Jump to content
RemedySpot.com

Re: Iodine and Hashimotos

Rate this topic


Guest guest

Recommended Posts

Guest guest

Its normal for TSH to be high when taking iodine, tsh is not an accurate test for thyroid function anyway. What do you mean your FT3 and FT4 were off? too high, too low? will answer the Hashimoto/Iodine question as she has before.

Iodine and Hashimotos

Hi everyone,

Well it happened again yesterday, or over the weekend, someone at this group published a quote from Brownstein saying people with Hashimoto's should not take Iodine or follow the Iodine Protocol and in fact he mentions it in his book. Also I am reading "Why Do I Still Have Thyroid Symptoms? by Kharrazian and he is emphatic about people with Hashimoto's not taking iodine, not even eating shrimp. I do seem to have thyroid antibodies, or did in 2007, and I keep upping my dose of Iodoral to 50 mg but whenever I do I read something negative about taking iodine in any way, shape or form. When I tested my Thyroid a few weeks after I started on the Iodine Protocol my TSH had gone up to 13.2 and my FT3 and FT4 were off. I haven't tested again because I don't want to pay right now, so I can't provide current numbers. Can anyone provide a source of reliable information that is positive for using iodine when you have Hashimoto's to counter all this negativity? I have to admit I am getting really concerned about continuing to take my Iodoral.

Kris

Link to comment
Share on other sites

Guest guest

I took iodine for over 18 months without any Hashi flare ups or problems. I am having a break at the moment, but I am hoping that when I re-start it may clear up my Hashis.

MacGilchrist

From: Kathleen Blake <kathleenblake@...>iodine Sent: Mon, 22 March, 2010 18:19:11Subject: Re: Iodine and Hashimotos

Its normal for TSH to be high when taking iodine, tsh is not an accurate test for thyroid function anyway. What do you mean your FT3 and FT4 were off? too high, too low? will answer the Hashimoto/Iodine question as she has before.

Iodine and Hashimotos

Hi everyone,

Well it happened again yesterday, or over the weekend, someone at this group published a quote from Brownstein saying people with Hashimoto's should not take Iodine or follow the Iodine Protocol and in fact he mentions it in his book. Also I am reading "Why Do I Still Have Thyroid Symptoms? by Kharrazian and he is emphatic about people with Hashimoto's not taking iodine, not even eating shrimp. I do seem to have thyroid antibodies, or did in 2007, and I keep upping my dose of Iodoral to 50 mg but whenever I do I read something negative about taking iodine in any way, shape or form. When I tested my Thyroid a few weeks after I started on the Iodine Protocol my TSH had gone up to 13.2 and my FT3 and FT4 were off. I haven't tested again because I don't want to pay right now, so I can't provide current numbers. Can anyone provide a source of reliable information that is positive for using iodine when you have Hashimoto's to counter all

this negativity? I have to admit I am getting really concerned about continuing to take my Iodoral.

Kris

Link to comment
Share on other sites

Guest guest

Kris, Dr Brownstein says in his book that iodine is good for

Hashi's. ANyone who says different hasn't read his books. Steph's

husband's Hashi's antibodies disappeared after a year on the iodine

protocol. I have Hashi's and I'm doing great.

I really think this is an area that people should just listen to Steph and

not be willing to listen to what other people say on it. Lots of people

have opinions, lots of people can pull a quote out of context or repeat

iodine-phobic dogma here, that doesn't make it correct. Steph cannot read

every single post in detail to check for accuracy, I know she tries but she

simply doesn't have the time.

Anyone who says iodine protocol is not for Hashis's people is trying to

keep those people sick. Dr Brownstein says Hashi's is from a lack of

iodine, period.

Please don't let yourself be pushed around by every stray comment that is

made. I know that can be difficult but it's the only way to sanity. There

are always people who will stand and say that one's protocol isn't

correct. In this case, those people are dead wrong and their statements

are not the message that this Iodine list is meant to be conveying.

hope this helps. best wishes, --

>Hi everyone,

>Well it happened again yesterday, or over the weekend, someone at this

>group published a quote from Brownstein saying people with Hashimoto's

>should not take Iodine or follow the Iodine Protocol and in fact he

>mentions it in his book. Also I am reading " Why Do I Still Have Thyroid

>Symptoms? by Kharrazian and he is emphatic about people with Hashimoto's

>not taking iodine, not even eating shrimp. I do seem to have thyroid

>antibodies, or did in 2007, and I keep upping my dose of Iodoral to 50 mg

>but whenever I do I read something negative about taking iodine in any

>way, shape or form. When I tested my Thyroid a few weeks after I started

>on the Iodine Protocol my TSH had gone up to 13.2 and my FT3 and FT4 were

>off. I haven't tested again because I don't want to pay right now, so I

>can't provide current numbers. Can anyone provide a source of reliable

>information that is positive for using iodine when you have Hashimoto's to

>counter all this negativity? I have to admit I am getting really concerned

>about continuing to take my Iodoral.

