Re: Hashi and iodine/iodide

[Guest guest]

the best solution to hashi is low dose naltrexone.dr cowan says that this is the only treatment he found to work

On Wed, Aug 10, 2011 at 9:08 PM, jjc1000us <jjc1000us@...> wrote:

 

I have read through most all of the files and I did not find an adaquate answer to a question that is bothering me. I also have read Brownstien's books on Iodine and thryoid.I have hashi and as I increase my iodine, should I measure my hashi antibodies to make sure they do not go too high. What is the stratagy to ensure that the attack does not get worse on the thyroid during the period of raised TSH?

If you don't have hashi it seems there is less to worry about.Thanks.

[Guest guest]

Ido, how/where do you get your LDN?

thank you,

>

> > **

> >

> >

> > I have read through most all of the files and I did not find an adaquate

> > answer to a question that is bothering me. I also have read Brownstien's

> > books on Iodine and thryoid.

> >

> > I have hashi and as I increase my iodine, should I measure my hashi

> > antibodies to make sure they do not go too high. What is the stratagy to

> > ensure that the attack does not get worse on the thyroid during the period

> > of raised TSH?

> > If you don't have hashi it seems there is less to worry about.

> >

> > Thanks.

> >

> >

> >

>

[Guest guest]

Here is the information from the new member document: 13. I have Hashimotos. Can I take Iodine?Yes, Dr. Brownstein believes that low iodine is one the key causes in autoimmune thyroiddisease. The following is an excerpt from Dr. Brownstein’s Iodine book.THE UNDERLYING CAUSE OF AUTOIMMUNE THYROID ILLNESSES:IODINE DEFICIENCY AND ANTIOXIDANT DEFICIENCYChapter 7 described the apoptotic (i.e., anticancer) effects of the iodinated form of lactone (δ- Iodolactone).δ- Iodolactone is not only important for preventing cancer, its production is also necessary to help regulate theoxidation of iodine. Figure 3 illustrates this regulatory step in the oxidation/organification of iodine.As previously mentioned, the oxidation of iodide to iodine occurs through the interaction of H2O2 and TPO.Iodine is a necessary product in order to provide the correct molecule in the cell so that organification canIodine Group New Member Document Rev 3/17/11iodine/5occur. If organification does not take place or is blocked, thyroid hormone and iodo-lipids will not be formed.As can be seen from Figure 3, this reaction is controlled by intracellular calcium levels and iodinated lipids—δ-iodolactone.Intracellular calcium stimulates this pathway. On the other hand, δ-iodolactone and other iodinated lipids act asa brake on the system. If there is not enough iodine in the cell to organify and produce adequate amounts of δ-iodolactone, it can set the stage for damage to the thyroid cell and the development of an autoimmune thyroiddisorder such as Hashimoto’s or Graves’ disease.A Proposed Mechanism for the Development ofAutoimmune Thyroid DisordersThe NADPH oxydase system is found in the mitochondria of our cells. The mitochondria are the energyproducingcells of our body. The mitochondria produce energy (i.e., ATP) through a complex process calledoxidative phosphorylation. All medical students (and most physicians) are familiar with oxidativephosphorylation because we have to memorize the many steps responsible for producing ATP. This productionof ATP requires many items including: oxygen, magnesium, ADP, and amino acids.Many people with chronic illnesses, such as fibromyalgia, chronic fatigue syndrome, and autoimmunedisorders, complain they have no energy. ATP is the molecule that stores energy for the body. The body isconstantly producing and utilizing ATP. Its production is a complex process that is beyond this book.However, there are two cofactors, Vitamins B2 (riboflavin) and B3 (niacin), that are integral to stimulatingoxidative phosphorylation and ATP production.Hydrogen peroxide is a byproduct of oxidative phosphorylation. It is this production of hydrogen peroxide thatis so critical to the oxidation process of iodine. Hydrogen peroxide and TPO help to oxidize iodide to formiodine.If there is a deficiency in iodine, which is common when ingesting the RDA for iodine, there will not be enoughsubstrate (i.e., iodine) to produce iodinated lipids. As can be seen from Figure 3, the lack of δ-iodolactone andother iodinated lipids results in a loss of the ‘brake’ in the pathway to oxidize iodide. This may result in atemporarily production of too much hydrogen peroxide. This excess hydrogen peroxide can damage the enzymeTPO.What Happens If TPO Is Damaged? Autoimmune Thyroid IllnessThe body’s response to TPO damage is to produce antibodies against TPO or anti-TPO antibodies. A diagnosisof Hashimoto’s disease requires the presence of anti-TPO antibodies. As the damage worsens, surroundingproteins can also be damaged such as thyroglobulin. Damaged thyroglobulin will result in the body producingantibodies against thyroglobulin—anti-thyroglobulin antibodies.Iodine Group New Member Document Rev 3/17/11iodine/6In most cases of Hashimoto’s disease, there are antibodies to both TPO and thyroglobulin present. AlthoughGraves’ disease may also possess these same antibodies, antibody production is not necessary to make thediagnosis of Graves’ disease. However, my clinical experience has shown that the treatment for bothHashimoto’s and Graves’ disease can follow a similar course with similar positive outcomes.How to Treat Autoimmune Thyroid Disorders1. Ingest enough iodine in order to provide adequate substrate to iodinate lipids.2. Take Vitamins B2 (Riboflavin) and B3 (Niacin) in amounts necessary to stimulate the NADPH systemto produce adequate amounts of H2O2.3. Correct oxidant stress in the thyroid gland and the mitochondria with antioxidants.4. Ensure adequate magnesium levels.5. Minimize oxidative stress in the body. Dr. Brownstein says in this that adequate amounts of iodine will treat your hashi. It is what heals it, not make is worse. Just make sure you get enough and take all the companion nutrients. Donna in IL From: iodine [mailto:iodine ] On Behalf Of jjc1000usSent: Wednesday, August 10, 2011 2:09 PMiodine Subject: Hashi and iodine/iodide I have read through most all of the files and I did not find an adaquate answer to a question that is bothering me. I also have read Brownstien's books on Iodine and thryoid.I have hashi and as I increase my iodine, should I measure my hashi antibodies to make sure they do not go too high. What is the stratagy to ensure that the attack does not get worse on the thyroid during the period of raised TSH?If you don't have hashi it seems there is less to worry about.Thanks

