Re: Food for thought... How does Iodine Aggravate Hashimotos?

[Guest guest]

Thanks, Cindi.

I was really wondering what iodine uses you were interested in as

most of your comments lean toward warnings regarding Hashimoto's and

in general. I agree that we should all be careful our

recommendations. I have Hashimoto's and also have trouble with

iodine, though I have not given up yet!

One area where I am confused is the mechanism for iodine to cause

Hashimoto' problems. If you have a tendency for this intolerance,

what happens with the iodine to cause your antibodies to attack your

thyroid? And if your thyroid is atrophied, why does it matter - why

would attacks on an already atrophied gland cause any symptoms?

Take care,

Sharon

> Sharon,

> I agree with just about everything Brownstein has written

> in " iodine " , to include the conditions he mentions...in fact I

> recently did another careful reading of his book and can't recall

at

> the moment anything I disagreed with. I do think lots of folks

have

> taken some of his speculative comments as definitive and forget he

> allows for dosing variation among individuals.

>

> I only mention I'm iodine sensitive as a thread calls out for it to

> be mentioned. I've noted quite a few non-Hashi's folks tend to

push

> iodine on Hashi's folks....and so sometimes it needs to be

mentioned

> that this sensitivity (and worsening health)with additional iodine

> exposure is not an unusual occurrence at all for autoimmune thyroid

> folks. Caution is needed.

>

> So no, for my particular situation (final stage atrophic Hashi's

> gland now on full replacement TH - no hypo symptoms) I would be

> hesitant to supplement iodine unless there were some health issues

> that seemed to warrant it...I didn't get this stable to go mess it

> up, ya know? But I'm certainly interested in how it might play a

> part in early stage Hashi's....and of course I've always wondered

> about those folks who had hypo labs but were not Hashi's...and

> definitely see a major role for iodine in those cases.

> Cindi

>

>

> >

> > >

> > Which specific conditions do you think iodine has a place in

> treating?

> > Have you learned anything you would share with the group?

> >

> > As I recall most of your posts are about not tolerating iodine

and

> of

> > course your Hashimoto's - I assume you are not exploring the

> treatment

> > of Hashimoto's with iodine?

> >

> > Thanks,

> > Sharon

> >

>

[Guest guest]

I'd like to think I don't just post that info as periodic general

warnings (smile), but rather in response to someone who is pushing

50 mg. of Iodoral on me.

Regarding the mechanism you mention...that's something else I have

mentioned numerous times...because it's one thing to say Hashi's

might be caused by iodine deficiency...and another thing if that

deficiency is caused by a genetic defect that doesn't allow us to

properly absorb it or utilize it or even to react to it. so I don't

know if it's so much anything to do with antibodies...but rather the

effect of iodine on that thyroid tissue or elsewhere? I'd love to

have more answers regarding Hashi's and iodine....Brownstein's book

doesn't really address this issue fully (although there is certainly

a lot of other medical literature on it), nor does he make any

comment regarding stage of disease on those hashi's folks he says

are using it. But I keep listening to the Hashi's experiences

here...

cindi

>

>

> I was really wondering what iodine uses you were interested in as

> most of your comments lean toward warnings regarding Hashimoto's

and

> in general. I agree that we should all be careful our

> recommendations. I have Hashimoto's and also have trouble with

> iodine, though I have not given up yet!

>

> One area where I am confused is the mechanism for iodine to cause

> Hashimoto' problems. If you have a tendency for this intolerance,

> what happens with the iodine to cause your antibodies to attack

your

> thyroid? And if your thyroid is atrophied, why does it matter -

why

> would attacks on an already atrophied gland cause any symptoms?

[Guest guest]

Cindi,

> Regarding the mechanism you mention...that's something else I have

> mentioned numerous times...because it's one thing to say Hashi's

> might be caused by iodine deficiency...and another thing if that

> deficiency is caused by a genetic defect that doesn't allow us to

> properly absorb it or utilize it or even to react to it. so I

don't

> know if it's so much anything to do with antibodies...but rather

the

> effect of iodine on that thyroid tissue or elsewhere?

So if I understand you, you're saying that perhaps:

an inability to properly use iodine causes 1) an intolerance to

iodine and 2) an iodine deficiency that then causes Hashimoto's? So

the intolerance to iodine is not caused by Hashimoto's itself but

just correlated with it - as both are caused by the iodine

deficiency?

I think I probably put a few words in your mouth there! Correct me if

I got it wrong!