>

>Kris

~~~ There is no way to peace; peace is the way ~~~~

--A.J. Muste

Link to comment
Share on other sites

Guest guest

he might have changed his mind ,but that is what is what he said

 

http://www.thyroid-info.com/articles/brownstein-hormones.htm

 

A. I agree that iodine can aggravate autoimmune thyroid conditions. Iodine supplementation in those that have an autoimmune thyroid problem can be akin to pouring gas over a fire. However, with hypothyroid conditions that are not autoimmune in nature, iodine-containing foods can actually help the thyroid function better.

On Mon, Mar 22, 2010 at 7:38 PM, Baker <vbaker@...> wrote:

 

Kris, Dr Brownstein says in his book that iodine is good for Hashi's. ANyone who says different hasn't read his books. Steph's husband's Hashi's antibodies disappeared after a year on the iodine

protocol. I have Hashi's and I'm doing great.I really think this is an area that people should just listen to Steph and not be willing to listen to what other people say on it. Lots of people have opinions, lots of people can pull a quote out of context or repeat

iodine-phobic dogma here, that doesn't make it correct. Steph cannot read every single post in detail to check for accuracy, I know she tries but she simply doesn't have the time.Anyone who says iodine protocol is not for Hashis's people is trying to

keep those people sick. Dr Brownstein says Hashi's is from a lack of iodine, period.Please don't let yourself be pushed around by every stray comment that is made. I know that can be difficult but it's the only way to sanity. There

are always people who will stand and say that one's protocol isn't correct. In this case, those people are dead wrong and their statements are not the message that this Iodine list is meant to be conveying.

hope this helps. best wishes, -->Hi everyone,>Well it happened again yesterday, or over the weekend, someone at this >group published a quote from Brownstein saying people with Hashimoto's

>should not take Iodine or follow the Iodine Protocol and in fact he >mentions it in his book. Also I am reading " Why Do I Still Have Thyroid >Symptoms? by Kharrazian and he is emphatic about people with Hashimoto's

>not taking iodine, not even eating shrimp. I do seem to have thyroid >antibodies, or did in 2007, and I keep upping my dose of Iodoral to 50 mg >but whenever I do I read something negative about taking iodine in any

>way, shape or form. When I tested my Thyroid a few weeks after I started >on the Iodine Protocol my TSH had gone up to 13.2 and my FT3 and FT4 were >off. I haven't tested again because I don't want to pay right now, so I

>can't provide current numbers. Can anyone provide a source of reliable >information that is positive for using iodine when you have Hashimoto's to >counter all this negativity? I have to admit I am getting really concerned

>about continuing to take my Iodoral.>>Kris ~~~ There is no way to peace; peace is the way ~~~~--A.J. Muste

Link to comment
Share on other sites

Guest guest

How many times do I have to say that this article was from before 1997 when he did not know what he knows now. He regrets saying this and has tried to correct it with his many interviews and his book. It is not how he feels now.

PLEASE STOP POSTING THIS as if it is how he feels now.

Re: Iodine and Hashimotos

he might have changed his mind ,but that is what is what he said

http://www.thyroid-info.com/articles/brownstein-hormones.htm

A. I agree that iodine can aggravate autoimmune thyroid conditions. Iodine supplementation in those that have an autoimmune thyroid problem can be akin to pouring gas over a fire. However, with hypothyroid conditions that are not autoimmune in nature, iodine-containing foods can actually help the thyroid function better.

On Mon, Mar 22, 2010 at 7:38 PM, Baker <vbakersstar> wrote:

Kris, Dr Brownstein says in his book that iodine is good for Hashi's. ANyone who says different hasn't read his books. Steph's husband's Hashi's antibodies disappeared after a year on the iodine protocol. I have Hashi's and I'm doing great.I really think this is an area that people should just listen to Steph and not be willing to listen to what other people say on it. Lots of people have opinions, lots of people can pull a quote out of context or repeat iodine-phobic dogma here, that doesn't make it correct. Steph cannot read every single post in detail to check for accuracy, I know she tries but she simply doesn't have the time.Anyone who says iodine protocol is not for Hashis's people is trying to keep those people sick. Dr Brownstein says Hashi's is from a lack of iodine, period.Please don't let yourself be pushed around by every stray comment that is made. I know that can be difficult but it's the only way to sanity. There are always people who will stand and say that one's protocol isn't correct. In this case, those people are dead wrong and their statements are not the message that this Iodine list is meant to be conveying.hope this helps. best wishes, -->Hi everyone,>Well it happened again yesterday, or over the weekend, someone at this >group published a quote from Brownstein saying people with Hashimoto's >should not take Iodine or follow the Iodine Protocol and in fact he >mentions it in his book. Also I am reading "Why Do I Still Have Thyroid >Symptoms? by Kharrazian and he is emphatic about people with Hashimoto's >not taking iodine, not even eating shrimp. I do seem to have thyroid >antibodies, or did in 2007, and I keep upping my dose of Iodoral to 50 mg >but whenever I do I read something negative about taking iodine in any >way, shape or form. When I tested my Thyroid a few weeks after I started >on the Iodine Protocol my TSH had gone up to 13.2 and my FT3 and FT4 were >off. I haven't tested again because I don't want to pay right now, so I >can't provide current numbers. Can anyone provide a source of reliable >information that is positive for using iodine when you have Hashimoto's to >counter all this negativity? I have to admit I am getting really concerned >about continuing to take my Iodoral.>>Kris ~~~ There is no way to peace; peace is the way ~~~~--A.J. Muste

Link to comment
Share on other sites

Guest guest

I have been diagnosed with Hashi's for 3 years now and I am on 12.5mg of Iodoral

since then. My aTPO was almost 3000. I just had my aTPO tested recently and it

535.1. I do not have any adverse affects from taking iodine. I have read Dr.

Brownsteins book.

Marilyn

>

> Hi everyone,

>

> Well it happened again yesterday, or over the weekend, someone at this group

> published a quote from Brownstein saying people with Hashimoto's should not

> take Iodine or follow the Iodine Protocol and in fact he mentions it in his

> book. Also I am reading " Why Do I Still Have Thyroid Symptoms? by Kharrazian

> and he is emphatic about people with Hashimoto's not taking iodine, not even

> eating shrimp. I do seem to have thyroid antibodies, or did in 2007, and I

> keep upping my dose of Iodoral to 50 mg but whenever I do I read something

> negative about taking iodine in any way, shape or form. When I tested my

> Thyroid a few weeks after I started on the Iodine Protocol my TSH had gone

> up to 13.2 and my FT3 and FT4 were off. I haven't tested again because I

> don't want to pay right now, so I can't provide current numbers. Can anyone

> provide a source of reliable information that is positive for using iodine

> when you have Hashimoto's to counter all this negativity? I have to admit I

> am getting really concerned about continuing to take my Iodoral.

>

>

>

> Kris

>

Link to comment
Share on other sites

Guest guest

That's great Marilyn!!!

Steph

Re: Iodine and Hashimotos

I have been diagnosed with Hashi's for 3 years now and I am on 12.5mg of Iodoral since then. My aTPO was almost 3000. I just had my aTPO tested recently and it 535.1. I do not have any adverse affects from taking iodine. I have read Dr. Brownsteins book.Marilyn>> Hi everyone,> > Well it happened again yesterday, or over the weekend, someone at this group> published a quote from Brownstein saying people with Hashimoto's should not> take Iodine or follow the Iodine Protocol and in fact he mentions it in his> book. Also I am reading "Why Do I Still Have Thyroid Symptoms? by Kharrazian> and he is emphatic about people with Hashimoto's not taking iodine, not even> eating shrimp. I do seem to have thyroid antibodies, or did in 2007, and I> keep upping my dose of Iodoral to 50 mg but whenever I do I read something> negative about taking iodine in any way, shape or form. When I tested my> Thyroid a few weeks after I started on the Iodine Protocol my TSH had gone> up to 13.2 and my FT3 and FT4 were off. I haven't tested again because I> don't want to pay right now, so I can't provide current numbers. Can anyone> provide a source of reliable information that is positive for using iodine> when you have Hashimoto's to counter all this negativity? I have to admit I> am getting really concerned about continuing to take my Iodoral.> > > > Kris>

Link to comment
Share on other sites

Guest guest

Iodine is pretty much trashed on the other thyroid groups I'm on. It's been my experience that those who bad mouth iodine really don't know much about it. A lot of people haven't even tried it and the majority of those that have, didn't do the research and didn't take it properly. I've found the same with folks "trying" a gluten free diet. They try it for a few weeks at most, don't do it correctly and then say that it didn't help at all. I honestly don't get it.Linn On Mar 22, 2010, at 2:27 PM, Baker wrote:

Thank you, Steph, I knew this wasn't the current information.

I think people would do really well to stop trying to find reasons NOT to

iodine and start reading Dr Brownstein's books and get with the

protocol. I'm looking at his IOdine book and I also have his Thyroid book

and they both are very clear that the disease is the result of LACK of

iodine and that IODINE is the treatment.

with wishes for the best of health,

--

At 02:00 PM 3/22/2010, you wrote:

>How many times do I have to say that this article was from before 1997

>when he did not know what he knows now. He regrets saying this and has

>tried to correct it with his many interviews and his book. It is not how

>he feels now.

>

>PLEASE STOP POSTING THIS as if it is how he feels now.

>

>

>

>

> Re: Iodine and Hashimotos

>

>

>

>

>he might have changed his mind ,but that is what is what he said

>

><http://www.thyroid-info.com/articles/brownstein-hormones.htm>http://www.thyroid-info.com/articles/brownstein-hormones.htm

>

>A. I agree that iodine can aggravate autoimmune thyroid conditions. Iodine

>supplementation in those that have an autoimmune thyroid problem can be

>akin to pouring gas over a fire. However, with hypothyroid conditions that

>are not autoimmune in nature, iodine-containing foods can actually help

>the thyroid function better.

>

>On Mon, Mar 22, 2010 at 7:38 PM, Baker

><<mailto:vbakersstar>vbakersstar> wrote:

>

>

>Kris, Dr Brownstein says in his book that iodine is good for

>Hashi's. ANyone who says different hasn't read his books. Steph's

>husband's Hashi's antibodies disappeared after a year on the iodine

>protocol. I have Hashi's and I'm doing great.

>

>I really think this is an area that people should just listen to Steph and

>not be willing to listen to what other people say on it. Lots of people

>have opinions, lots of people can pull a quote out of context or repeat

>iodine-phobic dogma here, that doesn't make it correct. Steph cannot read

>every single post in detail to check for accuracy, I know she tries but she

>simply doesn't have the time.

>

>Anyone who says iodine protocol is not for Hashis's people is trying to

>keep those people sick. Dr Brownstein says Hashi's is from a lack of

>iodine, period.

>

>Please don't let yourself be pushed around by every stray comment that is

>made. I know that can be difficult but it's the only way to sanity. There

>are always people who will stand and say that one's protocol isn't

>correct. In this case, those people are dead wrong and their statements

>are not the message that this Iodine list is meant to be conveying.

>

>hope this helps. best wishes, --