[Guest guest]

did you get your prescripion in Belgium? Do we have to take LDN for several

months or forever?

> > >

> > > > **

> > > >

> > > >

> > > > I have read through most all of the files and I did not find an

> > adaquate

> > > > answer to a question that is bothering me. I also have read

> > Brownstien's

> > > > books on Iodine and thryoid.

> > > >

> > > > I have hashi and as I increase my iodine, should I measure my hashi

> > > > antibodies to make sure they do not go too high. What is the stratagy

> > to

> > > > ensure that the attack does not get worse on the thyroid during the

> > period

> > > > of raised TSH?

> > > > If you don't have hashi it seems there is less to worry about.

> > > >

> > > > Thanks.

> > > >

> > > >

> > > >

> > >

> >

> >

> >

>

[Guest guest]

Thanks for the below info. As I stated I have read Dr. B's books on Iodine and

Thyroid. I have also read other related publications. If one's reason for

getting Hashi was iodine deficiency it makes sence to supplement aggressively

with iodine. Dr. B states other causes of Hashi may be toxicity, food

allergies, infections, and nutrional imbalances. It seems if these are also

issues, you would want to correct them before aggressivley using iodine.

I know this is controversial and I am not trying to stir the pot. Dr. B even

admits that iodine can cause flair ups in Hashimotos(IODINE page 240). He

attempts to minimize them with Salt, Vit C, selenium and magnesium. There is

some risk of further thyroid damage from flair ups. How is this risk managed? To

an earlier point a member made, how often should Antibodies be checked?

I think it would give confidence to hear from others who have Hashimoto's and

have been down this path of iodine supplementation. What was their experience

like?

Did they take Thyroid hormones and were they able to get off of them?

Did they have flair ups? etc.

Thanks in advance to all those who weigh in with Hashi or can speak for someone

with Hashi.

>

> Here is the information from the new member document:

>

> 13. I have Hashimotos. Can I take Iodine?

> Yes, Dr. Brownstein believes that low iodine is one the key causes in

autoimmune thyroid

> disease. The following is an excerpt from Dr. Brownstein's Iodine book.

> THE UNDERLYING CAUSE OF AUTOIMMUNE THYROID ILLNESSES:

> IODINE DEFICIENCY AND ANTIOXIDANT DEFICIENCY

> Chapter 7 described the apoptotic (i.e., anticancer) effects of the iodinated

form of lactone (δ- Iodolactone).

> δ- Iodolactone is not only important for preventing cancer, its production is

also necessary to help regulate the

> oxidation of iodine. Figure 3 illustrates this regulatory step in the

oxidation/organification of iodine.

> As previously mentioned, the oxidation of iodide to iodine occurs through the

interaction of H2O2 and TPO.

> Iodine is a necessary product in order to provide the correct molecule in the

cell so that organification can

> Iodine Group New Member Document Rev 3/17/11

> iodine/

> 5

> occur. If organification does not take place or is blocked, thyroid hormone

and iodo-lipids will not be formed.

