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" The Life Extension Foundation is constantly uncovering information to

provide the cancer patient with more ammunition to battle their disease.

A special website has been established for the purpose of updating

patients on new findings that directly pertain to the cancer protocols.

Whenever Life Extension discovers information that points to a better

way of treating cancer, it will be posted on the website

www.lefcancer.org " .

[From their recent newsletter]

Protocol

Breast Cancer

Breast cancer occurs when cells in the breast tissue divide and grow

without control. In a healthy body, the cell cycle is the natural

mechanism that regulates the creation, growth, and death of cells. It is

this mechanism that regulates cell division, allowing the creation of

new tissue as older cells die. When the normal cell regulators

malfunction and cells do not die at the proper rate, there is a failure

of apoptosis (programmed cell death) and cell growth goes unchecked. As

a result, cancer begins to develop as cells divide without control,

accumulating into a mass of extra tissue called a malignant tumor. As a

tumor grows, it elicits new capillary growth (angiogenesis) from the

surrounding normal tissues and diverts blood supply and nutrients away

from the tissue to feed itself. Unregulated angiogenesis can facilitate

the growth of cancer throughout the body.

An important aspect in any reproductive cancer is whether the tumor

growth is hormonally driven. Often breast cancer tumors require hormones

for growth. This is known as a receptor-positive tumor. This type of

cancer poses a unique problem because the hormones involved in tumor

growth are either estrogen or progesterone or both. Estrogen and

progesterone are naturally occurring hormones that the body produces in

varying amounts throughout one's lifetime. To complicate the situation

further, these hormones are essential for many other physiological

functions, such as bone integrity.

In 1991, researchers at the Institute for Hormone Research announced

that they had been able to induce the body to convert the stronger form

of estrogen (estradiol) into the weaker form (estriol) without using

drugs. Estriol is considered to be a more desirable form of estrogen. It

is less active than estradiol, so when it occupies the estrogen

receptor, it blocks estradiol's strong " grow " signals. It took only 1

week to prove that the conversion of estradiol to estriol could be

accomplished without drugs. Using a natural substance, researchers were

able to increase the conversion of estradiol to estriol by 50% in 12

healthy people (Michnovicz et al. 1991).

Next, they tested the natural substance in female mice prone to

developing breast cancer. The incidence of cancer and the number of

tumors fell significantly. The substance was indole-3-carbinol (IC3), a

phytochemical isolated from cruciferous vegetables (broccoli,

cauliflower, Brussels sprouts, turnips, kale, green cabbage, mustard

seed, etc.). I3C was then given to 17 men and women for 2 months. Again,

levels of strong estrogen declined, and levels of weak estrogen

increased. But more importantly, the level of an estrogen metabolite

associated with breast and endometrial cancer (16,alpha-hydroxyestrone)

fell (Bradlow et al. 1991).

This cancer protocol raises many issues that are subject to change as

new data emerge. Furthermore, cancer is still a disease with

unacceptably high mortality rates, and none of our suggested treatment

regimens can guarantee a cure.

Featured Products of the Week

Indole-3-carbinol

A summary of recent studies shows that this vegetable extract:

Increases the conversion of estradiol to ?weaker? (2OHE) estrogen, which

has been shown to reduce breast cancer incidence

Works in estrogen receptor negative breast cancer cells.

Stops human cancer cells from growing (54-61%) and provokes the cells to

self-destruct (apoptosis).

Inhibits MCF7 human breast cancer cells better than tamoxifen under

laboratory conditions.

Protects against the environmental toxin, dioxin.

Restores p21 tumor suppressor gene.

Provides antioxidant protection.

Super Green Tea Extract

The initial interest in studying green tea came from epidemiological

studies showing a lower rate of cancer in regions where green tea was

consumed. Life Extension Buyers Club?s 95% green tea extracts, in either

lightly caffeinated or decaffeinated form, are standardized to provide

35% of epigallocatechin gallate (EGCG). Published research shows that

EGCG is the most potent cancer cell suppressing polyphenol found in

green tea. Some noteworthy studies on green tea include:

When non-Hodgkins lymphoma cells were transplanted into mice, green tea

prevented 50% of the tumors from taking hold and significantly inhibited

growth of the tumors (Leukemia 2000 Aug;14(8):1477-82).

Green tea was shown to help protect against the progression of atrophic

gastritis in Japanese men (Scand J Gastroenterol 2000 May;35(5)476-81).

Phenol sulsotransferases are involved in cancer growth, and green tea

extract was shown to inhibit this activity in a human colon cancer call

line (Biol Pharm Bull 2000 Jun;23(6):695-9).