~~~ There is no way to peace; peace is the way ~~~~

--A.J. Muste

Link to comment
Share on other sites

Guest guest

Thanks and and Linn, I think after pausing to read the book

again (pages 135-138) I finally get it. The comments attributed to Brownstein

via Shomon were confusing and as there was no date referenced it was hard

to tell when they were said.

I am willing to stick to the Iodine Protocol but I know I need to have my facts

straight as I will soon face my doctor and have to give him the data I have

collected and reasons to not give up on Iodine just because my TSH is elevated.

Now that I know Brownstein is not in anyway against taking Iodoral if you have

Hashimoto's I guess my only question is if I should keep my level at 25 mg of

Iodoral or try to raise it to 50mg? Clearly my doctor knows a little but not a

lot about the Iodine Protocol and I need to know more than he does to treat the

issue efficiently and sufficiently, I will keep my eyes glued to the discussion

boards and I for one will not mention these outdated quotes again.

Kris

> >

> > >How many times do I have to say that this article was from before 1997

> > >when he did not know what he knows now. He regrets saying this and has

> > >tried to correct it with his many interviews and his book. It is not how

> > >he feels now.

> > >

> > >PLEASE STOP POSTING THIS as if it is how he feels now.

> > >

> > >

> > >

> > >

> >

> >

>

Link to comment
Share on other sites

Guest guest

Ok, the increase to 50 mg would be dictated by the need to reduce symptoms. Or in the case of low saturation levels with high bromide levels you know you need to get the bromide out and it seems that it is taking most people 50 mgs or more to effectively move it out. It also seems like the most benefits come from those taking 50 mgs or more. Just an observation at this point.

Stephl

Re: Iodine and Hashimotos

Thanks and and Linn, I think after pausing to read the book again (pages 135-138) I finally get it. The comments attributed to Brownstein via Shomon were confusing and as there was no date referenced it was hard to tell when they were said.I am willing to stick to the Iodine Protocol but I know I need to have my facts straight as I will soon face my doctor and have to give him the data I have collected and reasons to not give up on Iodine just because my TSH is elevated. Now that I know Brownstein is not in anyway against taking Iodoral if you have Hashimoto's I guess my only question is if I should keep my level at 25 mg of Iodoral or try to raise it to 50mg? Clearly my doctor knows a little but not a lot about the Iodine Protocol and I need to know more than he does to treat the issue efficiently and sufficiently, I will keep my eyes glued to the discussion boards and I for one will not mention these outdated quotes again. Kris> > > > >How many times do I have to say that this article was from before 1997 > > >when he did not know what he knows now. He regrets saying this and has > > >tried to correct it with his many interviews and his book. It is not how > > >he feels now.> > >> > >PLEASE STOP POSTING THIS as if it is how he feels now.> > >> > >> > >> > >> > > >>

Link to comment
Share on other sites

Guest guest

Just be prepared for the response. Personally, when I first talked to my daughter's endo about iodine, I thought his head was going to explode. :-) I'm not easily swayed though and we did manage to at least talk about it. I've found that empirical evidence works fairly well. LinnOn Mar 23, 2010, at 3:36 PM, kcpeterson55343 wrote:

Thanks and and Linn, I think after pausing to read the book again (pages 135-138) I finally get it. The comments attributed to Brownstein via Shomon were confusing and as there was no date referenced it was hard to tell when they were said.

I am willing to stick to the Iodine Protocol but I know I need to have my facts straight as I will soon face my doctor and have to give him the data I have collected and reasons to not give up on Iodine just because my TSH is elevated. Now that I know Brownstein is not in anyway against taking Iodoral if you have Hashimoto's I guess my only question is if I should keep my level at 25 mg of Iodoral or try to raise it to 50mg? Clearly my doctor knows a little but not a lot about the Iodine Protocol and I need to know more than he does to treat the issue efficiently and sufficiently, I will keep my eyes glued to the discussion boards and I for one will not mention these outdated quotes again.

Kris

> >

> > >How many times do I have to say that this article was from before 1997

> > >when he did not know what he knows now. He regrets saying this and has

> > >tried to correct it with his many interviews and his book. It is not how

> > >he feels now.

> > >

> > >PLEASE STOP POSTING THIS as if it is how he feels now.

> > >

> > >

> > >

> > >

> >

> >

>

Link to comment
Share on other sites

  • 8 months later...

Hi!You need to read the new member doc, but here is the excerpt from it: 1. I have hashimotos.  Can I take Iodine? (the following is an excerpt from Dr. Brownstien’s Iodine book)THE UNDERLYING CAUSE OF AUTOIMMUNE THYROID ILLNESSES: IODINE DEFICIENCY AND ANTIOXIDANT DEFICIENCYChapter 7 described the apoptotic (i.e., anticancer) effects of the iodinated form of lactone (δ- Iodolactone).  δ- Iodolactone is not only important for preventing cancer, its production is also necessary to help regulate the oxidation of iodine.    Figure 3 illustrates this regulatory step in the oxidation/organification of iodine.         As previously mentioned, the oxidation of iodide to iodine occurs through the interaction of H2O2 and TPO.  Iodine is a necessary product in order to provide the correct molecule in the cell so that organification can occur. If organification does not take place or is blocked, thyroid hormone and iodo-lipids will not be formed.  As can be seen from Figure 3, this reaction is controlled by intracellular calcium levels and iodinated lipids—δ-iodolactone.Intracellular calcium stimulates this pathway.  On the other hand, δ-iodolactone and other iodinated lipids act as a brake on the system. If there is not enough iodine in the cell to organify and produce adequate amounts of δ-iodolactone, it can set the stage for damage to the thyroid cell and the development of an autoimmune thyroid disorder such as Hashimoto’s or Graves’ disease. A Proposed Mechanism For The Development Of Autoimmune Thyroid DisordersThe NADPH oxydase system is found in the mitochondria of our cells. The mitochondria are the energy-producing cells of our body.  The mitochondria produce energy (i.e., ATP) through a complex process called oxidative phosphorylation.  All medical students (and most physicians) are familiar with oxidative phosphorylation because we have to memorize the many steps responsible for producing ATP.  This production of ATP requires many items including: oxygen, magnesium, ADP, and amino acids.Many people with chronic illnesses, such as fibromyalgia, chronic fatigue syndrome, and autoimmune disorders, complain they have no energy.  ATP is the molecule that stores energy for the body.  The body is constantly producing and utilizing ATP.  Its production is a complex process that is beyond this book.  However, there are two cofactors, Vitamins B2 (riboflavin) and B3 (niacin), that are integral to stimulating oxidative phosphorylation and ATP production.  Hydrogen peroxide is a byproduct of oxidative phosphorylation. It is this production of hydrogen peroxide that is so critical to the oxidation process of iodine.  Hydrogen peroxide and TPO help to oxidize iodide to form iodine.  If there is a deficiency in iodine, which is common when ingesting the RDA for iodine, there will not be enough substrate (i.e., iodine) to produce iodinated lipids.  As can be seen from Figure 3, the lack of δ-iodolactone and other iodinated lipids results in a loss of the ‘brake’ in the pathway to oxidize iodide.  This may result in a temporarily production of too much hydrogen peroxide. This excess hydrogen peroxide can damage the enzyme TPO. What Happens If TPO Is Damaged? Autoimmune Thyroid IllnessThe body’s response to TPO damage is to produce antibodies against TPO or anti-TPO antibodies.  A diagnosis of Hashimoto’s disease requires the presence of anti-TPO antibodies.  As the damage worsens, surrounding proteins can also be damaged such as thyroglobulin.  Damaged thyroglobulin will result in the body producing antibodies against thyroglobulin—anti-thyroglobulin antibodies.In most cases of Hashimoto’s disease, there are antibodies to both TPO and thyroglobulin present.  Although Graves’ disease may also possess these same antibodies, antibody production is not necessary to make the diagnosis of Graves’ disease.  However, my clinical experience has shown that the treatment for both Hashimoto’s and Graves’ disease can follow a similar course with similar positive outcomes. How to Treat Autoimmune Thyroid Disorders1. Ingest enough iodine in order to provide adequate     substrate to iodinate lipids.2.  Take Vitamins B2 and B3 in amounts necessary to stimulate the NADPH system to produce adequate amounts of H2O2.   3. Correct oxidant stress in the thyroid gland and the mitochondria with antioxidants.  4.   Ensure adequate magnesium levels.  5. Minimize oxidative stress in the body.  Donna in ILHello,I am new to the group and would like to know your advice on something. I have read both YES and NO that iodine is bad for people with hashimotos, yet some thyroid supplements contain kelp in them. I recently bought some supplements called " thyroid now " and trying to decide whether or not to take these. Can anyone shine some light on this subject?thank you