> As can be seen from Figure 3, this reaction is controlled by intracellular

calcium levels and iodinated lipids-

> δ-iodolactone.

> Intracellular calcium stimulates this pathway. On the other hand,

δ-iodolactone and other iodinated lipids act as

> a brake on the system. If there is not enough iodine in the cell to organify

and produce adequate amounts of δ-

> iodolactone, it can set the stage for damage to the thyroid cell and the

development of an autoimmune thyroid

> disorder such as Hashimoto's or Graves' disease.

> A Proposed Mechanism for the Development of

> Autoimmune Thyroid Disorders

> The NADPH oxydase system is found in the mitochondria of our cells. The

mitochondria are the energyproducing

> cells of our body. The mitochondria produce energy (i.e., ATP) through a

complex process called

> oxidative phosphorylation. All medical students (and most physicians) are

familiar with oxidative

> phosphorylation because we have to memorize the many steps responsible for

producing ATP. This production

> of ATP requires many items including: oxygen, magnesium, ADP, and amino acids.

> Many people with chronic illnesses, such as fibromyalgia, chronic fatigue

syndrome, and autoimmune

> disorders, complain they have no energy. ATP is the molecule that stores

energy for the body. The body is

> constantly producing and utilizing ATP. Its production is a complex process

that is beyond this book.

> However, there are two cofactors, Vitamins B2 (riboflavin) and B3 (niacin),

that are integral to stimulating

> oxidative phosphorylation and ATP production.

> Hydrogen peroxide is a byproduct of oxidative phosphorylation. It is this

production of hydrogen peroxide that

> is so critical to the oxidation process of iodine. Hydrogen peroxide and TPO

help to oxidize iodide to form

> iodine.

> If there is a deficiency in iodine, which is common when ingesting the RDA for

iodine, there will not be enough

> substrate (i.e., iodine) to produce iodinated lipids. As can be seen from

Figure 3, the lack of δ-iodolactone and

> other iodinated lipids results in a loss of the 'brake' in the pathway to

oxidize iodide. This may result in a

> temporarily production of too much hydrogen peroxide. This excess hydrogen

peroxide can damage the enzyme

> TPO.

> What Happens If TPO Is Damaged? Autoimmune Thyroid Illness

> The body's response to TPO damage is to produce antibodies against TPO or

anti-TPO antibodies. A diagnosis

> of Hashimoto's disease requires the presence of anti-TPO antibodies. As the

damage worsens, surrounding

> proteins can also be damaged such as thyroglobulin. Damaged thyroglobulin will

result in the body producing

> antibodies against thyroglobulin-anti-thyroglobulin antibodies.

> Iodine Group New Member Document Rev 3/17/11

> iodine/

> 6

> In most cases of Hashimoto's disease, there are antibodies to both TPO and

thyroglobulin present. Although

> Graves' disease may also possess these same antibodies, antibody production is

not necessary to make the

> diagnosis of Graves' disease. However, my clinical experience has shown that

the treatment for both

> Hashimoto's and Graves' disease can follow a similar course with similar

positive outcomes.

> How to Treat Autoimmune Thyroid Disorders

> 1. Ingest enough iodine in order to provide adequate substrate to iodinate

lipids.

> 2. Take Vitamins B2 (Riboflavin) and B3 (Niacin) in amounts necessary to

stimulate the NADPH system

> to produce adequate amounts of H2O2.

> 3. Correct oxidant stress in the thyroid gland and the mitochondria with

antioxidants.

> 4. Ensure adequate magnesium levels.

> 5. Minimize oxidative stress in the body.

>

>

> Dr. Brownstein says in this that adequate amounts of iodine will treat your

hashi. It is what heals it, not make is worse. Just make sure you get enough and

take all the companion nutrients.

>

> Donna in IL

>

> From: iodine [mailto:iodine ] On Behalf Of

jjc1000us

> Sent: Wednesday, August 10, 2011 2:09 PM

> iodine

> Subject: Hashi and iodine/iodide

>

>

>

> I have read through most all of the files and I did not find an adaquate

answer to a question that is bothering me. I also have read Brownstien's books

on Iodine and thryoid.

>

> I have hashi and as I increase my iodine, should I measure my hashi antibodies

to make sure they do not go too high. What is the stratagy to ensure that the

attack does not get worse on the thyroid during the period of raised TSH?

> If you don't have hashi it seems there is less to worry about.

>

> Thanks.

>

> ________________________________

>

[Guest guest]

thank you for all these informations, Ido!

about Berkson protocol for the liver, are you talking about " The Alpha Lipoic

Acid " book?

