Fw: Life Force Pyramid

November 10, 2004

[ ] Life Force Pyramid

>

> >

> > I have researched the Life Force organization (makers of Body Balance)

> tonight and it is nothing more than a typical pyramid scam quite like

> Sunrider Foods and many others. They have conventions, newsletters, chummy

> mentors, and all sorts of levels for their " independent " sales agents

(even

> a " Diamond Club " )... As for Juli Ferrante she needs to get her stories

> straight of the twenty different web sites I found for people selling this

> product they all list their calcium source as carbonate - additionally

this

> woman is all over the internet posting on different health boards for

> different products.. she claims to have fixes for everything from acid

> reflux to diabetes - she tells the same stories on different boards but

> changes the persons name. If you go to Amazon.com you can read her

> recommendation for a book she needed to help her learn enough about

science

> to get her through the entrance exam to study to be an RN. I would hardly

> consider myself an expert on vitamins or supplements but I do read what

the

> experts say. I think the whole of Ms. Ferrante's knowledge could easily be

> inscribed onto the head of a pin. It's simply laughable for her to call

> Marks scientific knowledge into question - Mark lectures to Doctors and

> other scientists on these subjects while Juli crams off the medical

> equivalent of Cliff Notes.

> >

> > As for her accusation that I am speaking out against her poison in order

> to sell my own products- she is talking out of her hat. I have never

> endorsed or sold anyone a vitamin or supplement in my life. In fact, I am

> deeply concerned about the unscrupulous carryings on of people like her

> because I happen to be in deep debt from throwing money down the sink hole

> of quackery that folks like Juli Ferrante are hoping to cash in on through

> parents like myself trying to find a way to stop my sons seizures. I have

> been fortunate in getting into a legitimate experimental program at s

> Hopkins University which has managed to completely stop my sons seizures

> without any medications.

> >

> > My reason for speaking out is not to gain money or notoriety for myself

> but to help other parents help their children without doing damage in the

> process.

> >

> > I wish only good things for Don and his son .

> >

November 10, 2004

RE: [ ] Life Force Pyramid

> For this guy's lack of information, I am in school getting my RN - hence

my

> reading the entrance exam book. What an idiot. He sure makes a lot of

> assumptions about someone he doesn't know.

>

> Who is this idiot? The stuff works, works for you, works for me, works

for

> lots and lots of people, so whats it matter this guy's PERSONAL opinion?

>

> Im sorry,. I learned a long time ago not to argue with idiots like this.

> There is NO winning. And who cares anyway. He will never know how good

it

> works because he will never try it, he will just be a TALKING HEAD till

the

> end of time. There is no hope with guy, and who cares anyway? We are

not

> looking to convince everyone that the products work, we're just looking

for

> the ones who want help and health.

>

> And this guy obviously have NO LIFE if he spends all his time arguing with

> people on the internet about products the has no intention of ever trying.

>

> Oh - I included the article about calcium OROTATE below!

>

> Get YUCKY people out of your life. Stop bothering with this creep.

> Really - your wasting your time. Find the people who WANT to get better,

> not those think-they-know-it-alls who just bitch all the time.

>

> ..juli <><...

>

> ****************

> Thanks for ordering this German study on the benefits of calcium orotate.

I

> think you will find it fascinating. The study bears out what we find in

the

> field with Life Force customers and members who consistently take

> OsteoProCare.

>

> We are one of the few companies in the US that uses calcium

> and oritic acid (calcium orotate) in a calcium supplement.

> It is the most expensive way to go which is why so many companies fall

back

> to calcium citrate or calcium carbonate. When you combine our use of this

> amazing ingredient (in liquid form no less) with other high quality

> ingredients like

> magnesium, glucosamine and chondroitin, it sets OsteoProCare

> apart from anything else currently in the marketplace.

>

> The study is fairly long and technical. It was translated from German, so

> the writing is choppy at best. But even a quick read through this data

will

> reveal to you the amazing properties of calcium orotate, especially as

> compared to the other top calcium supplements on the market. There are

some

> very specific and measurable reasons why orotate is the superior carrier

for

> calcium, this study lays out that fact in convincing fashion.

>

> Take your time and work your way through the whole study, it

> is definitely worth the effort.