Link to comment
Share on other sites

Ok... So i guess thats a yes? I cant comprehend that document...From: Donna Iler <Donna@...>Sent: Friday, December 10, 2010 2:10 PMiodine <iodine >Subject: RE: Iodine and Hashimotos Hi!You need to read the new member doc, but here is the excerpt from it: 1. I have hashimotos. Can I take Iodine? (the following is an excerpt from Dr. Brownstien’s Iodine book)THE UNDERLYING CAUSE OF AUTOIMMUNE THYROID ILLNESSES: IODINE DEFICIENCY AND ANTIOXIDANT DEFICIENCYChapter 7 described the apoptotic (i.e., anticancer) effects of the iodinated form of lactone (δ- Iodolactone). δ- Iodolactone is not only important for preventing cancer, its production is also necessary to help regulate the oxidation of iodine. Figure 3 illustrates this regulatory step in the oxidation/organification of iodine. As previously mentioned, the oxidation of iodide to iodine occurs through the interaction of H2O2 and TPO. Iodine is a necessary product in order to provide the correct molecule in the cell so that organification can occur. If organification does not take place or is blocked, thyroid hormone and iodo-lipids will not be formed. As can be seen from Figure 3, this reaction is controlled by intracellular calcium levels and iodinated lipids—δ-iodolactone.Intracellular calcium stimulates this pathway. On the other hand, δ-iodolactone and other iodinated lipids act as a brake on the system. If there is not enough iodine in the cell to organify and produce adequate amounts of δ-iodolactone, it can set the stage for damage to the thyroid cell and the development of an autoimmune thyroid disorder such as Hashimoto’s or Graves’ disease. A Proposed Mechanism For The Development Of Autoimmune Thyroid DisordersThe NADPH oxydase system is found in the mitochondria of our cells. The mitochondria are the energy-producing cells of our body. The mitochondria produce energy (i.e., ATP) through a complex process called oxidative phosphorylation. All medical students (and most physicians) are familiar with oxidative phosphorylation because we have to memorize the many steps responsible for producing ATP. This production of ATP requires many items including: oxygen, magnesium, ADP, and amino acids.Many people with chronic illnesses, such as fibromyalgia, chronic fatigue syndrome, and autoimmune disorders, complain they have no energy. ATP is the molecule that stores energy for the body. The body is constantly producing and utilizing ATP. Its production is a complex process that is beyond this book. However, there are two cofactors, Vitamins B2 (riboflavin) and B3 (niacin), that are integral to stimulating oxidative phosphorylation and ATP production. Hydrogen peroxide is a byproduct of oxidative phosphorylation. It is this production of hydrogen peroxide that is so critical to the oxidation process of iodine. Hydrogen peroxide and TPO help to oxidize iodide to form iodine. If there is a deficiency in iodine, which is common when ingesting the RDA for iodine, there will not be enough substrate (i.e., iodine) to produce iodinated lipids. As can be seen from Figure 3, the lack of δ-iodolactone and other iodinated lipids results in a loss of the ‘brake’ in the pathway to oxidize iodide. This may result in a temporarily production of too much hydrogen peroxide. This excess hydrogen peroxide can damage the enzyme TPO. What Happens If TPO Is Damaged? Autoimmune Thyroid IllnessThe body’s response to TPO damage is to produce antibodies against TPO or anti-TPO antibodies. A diagnosis of Hashimoto’s disease requires the presence of anti-TPO antibodies. As the damage worsens, surrounding proteins can also be damaged such as thyroglobulin. Damaged thyroglobulin will result in the body producing antibodies against thyroglobulin—anti-thyroglobulin antibodies.In most cases of Hashimoto’s disease, there are antibodies to both TPO and thyroglobulin present. Although Graves’ disease may also possess these same antibodies, antibody production is not necessary to make the diagnosis of Graves’ disease. However, my clinical experience has shown that the treatment for both Hashimoto’s and Graves’ disease can follow a similar course with similar positive outcomes. How to Treat Autoimmune Thyroid Disorders1. Ingest enough iodine in order to provide adequate substrate to iodinate lipids.2. Take Vitamins B2 and B3 in amounts necessary to stimulate the NADPH system to produce adequate amounts of H2O2. 3. Correct oxidant stress in the thyroid gland and the mitochondria with antioxidants. 4. Ensure adequate magnesium levels. 5. Minimize oxidative stress in the body. Donna in ILHello,I am new to the group and would like to know your advice on something. I have read both YES and NO that iodine is bad for people with hashimotos, yet some thyroid supplements contain kelp in them. I recently bought some supplements called "thyroid now" and trying to decide whether or not to take these. Can anyone shine some light on this subject?thank you

Link to comment
Share on other sites

Thank you for responding.  One of the symptoms of hashimotos is brain fog.  It would take a person with the highest IQ available to read and comprehend that entire email....  thanks again

 

are there any posts on how to determine how much to take?

On Fri, Dec 10, 2010 at 3:09 PM, ladybugsandbees <ladybugsandbees@...> wrote:

 

 Yes it is fine.  My husband is a patient of Dr. Brownstein and he was able to eliminate the hashi's antibodies with the iodine protocol. 

 

Dr. Brownstein is describing the mechanism by which hasi's is created from iodine deficiency.

 

 

RE: Iodine and Hashimotos 

Hi!

You need to read the new member doc, but here is the excerpt from it:

1.       I have hashimotos.  Can I take Iodine? (the following is an excerpt from Dr. Brownstien’s Iodine book)

THE UNDERLYING CAUSE OF AUTOIMMUNE THYROID ILLNESSES: IODINE DEFICIENCY AND ANTIOXIDANT DEFICIENCY

Chapter 7 described the apoptotic (i.e., anticancer) effects of the iodinated form of lactone (δ- Iodolactone).  δ- Iodolactone is not only important for preventing cancer, its production is also necessary to help regulate the oxidation of iodine.    Figure 3 illustrates this regulatory step in the oxidation/organification of iodine. 