> > > > >

> > > > > > **

> > > > > >

> > > > > >

> > > > > > I have read through most all of the files and I did not find an

> > > > adaquate

> > > > > > answer to a question that is bothering me. I also have read

> > > > Brownstien's

> > > > > > books on Iodine and thryoid.

> > > > > >

> > > > > > I have hashi and as I increase my iodine, should I measure my hashi

> > > > > > antibodies to make sure they do not go too high. What is the

> > stratagy

> > > > to

> > > > > > ensure that the attack does not get worse on the thyroid during the

> > > > period

> > > > > > of raised TSH?

> > > > > > If you don't have hashi it seems there is less to worry about.

> > > > > >

> > > > > > Thanks.

> > > > > >

> > > > > >

> > > > > >

> > > > >

> > > >

> > > >

> > > >

> > >

> >

> >

> >

>

[Guest guest]

Well, the loading dose made me happy yes, 50 mgs, not sure how much to take

daily tho? I did get several pimples on my neck the day after.

> > > > >

> > > > > > **

> > > > > >

> > > > > >

> > > > > > I have read through most all of the files and I did not find an

> > > > adaquate

> > > > > > answer to a question that is bothering me. I also have read

> > > > Brownstien's

> > > > > > books on Iodine and thryoid.

> > > > > >

> > > > > > I have hashi and as I increase my iodine, should I measure my hashi

> > > > > > antibodies to make sure they do not go too high. What is the

stratagy

> > > > to

> > > > > > ensure that the attack does not get worse on the thyroid during the

> > > > period

> > > > > > of raised TSH?

> > > > > > If you don't have hashi it seems there is less to worry about.

> > > > > >

> > > > > > Thanks.

> > > > > >

> > > > > >

> > > > > >

> > > > >

> > > >

> > > >

> > > >

> > >

> >

> >

>

[Guest guest]

do you still take NTH since LDN?

> > > > > > >

> > > > > > > > **

> > > > > > > >

> > > > > > > >

> > > > > > > > I have read through most all of the files and I did not find an

> > > > > > adaquate

> > > > > > > > answer to a question that is bothering me. I also have read

> > > > > > Brownstien's

> > > > > > > > books on Iodine and thryoid.

> > > > > > > >

> > > > > > > > I have hashi and as I increase my iodine, should I measure my

> > hashi

> > > > > > > > antibodies to make sure they do not go too high. What is the

> > > > stratagy

> > > > > > to

> > > > > > > > ensure that the attack does not get worse on the thyroid during

> > the

> > > > > > period

> > > > > > > > of raised TSH?

> > > > > > > > If you don't have hashi it seems there is less to worry about.

> > > > > > > >

> > > > > > > > Thanks.

> > > > > > > >

> > > > > > > >

> > > > > > > >

> > > > > > >

> > > > > >

> > > > > >

> > > > > >

> > > > >

> > > >

> > > >

> > > >

> > >

> >

> >

> >

>

[Guest guest]

,

Thanks for sharing your success story.

I hope others with Hashi will also weigh in and share their experience on an

Iodine/Iodide protocol and how they managed any flair ups.

> >

> > Here is the information from the new member document:

> >

> > 13. I have Hashimotos. Can I take Iodine?

> > Yes, Dr. Brownstein believes that low iodine is one the key causes in

> >autoimmune thyroid

> > disease. The following is an excerpt from Dr. Brownstein's Iodine book.

> > THE UNDERLYING CAUSE OF AUTOIMMUNE THYROID ILLNESSES:

> > IODINE DEFICIENCY AND ANTIOXIDANT DEFICIENCY

> > Chapter 7 described the apoptotic (i.e., anticancer) effects of the

iodinated

> >form of lactone (δ- Iodolactone).

> > δ- Iodolactone is not only important for preventing cancer, its

production is

> >also necessary to help regulate the

> > oxidation of iodine. Figure 3 illustrates this regulatory step in the

> >oxidation/organification of iodine.

> > As previously mentioned, the oxidation of iodide to iodine occurs through

the

> >interaction of H2O2 and TPO.

> > Iodine is a necessary product in order to provide the correct molecule in

the

> >cell so that organification can

> > Iodine Group New Member Document Rev 3/17/11

> > iodine/

> > 5

> > occur. If organification does not take place or is blocked, thyroid hormone

and

> >iodo-lipids will not be formed.

> > As can be seen from Figure 3, this reaction is controlled by intracellular

> >calcium levels and iodinated lipids-

> > δ-iodolactone.

