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Thank you, . I have been waiting for someone to explain that. I

didn't know they could do the split array until someone ,mentioned it here.

Good to know theres a choice for the surgeon if he needs it.

Betty

N22

Split array

>

> Can anyone describe the split array?

> Is it much the same type as the old N22 was made?

>

> Thanks

> Betty

> N22-Esprit 22

>

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Hi and Betty

Yes the N22 is a straight regular array.If you have heard my story you will

remember that I had chosen the Clarion but my Dr. was unable to insert the

array ,due to my ossification.He was a wise Dr. and also had the N22 in the

OR..he tried that and he got full insertion and I got wonderful hearing!!!!Then

,3 years later,after the n24C was FDA approved he again got full insertion and

because of the nerve hugging array i have even better hearing,

Our friend is in surgery today..she has ossification so her Dr. will

first try the regular array and if he is unsuccesful in inserting it he will go

with the split array.

Love Dora

Split array

Can anyone describe the split array?

Is it much the same type as the old N22 was made?

Thanks

Betty

N22-Esprit 22

________________________________________________________________

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  • 1 year later...

Our Dr. (Walter Belenky) at Children's Hospital in Detroit said he

is going to contact Cochlear about Peyton. He said he might be able

to use traditional implant in right ear but won't know till he

actually sees it. Dr. B wanted to also talk with another physician

because Peyton's entire hearing loss has baffled many. He has always

been tested at 20-25 db aided, even with an 85-90 db loss. His

audiograms show a flat loss to 4000. His speech never improved much

since initial diagnosis four years ago. We had extensive AVT for

almost 2 years. We have also had him in preschool for 2 years before

kindergarten. He was lucky to be born into a large, loud family who

supported him in every way. He is the youngest of 4. There seems to

be no reason that anyone can find as to why his speech didn't

improve. The possibility of AN has been discussed along with another

processing disorder. His hearing has dropped by 15 db in the last

year. I'm assuming because of the ossification. It wasn't there when

he had initial CT scan 4 years ago. He also never did have

meningitis. We have no family history of hearing loss and Peyton

wasn't detected till almost 2 years old. His initial dx was SNHL. We

also have seen him respond to questions and sounds unaided. The Dr's

of course thought I was insane. I have spoke to him from across the

room (normal speaking) and he has answered me. There is no way he

should have been able to hear me unaided. This has happened many

times with his back turned to me also. This si one of the resons AN

was suspected. I do wonder how can he hear with the ossification? If

his left ear is fully closed how is he able to hear me with only

that aid in? Everytime I think we have the answers to his hearing

loss something will change. As far as a bi-lat. implant goes, I

truly am not comfortable with the idea yet. The idea of getting one

has me so stressed out that I can't even imagine going for 2. The

Dr. did explain that after speaking with some collegues he would

have more answers for me as far as removing ossification. As for now

we are going with the assumption that the right ear will be

implanted and have the dbl array available if it is needed. I also

did read that sometimes it can be caused for chronic ear infections.

Peyton has had many since he started wearing HA's. He has had tubes

put in, adenoids removed and had tubes replaced. He also had a pic-

line for 3 weeks after last set of tubes. Now we dont treat ear

infections at all. They get better by themselves with or without

antibiotics in about 10 days. Since he never has pain or fever this

seemed the best way to go. His ENT was afraid of him having a

resistence down the road. Wow, sorry for the novel, LOL.

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,

It sounds as if you have had a very stressful journey with Peyton already. I'm

glad that Dr Belenky is planning to seek the opinion of another doctor for him

because that should help him narrow things down. Indeed, frequent ear

infections can cause scar tissue to form and that is one of my issues. I had

very frequent ear infections as a child, many of which went untreated and others

were. Do you know what kind of antibiotics were given to treat his ear

infections? There is a list of medications that can cause hearing loss called

otoxic drugs. It's a long list but I'll add it at the end of this message.

Since Peyton has had a pic-line, it sounds as if his body couldn't fight off

infections and perhaps he ran high temps. Has he ever seen a pediatric

neurologist because many times a neurologist can find a cause for hearing loss?

I'm sorry that you all have been thru so much. He's still a little boy and your

description of his ability at times to hear makes me feel that his hearing loss

fluctuates but of course he probably can't tell you that since he's so young.