        As previously mentioned, the oxidation of iodide to iodine occurs through the interaction of H2O2 and TPO.  Iodine is a necessary product in order to provide the correct molecule in the cell so that organification can occur. If organification does not take place or is blocked, thyroid hormone and iodo-lipids will not be formed.  As can be seen from Figure 3, this reaction is controlled by intracellular calcium levels and iodinated lipids—δ-iodolactone.

Intracellular calcium stimulates this pathway.  On the other hand, δ-iodolactone and other iodinated lipids act as a brake on the system.  If there is not enough iodine in the cell to organify and produce adequate amounts of δ-iodolactone, it can set the stage for damage to the thyroid cell and the development of an autoimmune thyroid disorder such as Hashimoto’s or Graves’ disease.

 A Proposed Mechanism For The Development Of Autoimmune Thyroid Disorders

The NADPH oxydase system is found in the mitochondria of our cells. The mitochondria are the energy-producing cells of our body.  The mitochondria produce energy (i.e., ATP) through a complex process called oxidative phosphorylation.  All medical students (and most physicians) are familiar with oxidative phosphorylation because we have to memorize the many steps responsible for producing ATP.  This production of ATP requires many items including: oxygen, magnesium, ADP, and amino acids.

Many people with chronic illnesses, such as fibromyalgia, chronic fatigue syndrome, and autoimmune disorders, complain they have no energy.  ATP is the molecule that stores energy for the body.  The body is constantly producing and utilizing ATP.  Its production is a complex process that is beyond this book.  However, there are two cofactors, Vitamins B2 (riboflavin) and B3 (niacin), that are integral to stimulating oxidative phosphorylation and ATP production. 

Hydrogen peroxide is a byproduct of oxidative phosphorylation.  It is this production of hydrogen peroxide that is so critical to the oxidation process of iodine.  Hydrogen peroxide and TPO help to oxidize iodide to form iodine. 

If there is a deficiency in iodine, which is common when ingesting the RDA for iodine, there will not be enough substrate (i.e., iodine) to produce iodinated lipids.  As can be seen from Figure 3, the lack of δ-iodolactone and other iodinated lipids results in a loss of the ‘brake’ in the pathway to oxidize iodide.  This may result in a temporarily production of too much hydrogen peroxide. This excess hydrogen peroxide can damage the enzyme TPO.

What Happens If TPO Is Damaged? Autoimmune Thyroid Illness

The body’s response to TPO damage is to produce antibodies against TPO or anti-TPO antibodies.  A diagnosis of Hashimoto’s disease requires the presence of anti-TPO antibodies.  As the damage worsens, surrounding proteins can also be damaged such as thyroglobulin.  Damaged thyroglobulin will result in the body producing antibodies against thyroglobulin—anti-thyroglobulin antibodies.

In most cases of Hashimoto’s disease, there are antibodies to both TPO and thyroglobulin present.  Although Graves’ disease may also possess these same antibodies, antibody production is not necessary to make the diagnosis of Graves’ disease.  However, my clinical experience has shown that the treatment for both Hashimoto’s and Graves’ disease can follow a similar course with similar positive outcomes.

How to Treat Autoimmune Thyroid Disorders

1.      Ingest enough iodine in order to provide adequate     substrate to iodinate lipids.

2.  Take Vitamins B2 and B3 in amounts necessary to stimulate the NADPH system to produce adequate amounts of H2O2.  

3. Correct oxidant stress in the thyroid gland and the mitochondria with antioxidants. 

4.       Ensure adequate magnesium levels. 

5.        Minimize oxidative stress in the body. 

  Donna in IL

Hello,I am new to the group and would like to know your advice on something. I have read both YES and NO that iodine is bad for people with hashimotos, yet some thyroid supplements contain kelp in them. I recently bought some supplements called " thyroid now " and trying to decide whether or not to take these. Can anyone shine some light on this subject?

thank you

Link to comment
Share on other sites

does your husband have hashimotos?  Does he take anything else besides iodine (thyroid)?

 

Is this discussion appropriate here, or do i need to email you sidebar?

 

thank you,

 

bruce

On Fri, Dec 10, 2010 at 7:13 PM, ladybugsandbees <ladybugsandbees@...> wrote:

 

 It makes total sense to me - but then again I helped Dr. Brownstein edit this book.  :)  I have been researching this since 2006 so sometimes I forget how far I have come. 

 

Let me see if this is easier.  The long and the short of it is this:

 

Iodine is converted to iodide in the thyroid..

 

H2O2 (hydrogen peroxide) is used to remove one molecule from iodine to make it iodide. 

 

Iodide is then used to bind with thyroglobulin to make T4 & T3 - which are the two major thyroid hormones - the number designating the number of iodine molecules.

 

The body has a self regulation process to not " over create " iodide.  This is controlled by the level of Iodine being supplied.

 

When enough iodine is supplied to make thyroid hormones AND iodolipids then the process works well.  Iodolipids are the " breaks " on the system.  If they are not available then the H202 burns out of control in the cell and mutates the cell.

 

This mutated cell is seen as an invader by the body's immune system and it creates antibodies.  There are two antibodies - one on the hydrogen peroxide process (TPO Ab) and the other against Thyroglobul (Tg Ab) you can have one or both to have Hashimotos.

 

--------------------------

 

There is no correct dose but my husband started on 37.5 mgs Iodoral and upped it to 50 mgs after 9 mos.  He was antibody free in a little over 1 year after starting the entire protocol (in the new members document you should have already read).

 

 

 

 

 

 

 

RE: Iodine and Hashimotos 

Hi!

You need to read the new member doc, but here is the excerpt from it:

1.       I have hashimotos.  Can I take Iodine? (the following is an excerpt from Dr. Brownstien’s Iodine book)

THE UNDERLYING CAUSE OF AUTOIMMUNE THYROID ILLNESSES: IODINE DEFICIENCY AND ANTIOXIDANT DEFICIENCY

Chapter 7 described the apoptotic (i.e., anticancer) effects of the iodinated form of lactone (δ- Iodolactone).  δ- Iodolactone is not only important for preventing cancer, its production is also necessary to help regulate the oxidation of iodine.    Figure 3 illustrates this regulatory step in the oxidation/organification of iodine. 

        As previously mentioned, the oxidation of iodide to iodine occurs through the interaction of H2O2 and TPO.  Iodine is a necessary product in order to provide the correct molecule in the cell so that organification can occur. If organification does not take place or is blocked, thyroid hormone and iodo-lipids will not be formed.  As can be seen from Figure 3, this reaction is controlled by intracellular calcium levels and iodinated lipids—δ-iodolactone.

Intracellular calcium stimulates this pathway.  On the other hand, δ-iodolactone and other iodinated lipids act as a brake on the system.  If there is not enough iodine in the cell to organify and produce adequate amounts of δ-iodolactone, it can set the stage for damage to the thyroid cell and the development of an autoimmune thyroid disorder such as Hashimoto’s or Graves’ disease.

 A Proposed Mechanism For The Development Of Autoimmune Thyroid Disorders

The NADPH oxydase system is found in the mitochondria of our cells. The mitochondria are the energy-producing cells of our body.  The mitochondria produce energy (i.e., ATP) through a complex process called oxidative phosphorylation.  All medical students (and most physicians) are familiar with oxidative phosphorylation because we have to memorize the many steps responsible for producing ATP.  This production of ATP requires many items including: oxygen, magnesium, ADP, and amino acids.