> > Intracellular calcium stimulates this pathway. On the other hand,

> >δ-iodolactone and other iodinated lipids act as

> > a brake on the system. If there is not enough iodine in the cell to organify

> >and produce adequate amounts of δ-

> > iodolactone, it can set the stage for damage to the thyroid cell and the

> >development of an autoimmune thyroid

> > disorder such as Hashimoto's or Graves' disease.

> > A Proposed Mechanism for the Development of

> > Autoimmune Thyroid Disorders

> > The NADPH oxydase system is found in the mitochondria of our cells. The

> >mitochondria are the energyproducing

> > cells of our body. The mitochondria produce energy (i.e., ATP) through a

> >complex process called

> > oxidative phosphorylation. All medical students (and most physicians) are

> >familiar with oxidative

> > phosphorylation because we have to memorize the many steps responsible for

> >producing ATP. This production

> > of ATP requires many items including: oxygen, magnesium, ADP, and amino

acids.

> > Many people with chronic illnesses, such as fibromyalgia, chronic fatigue

> >syndrome, and autoimmune

> > disorders, complain they have no energy. ATP is the molecule that stores

energy

> >for the body. The body is

> > constantly producing and utilizing ATP. Its production is a complex process

> >that is beyond this book.

> > However, there are two cofactors, Vitamins B2 (riboflavin) and B3 (niacin),

> >that are integral to stimulating

> > oxidative phosphorylation and ATP production.

> > Hydrogen peroxide is a byproduct of oxidative phosphorylation. It is this

> >production of hydrogen peroxide that

> > is so critical to the oxidation process of iodine. Hydrogen peroxide and TPO

> >help to oxidize iodide to form

> > iodine.

> > If there is a deficiency in iodine, which is common when ingesting the RDA

for

> >iodine, there will not be enough

> > substrate (i.e., iodine) to produce iodinated lipids. As can be seen from

> >Figure 3, the lack of δ-iodolactone and

> > other iodinated lipids results in a loss of the 'brake' in the pathway to

> >oxidize iodide. This may result in a

> > temporarily production of too much hydrogen peroxide. This excess hydrogen

> >peroxide can damage the enzyme

> > TPO.

> > What Happens If TPO Is Damaged? Autoimmune Thyroid Illness

> > The body's response to TPO damage is to produce antibodies against TPO or

> >anti-TPO antibodies. A diagnosis

> > of Hashimoto's disease requires the presence of anti-TPO antibodies. As the

> >damage worsens, surrounding

> > proteins can also be damaged such as thyroglobulin. Damaged thyroglobulin

will

> >result in the body producing

> > antibodies against thyroglobulin-anti-thyroglobulin antibodies.

> > Iodine Group New Member Document Rev 3/17/11

> > iodine/

> > 6

> > In most cases of Hashimoto's disease, there are antibodies to both TPO and

> >thyroglobulin present. Although

> > Graves' disease may also possess these same antibodies, antibody production

is

> >not necessary to make the

> > diagnosis of Graves' disease. However, my clinical experience has shown that

> >the treatment for both

> > Hashimoto's and Graves' disease can follow a similar course with similar

> >positive outcomes.

> > How to Treat Autoimmune Thyroid Disorders

> > 1. Ingest enough iodine in order to provide adequate substrate to iodinate

> >lipids.

> > 2. Take Vitamins B2 (Riboflavin) and B3 (Niacin) in amounts necessary to

> >stimulate the NADPH system

> > to produce adequate amounts of H2O2.

> > 3. Correct oxidant stress in the thyroid gland and the mitochondria with

> >antioxidants.

> > 4. Ensure adequate magnesium levels.

> > 5. Minimize oxidative stress in the body.

> >

> >

> > Dr. Brownstein says in this that adequate amounts of iodine will treat your

> >hashi. It is what heals it, not make is worse. Just make sure you get enough

and

> >take all the companion nutrients.

> >

> > Donna in IL

> >

> > From: iodine [mailto:iodine ] On Behalf Of

> >jjc1000us

> > Sent: Wednesday, August 10, 2011 2:09 PM

> > iodine

> > Subject: Hashi and iodine/iodide

> >

> >

> >

> > I have read through most all of the files and I did not find an adaquate

answer

> >to a question that is bothering me. I also have read Brownstien's books on

> >Iodine and thryoid.

> >

> > I have hashi and as I increase my iodine, should I measure my hashi

antibodies

> >to make sure they do not go too high. What is the stratagy to ensure that the

> >attack does not get worse on the thyroid during the period of raised TSH?

> > If you don't have hashi it seems there is less to worry about.

> >

> > Thanks.

> >

> > ________________________________

> >

>