>

> ***************************************************************

>

> Dr. H. A. Nieper, M.D.Paracelsus KlinikHannover,

> Germany

>

> Translated from " Zeitschrift fur praklinische

> GERIATRIE " Vol. 3, 4/73

>

> THE CLINICAL EFFECT OF CALCIUM OROTATE [1] ON

> CARTILAGE TISSUE

>

> [1 also known as calcium diorotate]

>

> A specific function in relation to pentose metabolism

> of bradytrophic tissue?

>

> We are presenting here an orientation into the

> clinical effect of calcium orotate. Calcium orotate is absolutely free of

> any side effects and in this respect, it is far superior to all the

> conventional calcium salts now being used. Because calcium orotate can

> penetrate the complex cell membranes, it can compensate for a disturbed

> calcium transport through these cell membranes. In addition, calcium

orotate

> has a special affinity for bradytrophic tissue - cartilage, for example -

> where it is metabolized. Parallel studies have shown that a defective

> calcium transport through the cell membrane is of great pathogenetical

> significance.

>

> Previous experience, up to now:

>

> We have employed calcium orotate extensively since

> 1968, hospitalized and ambulatory, to treat

> decalcification conditions and even in many cases, immunological diseases.

> The results accomplished here are in complete accord with what we

discovered

> in 1959 concerning the transmembrane transport complex.

>

> Calcium is first released as an ion, on the cytoplasma

> membrane level, because orotic acid is chiefly

> metabolized there, not in the outer cell membrane. The

> clinical results that we have seen up to now are very encouraging, with a

> minimum of side effects completely unparalleled with anything that we have

> seen in the entire field of calcium supplementation.

>

> And what is more, in suitable cases, this substance is

> a very satisfactory agent in the recalcification of

> metastatic defects in the skeletal system (1), (2). I

> reported this at the 1970 cancer congress in Houston.

> The results of calcium orotate therapy with juvenile decalcification and

> with osteoporosis of the aged, are completely satisfactory for the first

> time, especially in view of the absence of side effects. This finding was

> contrary to all my previous experience with recalcification therapy.

>

> The very remarkable results which we have achieved in

> hip joint plastic surgery, (3) are due to a hardening

> of the bone by preliminary treatment with calcium

> orotate. Previously, in this periodical, I reported

> the recalcification of bone metastases. Illustration

> 1. show another patient (f), in which a severe defect

> of the acetabular roof was so improved (recalcified),

> that hopeless immobilization was replaced by fully

> normal, pain-free locomotive ability. This was

> accomplished after about 2.5g calcium orotate tgl2 for

> 10 weeks. A long period of therapy with a conventional

> calcium seltzer, had been carried out previously, with

> no effect.

>

> 2tgl=daily (from the German " taglich " )

>

> Calcium Orotate and Liver:

>

> A several year search through many long term

> treatments with calcium orotate for side effects was

> absolutely negative. On the contrary, a whole series

> of positive observations came to light, which may be

> connected with the unique transmembrane calcium

> transport. We should mention here, that there is still

> quite a bit of clinical interest here. I will discuss

> this before I dwell on the main theme of the skeletal

> effect of calcium orotate.

>

> One noteworthy observation is that patients with

> chronic cholangitis and light cases of chronic

> hepatitis - and also in severe aggressive cases - show

> a considerable improvement in their ailments, their

> state of health, their gall function, and to some

> extent, their biochemical and biological findings.

> This is especially true under long term therapy. It

> appears to be associated with an anti-inflammatory

> effect on the mesenchymal stroma system in the liver.

> The carrier molecule - orotic acid - has an especially

> high affinity for mesenchymal tissue (4). Free orotic

> acid, magnesium orotate, or other calcium salts do not

> show this effect on the liver. We should refer here to

> the research of Deborah Doniach (10) on all

> immunological liver disease - with and without

> cirrhosis - concerning antimitochondrial (not

> anticellular) antibodies, as sickness motivation.

>

> Calcium orotate liberates its calcium ion - this the

> classic anti-inflammatory principle - at the level of

> the mitochondrial membrane. This explains the anti- inflammatory effect in

> the liver.

>

> Four patients who had taken, on the average, 3g of

> calcium orotate in stomach acid resistant capsules per

> day, for more than a year, submitted to a liver

> puncture. (For reasons which had nothing to do with

> the application of calcium orotate.) In all cases

> there was no sign of fatty liver development. Quite

> the contrary, three of the patients with stage 3 fatty

> liver showed a remarkable improvement under long term

> calcium orotate therapy.

>

> The first patient was a teamster - very heavy

> consumption of alcohol. His condition had been a

> constant stage III for six years. After one year of

> therapy - tgl 2g calcium orotate - not only were the

> complaints lessened, but a liver biopsy showed nothing

> but a normal amount of fat accumulation.