When I started iodine supplementation, I was slightly hypoT (taking

37.5mcg Synthroid) and had a low but positive level of thyroglobulin

ABs. Six months later I was very HYPERt and had a very high level of

antibodies. So for me, I think the iodine caused an increase in

antibodies, which then caused the hyperT problems. Does this fit with

your thinking? Or do you imagine two different paths for iodine to

aggravate Hashimoto's?

Thanks!

Sharon

[Guest guest]

Hello,

To explain a little about how Hashi's works, please read below:

This is from a doc currently doing research on Hashi's thyroiditis:

Dr. O says- " Hashimoto's thyroiditis, atrophic thyroiditis (the late

course of the disease) and postpartum thyroiditis (an other form which

usually begins after delivery) " ... They are all called " autoimmune

thyroiditis " (AIT).

In fact the triggering mechanisms are similar... Our immune cells (CD4

etc) are educated during the intrauterine life. So they can recognize

their own cells and the enemies (like bacterias, viruses, or abnormal

cells like cancer cells). Now, smoking (active or passive), additives,

pollutants or what ever they are... Many inciting agents change the

faces of our cells. They became abnormal... Your soldiers love you.

Inorder to defend you, they begin an attack to whatever does not seem

familiar. This war is mediated by many immune cells or the mediators

of them (antibodies)... Do you hate your immune cells still? Do not

please. They love you... "

To explain further:

So, what does this mean for us? Our thyroids are under attack by

antibodies that see our thyroids as invaders. The antibodies attack

the thyroid cells, causing inflammation and the release of peroxidase

from those damaged cells. Peroxidase is a substance found within the

cell wall only. When the cell wall is broken, the peroxidase is

released. When we have a TPO antibody test, we are testing for the

presence of Peroxidase antibodies there to clean up the peroxidase

which has been released upon the destruction of our cells. Release of

peroxidase can happen with Graves, Hashi's, or even a wound to the

throat area, so the test is not specific for which you have going on

at the time. It only indicates cell damage. Other more specific

tests can tell you which autoimmune you have going on (or both).

So what does this have to do with iodine? Iodine is the fuel for our

thyroids. When our thyroids are being damaged, they are not working

properly either because of what the different antibodies are doing to

it. There are receptor antibodies and blocking antibodies and each

fits into a different receptor and blocks a function of the thyroid

cell, either turning it on, or off. It's like a car with the gas

pedal stuck. In graves, the gas pedal is stuck on the floor, so the

more gas (iodine) we give, the faster the car is going to run.

Hashi's after destruction of the thyroid by the antibodies is like

having the gas pedal normal but the brake on all the time. We're

giving it the proper amt of gas, but we're not going fast enough.

Early Hashi's thyroiditis (the time before the thyroid burns out and

when it is still hyperactive) is like the pedal to the floor and the

brake to the floor at the same time. If we give more Iodine in such

cases, it creates more unbalance and we go hypER- the car goes faster.

This creates more inflammation because our brain (pituitary) is

telling our thyroid to slow down (brake pedal) but the gas pedal isn't

letting it. With more iodine, the gas pedal is going to win and you

will be hypER. With less iodine, the brake pedal will win and you

will be hypO. The balance is what's important because that keeps the

brain (and body) happy and from trying to re-regulate the thyroid with

TSH causing more disruption, damage and inflammation.

So what about Hashi's without thyroiditis? Some people say that

because the thyroid isn't acting hypER that adding iodine will help

them push the gas pedal a little closer to the normal speed to

counteract the fact that the brake is always on a bit. In some people

this works, usually because in the meantime they go into remission and

the antibodies quit fighting the cells. In others it creates more

inflammation and cell destruction within the gland as it tries in vain

to work harder. It is trying so hard to obey! This results in more

damage, which results in more inflammation and cell destruction and,

well, you can see where it goes from there... Other people say that

because we are pushing the gland to work harder it will burn out

faster, leaving us permanently hypO and without a functioning gland.

For most people this seems to be true, so pushing our thyroids into

exhaustion isn't really something we really want to do either. You

will receive temporary relief, but unless you go into remission, this

relief is only temporary and you will still end up with a burned out

thyroid, just a little faster. Once you achieve remission and your

thyroid is burned out, it no longer matters as there are no longer any

cells capable of doing their jobs and producing T4 and T3 and the

other T's.

