria in this form can easily

> be mistaken for fungi and yeast organisms.

>

> Giant-sized L-forms greatly resemble large-sized bodies. The

> century-old history of research into atypical growth forms of

> bacteria is reviewed in Lida Mattman's seminal text, Cell Wall

> Deficient Forms: Stealth Pathogens (1993). A knowledge of this

> somewhat esoteric branch of microbiology is essential to understand

> the proposed microbiology of cancer.

>

> The most impressive electron microscopic photographs I have ever

> observed of cell wall-deficient L-forms of mycobacteria were taken by

> the late C Xalabarder of Barcelona. In a series of papers and books

> (1953-1976) published in Spanish (with English-language summaries) by

> the Publicaciones del Instituto Antituberculoso " Francisco Moragas " ,

> Xalabarder totally transformed my concept about how tuberculosis-

> causing mycobacteria reproduce and grow and drastically change their

> appearance. In medical school we were taught that " simple " bacteria

> simply divide in two equal halves by " binary fission " . However,

> nothing could be further from the truth, and it is only by a

> refutation of this simplistic concept that a serious study of the

> microbiology of cancer can be undertaken.

>

> Tuberculosis and Cancer

>

> Because cancer is produced by a microbe similar to the bacteria that

> cause TB, much can be learned from experiments like those performed

> by Xalabarder in 1967. Using " atypical mycobacteria " grown from TB

> patients who had taken long courses of drug therapy, Xalabarder then

> injected these bacteria into guinea-pigs and rabbits. Amazingly, he

> was able to experimentally produce lesions which microscopically

> resembled cancer! He also produced experimental lesions

> characteristic of so called " collagen disease " - a type of lesion

> seemingly unrelated to cancer.

>

> During the 1960s I discovered unusual pleomorphic acid-fast bacteria

> in a collagen disease called scleroderma, and later in another

> collagen disease called lupus erythematosus. The germs I grew from

> these patients closely resembled scleroderma microbes that were

> reported by Virginia Livingston in 1947, and which subsequently led

> to her discovery of similar acid-fast microbes in cancer.

>

> In 1969 Xalabarder manipulated different developmental stages of TB

> bacteria and inoculated them into one thousand guinea pigs. In the

> process, he produced the microscopic picture of sarcoidosis in the

> animals. Sarcoidosis is a human disease closely related to TB but one

> in which TB germs cannot be found. Xalabarder's most impressive

> sarcoid lesions were produced by inoculating sputum specimens from TB

> patients who " converted " , meaning that their TB bacteria could no

> longer be cultured from their sputum. Controversy over the cause of

> sarcoidosis is still not settled, although I reported bacteria

> similar to cancer microbes in this disease in the 1980s.

>

> The most spectacular electron microphotographs of cell wall-deficient

> mycobacteria are presented in Xalabarder's L-forms of mycobacteria

> and chronic nephritis (1970). In the earliest growth stages of

> mycobacteria in culture the smallest elements appear as tiny

> submicroscopic forms visualized only with the electron microscope.

> These filterable forms of tuberculosis bacteria - the so-

> called " tuberculosis virus " - have been known to cause cancer in

> animals since the 1920s. By adding antibiotics to the lab culture

> media Xalabarder was able to induce many unusual growth forms of

> tuberculosis bacteria. Using serial images, he was able to trace the

> development of these tiny submicroscopic forms up to the size of

> ordinary cocci - and then up to the size of " large body " forms

> reaching and even surpassing the size of red blood cells. Some of the

> large bodies of mycobacteria also exhibit internal structure, similar

> to what Gaylord noted in his body research.

>

> Cancer and Bacteria

>

> Although the idea of a cancer microbe is medical heresy, there is

> ample data to show that cancer patients are highly prone to bacterial

> infection. A PubMed computer search

> (http://www.ncbi.nlm.nih.gov/entrez/query.fcgi)

> of " bacteria + cancer " elicits 49, 244 citations contained within

> 2,463 web pages. According to a 2003 article by Vento and Cainelli,

> patients with cancer who are undergoing chemotherapy are highly

> susceptible to almost any type of bacterial or fungal infection.

> Why are physicians, and especially pathologists and bacteriologists,

> so unaware, so disinterested, or so antagonistic to credible cancer

> microbe research? Why have pathologists failed to consider

> bodies as large forms of bacteria?

> For over 30 years I studied various forms of cancer and skin

> diseases " of unknown origin " , as well as autopsy cases of cancer,

> lupus, scleroderma, and AIDS. In all these diseases I was able to

> detect bacteria, although pathologists would never mention bacteria

> in any of their official biopsy reports. In my experience, they

> simply could not conceive of cancer and collagen disease (and AIDS)

> as a bacterial infection, nor did they seem to be aware of

> bacteriology reports pertaining to " large bodies " and pathologic

> effects produced by the " tuberculosis virus. " In short, they were

> trained to see and report only the typical rod-shaped acid-fast (red-

> stained) " typical " form of mycobacteria, , but they were not trained

> to look for or to recognize other growth forms of the same bacteria

> that might be hidden in their pathologic tissue specimens.