>

> Another patient - U.S. American (f) - came to us for

> treatment for a severe case of fatty liver. In just

> ten days, after tgl 6g calcium orotate therapy, the

> complaint was improved and also a mild case of

> jaundice was cleared up, which had been with her for

> three years, following an influenza attack. After

> three weeks, a biopsy showed a dramatic improvement.

>

> Every possible liver therapy had been tried earlier,

> both in the United States, and here in Germany, with

> no success whatsoever. A third case of fatty liver

> III, showed a similar improvement after

> hyperalimentation.

>

> It is absolutely necessary that we conduct a very

> thorough investigation on this effect of calcium

> orotate on fatty liver. We must remember that the

> lipase enzymes which are necessary for the

> mobilization of stored fat, are activated by Ca++. I

> might assume, in this connection, that the primary

> source of fatty liver is a defective calcium transit

> in the liver cell. A magnesium washout caused by

> chronic alcoholism, in the liver cell membrane, could

> explain this. Mg++ is necessary in the cell membrane

> for Ca-transit into the cell. (6a) Anything which

> disturbs or injures the cell membrane function can

> affect calcium transit through the cell membrane. So

> there are possibly some other significant

> considerations, such as essential hypertonia or the

> ingestion of detergents which stick to kitchen

> utensils after washing. Also a fare too rich in

> carbohydrates puts a strain on the P-pools and

> likewise interrupts the Ca-transport through the

> membranes. Free orotic acid has no effect on the fatty

> liver condition. Likewise the same is true for

> magnesium orotate.

>

> Effect on Heart and Circulation

>

> Another interesting observation is of a moderate, to

> often marked, dropping of the blood pressure with

> fixed hypertonia. This treatment will elevate the

> lowered blood pressure both in the chronic, as well as

> the light form of renal hypertonia. Normal blood

> pressure is not affected. What is especially

> convincing is the disappearance of angina pains,

> especially with a hypertrophied heart. In addition,

> the improvement is remarkably good with infarct

> anamnesis with hypertonia and a distinct sclerosis of

> the heart. Digitalis or Strophanthin tolerance appears

> to be better. With a dosage of 10g calcium diorotate

> tgl., no incompatibility of Digitalis can be observed,

> in the EKG, subjectively, or otherwise. Probably

> because no membraneous calcium ions are created. In

> this connection, I must point out that some time ago,

> Kaufmann and coworkers (Kaufmann and Mitarbeit) (11), researched the

problem

> of defective or insufficient calcium transit in hypertrophia of the heart

> muscles.

>

> They conclude that this is the reason for contractile insufficiency, in

> hypertrophy and other metabolic problems of the hypertrophic heart.

Special

> reference must be made to the papers of the Kaufmann group. We have made a

> series of tests with 14 year old dogs on a 14 percent incline. Our results

> were a very good confirmation of the pathogenic mechanism that Kaufmann

> portrayed. We will give you the details in a later article.

>

> Rilling (12) has verified the research of Zondek (13)

> and Kylin (14) with his comprehensive spectrographic

> studies. These authors maintain that essential

> hypertonia is a cellular calcium-deficient

> pathognomonic condition. This interpretation is much

> the same as Kaufmann's explanation of heart muscle

> hypertrophy and would explain the blood pressure-

> lowering effect of calcium orotate. At the time that

> Nieper and Laborit were doing clinical research on

> potassium magnesium aspartate, we hoped to be able to

> correct disturbances of the heart action regulation

> system. Except for a suppression of ventricular and supraventricular

> extrastole, this hope was not realized. (15)

>

> As you know, the heart action regulation system is

> musculature in nature, which consists of a different

> type of metabolism, the direct oxygen or " pentose

> pathway " . The stimulatory regulation of ganglion

> tissue provide especially good protection against

> oxygen deficiency intrafetally and later. Orotic acid

> plays an essential role in the pentose pathway, and so

> appears to be the electrolyte transporter needed for

> that tissue. In view of that, it should not be

> surprising to learn that we were able to normalize

> apparently therapy-resistant tachyarrhythmia with

> auricle flutter, in three doses, with a dosage of 5g

> calcium tgl.

>

> After this excursus, I would like to mention the immune-inhibiting effect

> (as already reported) (4), of calcium orotate on a succession of

> bradytrophic tissues.

