Re: improve platelet count

[Guest guest]

I was diagnosed with Leukemia over six years ago and my platelet count has

dropped below the normal range several times. I can seem to always raise it

back up by paying attention to the following:

Eating proper foods only

Water intake

Colon cleansing

Parasite cleansing

Blood cleansing - Red Clover formula

Blue Green Algae

Chlorophyll

Cat's Claw

Minerals - all of the trace ones

I don't know if any one of them raise it by themselves but combined I have

great success.

Tim

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[Guest guest]

I was diagnosed with Leukemia over six years ago and my platelet count has

dropped below the normal range several times. I can seem to always raise it

back up by paying attention to the following:

Eating proper foods only

Water intake

Colon cleansing

Parasite cleansing

Blood cleansing - Red Clover formula

Blue Green Algae

Chlorophyll

Cat's Claw

Minerals - all of the trace ones

I don't know if any one of them raise it by themselves but combined I have

great success.

Tim

------------------------------------------------------------------------

[Guest guest]

I was diagnosed with Leukemia over six years ago and my platelet count has

dropped below the normal range several times. I can seem to always raise it

back up by paying attention to the following:

Eating proper foods only

Water intake

Colon cleansing

Parasite cleansing

Blood cleansing - Red Clover formula

Blue Green Algae

Chlorophyll

Cat's Claw

Minerals - all of the trace ones

I don't know if any one of them raise it by themselves but combined I have

great success.

Tim

------------------------------------------------------------------------

[Guest guest]

Otto Korb wrote:

>

> Can somebody tell me how I do to increase the number of platelet count

> in blood?

>

> Otto Korb

do you know what the count is?

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[Guest guest]

Otto Korb wrote:

>

> Can somebody tell me how I do to increase the number of platelet count

> in blood?

>

> Otto Korb

you may need a transfusion if the count goes too low.....

the transfusion sure beats dying!

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[Guest guest]

At 20:52 13.04.99 -0300, you wrote:

> Can somebody tell me how I do to increase the number of platelet count

>in blood? Otto Korb

It is a symptom of nutritional deficiency.

Better diet, better lifestyle, beter thoughts will help.

Dr.Budwig Diet, Vegetable juices, more seaweed in diet, fish, diet adjusted

for your blood type, bowel cleanse, liver cleanse, dental cleanse ,

physical activity, ...

Dusan

http://home.sol.no/~dusan/A_Health_General_Program.html

______________________

Platelet Dysfunction

Author: Philip Cumpston { P.Cumpston@... }.

Platelets are an important part of the body's mechanisms for forming blood

clots when necessary. There are a number of reasons why the platelet count

might drop. Common causes may include the following: platelet counts often

decline after a viral illness; sometimes low platelet counts are related to

medication; and a more long-term low platelet condition called ITP

(idiopathic thrombocytopenic purpura) may be involved. The treatment to

raise low platelets depends on why platelets are low, how long they've been

low, and other medical conditions.

Disorders associated with platelet dysfunction

Myeloproliferative disorders.

Acquired storage pool or release defects.

Renal disease

Dysproteinemias

Fibrinogen and Fibrin Degradation Products

Liver disease

Endocrine disease

Lipid metabolism

Thromboembolic disorders

Other diseases

Drugs

Drugs and their effects on platelets

Many drugs have the potential to cause thrombocytopenia.

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Others have specific actions on platelet function - some rendering them

less active, others increasing their capacity to respond to stimuli.

Classes of Drugs and agents

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Platelet cyclo-oxygenase inhibitors

Platelet c-AMP Phosphodiesterase inhibitors

Prostaglandins

Thromboxane synthetase inhibitors

Thromboxane inhibitors

Membrane-active drugs

Miscellaneous agents (various or unknown actions)

Aspirin

It has long been known that aspirin ingestion is associated with an

increased risk of significant bleeding. The mechanism of this effect was

clarified when aspirin was shown to inhibit collagen-induced platelet

aggregation and the second wave of DP-induced platelet aggregation, as well

as blocking the release of ADP from platelets. ADP is known to be a potent

platelet-aggregating substance.

Aspirin and other non-steroidal anti-inflammatory analgesics have been

shown to interfere with platelet prostaglandin synthesis by inhibiting

platelet fatty acid cyclo-oxygenase. This in turn reduces synthesis of

labile endoperoxides PGG2 and PGH2 from platelet membrane arachidonic acid,

in turn reducing the production of TXA2 (Thromboxane A2), the extremely

potent mediator of platelet aggregation.

It is now known that the interference with endoperoxide production inhibits

stimulus-induced ADP release, so it is probably the inhibition of

cyclo-oxygenase that is the root cause of aspirin-induced platelet

dysfunction.

Aspirin acts on cyclo-oxygenase by causing irreversible acetylation of the

enzyme, and therefore the effect is irreversible for the life of that

platelet (7-10 days). Other non-steroidal anti-inflammatory analgesics have

a reversible action on that enzyme, and hence only act until the drug is

cleared from the circulation.

Aspirin also inhibits cyclo-oxygenase in the endothelial cells of blood

vessels. This action results in inhibition of PGI2 production. PGI2 is the

most potent inhibitor of platelet aggregation know. However, endothelial

cells can actively synthesise cyclo-oxygenase, so recovery of PGI2

synthesis is possible with intermittent aspirin ingestion.

Uremia

defective availabilitty of platelet factor 3 (platelet membrane phospholipid)

impairment of platelet adheerence to foreign surfaces

defective platelet-to-platelet interaction (aggregation) in response to

ADP, adrenaline, collagen.

Penicillins and cephalosporins

Carbenicillin and Penicillin G in high doses are the most likely to cause

problems. A bleeding tendency, manifested by prolonged bleeding time and

impaired platelet aggregation has been reported. Most other penicillins,

including ampicillin, nafcillin, oxacillin, and to a lesser extent,

methicillin and ticarcillin can effect similar, dose-dependent effects.

Cephalosporins can also exert these effects.

These substances act by inhibiting most platelet reactions, such as shape

change; adhesion to collagen-coated or subendothelial surfaces; platelet

aggregation induced by ADP, adrenaline, collagen, thrombin; platelet

release of stored substances; generation of platelet factor 3; and clot

retraction.

These antibiotics may also intercalate into the bilipid layer of the

platelet plasma membrane and interfere with binding of various agonists to

receptor sites. Such a mechanism would be consistent with the global

effects of these drugs on platelet functions, and the observed relationship

between the lipid solubility of various drugs and their antiplatelet effects.

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References

1) Sakariassen KS, Bolhuis PA, Sixma JJ. Human blood platelet adhesion to

artery subendothelium is mediated by factor VII:von Willebrand factor bound

to the subendothelium. Nature 279:636,1979

2) Connor AM, Laposta M. A rapid assay for platelet thromboxane production

and its use in assessing prior aspirin ingestion. Am J Clin Pathol

1988;89:216-221

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[Guest guest]

You might be told to take iron supplements, but you might consult Dr. Leo

Galland's book " The Four Pillars of Healing " . He explains how taking iron

can be risky business. Excellent comprehensive review of nutrition therapy.

This doc is in NYC. The best.

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