Guest guest Posted September 23, 2006 Report Share Posted September 23, 2006 Hi Everyone, I decided to post this after 2 years of just listening and learning from everyone on this site, just to help those still deciding on LDN. I originally experienced symptoms in 2003, after my first MRI on 6/2003 doctors were not sure what was going on. I had partial paryalsis on my left side, lost the hearing in one ear and double vision. While the weakness subsided, hearing returned and my facial paralasis cleared, I never felt normal, there was continuing brain fog. After a lot of research, I figured I had MS. A new series of symptoms resurfaced and another MRI 6/2004 a diagnosis was given of MS. I was on LDN by August of 2004. Almost immediately the brain fog lifted. However, there was literally an awakening of every symptom I had ever experienced which lasted about 6 months, even though much milder. Timeline July 2004 Blood Type Diet and Supplements August 2004 Started LDN (brain Fog lifted immediately) October 2004 Started Copoxane daily January 2005 All Symptoms diasppeared March 2006 Reduced Copoxane to every other day June 2006 Added Colloadial Silver from a quality company 20ppm ((Expensive)The next 5 weeks had a reawakening of symptoms even though very mild) September 2006 MRI PALO ALTO MEDICAL FOUNDATION HEALTH CARE DIVISION/PALO ALTO MEDICAL CLINIC 795 El Camino Real Palo Alto, CA 94301 IMAGING REPORT Patient Name: XXXXXX,XXXX MRN: XXXXXXXXXX DOB: XX/XX/XXXX Telephone #:XXXXXXXXXX Referring MD: Exam Date: 09/07/2006 Examination: BRAIN MRI WITH AND WITHOUT GADOLINIUM Indication: History of multiple sclerosis, on therapy for 2 years. Symptoms have improved. Comparison: Comparison with outside hospital MRI dated 06/09/2004 from Fremont Sports Imaging. Technique: 5 mm sagittal T1, 5 mm FLAIR coronal, 5 mm axial proton density, 5 mm axial T2. Additional axial T1 pre and post-gadolinium, coronal T1 post-gadolinium, sagittal T2 sequences through the brain. Report: In comparison with 06/09/2004, there is no significant change in number or size of the multiple, periventricular and subcortical white matter T2 hyperintense lesions, some of which are oriented perpendicular to the corpus callosum. A few of these lesions appear to involve the corpus callosum. There are also small T2 hyperintense lesions within the right thalamus and possibly left basal ganglia, also unchanged. Previously seen enhancement has resolved, involving the lesions adjacent to the lateral ventricles. There are no new lesions. The ventricles and sulci are within normal limits for age. The major intracranial flow voids are unremarkable. There are no extraaxial fluid collections, midline shift. Visualized orbits, optic nerves, paranasal sinuses, mastoid air cells and posterior fossa are unremarkable. IMPRESSION: In comparison with 06/09/2004, the previously seen enhancing lesions adjacent to the lateral ventricles have resolved, otherwise there is no significant change with persistent T2 hyperintense lesions in the periventricular and subcortical white matter, right thalamus, and possibly left basal ganglia. Some of these lesions involve the corpus callosum. These findings are nonspecific, but consistent with the reported history of multiple sclerosis. Anyway, I hope this information helps those that are still deciding. Quote Link to comment Share on other sites More sharing options...
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