>

> Specific Effect On Cartilage:

>

> It was first reported by White towards the end of

> 1969, that calcium orotate showed an astounding

> curative effect on the Tietze syndrome. These reports

> were repeated over and over during 1970 and 1971, so

> that we were induced to try calcium orotate in three

> cases of stubborn Tietze syndrome. The effect of the

> calcium orotate was indeed surprising - all details of

> the White article were fully verified.

>

> Tietze syndrome, according to our information, is much

> more common in the US than here in Germany. According

> to White, the syndrome is suppressed by very low doses

> - down to 1g/week, which we could verify. A dosage of

> 500mg/day is fully effective. It is highly significant

> that there is no effect whatsoever from calcium EAP,

> calcium-L dl aspartate (calciretard), calcium

> gluconate, calcium citrate, magnesium orotate, and K- MG-aspartate, upon

the

> Tietze syndrome.

>

> On the basis of our knowledge of the effect of calcium

> orotate on the Tietze syndrome, we must conclude, that

> a favorable trophic effect on the cartilaginous

> intervertebral substance is the reason for the not

> infrequently spectacular improvement of the patient.

>

> This fully specific effect of calcium orotate on

> cartilage, as evidenced by the Tietze syndrome

> experience, appears now to be of tremendous clinical significance.

> Unfortunately, we only learned this after the repeated reports of White.

> While we were treating patients with spinal column syndrome and

> calcification damage, it had been apparent, for a long time, that the

> reported and verified improvement of their condition, must be attributed

to

> more than simply an influence on the bone tissue.

>

> For example, we had five patients (f) and one patient

> (m) from 26 to 76 years, with symptoms of weakness and

> painful sensitivity in the wrists. In three cases, it

> could be observed only with a sphygmomanometer, and in

> three cases observation was not possible at all. In

> every case, the complaints disappeared with calcium

> orotate therapy. Upon removal of the therapy, or when

> the dosage was insufficient (less than about

> 1.6g/week) the complaints returned.

>

> Especially puzzling are the findings for 18 patients

> from our files, in which there were severe dislocating alterations of the

> spinal column. Treated with calcium orotate, these patients became

> exceptionally free of complaints. Other medications - Butazonderivate,

> cortisone, indomethacin, physical therapy, gold medication - had failed.

> Even intensive treatment with calcium EAP, and calciretard along with

> calcium-sandoz and K-Mg-aspartate were ineffectual.

>

> CASE HISTORY:

>

> NOTE: The illustrations which are x-ray images cannot

> be reproduced by our copy machine, so they are omitted

> here. They are in the German manuscript however.

>

> Illustration 1, Frau K, 35 years old,

> Before and after therapy with calcium orotate. Metast. Mamma-carcinoma

>

> Illustration 2a, patient (f) H. Sch. 64 years

>

> Severe complaint complex with LWS syndrome. Every

> imaginable therapeutic preventive measure taken to no

> avail. Infusion treatment with calcium-L-dl aspartate

> calciretard) plus calcium EAP for more than six months. Complete

stationary

> immobilization for three months 1970/71 to no avail. The pain associated

> with the LWS syndrome had forced her almost to the point of complete

> immobility. About three to five weeks after starting calcium orotate

> therapy - an average of 3g/daily - the patient was complaint free and

> remained so for 16 months - practically normal movement and walking

ability.

> In contrast to calcium orotate, other calcium transporters such as calcium

> EAP and calciretard were fully ineffective. Is the improvement to be

sought

> in a structurally favorable influence of the intervertebral tissue?

>

> Illustration 2b,

>

> Almost complaint free after six weeks intensive

> treatment with calcium orotate. reconstruction mainly

> in the gap between 3 and 4. LWK Both developments are stabilized. These

> findings are not the only ones which gave rise to our questions about the

> effectiveness of calcium orotate on cartilage.

>

> In 1968, we started using calcium orotate to treat

> patients with definite Bechterewschen disease. There

> were, in all, five such patients and in all the cases

> the very intense pain in the spinal column disappeared

> for the most part. All previous treatments -

> phenylbutazone, cortisone, gold therapy and healing

> baths - had been ineffective.

>

> Illustration 3, patient (f) N.N. 56 years

>

> In 1963, the disease has progressed so far in this 56

> year old patient, that she was almost completely

> immobilized. In spite of standard therapies and

> seltzer water cure, the progression continued. By

> 1968, the face was almost in a fixed position,

> movement and sitting were almost impossible. 1.5g

> calcium orotate was prescribed daily for 54 months continuously. No more

> complaints. The face could be moved normally and the patient resumed her

> household duties.