>

> Photo #7: Pleomorphic growth forms (L-forms) of tuberculosis

> mycobacteria photographed with an electron microscope. Note the

> darker staining tiny coccal forms (similar in size to ordinary

> staphylococci) and the larger clear balloon-sized " ghost " forms

> similar in size and shape to bodies found in tissue. These

> forms are all characteristic of " cell wall-deficient bacteria " and

> totally unlike the well-known " typical " acid-fast rod forms of

> Mycobacterium tuberculosis. Reproduced from L-forms of Mycobacteria

> and Chronic Nephritis (1970), by Dr. C. Xalabarder P., page 51.

>

> When objects like bodies are observed in a wide variety of

> diseases and in " normal " tissue, the significance is lessened.

> Doctors expect " normal " tissue to be free of microbes. I suppose they

> also conclude that bodies cannot be an infectious agent

> because it would be impossible for an infectious agent to appear in

> so many different kinds of diseases and in so many different forms of

> cancer.

>

> For most of the last century stomach ulcers were thought to be non-

> infectious because pathologists could not identify bacteria in the

> ulcers and because doctors believed bacteria could not live in the

> acid environment of the stomach. This thinking all changed gradually

> after 1982 when Barry Marshall, an Australian physician, proved most

> stomach ulcers were caused by a microbe called Helicobacter pylori,

> which could be identified microscopically with special tissue

> staining techniques in ulcer tissue. On the other hand, many people

> normally carry this stomach microbe without any ill effects. Not

> surprisingly, pathologists are now reporting numerous bodies

> in plasma cells in some ulcer patients, giving rise to a previously

> unrecognized tissue reaction called " cell gastritis. "

>

> bodies and bacteria

>

> When bacteria are threatened by the immune system or by antibiotics

> they may lose their cell-wall and assume a different growth form that

> renders them less susceptible to attack by the immune system. Some

> bodies elicit little or no inflammatory cell response. This

> lack of cellular response is yet another reason why physicians have a

> hard time believing bodies could be microbes.

>

> I have observed the largest and most complex bodies in tissue

> where there was almost a total lack of inflammation. My photographs

> of such " large bodies " , some with obvious internal structure, that I

> observed in patients with scleroderma and pseudoscleroderma, were

> published in the American Journal of Dermatopathology in 1980. The

> first case of fatal scleroderma I studied in 1963 had numerous " large

> bodies " in the fat layer of the diseased skin that were unlike

> anything ever seen in dermatology. The patient had been hospitalized

> for pulmonary tuberculosis 7 years before developing scleroderma. The

> mystery of these " yeast-like " bodies deep in his skin was solved

> years later when I first learned about the existence of " large body "

> forms of Mycobacterium tuberculosis. When this patient died,

> Mycobacterium fortuitum, an " atypical " form of mycobacteria was

> cultured from his scleroderma tissue.

>

> Bacteria are vital for our survival. They are hardy and the bacteria

> we carry will surely outlive us. The bacteria that cause cancer are

> the " simple " bacteria we carry with us. The cancer microbe is not an

> exotic microbe nor a rare one. However, bacteria can change form as

> the environment in our bodies changes. There is indeed a delicate

> balance between our bacteria and our immune system which allows these

> bacteria to live in harmony with us.

>

> But when dis-ease occurs these microbes become aggressive, giving

> rise to a host of diseases, some of which are cancerous, and others

> that are inflammatory, degenerative, or simply transitory. Another

> reason for physicians to doubt that a single type of germ could cause

> such a variety of pathologic effects.

> Bacteria are ubiquitous and so are bodies. And if

> bodies prove to be bacteria, the reason for this becomes obvious.

>

> The body and the origin of cancer

>

> In 1981 King and Eisenberg's article on " 's fuchsin body: 'The

> characteristic organism of cancer' " appeared in the American Journal

> of Dermatopathology. They reconfirmed that " bodies have now

> been shown to be immunoglobulins. " They remarked that was not

> the first to describe them; and that similar bodies were reported by

> Cornil and Alvarez in rhinoscleroma five years earlier in a French

> journal in 1885. Declaring it ironic that these " bodies should bear

> the name of a man who so thoroughly misunderstood them " , the authors

> ended by stating: " Hence, when the term body is used today,

> one should be aware that the eponym is as inaccurate as was 's

> perception of their significance. "

>

> Unlike King and Eisenberg, I believe was right on the mark.

> There is a parasite in cancer. It has been studied and reported by

> various scientists throughout the world for many decades, and a

> wealth of scientific information on the cancer microbe is available

> in medical libraries. For those with Internet capability, the

> words " cancer microbe " typed into Google.com will give instant access

> to a treasure trove of information on the subject.

> There is no secret to cancer. In my view, the cause is staring us

> right in the face in the form of the body.

> understood very well in the nineteenth century what medical science

> in the twenty-first century has yet to discover.

>

> Alan Cantwell, M.D. is a retired dermatologist and cancer researcher.

> His book, The Cancer Microbe, is available through Internet sources.

> A number of his full-length papers on the microbiology of cancer can

> be found on the net at the Journal of Independent Medical Research

> web site (www.joimr.org/) Email: alanrcan@...

>

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December 15, 2003