So what is remission? Remission is when suddenly our bodies recognize

that thyroid tissue as our own and stop pumping out antibodies to

attack it. It is a " CeaseFire " . The cause of the disease

(autoimmune) has reversed itself and the inflammation and cell

destruction ceases and our thyroids are now able to drive themselves

(based on TSH) and stay at the correct level for our own personal

bodies. Remission therefore, is our goal. Most people can achieve

remission if given the right circumstances and enough time. In Graves

this is probably easier, as we can give Anti Thyroid Medication or

ATD's to slow or halt the action of our thyroids (stops the conversion

of iodine to thyroid hormone in the cell) and give the gland a rest to

try to heal itself. In the meantime, we might try to really give it a

good rest by blocking the action of iodine completely and replacing

the thyroid hormone artificially for a time. This is called B & R or

Block and Replace. Most statistics show that the majority (I think

the number is around 80%) of people will achieve remission in about 4

yrs by giving their glands a rest with ATD's. Sometimes it takes much

longer than this, but remission usually can happen if given the

chance. Most docs don't give patients this option. RAI is much easier

for them. Their malpractice insurance is based on how " safe a risk "

their patient base is. If you have a lot of high risk patients, you

pay a lot more in malpractice insurance. Those undergoing a treatment

that requires close monitoring such as ATD's are not as good a risk as

someone who has had the " problem gland " (NOT!) removed from the

equation and the patient placated with an " easily tolerated tablet " .

Also, I'm sure the pressure of the drug companies and the kickbacks

play a large part in these physician choices as well.

Some people feel that B & R with early Hashi's can help them achieve

remission as well, and indeed many people with thyroiditis are treated

as if they have Graves because they are in the hypER stage and the

docs never take the time to do the correct antibody testing to find

out which disease the patient really has as the treatment will be the

same. Many docs feel that there is no difference between Graves and

Hashi's- that they are the same disease. When these patients go into

remission it will be impossible to tell (lack of antibodies) which

disease they had before treatment. We only know that they go into

remission. The jury is still out on this one apparently. The best

goal for anyone with thyroid autoimmune issues is not to provoke the

antibodies. This means keeping our thyroids at a very stable, normal

(for our body at our own personal set-point) level in order not to

create more inflammation and the body's perceived need for more

antibodies to play " clean-up.

Whew! There really is a lot more to it than that. That is the

simplified version, but I hope it's understandable. Those of you who

have read my personal posts know that I am a Graves patient (diagnosed

without proper antibody testing). Iodine is definitely

contraindicated in my hypER disease. However, my daughter is

suffering from Hashi's- at least we know that she has the antibodies

for Hashi's. I can't get the docs to test to see if she has Graves

antibodies as well (both blocking and receptor antibodies). Her

levels are not stable. There are some docs who think that low iodine

can cause inflammation resulting in the production of antibodies and

autoimmune Hashi's. This is not to say that simply giving a

supplement will eliminate the problem- the antibodies are now a part

of our bodies and any fluctuations will cause inflammation which will

cause problems. Perhaps in some people it may help them to stabilize,

or even if caught very early on, to eliminate the antibodies by

eliminating the inflammation caused because of the lack of I that

starts the whole process. I know I am here because I want to learn

all that there is to learn about this entire process from start to

finish. There is too much we just don't understand about the human

body and how it works. Autoimmune tendencies have been shown to be

genetic, as it is in my own family. Why? I can't answer that, but

maybe someday someone can. This means that what I learn today may not

help me. It may not help my daughter. But what about the daughter

she may someday have? Or her daughter??? What about my friend who is

showing abnormalities in her bloodwork today? I am here to learn for

them as well. I cannot go back with my own disease, but I can make it

my mission to make sure that no one else is mis-informed or misled by

docs thinking that they are receiving proper treatment when they are

not, ending up in catastrophe when a vital gland is destroyed either

by mistreatment or intentionally with surgery or RAI.

E (Ellen in Missouri)

> > >

> > > >

> > > Which specific conditions do you think iodine has a place in

> > treating?

> > > Have you learned anything you would share with the group?

> > >

> > > As I recall most of your posts are about not tolerating iodine

> and

> > of

> > > course your Hashimoto's - I assume you are not exploring the

> > treatment

> > > of Hashimoto's with iodine?