>

> The second picture is the patient (f) with Morbus

> Bechterew after 54 months continuous treatment with

> calcium orotate.

>

> Illustration 4, patient (f) M.B. 71 years

>

> Most severe complaints for three years as a result of spondylitic

arthrosis

> deformation. Treatment with phenylbutazone and also in combination with

> cortisone and with pyrazolone showed only slight palliative effect.

> Calcium-sandoz forte tgl. 3 tabl. for over four months without any effect.

> After 10 days tgl. 5g calcium orotate, the patient was almost complaint

> free, and has remained that way.

>

> Illustration 5, patient (f) N.B. 81 years

>

> A sister of Frau M.B. above. 10 years older. Xrays and

> symptom complex indicate a very severe case. After

> being treated with calcium orotate tgl. 5g, she became complaint free,

that

> she again took up vigorous employment after previous inactivity.

>

> Illustration 6, patient R. 79 years (f)

>

> Frau R., mentally and bodily vigorous. Had an acute

> attack of a long time existing osteochondrosis. Quite comfortable lying

> down, but experienced immediate unbearable pain in the spinal column upon

> standing up. After treatment with 5g calcium orotate daily, the patient

> became complaint free within three weeks. The pain had previously worsened

> under cortisone therapy. Calcium-gluconate-citrate, phenylbutzone and

> indomethazine had no effect whatsoever. The curative effect of calcium

> orotate was permanent (now over 14 months).

>

> Illustration 7, patient (m) St. 55 years

>

> Acute monarthritis of the right tibiotarsal joint,

> forcing him almost to the point of immobilization.

> Three tablets realin daily were of very little effect. Soludecortin H 50mg

> likewise. Volon 80 brought limited relief for 36 hours. Calcium orotate 8

> tabl. each .5g daily brought improvement after 2 days and corrected the

> condition in 6. There are four similar cases.

>

> Latest findings

>

> By the end of 1972, we had experience with 21 cases of deforming

> spondylarthosis - five men, sixteen women, from 58 years up. Clinical and

> objective findings were consistent with the cases mentioned previously. 14

> out of 16 women and 4 out of 5 men were helped by calcium orotate alone.

> They were observed for over a year. The remarkable effect of calcium

orotate

> (called Ca orotate for short) with Tietze syndrome in the intervertebral

> disk (and possibly in the ligamentous

> apparatus) causes us to again evaluate the metabolic specificity of

> cartilage and other bradytrophic tissue. Here the pentose pathway

(so-called

> direct

> oxidation) plays a very crucial role - an extremely

> old phylogenetic-metabolic pathway, which is not

> dependent upon the erythrocyte oxygen donation. In the

> pentose pathway, the ribose is activated through

> orotate coupling. For this reason, orotic acid plays a

> very essential role in the pentose pathway.

>

> Here we are concerned with a large variety of tissues.

> Besides cartilage and ligamentous tissue, there is the connective tissue,

> the skin, the walls of the blood vessels, a specific section of the

venules

> in the blood-brain barrier, the heart stimulation-regulation tissue and

> keratin building tissue (hair, nails, etc.). Also the pentose pathway

plays

> a very important role in the bone matrix, in the heart muscle and in the

> liver (both in the liver cells and in the stroma).

>

> The aromatic structure of the orotic acid is

> responsible for the high complex stability of the

> salts, and the already mentioned highly complex

> passage through the cell membrane typical of orotic

> acid, plainly make the orotate an ideal mineral

> transporter. (4)

>

> We are indebted to the tireless effort of Laborit (8)

> and his scholars (7) who made public the results of

> their year-long research which disclosed the vital

> effect of the pentose pathway in the function and

> structure of bradytrophic tissue. Life would not be

> possible without it. Previously, cartilage and

> connective tissue had been considered uninfluencable,

> for the most part.

>

> If calcium orotate can bring about a change here, as

> was demonstrated, then there is not much value in the

> geriatric therapeutic concept.

>

> Summary

>

> An orientation is given concerning the clinical

> effects of calcium orotate (called Ca orotate for

> short). Because calcium orotate is free from side

> effects, it is superior to conventional calcium salts,

> which have certain problems when applied in

> osteoporosis with concomitant arteriosclerosis of the

> abdominal aorta. Calcium orotate, on the other hand,

> protects the body from arteriosclerosis.