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Share on other sites

" The Life Extension Foundation is constantly uncovering information to

provide the cancer patient with more ammunition to battle their disease.

A special website has been established for the purpose of updating

patients on new findings that directly pertain to the cancer protocols.

Whenever Life Extension discovers information that points to a better

way of treating cancer, it will be posted on the website

www.lefcancer.org " .

[From their recent newsletter]

Protocol

Breast Cancer

Breast cancer occurs when cells in the breast tissue divide and grow

without control. In a healthy body, the cell cycle is the natural

mechanism that regulates the creation, growth, and death of cells. It is

this mechanism that regulates cell division, allowing the creation of

new tissue as older cells die. When the normal cell regulators

malfunction and cells do not die at the proper rate, there is a failure

of apoptosis (programmed cell death) and cell growth goes unchecked. As

a result, cancer begins to develop as cells divide without control,

accumulating into a mass of extra tissue called a malignant tumor. As a

tumor grows, it elicits new capillary growth (angiogenesis) from the

surrounding normal tissues and diverts blood supply and nutrients away

from the tissue to feed itself. Unregulated angiogenesis can facilitate

the growth of cancer throughout the body.

An important aspect in any reproductive cancer is whether the tumor

growth is hormonally driven. Often breast cancer tumors require hormones

for growth. This is known as a receptor-positive tumor. This type of

cancer poses a unique problem because the hormones involved in tumor

growth are either estrogen or progesterone or both. Estrogen and

progesterone are naturally occurring hormones that the body produces in

varying amounts throughout one's lifetime. To complicate the situation

further, these hormones are essential for many other physiological

functions, such as bone integrity.

In 1991, researchers at the Institute for Hormone Research announced

that they had been able to induce the body to convert the stronger form

of estrogen (estradiol) into the weaker form (estriol) without using

drugs. Estriol is considered to be a more desirable form of estrogen. It

is less active than estradiol, so when it occupies the estrogen

receptor, it blocks estradiol's strong " grow " signals. It took only 1

week to prove that the conversion of estradiol to estriol could be

accomplished without drugs. Using a natural substance, researchers were

able to increase the conversion of estradiol to estriol by 50% in 12

healthy people (Michnovicz et al. 1991).

Next, they tested the natural substance in female mice prone to

developing breast cancer. The incidence of cancer and the number of

tumors fell significantly. The substance was indole-3-carbinol (IC3), a

phytochemical isolated from cruciferous vegetables (broccoli,

cauliflower, Brussels sprouts, turnips, kale, green cabbage, mustard

seed, etc.). I3C was then given to 17 men and women for 2 months. Again,

levels of strong estrogen declined, and levels of weak estrogen

increased. But more importantly, the level of an estrogen metabolite

associated with breast and endometrial cancer (16,alpha-hydroxyestrone)

fell (Bradlow et al. 1991).

This cancer protocol raises many issues that are subject to change as

new data emerge. Furthermore, cancer is still a disease with

unacceptably high mortality rates, and none of our suggested treatment

regimens can guarantee a cure.

Featured Products of the Week

Indole-3-carbinol

A summary of recent studies shows that this vegetable extract:

Increases the conversion of estradiol to ?weaker? (2OHE) estrogen, which

has been shown to reduce breast cancer incidence

Works in estrogen receptor negative breast cancer cells.

Stops human cancer cells from growing (54-61%) and provokes the cells to

self-destruct (apoptosis).

Inhibits MCF7 human breast cancer cells better than tamoxifen under

laboratory conditions.

Protects against the environmental toxin, dioxin.

Restores p21 tumor suppressor gene.

Provides antioxidant protection.

Super Green Tea Extract

The initial interest in studying green tea came from epidemiological

studies showing a lower rate of cancer in regions where green tea was

consumed. Life Extension Buyers Club?s 95% green tea extracts, in either

lightly caffeinated or decaffeinated form, are standardized to provide

35% of epigallocatechin gallate (EGCG). Published research shows that

EGCG is the most potent cancer cell suppressing polyphenol found in

green tea. Some noteworthy studies on green tea include:

When non-Hodgkins lymphoma cells were transplanted into mice, green tea

prevented 50% of the tumors from taking hold and significantly inhibited

growth of the tumors (Leukemia 2000 Aug;14(8):1477-82).

Green tea was shown to help protect against the progression of atrophic

gastritis in Japanese men (Scand J Gastroenterol 2000 May;35(5)476-81).

Phenol sulsotransferases are involved in cancer growth, and green tea

extract was shown to inhibit this activity in a human colon cancer call

line (Biol Pharm Bull 2000 Jun;23(6):695-9).

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