The double array does sound like the one that would be most suitable for him and

is successful for those who need it. It will be interesting to follow his

journey to sound and your courage as a parent is obvious. I'm glad you found

the list and hope you'll keep posting as you have more information. I wish you

all great success. We are here to provide support and as much information as we

can but your medical team is your ultimate source. Alice

Here's the list of otoxic drugs:

Drugs that can cause hearing loss

Salicylates

* aspirin and aspirin-

containing products

* salicylates and methyl

salicylates (linaments)

(Toxic effects appear to be dose related and are almost always reversible once

medications are discontinued.)

Non-Steroidal Anti-Inflammatory

Drugs (NSAIDS)

(Most NSAIDS have the potential for causing hearing loss and/or tinnitus.

Because new drugs are being frequently approved for use it is important that you

check with your doctor or pharmacist to determine jf the drug you were

prescribed can cause a problem.)

* diclofenac (Voltaren)

* etocolac (Lodine)

* ibuprofen (Nalfon)

* ibuprofen (Motrin, Advil, Nuprin, etc.)

* indomethacin (Indocin)

* naproxen (Naprosyn, Anaprox Aleve)

* piroxicam (Feldene)

* sulindac (Clinoril)

(Toxic effects are dose related and are almost always reversible once

medications are discontinued)

Antibiotics

* aminoglycosides

* amikacin (Amakin)

* gentamycin (Garamycin)

* kanamycin (Kantrex)

* neomycin (Found in many over-the-counter antibiotic ointments)

* netilmicin (Netromycin)

* streptomycin

* tobramycin (Nebcin)

(Of particular interest is that topical ear drop medications containing

gentamycin or

neomycin do not appear to be ototoxic in humans unless the tympanic membrane

(ear drum ) is perforated. When a solution of an aminoglycoside antibiotic is

used on the skin together with an aminoglycoside antibiotic used intravenously

there is a risk of an increase of the ototoxic effect especially if the

solution is used on a wound that is open or raw or the patient has underlying

kidney damage. Neomycin is the drug that is most toxic to the structure

involved in hearing, the cochlea so it is recommended for topical use only. But

even topical therapy has resulted in hearing loss when large areas were treated

which allowed for large amounts of the drug to be absorbed into the body.

Hearing loss caused by this class of antibiotics is usually permanent.)

* erythromycin

* (EES)

* (E-mycin)

* (Ilosone)

* (Eryc)

* (Pediazole)

* (Biaxin)

* (Zithromax)

(Usually ototoxic when given in

intravenous doses of 2-4 grams per 24 hours especially if there is underlying

kidney failure)

* vancomycin (Vancocin)

(Similar to aminoglycosides in that it may be ototoxic when used intravenously

in life-threatening infections. The fact that

aminoglycosides and vancomycin are often used together intravenously when

treating

life-threatening infections further exaggerates the problem)

* minocycline (Minocin)

(Similar to erythromycin)

* polymixin B & amphotericin B

(Antifungal preparations)

* capreomycin (Capestat)

(Anti-tuberculosis medication)

Diuretics

* bendroflumethazide (Corzide)

* bumetanide (Bumex)

* chlor-thalidone (Tenoretic)

* ethacrynic acid (Edecrin)

* furosemide (Lasix)

(These are usually ototoxic when given intravenously for acute kidney failure

acute hypertensive crisis or acute pulmonaty edema/congestive heart failure.

Rare cases of ototoxicity have been found when these medications are taken

orally in high doses by people with chronic

kidney disease)

Chemotherapeutic Agents

* bleomycine (Blenoxane)

* bromocriptine (Parlodel)

* carboplatinum (Carboplatin)

* cisplatin (Platinol)

* methotrexate (Rheumatrex)

* nitrogen mustard (Mustargen)

* vinblastin (Velban)

* vincristine (Oncovin)

(The ototoxic effects can be minimized by carefully monitoring blood levels)

Quinine

* chioroquine phosphate (Aralen)

* quinacrine hydrochloride

(Atabrine)

* quinine sulfate (Quinam)

(The ototoxic effects are very similar to those of aspirin)

Mucosal Protectant

* misoprostol (Cytotec)

Narcotic Analgesics

* hydrocodone (Lorcet, Vicodin)

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