> > >

> > > Thanks,

> > > Sharon

> > >

> >

>

[Guest guest]

that may have been what i said.

let's say it another way...obviously in Hashi's, the thyroid gland

starts to malfunction, often because of a hormonal change like

childbirth or enviromental trigger like mercury. and I also think

it's important the remember the disease process (damage to the

thyroid gland) can be there even without antibodies according to

Werner & Ingbar's The Thyroid...and I believe that as every Hashi's

person does Not have TPO or Antithyroglobulin antibodies. this is

one reason why the " autoimmune " aspect of it sometimes confuses

me...as I can clearly see in my family that this is a genetic

disease that has been passed down through each generation...just

like any other genetic disease. I also think there may be other

genetic defects in the HPT axis associated with the whole Hashi's

thing.

Now..does Hashi's the disease include a genetic component where we

don't absorb iodine properly and this is why our thyroid gland

starts going haywire? which makes ya think about the hashi's thing

of hypo to hyper to hypo cycling. what is that about? does it have

anything to do with iodine?

i guess bottom line is...I don't know...all I really know is that

there is a vast amount of medical literature that clearly indicates

that autoimmune thyroid disease can be aggravated by iodine...just a

your own experience indicates...and my experience too.

So I guess I just need to hear a bit more from the iodine docs about

autoimmune thyroid diseae and iodine....and exactly what iodine

supplementation does for that condition. It apparently doesn't

dramatically impact oral thyroid hormone supplementation and it

doesn't cure the condition...so I'm assuming that perhaps in early

stage Hashi's the expectation might be that is slows the disease

process? but is that true if it increases antibodies?

I can understand the general benefits of iodine to the body with no

problem...and for its use in cases of hypo not causes by

Hashi's...but when a person has a condition known to be aggravated

by iodine...this is where I have to keep my guard up...and feel I

need a bit more information. And so I keep listening for some

answers...

cindi

>

> Cindi,

>

> > Regarding the mechanism you mention...that's something else I

have

> > mentioned numerous times...because it's one thing to say Hashi's

> > might be caused by iodine deficiency...and another thing if that

> > deficiency is caused by a genetic defect that doesn't allow us

to

> > properly absorb it or utilize it or even to react to it. so I

> don't

> > know if it's so much anything to do with antibodies...but rather

> the

> > effect of iodine on that thyroid tissue or elsewhere?

>

> So if I understand you, you're saying that perhaps:

> an inability to properly use iodine causes 1) an intolerance to

> iodine and 2) an iodine deficiency that then causes Hashimoto's?

So

> the intolerance to iodine is not caused by Hashimoto's itself but

> just correlated with it - as both are caused by the iodine

> deficiency?

>

> I think I probably put a few words in your mouth there! Correct me

if

> I got it wrong!

>

> When I started iodine supplementation, I was slightly hypoT

(taking

> 37.5mcg Synthroid) and had a low but positive level of

thyroglobulin

> ABs. Six months later I was very HYPERt and had a very high level

of

> antibodies. So for me, I think the iodine caused an increase in

> antibodies, which then caused the hyperT problems. Does this fit

with

> your thinking? Or do you imagine two different paths for iodine to

> aggravate Hashimoto's?

[Guest guest]

What are your sources for the the stages of Hashimoto's? Could you post

please?

--- cindi22595 <cindi22595@...> wrote:

> I'd like to think I don't just post that info as periodic general

> warnings (smile), but rather in response to someone who is pushing

> 50 mg. of Iodoral on me.

>

> Regarding the mechanism you mention...that's something else I have

> mentioned numerous times...because it's one thing to say Hashi's

> might be caused by iodine deficiency...and another thing if that

> deficiency is caused by a genetic defect that doesn't allow us to

> properly absorb it or utilize it or even to react to it. so I don't

> know if it's so much anything to do with antibodies...but rather the

> effect of iodine on that thyroid tissue or elsewhere? I'd love to

> have more answers regarding Hashi's and iodine....Brownstein's book

> doesn't really address this issue fully (although there is certainly

> a lot of other medical literature on it), nor does he make any

> comment regarding stage of disease on those hashi's folks he says

> are using it. But I keep listening to the Hashi's experiences

> here...

> cindi

>

>

>

>

> >

> >

> > I was really wondering what iodine uses you were interested in as

> > most of your comments lean toward warnings regarding Hashimoto's

> and

> > in general. I agree that we should all be careful our

> > recommendations. I have Hashimoto's and also have trouble with

> > iodine, though I have not given up yet!

> >

> > One area where I am confused is the mechanism for iodine to cause

> > Hashimoto' problems. If you have a tendency for this intolerance,

> > what happens with the iodine to cause your antibodies to attack

> your

> > thyroid? And if your thyroid is atrophied, why does it matter -

> why

> > would attacks on an already atrophied gland cause any symptoms?