>

> Calcium, for this reason, is of value as a food

> supplement when used in the form of calcium orotate,

> which can penetrate the cell membranes as a complex

> form, compensating for defective calcium transit into

> the cells. In addition, calcium orotate has a special

> affinity for cartilage and other bradytrophic tissue,

> where it is metabolized.

>

> Not only is the basic principle of action quite

> simple, but the long time therapeutic effects are of considerable

interest.

> A new dimension of therapy now appears with the improvements in

> osteochondrosis and disk degeneration treatment. Far better than the

present

> therapeutic possibilities. Much the same observations seem to apply to

> osteoporosis.

>

> Parallel investigations point to the important

> pathogenetic significance of a defective calcium

> transport through the cell membrane. This is the case,

> for example, in hypertensions - especially essential hypertension - in

fatty

> liver, in disturbances of the ductile of the heart, and in contractile and

> metabolic insufficiency of the hypertrophic myocardium. In respect to all

of

> these indications, calcium seems to bring about the most promising

> therapeutic results, when combined with the carrier orotic acid for better

> transmembrane transport, in the form of calcium orotate.

>

> Literature

>

> 1. Nieper, H.A.: Recalcification of Bone Metastases by

> Calcium Di-orotate, Agressologie II, 6, 495-502

> (1970);

>

> 2.Nieper, H.A.: A Clinical Study of the Calcium

> Transport Substances Ca-di-aspartate and Ca-2-

> aminoethanol Phosphate as Potent Agents Against

> Autoimmunity and Other Anticytological Aggressions, Agressologie VIII, 4

> (1967);

>

> 3.Al Haddad, M.: Pers. Mitteilung;

>

> 4.Nieper, H.A.: The Anti-inflamm. and Immune

> Inhibiting Effects of Calcium Orotate on Bradytrophic

> Tissues, Agressologie X, 3, 349-353 (1969);

>

> 5. White, M.D.: In Preparat. und pers. Mitteilung;

> 6.Nieper, H.A.: Metabolisme du Calcium et du Phosphore

> des patients traites pa l'Orotate de Calcium,

> Agressologie (Paris) 1971, 12,

>

> 6, 401-408; 6a.Nieper, H.A.: Bilanzuntersuchung des

> Calcium- und Phosphatstoffwechsels bei Patientin, die

> mit Calcium-Orotat behandelt werden, Z. praklin.

> Geriatrie (1197) 9, 184-191;

>

> 7.Sonka, J. (Prag): Voies de Pentoses-Physiologie et Physiopathogenie,

> Agressologie VII, 5, 461-477 (1966);

>

> 8.Laborit, H.: Regulations metaboliques dans le Tissu Conjonctif,

> Agressologie 1969, 10, 1, p.11-26; Silberberg, R.: Gesamtband Act

> Rheumatologica, Documenta Geigy, No. 26 (1972), " Wachstum und Alterung des

> Skelettes " ;

>

> 10.Doniach, D.: Die klinische Bedeutung der

> antimitochondrialen Antikorper, Deutch in: Triangel-

> Sandox, Band 11, 1, 29-34 (1972);

>

> 11.Kaufmann, R.I., Homburger, H., und H. Wirth:

> Disorder in Excitation-Contraction Coupling of Cardiac

> Muscle from Cats with Exptl. Produced Right Ventric, Hypertrophy

Circulation

> Research, Vol. XXVIII, 346-357 (1971);

>

> 12.Rilling, S.: Gesamtdokumentation Bayer Spektromat Laboratorien,

> Stuttgart, Klagenfurter Str. 4 (1972);

>

> 13.Zondek, R.: " Die Elektrolyte " , Springer, Berlin

> (1927), p. 299-300;

>

> 14.Kylin, E.: Die Hypertoniekrankheiten, J. Springer,

> Berlin (1926); 15.Nieper, H.A. und Kj. Blumberger:

> Wirkung von Elekrolyutschlepper - Verbindungen (K-Mg-

> Asparaginat) auf den Herzstoffwechsel, Beh. von

> Extrasystolien Verh. dt. Ges. Kreislaufforsch. 27,

> 238-242 (1961).

>

> ***************************************************************

>

November 10, 2004

Don,

I must say that if I had any doubts before, just made up my mind not to

try this product. Juli Ferrante's attack on Mark and Mike, whether they are

right or wrong is uncalled for and tells me something just ain't right.

Grace

Don Vance <dvance@...> wrote: