Mad Cow Disease traced to Insecticide Spray

[Guest guest]

Insecticide Causes Mad Cow Disease

> >

> >

> >http://www.mercola.com/2000/dec/17/bovine_spongiform_disease.htm

> >

> >

> >

> > Insecticide Causes Mad Cow Disease

> >

> > by Fintan Dunne

> > Research by Kathy McMahon

> >

> > Reprinted from eionews.com, email - news@...

> >

> >

>

>--------------------------------------------------------------------------

> >

> > Pharmaceutical interests in the UK are ignoring new scientific

> >research that shows the insecticide used in the UK government's own

> >warble-fly campaigns triggered the UK surge of 'Mad Cow' disease.

> >

> > Latest experiments by Cambridge University prion specialist,

R.

> >Brown, have shown that manganese bonds with prions. Other researchers

work

> >shows that prions in the bovine spine -- along which insecticides are

> >applied -- can be damaged by ICI's Phosmet organophosphate(OP)

> >insecticide -causing the disease.

> >

> > British scientists have led the current theory that an infectious

> >prion in bonemeal fed to cattle causes bovine spongiform disease (BSE).

> >

> > Infectious prions are also claimed to cause new variant

> >Creutzfeld-Jakob Disease (CJD) in humans -from ingesting beef. But the

> >infectious prion theory serves to obscure a tragic chemical poisoning

> >scandal behind the majority of BSE cases.

> >

> > The new work proves that the prions can bond with manganese in

animal

> >feeds or mineral licks. These manganese prions cause the neurological

> >degeneration seen in BSE. By a similar process, prions in human brains

are

> >damaged by lice lotions containing organophosphate. This can result in

> >neurological diseases like CJD and Alzheimers -later in life.

> >

> > Many might be surprised to hear that organophosphates were

developed

> >by Nazi chemists during the course World War Two,

> > as a chemical weapon nerve agent. One formulation of the

> >insecticide -- Maneb, or Mancozeb -- actually contains manganese in

addition

> >to organophosphate.

> >

> > The marginalized research has devestating financial implications

for

> >ICI. It would provide a firm basis for litigants -who could include CJD

> >sufferers, farmers across the world and families of the many British

farmers

> >who committed suicide during this BSE debacle.

> >

> > Phosmet organophosphate has been used at high doses in British

warble

> >fly campaigns. In 1996, ICI subsidiary Zeneca sold the phosmet patent to

a

> >PO Box company in Arizona called Gowan -just one week before the UK

> >government admitted to a link between BSE and nvCJD.

> >

> > The politically well-connected British pharmaceuticals group, ICI

has

> >the financial and political clout to block research into any cause other

> >than the infective model. Indeed no substantive alternative research has

> >been done. British BSE disease management and research bodies have taken

> >decisions that do not seem guided by spirited scientific enquiry.

Mysterious

> >prions that jump species is the preferred research arena.

> >

> > Scientist and organic farmer, Mark Purdey gave evidence to the UK

BSE

> >inquiry, that warble fly insecticide was the cause of the disease. The

> >scientist wheeled out to rubbish Purdy's evidence -Dr. Ray, later

> >turned out to have been receiving funding from the insecticide

manufacturer

> >ICI.

> >

> > A lobby group that includes Bayer, Monsanto, Novartis, Pfizer,

Roche

> >and Schering-Plough was behind the effort to discredit Purdey. In

December

> >1999, the same Ray was appointed to the UK Veterinary Products

> >Committee (VPC) -a government body that licences animal medicines.

> >

> > Purdey has been consistently denied even exploratory funding to

extend

> >his privately supported research. Yet the Purdey/Brown chemical poisoning

> >model matches with the epidermiological spread of CJD clusters in humans.

It

> >also predicts the incidence of BSE-type diseases in animals. The accepted

> >infectious model fits neither.

> >

> > The pharmaceutical industry is all the more determined to hide the

> >chemical source of BSE and CJD, because a spotlight on chemicals would

> >expose the role the insecticides in Alzheimer's -- another

neurodegenerative

> >disease -- that might lead to claims which would dwarf those from BSE and

> >CJD litigants. In fact, two leading brain researchers into CJD and

> >Alzheimers have died in suspicious circumstances in recent years.

> >

> > In the United States, the Environmental Protection Agency is

already

> >reviewing Phosmet's safety. The Centers for Disease Control in the US has

> >recently conducted experiments on mice that confirm the organophosphate

> >risk.

> >

> > Not only is the EC beef slaughter campaign futile -because BSE

disease

> >is mostly non-infectious, but unless the underlying chemical cause is

> >addressed, BSE will simply reappear from chemical causes. A new warble

fly

> >campaign is already underway in France using the organophosphate

> >insecticide.

> >

> > Of greater concern is that some lotions for scabies and head lice

are

> >now priming children and adults, for CJD and Alzheimers in later life.

> >

> > Bonding The Prion

> >

> > Cambridge University prion biochemist, R. Brown is dismissive

of

> >the science behind the infectious model of BSE. He terms it " a very

limited

> >amount of science by a few assumed- reputable scientists. " He insists

there

> >is " no evidence an infectious agent is present in either meat or milk. "

> >

> > " Simple tests on udder walls of cows -- which could easily detect

an

> >infectious prion -- have not been done, why I don't understand. "

> >

> > A number of researchers have found that organophosphate(OP) in

> >systemic warble fly insecticide can deform the prion molecule, rendering

it

> >ineffective at buffering free radical effects in the body. Worse still,

the

> >prion is then partial to bond with manganese and become a 'rogue' prion.

A

> >chain reaction whereby rogue prions turn others to rogues also, can

explain

> >the bovine spongiform disease mechanism.

> >

> > Brown showed how prion protein bonds benignly with copper, but

> >lethally with manganese. Even natural variations in relative

environmental

> >availability of manganese versus copper can trigger prion degradation.

> >

> > The CJD and BSE symptoms mirror 'manganese madness', an

irreversible

> >fatal neuro-psychiatric degenerative syndrome that plagued manganese

miners

> >in the first half of the last century

> >

> > Shining a Light on Spongiform

> >

> > Organic dairy farmer and peer-review-published independent

scientist,

> >Mark Purdey, says the accepted theory of transmission from BSE-infected

> >cattle to human CJD -by bonemeal or meat, is dependent on a mutant prion

> >that has never been isolated under the scientific protocol called Koch's

> >postulates.

> >

> > Purdey's insistence on sticking to the letter of this scientific

law

> >earned him the condemnation of UK officialdom when he first mooted his

> >theory. But Purdey pointed to CJD clusters downwind of a British Phosmet

> >production plant to back his case.

> >

> > He gave evidence to the UK Government BSE inquiry and was supported

by

> >Conservative MP, Thessa Gorman. His views were discounted, but his

> >subsequent research and the new Cambridge prion work have confirmed the

> >alternative theory. Despite this, and the backing of a British peer, he

is

> >denied even exploratory funding.

> >

> > Speaking from his rural English Somerset farm yesterday -as plans

> >forge ahead for the European cattle cull, he asks:

> >

> > " Why does CJD degeneration in humans begin in the retina, and why

are

> >CJD disease clusters found in high altitude locations? "

> >

> > The question is rhetorical, and Purdey has an eye-opening answer.

He

> >argues that the prion molecule has a known natural role as a shock

adsorber

> >of damaging energy from ultraviolet rays and other oxidizing agents.

> >

> > Once this prion defence system is rendered ineffective by

> >organophosphates - for example in human head lice lotions, these

oxidizing

> >effects have an unmediated impact on tissues. Eventually, UV radiation

> >damages the retina and oxidative stress destroys the brain tissues of CJD

> >patients. This theory would expect to find higher CJD incidence in

mountain

> >regions -where UV radiation levels are elevated. That prediction holds

true.

> >

> > A similar but accelerated mechanism could be driving BSE. ICI's

> >Phosmet organophosphate warble fly insecticide -applied on the backs of

> >animals along the spinal column, similarly degrades prions. " Systemic

> >versions of the insecticide are designed to make the entire cow carcass

> >toxic to warble fly, " explains Purdey. " Unfortunately it's toxic to

prions

> >too -especially those prions located just millimeters from the point of

> >application. "

> >

> > The damaged prions are then ready to react with manganese in animal

> >feed, or manganese sprayed on land or in mineral licks -to become the

> >driving force of BSE neurodegeneration. Purdey says manganese-tipped

prions

> >set off lethal chain reactions that neurologically burn through the

animal.

> >

> > Chickens notoriously excrete most of the supplements fed to

> >them -including manganese. And their manganese-rich excreta have been

> >blended into cattle feed in the UK. Natural variations in the relative

> >environmental availability of copper and manganese can also spur prion

> >degeneration says Purdey.