>

>

__________________________________________________

[Guest guest]

This could only be determined (imo) by looking at the individual's

history combined with their clinical presentation combined with

their labs. Even antibody counts can be useful if one knows they can

decline in late stage Hashi's. But there is no one marker for

determining " stage " .

So for example, a person who presents in myxedematous cachexia with

a history of 15-25 years of progressing hypo symptoms whose Free

levels are below range with atrophied thyroid gland and antibodies

of say 200 can pretty well be determined to be someone whose Hashi's

has run its course. Compare that with a 23 year old who has a bout

of postpartum thyroiditis after childbirth...which may or may not be

the onset of Hashi's....time will tell...they should be monitored.

and then there can be the 35 year old with TSH of 1.5 and Frees

still at mid range and antibodies of 1000 and swollen thyroid

gland....this is someone in active throes of Hashi's, but not late

stage. Just some examples...the history is probably more important

than anything else imo...as Hashi's can fluctuate.

There might be some useful info here:

http://www.thyroidmanager.org/chapter9/ch_9_3.htm

Cindi

>

> What are your sources for the the stages of Hashimoto's? Could

you post

> please?

[Guest guest]

Are there certain 'set' stages of hashis and are you

saying that from researching, you can identify at what

stage a person can take iodine and benefit and/or take

iodine and get worse?

I'm not quite understanding your example, either. In

your first-case scenerio, you seem to indicate a

person that has had hashis for 15-20 years with

antibodies at 200 is pretty much 'done.' Is that

'done' from taking iodine? Or the person is beyond

hope in taking armour?

What are you basing this on?

--- cindi22595 <cindi22595@...> wrote:

> This could only be determined (imo) by looking at

> the individual's

> history combined with their clinical presentation

> combined with

> their labs. Even antibody counts can be useful if

> one knows they can

> decline in late stage Hashi's. But there is no one

> marker for

> determining " stage " .

>

> So for example, a person who presents in

> myxedematous cachexia with

> a history of 15-25 years of progressing hypo

> symptoms whose Free

> levels are below range with atrophied thyroid gland

> and antibodies

> of say 200 can pretty well be determined to be

> someone whose Hashi's

> has run its course. Compare that with a 23 year old

> who has a bout

> of postpartum thyroiditis after childbirth...which

> may or may not be

> the onset of Hashi's....time will tell...they should

> be monitored.

> and then there can be the 35 year old with TSH of

> 1.5 and Frees

> still at mid range and antibodies of 1000 and

> swollen thyroid

> gland....this is someone in active throes of

> Hashi's, but not late

> stage. Just some examples...the history is probably

> more important

> than anything else imo...as Hashi's can fluctuate.

>

>

> There might be some useful info here:

> http://www.thyroidmanager.org/chapter9/ch_9_3.htm

>

> Cindi

>

>

>

>

> >

> > What are your sources for the the stages of

> Hashimoto's? Could

> you post

> > please?

>

>

________________________________________________________________________________\

____

Any questions? Get answers on any topic at www.Answers.. Try it now.

[Guest guest]

Regis <vegan_mamma@...> wrote:

> Are there certain 'set' stages of hashis and are you

> saying that from researching, you can identify at what

> stage a person can take iodine and benefit and/or take

> iodine and get worse?

I don't think there are any general rules. I've had Hashi

for at least 16 yrs now, possibly several years longer, and

my antibodies at the time of hashi crisis early on were only

just outside of normal range, yet with severe symptoms.

Iodine and selenium tend to greatly aggravate and inflame

my thyroid and disrupt stability on Armour. This reaction to

iodine and selenium has not changed over 16 yrs. Every so

often I test it again, but same result. My point here is

that antibody numbers may not correlate to severity of

autoimmune thyroid disease, and it's unpredictable how

different thyroid patients will react over time to various

amounts of iodine or selenium. Everyone has to find his

own limits. It would be nice if there were rules and it was

all very tidy, but that doesn't seem to be the case.

Carol

[Guest guest]

no, I'm not saying a thing about when or whether a Hashi's person

can take iodine. I was just answering a question someone asked

about determining " stage " of Hashi's....which I think has to be

looked at on an individual basis with consideration of several

factors. As to your question, " done " would just mean the natural

progression of the disease has run its course (see the link that was

provided regarding hypothyroidism progression into myxedema

cachexia)....hopefully most folks would receive thyroid hormone

replacement before they actually die of hypothyroidism.

cindi

>

> Are there certain 'set' stages of hashis and are you

> saying that from researching, you can identify at what

> stage a person can take iodine and benefit and/or take

> iodine and get worse?