Many people with chronic illnesses, such as fibromyalgia, chronic fatigue syndrome, and autoimmune disorders, complain they have no energy.  ATP is the molecule that stores energy for the body.  The body is constantly producing and utilizing ATP.  Its production is a complex process that is beyond this book.  However, there are two cofactors, Vitamins B2 (riboflavin) and B3 (niacin), that are integral to stimulating oxidative phosphorylation and ATP production. 

Hydrogen peroxide is a byproduct of oxidative phosphorylation.  It is this production of hydrogen peroxide that is so critical to the oxidation process of iodine.  Hydrogen peroxide and TPO help to oxidize iodide to form iodine. 

If there is a deficiency in iodine, which is common when ingesting the RDA for iodine, there will not be enough substrate (i.e., iodine) to produce iodinated lipids.  As can be seen from Figure 3, the lack of δ-iodolactone and other iodinated lipids results in a loss of the ‘brake’ in the pathway to oxidize iodide.  This may result in a temporarily production of too much hydrogen peroxide. This excess hydrogen peroxide can damage the enzyme TPO.

What Happens If TPO Is Damaged? Autoimmune Thyroid Illness

The body’s response to TPO damage is to produce antibodies against TPO or anti-TPO antibodies.  A diagnosis of Hashimoto’s disease requires the presence of anti-TPO antibodies.  As the damage worsens, surrounding proteins can also be damaged such as thyroglobulin.  Damaged thyroglobulin will result in the body producing antibodies against thyroglobulin—anti-thyroglobulin antibodies.

In most cases of Hashimoto’s disease, there are antibodies to both TPO and thyroglobulin present.  Although Graves’ disease may also possess these same antibodies, antibody production is not necessary to make the diagnosis of Graves’ disease.  However, my clinical experience has shown that the treatment for both Hashimoto’s and Graves’ disease can follow a similar course with similar positive outcomes.

How to Treat Autoimmune Thyroid Disorders

1.      Ingest enough iodine in order to provide adequate     substrate to iodinate lipids.

2.  Take Vitamins B2 and B3 in amounts necessary to stimulate the NADPH system to produce adequate amounts of H2O2.  

3. Correct oxidant stress in the thyroid gland and the mitochondria with antioxidants. 

4.       Ensure adequate magnesium levels. 

5.        Minimize oxidative stress in the body. 

  Donna in IL

Hello,I am new to the group and would like to know your advice on something. I have read both YES and NO that iodine is bad for people with hashimotos, yet some thyroid supplements contain kelp in them. I recently bought some supplements called " thyroid now " and trying to decide whether or not to take these. Can anyone shine some light on this subject?

thank you

Link to comment
Share on other sites

Bruce-- Did you read the New Member document? It describes the Iodine

protocol which is the iodine plus a short list of companion nutrients that

the body needs in combination with the iodine.

I have Hashi's, I take NatureThroid and a number of supplements of various

types, and I take the Iodine protocol.

I'm attaching the New Member document here.

--

At 07:16 PM 12/10/2010, you wrote:

>does your husband have hashimotos? Does he take anything else besides

>iodine (thyroid)?

>Is this discussion appropriate here, or do i need to email you sidebar?

>thank you,

>bruce

>

>On Fri, Dec 10, 2010 at 7:13 PM, ladybugsandbees

><<mailto:ladybugsandbees@...>ladybugsandbees@...> wrote:

>

>

>It makes total sense to me - but then again I helped Dr. Brownstein edit

>this book. :) I have been researching this since 2006 so sometimes I

>forget how far I have come.Â

>Let me see if this is easier. The long and the short of it is this:

>Iodine is converted to iodide in the thyroid..

>H2O2 (hydrogen peroxide) is used to remove one molecule from iodine to

>make it iodide.Â

>Iodide is then used to bind with thyroglobulin to make T4 & T3 - which are

>the two major thyroid hormones - the number designating the number of

>iodine molecules.

>The body has a self regulation process to not " over create " iodide. This

>is controlled by the level of Iodine being supplied.

>When enough iodine is supplied to make thyroid hormones AND iodolipids

>then the process works well. Iodolipids are the " breaks " on the

>system. If they are not available then the H202 burns out of control in

>the cell and mutates the cell.

>This mutated cell is seen as an invader by the body's immune system and it

>creates antibodies. There are two antibodies - one on the hydrogen

>peroxide process (TPO Ab) and the other against Thyroglobul (Tg Ab) you

>can have one or both to have Hashimotos.

>--------------------------

>There is no correct dose but my husband started on 37.5 mgs Iodoral and

>upped it to 50 mgs after 9 mos. He was antibody free in a little over 1

>year after starting the entire protocol (in the new members document you

>should have already read).

>

> RE: Iodine and Hashimotos

>

>

>Hi!

>

>You need to read the new member doc, but here is the excerpt from it:

>

>1.      I have hashimotos. Can I take Iodine? (the following is

>an excerpt from Dr. Brownstien’s Iodine book)

>

>THE UNDERLYING CAUSE OF AUTOIMMUNE THYROID ILLNESSES: IODINE DEFICIENCY

>AND ANTIOXIDANT DEFICIENCY

>

>Chapter 7 described the apoptotic (i.e., anticancer) effects of the

>iodinated form of lactone (δ- Iodolactone). δ- Iodolactone is not only

>important for preventing cancer, its production is also necessary to help

>regulate the oxidation of iodine.   Figure 3 illustrates this

>regulatory step in the oxidation/organification of iodine.Â

>

>Â Â Â Â Â Â Â As previously mentioned, the oxidation of iodide to iodine

>occurs through the interaction of H2O2 and TPO. Iodine is a necessary

>product in order to provide the correct molecule in the cell so that

>organification can occur. If organification does not take place or is

>blocked, thyroid hormone and iodo-lipids will not be formed. As can be

>seen from Figure 3, this reaction is controlled by intracellular calcium

>levels and iodinated lipids—δ-iodolactone.

>

>Intracellular calcium stimulates this pathway. On the other hand,

>δ-iodolactone and other iodinated lipids act as a brake on the

>system. If there is not enough iodine in the cell to organify and

>produce adequate amounts of δ-iodolactone, it can set the stage for

>damage to the thyroid cell and the development of an autoimmune thyroid

>disorder such as Hashimoto’s or Graves’ disease.

>

>Â A Proposed Mechanism For The Development Of Autoimmune Thyroid Disorders

>

>The NADPH oxydase system is found in the mitochondria of our cells. The

>mitochondria are the energy-producing cells of our body. The

>mitochondria produce energy (i.e., ATP) through a complex process called

>oxidative phosphorylation. All medical students (and most physicians)

>are familiar with oxidative phosphorylation because we have to memorize

>the many steps responsible for producing ATP. This production of ATP

>requires many items including: oxygen, magnesium, ADP, and amino acids.

>

>Many people with chronic illnesses, such as fibromyalgia, chronic fatigue

>syndrome, and autoimmune disorders, complain they have no energy. ATP is

>the molecule that stores energy for the body. The body is constantly

>producing and utilizing ATP. Its production is a complex process that is

>beyond this book. However, there are two cofactors, Vitamins B2

>(riboflavin) and B3 (niacin), that are integral to stimulating oxidative

>phosphorylation and ATP production.Â

>

>Hydrogen peroxide is a byproduct of oxidative phosphorylation. It is

>this production of hydrogen peroxide that is so critical to the oxidation

>process of iodine. Hydrogen peroxide and TPO help to oxidize iodide to

>form iodine.Â

>

>If there is a deficiency in iodine, which is common when ingesting the RDA

>for iodine, there will not be enough substrate (i.e., iodine) to produce

>iodinated lipids. As can be seen from Figure 3, the lack of

>δ-iodolactone and other iodinated lipids results in a loss of the

>‘brake’ in the pathway to oxidize iodide. This may result in a

>temporarily production of too much hydrogen peroxide. This excess hydrogen

>peroxide can damage the enzyme TPO.