> >

> > From this research, any prudent person would conclude there is a

> >significant risk attaching to the use of organophosphate in humans.

> >Preparations for head lice and scabies are known to be overused in

practice

> >and might be priming users for CJ disease.

> >

> > Purdey believes his bias for field work is the key to his success.

He

> >bemoans the " reductionism " of much lab-centered science. " I have traveled

> >the world to investigate known clusters of spongiform disease -something

> >mainstream researchers don't seem remotely interested in doing. "

> >

> > Since first postulating an environmental -rather than infectious-

> >theory of spongiform diseases, Purdey has built evidence from around the

> >world that explains and predicts the incidence in humans and animals: a

> >cluster of CJD in Slovakia, Eastern Europe -around a manganese plant;

Rocky

> >Mountain deer with Chronic Wasting Disease (CWD), who were found to be

> >eating pine needles rich in manganese; the futile slaughter of sheep in

> >Cyprus -only for BSE to reemerge within years.

> >

> > " The reappearance of BSE in Cyprus obviously points to an

> >environmental cause, " says Purdey, who is sanguine when reflecting on the

> >condemnation of him by mainstream scientists.

> >

> > " I suppose they have mortgages and kids who need to go to

university, "

> >he muses. " Privately, some were agreeing with me, but then they would

> >denounce me publicly. It was quite strange really. "

> >

> > The Money Trail

> >

> > Critical scientists like Purdey are unlikely to prevail. The

> >pharmaceutical industry holds most research purse strings, and would

hardly

> >energetically explore an avenue of research that could expose them to

> >litigation for causing BSE. The official theory is lavishly funded,

> >alternative theories rarely, if at all.

> >

> > There are more explosive implications to his -and other's latest

> >research. Purdey says similar organophosphate-induced protein deformation

> >could also underlie Alzheimer's disease. If that were true, the

litigation

> >fallout would destroy some pharmaceutical giants, and a lot of very

> >influential noses would be out of joint.

> >

> > Disturbingly, Purdey and other brain researchers seem to have had

an

> >undue share of unfortunate accidents. Purdey's house was burned down and

his

> >lawyer who was working with him on Mad Cow Disease was driven off the

road

> >by another vehicle and subsequently died. The veterinarian on the case

also

> >died in a car crash -locally reported as: 'Mystery Vet Death Riddle.'

> >

> > Dr. C. Bruton, a CJD specialist -- who had just produced a paper on

a

> >new strain of CJD -- was killed in a car crash before his work was

announced

> >to the public. Purdey speculates that Bruton might have known more than

what

> >was revealed in his last scientific paper.

> >

> > In 1996, leading Alzheimer's researcher Tsunao Saitoh, 46 and his

> >13 -year-old daughter were killed in La Jolla, California, in what a

Reuters

> >report described as a " very professionally done " shooting.

> >

> > What Alzheimer's Disease, Mad Cow Disease, and CJ Disease have in

> >common, is abnormal brain proteins and a putative link to

organophosphates.

> >Even Gulf War syndrome among returning veterans has been attributed, in

part

> >to the insecticide. But the sidelined scientists' suspicions are still

> >largely ignored.

> >

> > In their favour at the moment, is a growing unease on the part of

the

> >public. As BSE forges on and Governments panic, Science may be out to

lunch

> >on BSE, compromised by bovine spongythinking myopathy.

> >

> >

>

>--------------------------------------------------------------------------

> >

> > Do Not Use Systemic Organophosphate Insecticides

> >

> > Do NOT treat children with OP head lice products - they may cause

CJD

> >and Alzheimer's

> >

> > Do NOT treat your pets with OP anti-flea products

> >

> > Do NOT treat cattle or animals with OP products - they may cause

BSE

> >

> > Do NOT give manganese to cattle previously dosed with a systemic OP

> >

> > The relative availability of the metals copper and manganese in you

> >local environment is a major factor in BSE & CJD

> >

> >

>

>--------------------------------------------------------------------------

> >

> > Useful Links

> >

> > EPA on Phosmet

> > BSE & CJD Researchers

> > Insecticides Classification

> > US Gov Pesticide resources

> >

> >

> >

> >

> >

> >

> >Mike Callicrate

> >P.O. Box 748

> >St. Francis, KS 67756

> >785-332-3344

> >mike@...

> >www.nobull.net

> >

> >www.ranchfoodsdirect.com

> >

> >

>

> --

> W

>

>

>

>

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[Guest guest]

Insecticide Causes Mad Cow Disease

> >

> >

> >http://www.mercola.com/2000/dec/17/bovine_spongiform_disease.htm

> >

> >

> >

> > Insecticide Causes Mad Cow Disease

> >

> > by Fintan Dunne

> > Research by Kathy McMahon

> >

> > Reprinted from eionews.com, email - news@...

> >

> >

>

>--------------------------------------------------------------------------

> >

> > Pharmaceutical interests in the UK are ignoring new scientific

> >research that shows the insecticide used in the UK government's own

> >warble-fly campaigns triggered the UK surge of 'Mad Cow' disease.

> >

> > Latest experiments by Cambridge University prion specialist,

R.

> >Brown, have shown that manganese bonds with prions. Other researchers

work

> >shows that prions in the bovine spine -- along which insecticides are

> >applied -- can be damaged by ICI's Phosmet organophosphate(OP)

> >insecticide -causing the disease.

> >

> > British scientists have led the current theory that an infectious

> >prion in bonemeal fed to cattle causes bovine spongiform disease (BSE).

> >

> > Infectious prions are also claimed to cause new variant

> >Creutzfeld-Jakob Disease (CJD) in humans -from ingesting beef. But the

> >infectious prion theory serves to obscure a tragic chemical poisoning

> >scandal behind the majority of BSE cases.

> >

> > The new work proves that the prions can bond with manganese in

animal

> >feeds or mineral licks. These manganese prions cause the neurological

> >degeneration seen in BSE. By a similar process, prions in human brains

are

> >damaged by lice lotions containing organophosphate. This can result in

> >neurological diseases like CJD and Alzheimers -later in life.

> >

> > Many might be surprised to hear that organophosphates were

developed

> >by Nazi chemists during the course World War Two,

> > as a chemical weapon nerve agent. One formulation of the

> >insecticide -- Maneb, or Mancozeb -- actually contains manganese in

addition

> >to organophosphate.

> >

> > The marginalized research has devestating financial implications

for

> >ICI. It would provide a firm basis for litigants -who could include CJD

> >sufferers, farmers across the world and families of the many British

farmers

> >who committed suicide during this BSE debacle.

> >

> > Phosmet organophosphate has been used at high doses in British

warble

> >fly campaigns. In 1996, ICI subsidiary Zeneca sold the phosmet patent to

a

> >PO Box company in Arizona called Gowan -just one week before the UK

> >government admitted to a link between BSE and nvCJD.

> >

> > The politically well-connected British pharmaceuticals group, ICI

has

> >the financial and political clout to block research into any cause other

> >than the infective model. Indeed no substantive alternative research has

> >been done. British BSE disease management and research bodies have taken

> >decisions that do not seem guided by spirited scientific enquiry.

Mysterious

> >prions that jump species is the preferred research arena.

> >

> > Scientist and organic farmer, Mark Purdey gave evidence to the UK

BSE

> >inquiry, that warble fly insecticide was the cause of the disease. The

> >scientist wheeled out to rubbish Purdy's evidence -Dr. Ray, later

> >turned out to have been receiving funding from the insecticide

manufacturer

> >ICI.

> >

> > A lobby group that includes Bayer, Monsanto, Novartis, Pfizer,

Roche

> >and Schering-Plough was behind the effort to discredit Purdey. In

December

> >1999, the same Ray was appointed to the UK Veterinary Products

> >Committee (VPC) -a government body that licences animal medicines.

> >

> > Purdey has been consistently denied even exploratory funding to

extend

> >his privately supported research. Yet the Purdey/Brown chemical poisoning

> >model matches with the epidermiological spread of CJD clusters in humans.

It

> >also predicts the incidence of BSE-type diseases in animals. The accepted

> >infectious model fits neither.

> >

> > The pharmaceutical industry is all the more determined to hide the

> >chemical source of BSE and CJD, because a spotlight on chemicals would

> >expose the role the insecticides in Alzheimer's -- another

neurodegenerative

> >disease -- that might lead to claims which would dwarf those from BSE and

> >CJD litigants. In fact, two leading brain researchers into CJD and

> >Alzheimers have died in suspicious circumstances in recent years.

> >

> > In the United States, the Environmental Protection Agency is

already

> >reviewing Phosmet's safety. The Centers for Disease Control in the US has

> >recently conducted experiments on mice that confirm the organophosphate

> >risk.

> >

> > Not only is the EC beef slaughter campaign futile -because BSE

disease

> >is mostly non-infectious, but unless the underlying chemical cause is

> >addressed, BSE will simply reappear from chemical causes. A new warble

fly

> >campaign is already underway in France using the organophosphate

> >insecticide.

> >

> > Of greater concern is that some lotions for scabies and head lice

are

> >now priming children and adults, for CJD and Alzheimers in later life.

> >

> > Bonding The Prion

> >

> > Cambridge University prion biochemist, R. Brown is dismissive

of

> >the science behind the infectious model of BSE. He terms it " a very

limited

> >amount of science by a few assumed- reputable scientists. " He insists

there

> >is " no evidence an infectious agent is present in either meat or milk. "

> >

> > " Simple tests on udder walls of cows -- which could easily detect

an

> >infectious prion -- have not been done, why I don't understand. "

> >

> > A number of researchers have found that organophosphate(OP) in

> >systemic warble fly insecticide can deform the prion molecule, rendering

it

> >ineffective at buffering free radical effects in the body. Worse still,

the

> >prion is then partial to bond with manganese and become a 'rogue' prion.

A

> >chain reaction whereby rogue prions turn others to rogues also, can

explain

> >the bovine spongiform disease mechanism.

> >

> > Brown showed how prion protein bonds benignly with copper, but

> >lethally with manganese. Even natural variations in relative

environmental

> >availability of manganese versus copper can trigger prion degradation.

> >

> > The CJD and BSE symptoms mirror 'manganese madness', an

irreversible

> >fatal neuro-psychiatric degenerative syndrome that plagued manganese

miners

> >in the first half of the last century

> >

> > Shining a Light on Spongiform

> >

> > Organic dairy farmer and peer-review-published independent

scientist,

> >Mark Purdey, says the accepted theory of transmission from BSE-infected

> >cattle to human CJD -by bonemeal or meat, is dependent on a mutant prion

> >that has never been isolated under the scientific protocol called Koch's

> >postulates.

> >

> > Purdey's insistence on sticking to the letter of this scientific

law

> >earned him the condemnation of UK officialdom when he first mooted his

> >theory. But Purdey pointed to CJD clusters downwind of a British Phosmet

> >production plant to back his case.

> >

> > He gave evidence to the UK Government BSE inquiry and was supported

by

> >Conservative MP, Thessa Gorman. His views were discounted, but his

> >subsequent research and the new Cambridge prion work have confirmed the

> >alternative theory. Despite this, and the backing of a British peer, he

is

> >denied even exploratory funding.

> >

> > Speaking from his rural English Somerset farm yesterday -as plans

> >forge ahead for the European cattle cull, he asks:

> >

> > " Why does CJD degeneration in humans begin in the retina, and why

are

> >CJD disease clusters found in high altitude locations? "

> >

> > The question is rhetorical, and Purdey has an eye-opening answer.

He

> >argues that the prion molecule has a known natural role as a shock

adsorber

> >of damaging energy from ultraviolet rays and other oxidizing agents.

> >

> > Once this prion defence system is rendered ineffective by

> >organophosphates - for example in human head lice lotions, these

oxidizing

> >effects have an unmediated impact on tissues. Eventually, UV radiation

> >damages the retina and oxidative stress destroys the brain tissues of CJD

> >patients. This theory would expect to find higher CJD incidence in

mountain

> >regions -where UV radiation levels are elevated. That prediction holds

true.

> >

> > A similar but accelerated mechanism could be driving BSE. ICI's

> >Phosmet organophosphate warble fly insecticide -applied on the backs of

> >animals along the spinal column, similarly degrades prions. " Systemic

> >versions of the insecticide are designed to make the entire cow carcass

> >toxic to warble fly, " explains Purdey. " Unfortunately it's toxic to

prions

> >too -especially those prions located just millimeters from the point of

> >application. "

> >

> > The damaged prions are then ready to react with manganese in animal

> >feed, or manganese sprayed on land or in mineral licks -to become the

> >driving force of BSE neurodegeneration. Purdey says manganese-tipped

prions

> >set off lethal chain reactions that neurologically burn through the

animal.

> >

> > Chickens notoriously excrete most of the supplements fed to

> >them -including manganese. And their manganese-rich excreta have been

> >blended into cattle feed in the UK. Natural variations in the relative

> >environmental availability of copper and manganese can also spur prion

> >degeneration says Purdey.

> >

> > From this research, any prudent person would conclude there is a

> >significant risk attaching to the use of organophosphate in humans.

> >Preparations for head lice and scabies are known to be overused in

practice

> >and might be priming users for CJ disease.

> >

> > Purdey believes his bias for field work is the key to his success.

He

> >bemoans the " reductionism " of much lab-centered science. " I have traveled

> >the world to investigate known clusters of spongiform disease -something

> >mainstream researchers don't seem remotely interested in doing. "

> >

> > Since first postulating an environmental -rather than infectious-

> >theory of spongiform diseases, Purdey has built evidence from around the

> >world that explains and predicts the incidence in humans and animals: a

> >cluster of CJD in Slovakia, Eastern Europe -around a manganese plant;

Rocky

> >Mountain deer with Chronic Wasting Disease (CWD), who were found to be

> >eating pine needles rich in manganese; the futile slaughter of sheep in

> >Cyprus -only for BSE to reemerge within years.

> >

> > " The reappearance of BSE in Cyprus obviously points to an

> >environmental cause, " says Purdey, who is sanguine when reflecting on the

> >condemnation of him by mainstream scientists.

> >

> > " I suppose they have mortgages and kids who need to go to

university, "

> >he muses. " Privately, some were agreeing with me, but then they would

> >denounce me publicly. It was quite strange really. "

> >

> > The Money Trail

> >

> > Critical scientists like Purdey are unlikely to prevail. The

> >pharmaceutical industry holds most research purse strings, and would

hardly

> >energetically explore an avenue of research that could expose them to

> >litigation for causing BSE. The official theory is lavishly funded,

> >alternative theories rarely, if at all.

> >

> > There are more explosive implications to his -and other's latest

> >research. Purdey says similar organophosphate-induced protein deformation

> >could also underlie Alzheimer's disease. If that were true, the

litigation

> >fallout would destroy some pharmaceutical giants, and a lot of very

> >influential noses would be out of joint.

> >

> > Disturbingly, Purdey and other brain researchers seem to have had

an

> >undue share of unfortunate accidents. Purdey's house was burned down and

his

> >lawyer who was working with him on Mad Cow Disease was driven off the

road

> >by another vehicle and subsequently died. The veterinarian on the case

also

> >died in a car crash -locally reported as: 'Mystery Vet Death Riddle.'

> >

> > Dr. C. Bruton, a CJD specialist -- who had just produced a paper on

a

> >new strain of CJD -- was killed in a car crash before his work was

announced

> >to the public. Purdey speculates that Bruton might have known more than

what

> >was revealed in his last scientific paper.

> >

> > In 1996, leading Alzheimer's researcher Tsunao Saitoh, 46 and his

> >13 -year-old daughter were killed in La Jolla, California, in what a

Reuters

> >report described as a " very professionally done " shooting.

> >

> > What Alzheimer's Disease, Mad Cow Disease, and CJ Disease have in

> >common, is abnormal brain proteins and a putative link to

organophosphates.

> >Even Gulf War syndrome among returning veterans has been attributed, in

part

> >to the insecticide. But the sidelined scientists' suspicions are still

> >largely ignored.

> >

> > In their favour at the moment, is a growing unease on the part of

the

> >public. As BSE forges on and Governments panic, Science may be out to

lunch

> >on BSE, compromised by bovine spongythinking myopathy.

> >

> >

>

>--------------------------------------------------------------------------

> >

> > Do Not Use Systemic Organophosphate Insecticides

> >

> > Do NOT treat children with OP head lice products - they may cause

CJD

> >and Alzheimer's

> >

> > Do NOT treat your pets with OP anti-flea products

> >

> > Do NOT treat cattle or animals with OP products - they may cause

BSE

> >

> > Do NOT give manganese to cattle previously dosed with a systemic OP

> >

> > The relative availability of the metals copper and manganese in you

> >local environment is a major factor in BSE & CJD

> >

> >

>

>--------------------------------------------------------------------------

> >

> > Useful Links

> >

> > EPA on Phosmet

> > BSE & CJD Researchers

> > Insecticides Classification

> > US Gov Pesticide resources

> >

> >

> >

> >

> >

> >

> >Mike Callicrate

> >P.O. Box 748

> >St. Francis, KS 67756

> >785-332-3344

> >mike@...

> >www.nobull.net

> >

> >www.ranchfoodsdirect.com

> >

> >

>

> --

> W

>

>

>

>

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