>

> I'm not quite understanding your example, either. In

> your first-case scenerio, you seem to indicate a

> person that has had hashis for 15-20 years with

> antibodies at 200 is pretty much 'done.' Is that

> 'done' from taking iodine? Or the person is beyond

> hope in taking armour?

> What are you basing this on?

[Guest guest]

I so totally agree with this statement.

I've seen young folks with antibodies in the thousands with hardly any

hypo symptoms and those with antibodies of 150 who were about dead from

untreated hypo.

>

> My point here is

> that antibody numbers may not correlate to severity of

> autoimmune thyroid disease, and it's unpredictable how

> different thyroid patients will react over time to various

> amounts of iodine or selenium. Everyone has to find his

> own limits. It would be nice if there were rules and it was

> all very tidy, but that doesn't seem to be the case.

[Guest guest]

"I've seen young folks with antibodies in the thousands with hardly any hypo symptoms "

Does the type of antibodies make a difference? TPOAB and TgAB?

With TgAB over 3000, and TPOAB is just 12, is there anything I need to concern?

I do take Armour .5 grain daily. And, I used to take Iodoral 1 tablet daily, now stopped.

mt

[Guest guest]

Hashi's folks can have one - or both - of the antibodies used to

diagnose it. The TPO antibodies are slightly more common.

My concern with antibodies of 3000 would that the person feels

miserable with hypo symptoms....as the thyroid gland is " under

attack " so to speak.

I'd be looking at Free levels and wanting to get Free levels at an

optimal level.

Suppressive TSH therapy was once listed in Werner & Ingbar's, The

Thyroid...as a treatment for Hashi's. It sure has helped me. My

TSH is is non-detectable and I take 4 grains of Armour....and Free

levels are near the top of the range for Free T4 and slightly over

for Free T3. I have no hypo symptoms and I'd describe my Hashi's as

under control and stable and being managed by oral thyroid hormone

replacement.

cindi

>

> " I've seen young folks with antibodies in the thousands with

hardly any

> hypo symptoms "

>

> Does the type of antibodies make a difference? TPOAB and TgAB?

>

> With TgAB over 3000, and TPOAB is just 12, is there anything I

need to concern?

>

> I do take Armour .5 grain daily. And, I used to take Iodoral 1

tablet daily, now stopped.

[Guest guest]

I should clarify my young folks statement...the young folks I've seen

with high antibodies have had severe psychological/psychiatric symptoms

of Hashi's...but not the more common hypo physical symptoms like weight

gain. Hashi's is notorious for sometimes presenting with mental

symptoms.

One problem is that there is no good way to determine if a person just

has " brain hypothyroidism " (TSH is useless imo)...other than

symptoms....and perhaps knowing they have thyroid antibodies.

cindi

-- In iodine , gs1 <grapeseed1@...> wrote:

>

> " I've seen young folks with antibodies in the thousands with hardly

any

> hypo symptoms "

[Guest guest]

Yes, those antibodies are confusing. I think that must be why a lot

of docs consider the two diseases to be the same, just with different

receptors. Those Frees are the key to it all. TSH can sometimes be

suppressed with blocking antibodies present, giving you a false

reading. A lot of docs still treat based on TSH alone. Too bad for

their patients.

I hope you keep feeling good. Keeping your frees high will keep your

thyroid from having to work at all and that's a good thing!!

Congrats! Whatever you're doing is working! I pray you will be one

of the lucky ones and end up in remission...

Ellen

> >

> > " I've seen young folks with antibodies in the thousands with

> hardly any

> > hypo symptoms "

> >

> > Does the type of antibodies make a difference? TPOAB and TgAB?

> >

> > With TgAB over 3000, and TPOAB is just 12, is there anything I

> need to concern?

> >

> > I do take Armour .5 grain daily. And, I used to take Iodoral 1

> tablet daily, now stopped.

>

[Guest guest]

This can be quite common in the early stages of the disease. Cindi is

right tho- it can often present itself in very odd ways including

neurological problems. It's called Hashimoto's Encephalopathy. It

happens in Graves also and is called Graves encephalopathy.

E (Ellen in Missouri)

> > " I've seen young folks with antibodies in the thousands with hardly

> any

> > hypo symptoms "

>