>

>What Happens If TPO Is Damaged? Autoimmune Thyroid Illness

>

>The body’s response to TPO damage is to produce antibodies against TPO

>or anti-TPO antibodies. A diagnosis of Hashimoto’s disease requires

>the presence of anti-TPO antibodies. As the damage worsens, surrounding

>proteins can also be damaged such as thyroglobulin. Damaged

>thyroglobulin will result in the body producing antibodies against

>thyroglobulin—aanti-thyroglobulin antibodies.

>

>In most cases of Hashimoto’s disease, there are antibodies to both TPO

>and thyroglobulin present. Although Graves’ disease may also possess

>these same antibodies, antibody production is not necessary to make the

>diagnosis of Graves’ disease. However, my clinical experience has

>shown that the treatment for both Hashimoto’s and Graves’ disease can

>follow a similar course with similar positive outcomes.

>

>How to Treat Autoimmune Thyroid Disorders

>

>1.     Ingest enough iodine in order to provide adequate  Â

>Â substrate to iodinate lipids.

>

>2. Take Vitamins B2 and B3 in amounts necessary to stimulate the NADPH

>system to produce adequate amounts of H2O2. Â

>

>3. Correct oxidant stress in the thyroid gland and the mitochondria with

>antioxidants.Â

>

>4.      Ensure adequate magnesium levels.Â

>

>5.       Minimize oxidative stress in the body.Â

>

>Â Donna in IL

>

>Hello,

>

>I am new to the group and would like to know your advice on something. I

>have read both YES and NO that iodine is bad for people with hashimotos,

>yet some thyroid supplements contain kelp in them. I recently bought some

>supplements called " thyroid now " and trying to decide whether or not to

>take these. Can anyone shine some light on this subject?

>

>thank you

>

>

>

>

>

>

~~~ There is no way to peace; peace is the way ~~~~

--A.J. Muste

1 of 1 File(s)

Iodine New Member.doc

Link to comment
Share on other sites



I am not sure how I can help you because that "IS" the beginners guide. Try reading Dr. Brownstein's book when you get it and see if it makes more sense then.

Steph

RE: Iodine and Hashimotos>>Â>>Hi!>>You need to read the new member doc, but here is the excerpt from it:>>1.      I have hashimotos. Can I take Iodine? (the following is >an excerpt from Dr. Brownstien’s Iodine book)>>THE UNDERLYING CAUSE OF AUTOIMMUNE THYROID ILLNESSES: IODINE DEFICIENCY >AND ANTIOXIDANT DEFICIENCY>>Chapter 7 described the apoptotic (i.e., anticancer) effects of the >iodinated form of lactone (δ- Iodolactone). δ- Iodolactone is not only >important for preventing cancer, its production is also necessary to help >regulate the oxidation of iodine.   Figure 3 illustrates this >regulatory step in the oxidation/organification of iodine.Â>>       As previously mentioned, the oxidation of iodide to iodine >occurs through the interaction of H2O2 and TPO. Iodine is a necessary >product in order to provide the correct molecule in the cell so that >organification can occur. If organification does not take place or is >blocked, thyroid hormone and iodo-lipids will not be formed. As can be >seen from Figure 3, this reaction is controlled by intracellular calcium >levels and iodinated lipids—δ-iodolactone.>>Intracellular calcium stimulates this pathway. On the other hand, >δ-iodolactone and other iodinated lipids act as a brake on the >system. If there is not enough iodine in the cell to organify and >produce adequate amounts of δ-iodolactone, it can set the stage for >damage to the thyroid cell and the development of an autoimmune thyroid >disorder such as Hashimoto’s or Graves’ disease.>> A Proposed Mechanism For The Development Of Autoimmune Thyroid Disorders>>The NADPH oxydase system is found in the mitochondria of our cells. The >mitochondria are the energy-producing cells of our body. The >mitochondria produce energy (i.e., ATP) through a complex process called >oxidative phosphorylation. All medical students (and most physicians) >are familiar with oxidative phosphorylation because we have to memorize >the many steps responsible for producing ATP. This production of ATP >requires many items including: oxygen, magnesium, ADP, and amino acids.>>Many people with chronic illnesses, such as fibromyalgia, chronic fatigue >syndrome, and autoimmune disorders, complain they have no energy. ATP is >the molecule that stores energy for the body. The body is constantly >producing and utilizing ATP. Its production is a complex process that is >beyond this book. However, there are two cofactors, Vitamins B2 >(riboflavin) and B3 (niacin), that are integral to stimulating oxidative >phosphorylation and ATP production.Â>>Hydrogen peroxide is a byproduct of oxidative phosphorylation. It is >this production of hydrogen peroxide that is so critical to the oxidation >process of iodine. Hydrogen peroxide and TPO help to oxidize iodide to >form iodine.Â>>If there is a deficiency in iodine, which is common when ingesting the RDA >for iodine, there will not be enough substrate (i.e., iodine) to produce >iodinated lipids. As can be seen from Figure 3, the lack of >δ-iodolactone and other iodinated lipids results in a loss of the >‘brake’ in the pathway to oxidize iodide. This may result in a >temporarily production of too much hydrogen peroxide. This excess hydrogen >peroxide can damage the enzyme TPO.>>What Happens If TPO Is Damaged? Autoimmune Thyroid Illness>>The body’s response to TPO damage is to produce antibodies against TPO >or anti-TPO antibodies. A diagnosis of Hashimoto’s disease requires >the presence of anti-TPO antibodies. As the damage worsens, surrounding >proteins can also be damaged such as thyroglobulin. Damaged >thyroglobulin will result in the body producing antibodies against >thyroglobulin—aanti-thyroglobulin antibodies.>>In most cases of Hashimoto’s disease, there are antibodies to both TPO >and thyroglobulin present. Although Graves’ disease may also possess >these same antibodies, antibody production is not necessary to make the >diagnosis of Graves’ disease. However, my clinical experience has >shown that the treatment for both Hashimoto’s and Graves’ disease can >follow a similar course with similar positive outcomes.>>How to Treat Autoimmune Thyroid Disorders>>1.     Ingest enough iodine in order to provide adequate   > substrate to iodinate lipids.>>2. Take Vitamins B2 and B3 in amounts necessary to stimulate the NADPH >system to produce adequate amounts of H2O2. Â>>3. Correct oxidant stress in the thyroid gland and the mitochondria with >antioxidants.Â>>4.      Ensure adequate magnesium levels.Â>>5.       Minimize oxidative stress in the body.Â>> Donna in IL>>Hello,>>I am new to the group and would like to know your advice on something. I >have read both YES and NO that iodine is bad for people with hashimotos, >yet some thyroid supplements contain kelp in them. I recently bought some >supplements called "thyroid now" and trying to decide whether or not to >take these. Can anyone shine some light on this subject?>>thank you>>>>>>

~~~ There is no way to peace; peace is the way ~~~~ --A.J. Muste

Link to comment
Share on other sites

Bruce-- I found that once I had Dr Brownstein's book, everything made a lot

more sense. It is very well organized and written for the layperson, but

at the same time is full of source notes for those who want to pursue the

literature further. He makes things really really clear.

