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2 Years on LDN MRI Results

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Hi Everyone,

I decided to post this after 2 years of just listening and learning

from everyone on this site, just to help those still deciding on LDN.

I originally experienced symptoms in 2003, after my first MRI on

6/2003 doctors were not sure what was going on. I had partial

paryalsis on my left side, lost the hearing in one ear and double

vision.

While the weakness subsided, hearing returned and my facial

paralasis cleared, I never felt normal, there was continuing brain

fog. After a lot of research, I figured I had MS. A new series of

symptoms resurfaced and another MRI 6/2004 a diagnosis was given of

MS.

I was on LDN by August of 2004. Almost immediately the brain fog

lifted. However, there was literally an awakening of every symptom

I had ever experienced which lasted about 6 months, even though much

milder.

Timeline

July 2004 Blood Type Diet and Supplements

August 2004 Started LDN (brain Fog lifted immediately)

October 2004 Started Copoxane daily

January 2005 All Symptoms diasppeared

March 2006 Reduced Copoxane to every other day

June 2006 Added Colloadial Silver from a quality company 20ppm

((Expensive)The next 5 weeks had a reawakening of symptoms even

though very mild)

September 2006 MRI

PALO ALTO MEDICAL FOUNDATION

HEALTH CARE DIVISION/PALO ALTO MEDICAL CLINIC

795 El Camino Real

Palo Alto, CA 94301

IMAGING REPORT

Patient Name: XXXXXX,XXXX

MRN: XXXXXXXXXX

DOB: XX/XX/XXXX Telephone #:XXXXXXXXXX

Referring MD:

Exam Date: 09/07/2006

Examination: BRAIN MRI WITH AND WITHOUT GADOLINIUM

Indication: History of multiple sclerosis, on therapy

for

2 years. Symptoms have improved.

Comparison: Comparison with outside hospital MRI dated

06/09/2004 from Fremont Sports Imaging.

Technique: 5 mm sagittal T1, 5 mm FLAIR coronal, 5

mm

axial proton density, 5 mm axial T2. Additional axial T1 pre and

post-gadolinium, coronal T1 post-gadolinium, sagittal T2

sequences through the brain.

Report: In comparison with 06/09/2004, there is no

significant change in number or size of the multiple,

periventricular and subcortical white matter T2 hyperintense

lesions, some of which are oriented perpendicular to the corpus

callosum. A few of these lesions appear to involve the corpus

callosum. There are also small T2 hyperintense lesions within

the right thalamus and possibly left basal ganglia, also

unchanged. Previously seen enhancement has resolved, involving

the lesions adjacent to the lateral ventricles. There are no

new

lesions.

The ventricles and sulci are within normal limits for age. The

major intracranial flow voids are unremarkable. There are no

extraaxial fluid collections, midline shift. Visualized orbits,

optic nerves, paranasal sinuses, mastoid air cells and posterior

fossa are unremarkable.

IMPRESSION:

In comparison with 06/09/2004, the previously seen enhancing

lesions adjacent to the lateral ventricles have resolved,

otherwise there is no significant change with persistent T2

hyperintense lesions in the periventricular and subcortical

white

matter, right thalamus, and possibly left basal ganglia. Some of

these lesions involve the corpus callosum. These findings are

nonspecific, but consistent with the reported history of

multiple sclerosis.

Anyway, I hope this information helps those that are still deciding.

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