However, your note that you can't tolerate thyroid meds is not relevant,

because the iodine protocol does not involved thyroid meds. It's just that

so many people have thyroid problems in conjunction with thyroid deficiency

that they get talked about a lot together.

Here's the list of companion nutrients that must be taken with iodine:

1. Supporting Nutrients for taking Iodine -

a. Vit C – 2,000 – 5,000 mgs / day

b. Selenium – 200 – 400 mcg / day

c. Unrefined salt (Celtic) – ½ tsp / day

d. Magnesium – 400 mgs / day

e. Optional – ATP CoFactors -1 tablet 2x / day

There are two forms of iodine that we recommend. One is Lugol's liquid,

and one is Iodoral tablets-- both have the same ratio of elemental iodine

and potassium iodide, both of which are required by the body. If you start

with Lugol's, you can increase your dose a drop at a time, very

slowly. The Iodoral tablets can be split-- they are 12.5mg.

Does this help at all?

--

At 09:34 PM 12/10/2010, you wrote:

>

>

>I am not sure how I can help you because that " IS " the beginners

>guide. Try reading Dr. Brownstein's book when you get it and see if it

>makes more sense then.

>

>Steph

>

> RE: Iodine and Hashimotos

>> >

>> >Â

>> >

>> >Hi!

>> >

>> >You need to read the new member doc, but here is the excerpt from it:

>> >

>> >1.      I have hashimotos. Can I take Iodine? (the

>> following is

>> >an excerpt from Dr. Brownstien̢۪s Iodine ne book)

>> >

>> >THE UNDERLYING CAUSE OF AUTOIMMUNE THYROID ILLNESSES: IODINE DEFICIENCY

>> >AND ANTIOXIDANT DEFICIENCY

>> >

>> >Chapter 7 described the apoptotic (i.e., anticancer) effects of the

>> >iodinated form of lactone (δ- Iodolactone). δ- Iodolactone is

>> not only

>> >important for preventing cancer, its production is also necessary to help

>> >regulate the oxidation of iodine.   Figure 3 illustrates this

>> >regulatory step in the oxidation/organification of iodine.Â

>> >

>> >Â Â Â Â Â Â Â As previously mentioned, the oxidation of iodide

>> to iodine

>> >occurs through the interaction of H2O2 and TPO. Iodine is a necessary

>> >product in order to provide the correct molecule in the cell so that

>> >organification can occur. If organification does not take place or is

>> >blocked, thyroid hormone and iodo-lipids will not be formed. As can be

>> >seen from Figure 3, this reaction is controlled by intracellular calcium

>> >levels and iodinated lipids—δ-iodolactone.

>> >

>> >Intraacellular calcium stimulates this pathway. On the other hand,

>> >δ-iodolactone and other iodinated lipids act as a brake on the

>> >system. If there is not enough iodine in the cell to organify and

>> >produce adequate amounts of δ-iodolactone, it can set the stage for

>> >damage to the thyroid cell and the development of an autoimmune thyroid

>> >disorder such as Hashimoto’s or Gravesâ€â¢â‚¬â„¢ disease.

>> >

>> >Â A Proposed Mechanism For The Development Of Autoimmune Thyroid Disorders

>> >

>> >The NADPH oxydase system is found in the mitochondria of our cells. The

>> >mitochondria are the energy-producing cells of our body. The

>> >mitochondria produce energy (i.e., ATP) through a complex process called

>> >oxidative phosphorylation. All medical students (and most physicians)

>> >are familiar with oxidative phosphorylation because we have to memorize

>> >the many steps responsible for producing ATP. This production of ATP

>> >requires many items including: oxygen, magnesium, ADP, and amino acids.

>> >

>> >Many people with chronic illnesses, such as fibromyalgia, chronic fatigue

>> >syndrome, and autoimmune disorders, complain they have no energy. ATP is

>> >the molecule that stores energy for the body. The body is constantly

>> >producing and utilizing ATP. Its production is a complex process that is

>> >beyond this book. However, there are two cofactors, Vitamins B2

>> >(riboflavin) and B3 (niacin), that are integral to stimulating oxidative

>> >phosphorylation and ATP production.Â

>> >

>> >Hydrogen peroxide is a byproduct of oxidative phosphorylation. It is

>> >this production of hydrogen peroxide that is so critical to the oxidation

>> >process of iodine. Hydrogen peroxide and TPO help to oxidize iodide to

>> >form iodine.Â

>> >

>> >If there is a deficiency in iodine, which is common when ingesting the RDA

>> >for iodine, there will not be enough substrate (i.e., iodine) to produce

>> >iodinated lipids. As can be seen from Figure 3, the lack of

>> >δ-iodolactone and other iodinated lipids results in a loss of the

>> >‘brakeÃe’ in the pathway to oxidize iodide. This may result in a

>> >temporarily production of too much hydrogen peroxide. This excess hydrogen

>> >peroxide can damage the enzyme TPO.

>> >

>> >What Happens If TPO Is Damaged? Autoimmune Thyroid Illness

>> >

>> >The body̢۪s responponse to TPO damage is to produce antibodies

>> against TPO

>> >or anti-TPO antibodies. A diagnosis of Hashimoto’s disease requires

>> >the presresence of anti-TPO antibodies. As the damage worsens,

>> surrounding

>> >proteins can also be damaged such as thyroglobulin. Damaged

>> >thyroglobulin will result in the body producing antibodies against

>> >thyroglobulin—aanti-thyroglobulin antibodies.

>> >>In most cases of Hashimoto̢۪s disease, there are antibodiodies to

>> both TPO

>> >and thyroglobulin present. Although Graves’ di disease may also

>> possess

>> >these same antibodies, antibody production is not necessary to make the

>> >diagnosis of Graves’ disease.  However, my clinical experience has

>> >shown that the treatment for both Hashimoto‬™s and Graves’ „¢

>> disease can

>> >follow a similar course with similar positive outcomes.

>> >

>> >How to Treat Autoimmune Thyroid Disorders

>> >

>> >1.     Ingest enough iodine in order to provide adequate  Â

>> >Â substrate to iodinate lipids.

>> >

>> >2. Take Vitamins B2 and B3 in amounts necessary to stimulate the NADPH

>> >system to produce adequate amounts of H2O2. Â

>> >

>> >3. Correct oxidant stress in the thyroid gland and the mitochondria with

>> >antioxidants.Â

>> >

>> >4.      Ensure adequate magnesium levels.Â

>> >

>> >5.       Minimize oxidative stress in the body.Â

>> >

>> >Â Donna in IL

>> >

>> >Hello,

>> >

>> >I am new to the group and would like to know your advice on something. I

>> >have read both YES and NO that iodine is bad for people with hashimotos,

>> >yet some thyroid supplements contain kelp in them. I recently bought some

>> >supplements called " thyroid now " and trying to decide whether or not to

>> >take these. Can anyone shine some light on this subject?

>> >

>> >thank you

>> >

>> >

>> >

>> >

>> >

>> >

>>

>>~~~ There is no way to peace; peace is the way ~~~~

>> --A.J. Muste

>

>

>

>

>

~~~ There is no way to peace; peace is the way ~~~~

--A.J. Muste

Link to comment
Share on other sites

Join the conversation

You are posting as a guest. If you have an account, sign in now to post with your account.
Note: Your post will require moderator approval before it will be visible.

Guest
Reply to this topic...

×   Pasted as rich text.   Paste as plain text instead

  Only 75 emoji are allowed.

×   Your link has been automatically embedded.   Display as a link instead

×   Your previous content has been restored.   Clear editor

×   You cannot paste images directly. Upload or insert images from URL.

Loading...
×
×
  • Create New...